Archive for the ‘vaccines’ Category

Study Shows 630% More Aerosolized Flu Virus Particles Emitted by Flu-Vaccinated – A Message to Ethical MD’s

Infectious virus in exhaled breath of symptomatic seasonal influenza cases from a college community

Jing YanMichael GranthamJovan PantelicP. Jacob Bueno de MesquitaBarbara AlbertFengjie LiuSheryl EhrmanDonald K. Milton and EMIT Consortium
  1. Edited by Peter Palese, Icahn School of Medicine at Mount Sinai, New York, NY, and approved December 15, 2017 (received for review September 19, 2017)


Lack of human data on influenza virus aerosol shedding fuels debate over the importance of airborne transmission. We provide overwhelming evidence that humans generate infectious aerosols and quantitative data to improve mathematical models of transmission and public health interventions. We show that sneezing is rare and not important for—and that coughing is not required for—influenza virus aerosolization. Our findings, that upper and lower airway infection are independent and that fine-particle exhaled aerosols reflect infection in the lung, opened a pathway for a deeper understanding of the human biology of influenza infection and transmission. Our observation of an association between repeated vaccination and increased viral aerosol generation demonstrated the power of our method, but needs confirmation.


Little is known about the amount and infectiousness of influenza virus shed into exhaled breath. This contributes to uncertainty about the importance of airborne influenza transmission. We screened 355 symptomatic volunteers with acute respiratory illness and report 142 cases with confirmed influenza infection who provided 218 paired nasopharyngeal (NP) and 30-minute breath samples (coarse >5-µm and fine ≤5-µm fractions) on days 1–3 after symptom onset. We assessed viral RNA copy number for all samples and cultured NP swabs and fine aerosols. We recovered infectious virus from 52 (39%) of the fine aerosols and 150 (89%) of the NP swabs with valid cultures. The geometric mean RNA copy numbers were 3.8 × 104/30-minutes fine-, 1.2 × 104/30-minutes coarse-aerosol sample, and 8.2 × 108 per NP swab. Fine- and coarse-aerosol viral RNA were positively associated with body mass index and number of coughs and negatively associated with increasing days since symptom onset in adjusted models. Fine-aerosol viral RNA was also positively associated with having influenza vaccination for both the current and prior season. NP swab viral RNA was positively associated with upper respiratory symptoms and negatively associated with age but was not significantly associated with fine- or coarse-aerosol viral RNA or their predictors. Sneezing was rare, and sneezing and coughing were not necessary for infectious aerosol generation. Our observations suggest that influenza infection in the upper and lower airways are compartmentalized and independent.

The association of current and prior year vaccination with increased shedding of influenza A might lead one to speculate that certain types of prior immunity promote lung inflammation, airway closure, and aerosol generation. This first observation of the phenomenon needs confirmation. If confirmed, this observation, together with recent literature suggesting reduced protection with annual vaccination, would have implications for influenza vaccination recommendations and policies.

___________  (Please read entire article here by James Lyons Weiler)

Letter to ethical MD’s (snippets below):

The last time the flu vaccine was 60-70% effective was eight years ago.


“This is the CDC’s data  Clearly, Gupta’s “Years” is, in immunological memory, a singular “Year”. Only once out of the last 14 years was the flu vaccine above 59% – that the value was not 60-70%, it was 60%.

This type of misrepresentation is a consistent penchant within the media and of course from the CDC to exaggerate and highly emphasize only positive views and diminish, dismiss, or ignore any negative views on the safety and efficacy of vaccines.

The Jury is In: The Flu Vaccine Reduces its Own Efficacy

Too many studies now exist that have independently come to the same conclusion: increases in the uptake of flu vaccine reduces that vaccine’s effectiveness in the following year – and some studies show the negative effects of mass influenza vaccination last two years.

The studies reporting those results are reviewed in my article, “Diseases with Unknown Etiology Trace Back to Mass Vaccination Against Influenza in 1976“, and they are extensive and damning.

Patients have a right to know the specific nature of their infections, and survivors in families of those who die from respiratory infections deserve an accurate cause of death. Coroners should certainly be required to provide an accurate cause of death in so-called “flu” mortalities. Health departments should be required to count only deaths due to confirmed influenza infection as “flu” – otherwise their numbers perpetuate misperception on the risk of influenza infection, and cause fear leading to increased vaccination. How is this seen as a good thing? The population deserves good and honest doctors and stewards of public health.

HHS could demand swab results for all suspected cases of “flu deaths” with a press release and enforce them with random audits. This annual ritual of fear-mongering over “flu-deaths” hides the fact that as long as thimerosal is injected into patients, they are at increased risk of other infections. And due to heterologous immunity, even without thimerosal, flu vaccines can confuse the immune system and muddle up ineffective immune response by trying to re-purpose B-cells trained on the wrong virus, hobbling the immune system making it unresponsive to similar viruses. Such as next year’s flu strain.

We do need objectivity to arise immediately throughout the public health system in the US, starting with HHS, then to CDC and to all Health Departments around the country. Many studies have also found problems with Tamiflu. But no emergency epidemiological study is addressing the question – why are so many young people dying from “flu”? Many of the reports I’ve seen include mention that they person had not only been vaccinated, they also had taken Tamiflu. And many had taken Tylenol. It’s time to ask the tough questions. The science is there on problems with Tylenol for vaccine-induced fever, and it must be taken into consideration. Fever due to respiratory infections after flu vaccination is still vaccine-induced.

A look at the issues with Tamiflu (see primary scientific literature reviewed here) shows that we cannot ignore the possibility that the human immune system is not infinitely resilient, and that medicine’s approaches to tackling “the flu” is imprecise, not evidence-based, and self-defeating. I’m not talking about the number of antigens the human body can take; I’m talking about the amount of tweaking it can tolerate, especially given the aluminum-dense childhood vaccination schedule. The allopathic medical community would do very well to heed the studies that show that Vitamin D helps alleviate both vaccine injury and severity of viral infections. It helps resolve the unfolded protein response without killing the cells. And the science of ER stress (endoplasmic reticulum stress) shows that Thimerosal is, after all, not safe for human use. Same for aluminum.

Real Reform is Coming – It’s a Mathematical Certainty

Vaccines injure people every day, and kill people every week. Each injury and death informs family members, co-workers, and schoolmates. The flaws in vaccines, combined with misinformation campaigns on safety, fuel the fire and build the vaccine risk aware army. It’s a peaceful army, filled with individuals who are hurt so badly, they do not want others to suffer the same fate. They are altruistic. And under informed, ethical and distributed leadership, they are finding their momentum.

Vaccine safety science reform means removing those in the CDC and HHS that perpetuated the debacle as it grew to proportions that even they could no longer easily deny it. And that’s fine. Let them go. There are many excellent professionals capable of replacing them – people who have not been involved in cooking studies to alter the public’s perception of vaccine risk. People who have withstood unwarranted and unfair criticism by those who live in cowardice of reality. People who now no longer afraid to publish their views. An important question is who among my colleagues in Academic Public Health, and which doctors in Pediatric medicine are willing to #bebrave and take on a debacle as huge as a failed national immunization program? Who will stand up to the AAP and tell them they are wrong?

If you are that type of doctor, it will be easier if you trust those who have worked at this for years. Read Dr. Paul Thomas’ book, The Vaccine Friendly Plan. After the resignations, have him come and teach the entire CDC and HHS what he knows. Consider Dr. Alvin Moss’s wisdom – ask him to create a Conflicts of Interest Policy for CDC and HHS, as he has done for the rest of academic medicine. Bring in Dr. Bob Sears from California, who was willing to stare down threats of the loss of his license to practice medicine because he dared to continue to practice medicine in the face of wanton misinformation and pressure from the AAP. Consider Dr. Richard Frye, and Dr. Chris Exley from the UK, who care first and foremost about the truths that impact total health. Dr. Frye would be great as the new NIH Director, in my opinion. Let these people form a new national public health direction that overrides existing contracts. There are others. Like Dr. Judy Mikovits whose character stands much taller than those who tried – and failed – to silence her – on the issue of adventitious agents in viral vaccines (specifically and quite problematic: retroviruses). Ask Dr. Ted Fogarty about Ethical Vaccinomics, and testing for vaccine injuries. Bring in Dr. John Piesse from Australia and end his persecution there, and put his good will toward safety to work here. We would be lucky to have him.

Create a Manhattan Project focused on reducing vaccine injuries, not on making currently licensed vaccines safer. They are old, and stale, and tired, and they, too, need to go. Bring in exciting new developments in artificial immunization like microneedle patches. Bring in Dr. Kanduc to screen epitopes that are unsafe. Drop aluminum, as many have now called for, and bring in calcium carbonate – if needed at all. Let those pharmaceutical companies who created the disaster make good on their promises to stop making their vaccines. Then we will see new approaches to artificial immunization that compete on the platform of safety.

Don’t just end COIs at ACIP: End ACIP. Create a Vaccine Safety Commission that enforces Science Integrity. Open up the markets. Let ideas thrive. Let consumers choose. Let the FDA do its job. Let the people’s experiences be heard. Establish a paradigm in which the end consumer has a say in the quality of the product. Strip the CDC of the ability to hold patents. End the CDC Foundation. End the differences between drugs and biologics and require randomized clinical trials – with proper placebos, not aluminum hydroxide – for vaccines. Repeal the 1986 Act that protects drug companies from liability for faulty vaccines. Perform random spot checks of vaccines in practices for contamination. The total sum of policies in the National Immunization Program, and the burden of morbidity on the population is a serious threat to our National Security.

Let some new faces and voices drive this reform. Bring in Dr. Dan Neides who had to escape the Cleveland Clinic after speaking his conscience. Let him oversee the transition. Bring in Dr. Brian Hooker to personally issue the pink slips to those who must now go from the CDC. Let all of those named here share his or her experience with Congress. Have Dr. Thompson testify. We need truth and reconciliation. And we need it 42 years ago.

There are MDs who sit in the shadows, silent, and afraid of job loss, sanction, ridicule. Step up. Let your views be known to the current Administration. Join Physicians for Informed Consent. You are not alone. You can help be part of the solution. Attend Health Department meetings and speak up for Informed Consent. Speak up for vaccine exclusions for kids in homes with high lead levels. Speak up for spacing out vaccines and skipping them. Speak up for tolerance and understanding of the pain and anguish parents of kids with autism experience when they are told it’s genetic, they know it’s environmental, and they are told they have to vaccinate their other babies. Speak up against calling CPS for parents who want to take the time they have under the law to consider vaccinations. And, of course, do right by your patients. Listen to their concerns. Inform them of both risks and benefits, as required by Federal Regulations. Let them know they are enrolling themselves or their children (and unborn baby) in post-licensure vaccine safety clinical trials (as required by Federal Regulations). Provide medical and philosophical exemptions for school waivers as required by the laws of your state and the rule of your own conscience. The AAP does not represent the rights and will of the people of the United States of America. Our legislation does.

Let’s aim to not make 2020 vaccination look anything like 2019. We have solutions. We’re now aiming for Healthy People 2050, and the current vaccines have very little to do with our vision. By the way, these ideas don’t come (exclusively) from me. They are shared by hundreds of thousands of American citizens, many of whom have been made sick or lost loved ones to vaccines. #werenotgoingaway #releasetheothermemos #hearthiswell #notmine #Vaxxed #cdctruth #saveourbabies #bebrave #ipak #cdcwhistleblower #rfkcommission #educatebeforeyouvaccinate #vaxxed #learntherisk #wedid #cdclied #stopmandatoryvaccination #learntherisk.”



More just keeps popping out of Pandora’s Box regarding vaccines.

This recent talk shows how vaccines are causing Lyme/MSIDS patients to relapse as well as worsen:  Scottish doctor treating a number of young women who fell ill after their HPV vaccination, which seems to have stimulated a latent Lyme infection to reactivate.  Asymptomatic girls after receiving Gardasil activated dormant Bartonella which was confirmed by testing.

Great video on the flu vaccine’s ineffectiveness:

I could go on and on to infinity.  Something must be done.  Be a part of the solution.







Dr. Muth – Immune Issues and Lyme/MSIDS

Published January, 2018

Dr. Debra Muth, ND, WHNP, BSNH, MSNH, ARNP, BAAHP spoke at the Madison Area Lyme Support Group  She discusses various bacterial, viral and immunological effects that activate the immune system and cause symptoms to increase, as an over active immune system can mimic Lyme/MSIDS symptoms. She believes it’s important to look at all angles of the immune system and how it affects patients.

She reviews how Marcon’s, Viruses, Heavy Metals and vaccines activate the immune system and cause increased brain inflammation. She discusses treatment options available to date, including the RK protocol and how it can assist with Lyme treatment, as well as how it fits in with treatment successes in her practice.

Vaccne and inlammation Madison Support Group 2018  Accompanying pdf

Cure a Cold Fast – It’s Not the Flu Vaccine

Lyme/MSIDS patients have impaired immune systems due to serious bacterial and often viral and fungal infections, so when a cold or flu strikes, we are sitting ducks.  Recent research shows that just breathing is enough to spread the flu:  This information is being used to frighten and push the flu vaccine which Dr. Mercola explains is about 10% effective this year due to viral mismatches and growth substrates where viruses can mutate:

Then there’s that pesky detail of not having enough studies on the effectiveness of formaldehyde killing the virus making it “inactive” which explains why some get the flu after getting the vaccine:  This happened before when 40,000 kids were given the Salk IPV and got polio.  OOPS!

What authorities don’t tell you about vaccines for the immunocompromised, such as Lyme/MSIDS patients, is that a vaccine which is created to lower your immune system so that your body mounts an immune response can activate latent infections.  Within this link you will also see a connection between the HPV vaccine and Bartonella.  I just finished “Lyme Madness,” by Lori Dennis.  Her son’s Lyme/MSIDS activated after a flu shot.  I hear these stories over and over.  

And then there’s the damning retroviral connection:

Often the first sign of immune dysregulation is an adverse reaction to a vaccine:  All it took was a tetanus shot for engineer Eric Gladen to get major neurological symptoms.  After going on a protocol for about 6 months to remove mercury, his symptoms vanished.  This led him on a journey and gave us the documentary “Trace Amounts,” which is about mercury-based thimerosal & autism.

The flu vaccine in particular often contains high amounts of thimerosal, which is 50% mercury.  Many vaccines also contain aluminum, a neurotoxin.  Research is growing on how these metals accumulate in the brain over time.  Great article on aluminum toxicity.

So rather than be frightened and reach for what seems like an easy button, this article will hopefully give you practical tools to use at home to lower your flu risk or lessen infection time.

The following video by NPR shows how viruses turn our cells into mini viral reproductive factories.

 Approx. 3.5 Min

Approx. 6 Min

In this video Dr. Axe shares his top seven remedies to cure a cold fast. These specific remedies can help you get rid of your cold in 24 hours or less.  They are most effective when taken upon initial flu/cold symptoms.

1. Echinacea

The research using Echinacea tea is not consistent. The studies that demonstrated the best effect at shortening the length of a cold occurred when you drink two to three cups of Echinacea tea per day starting on the first or second day of your cold.

2. Elderberry Syrup or Extract

In one study, elderberry syrup reduced the severity of flu symptoms and shortened their duration by about four days. Elderberry extract is also known for inducing sweating, and helps relieve congestion.
3. Vitamin C (1000mg 5x day)
A very potent antioxidant; use a natural form such as acerola, which contains associated micronutrients.  You can take several grams every hour (use the liposomal form so you don’t get loose stools), till you are better. 

4. Garlic (capsules or cloves)

In the singular study identified by the Cochrane group, those who took garlic daily for three months had fewer colds than those who took a placebo, and, when they did come down with a cold, the duration of illness was shorter — an average of 4.5 days compared to 5.5 days for the placebo group.

5. Oregano Oil/ Oil of Oregano (4-5 drops in water)
The higher the carvacrol concentration, the more effective and most active antimicrobial agent in oregano oil which has potent antibacterial and antiviral effects. In one study, researchers found when in spray form in combination with four other aromatic plants it could immediately reduce the side effects of a cold. This treatment was no longer effective after three days of use. Oil of oregano should not be used by children, women who are pregnant or nursing or who plan to become pregnant.
6. Zinc (100mg a day)
A Cochrane Database Review of the medical research on zinc found that when taken within one day of the first symptoms, zinc can cut down the time you have a cold by about 24 hours.  Zinc was also found to greatly reduce the severity of symptoms. Zinc was not recommended for anyone with an underlying health condition, like lowered immune function, asthma or chronic illness.  Dr. Mercola does not recommend taking more than 50 mg a day, and does not recommend taking zinc on a daily basis for preventive purposes as you could easily develop a copper imbalance that way.

7. Apple Cider Vinegar (2 Tbsp in water 3x a day)

Cold viruses increase the acidity of your body.  Apple Cider vinegar reduces the acidity and has acetic acid that helps prevent the growth of viruses.
Eliminate sugars from your diet, add in a real vegetable soup a day, consume chicken broth, and citrus fruits. For more information on getting rid of a cold check out this article:
Since it’s a fat-soluble vitamin, it can be toxic, which is why it’s important to test your level at least yearly.  It’s also more effective than the flu-vaccine:  Dr. Mercola adds to this list:

1. Meditation – has significant positive effects on heart rate, brain function, stress reduction and blood pressure.  Research also has demonstrated lasting positive effects on brain function and immune system.  Imaging demonstrated an increase in activation in the left frontal region of the brain associated with lower anxiety, and blood work showed larger increases in antibody production in participants who meditated in the study. 

2. Exercise – if all symptoms are above the neck, such as sneezing, sniffling and watery eyes, then breaking out in a sweat is generally considered safe. The immune system functions better when you exercise regularly and is a good preventative measure.  Walking, jogging, yoga and slow biking are among the best exercises when you have a cold, while endurance sports, team sports, weightlifting and exercising in the cold weather are among the worst.  Exercise may help you feel better but may not shorten the length of your cold. If you are involved in strenuous exercise it depletes the energy needed to fight the virus and can actually make your symptoms worse.

3. Sleep – Sleep has a strong regulatory influence on your immune system and promotes the influence of cytokines stimulating the interaction between antigen-presenting cells and T-helper cells necessary for your body to fight virus infections.  Most people need between about eight hours of sleep a night and plenty of rest during the day.

4. Nasal Saline Rinse – Although researchers can only speculate how saline nasal washes are effective in treating and preventing virus infections and recurrences, the fact is they are effective.  Use only sterile normal saline water in the rinse.  Tap water can increase the inflammatory response in the sinus passages and carry parasites that can infect your brain.

5. Hydrogen Peroxide – In 1928, Dr. Richard Simmons hypothesized that the cold virus entered your body through the ear canal and not the nose. His theory was dismissed by the medical community; however, in 1938, German researchers had great success using hydrogen peroxide in the ear canal to treat colds and the flu.  You must start treatment in the first 24 hours to have a significant impact on reducing the length of the cold. Take a capful of hydrogen peroxide and while lying on your side, pour it in, letting it bubble for a few minutes.  Switch sides and repeat.

6. Chicken Soup – A team of researchers from the University of Nebraska Medical Center found evidence that chicken soup — both homemade and from the can — had anti-inflammatory properties that could prevent the side effects of a cold.  Avoid the canned varieties in favor of a home-cooked version with homemade broth.

9. Coconut Oil – has both antibacterial and antiviral properties.  Rub coconut oil over your skin. It is readily absorbed into your body and, as an added benefit, will soften your skin too. Add one-half teaspoon to your coffee or tea when you have a cold and cook with it.

10. Baking Soda – reduces the acidity of your body in the treatment of colds and flu. However, I have pushed the body pH in the opposite direction and achieved the same results, shortening the length of an infection.  The administration of baking soda is simple, relatively harmless and easy to test on your own cold. Simply dissolve the recommended amount of baking soda in a glass of cold water and drink it. Recommended dosages from the Arm & Hammer Company for colds and influenza back in 1925 were:

Day 1 — Take six doses of one-half teaspoon of baking soda in glass of cool water, at about two-hour intervals
Day 2 —Take four doses of one-half teaspoon of baking soda in glass of cool water, at the same intervals
Day 3 — Take two doses of one-half teaspoon of baking soda in glass of cool water morning and evening, and thereafter ½ teaspoon in glass of cool water each morning until cold symptoms are gone
This should only be used as an occasional (not chronic) treatment, however, and be careful not to consume excessive amounts, which can cause serious electrolyte and acid/base imbalances.

11. Lifestyle Choices – eliminate or drastically reduce your alcohol intake and smoking. Both of these factors negatively impact your immune system, making it more difficult for your body to fight the viral infection.

12. Steam – will not shorten the length of your cold, but it will help to break up the mucous secretions in your sinuses, reduce the inflammation in your nasal passages and help you to breathe better.

13. Stress Reduction – Practicing meditation, yoga or Emotional Freedom Techniques (EFT) are simple and effective practices to support your immune system and prevent other damage caused by stress.

14. Hand Washing – is a deterrent for infection by viruses and against further infection while you are sick. It will also reduce the spread of the virus to other family members, but it will not shorten the length of your cold. Remember that too much hand washing is almost as bad as not enough. Frequent washing strips your skin of protective oils, causing the skin to crack and bleed.

15. Eat Real Food – and avoiding processed foods will give your body the necessary tools to fight a viral infection. It will also reduce the potential you’ll suffer a quick recurrence of the infection.

In this article Dr. Rawls gives some more great advice:

  1. Isolate the sick & stay in bed
  2. Wash your hands
  3. Maintain a healthy immune system
  4. Drink lots of fluids
  5. Take Vitamin C (1,000 mg up to 4X/day)
  6. Take herbal therapy to support your immune system
  7. Apple cider vinegar (2 Tbs with 8oz water and 1-2 tsp local honey – drink several X/day)
  8. Reduce food intake
  9. Inhale steam before bed (can use essential oils in steam)
  10. Antiviral drugs can reduce severity and duration  Please see this news story first:



Calling Out Those Who Bully Health Professionals Who Don’t Ascribe to the Narrative


Approx. 19.30 Min (Scroll to 8:30 to hear the censored threat)

In the last 48 hours, Dr. Suzanne Humphries, creator of the Honesty vs. Policypresentation and author of Dissolving Illusions — which expertly dismantles the most persistent myths of the pseudoscience vaccine industry — received a shockingly detailed death threat that promises a “suicide mission” mass shooting at a public autism event.

The bullying has to stop.

Erin Elizabeth of Health Nut News states there are currently 80 suspicious deaths of holistic practitioners who didn’t toe the main-stream medical line:

Number 66 on the list is Wisconsin’s very own Dr. Hoffman – the most experienced Lyme/MSIDS doctor in the state who unfortunately is no longer with us.

66) May 7, 2017 – Dr. John Greg Hoffmann, who at one point lost his medical license but got it back for his alternative practices as an MD, died from injuries sustained in a single car accident that went off a cliff, as he was returning home from his cabin. Some close to him tell us there were no tire marks and they find it suspicious. We will update you as more information becomes available.

Article here:

To hear Dr. Hoffman’s talk he gave our support group:

Much has been written about Fake Science recently and that all is not as it seems:  This trolling happened to me recently on LinkedIn when a child/adolescent psychologist told me I was “scientifically illiterate, a fear-monger, and a fringe nut,” because I reposted an incredibly important article on Microbiologist Judy Mikovitz’s discovery of vaccines being contaminated with retroviruses for 30 years:  Doing my homework, I looked the good doctor up to discover his license expired in June, so I reported him for masquerading as a licensed doctor and for abusive comments. Within about 15 minutes his entire profile came down.  Imagine being a vulnerable child in that man’s office.

Bullies beware. We are on to you and we are calling you out.




A Strange Itch, Trouble Breathing, Then Anaphylactic Shock

A Strange Itch, Trouble Breathing, Then Anaphylactic Shock

By LISA SANDERS, M.D. JAN. 4, 2018

“I can’t breathe,” the woman panted, her voice a husky monotone. Her sister looked anxiously at the clerk at the triage desk at the University of Iowa hospital emergency room. The woman’s breath was rapid and coarse. Her chest heaved with the work of simply breathing. She pulled at the neck of her sweatshirt — suddenly it was too tight. She pulled it over her head and dropped it to the floor. She was naked beneath the top; she had been in bed when this attack came on.

The 54-year-old woman was helped into a wheelchair and whisked into the inner sanctum of the E.R. What followed was a blur of concerned faces, needles and medical data. Her blood pressure was dangerously low; her heart was racing. She was given epinephrine and steroids, but it was hours before she could explain what had happened that night.

She was staying at her mother’s house in rural Iowa, she told the doctors. Just as she was going to bed, she felt a sudden tingling in the palms of her hands. She recognized the sensation immediately: Twice in the past eight years, she had felt the same strange itch on her hands and sometimes her feet. Each time it was quickly followed by a terrifying sense of her throat closing.

Anaphylactic Shock

She had driven herself to her sister’s house, several miles away, and her sister drove her the rest of the way to the hospital. She opened the car window to let in the frigid winter night air. She struggled to breathe. Black spots swam before her eyes, but she willed herself not to pass out.

She had had this kind of allergic reaction twice before but never as severely. She knew from her own research that this was anaphylactic shock — a potentially deadly allergic reaction. After she got the medications, the woman’s symptoms resolved. She stayed in the hospital overnight, and when it was clear that the episode was over, she went back to her mother’s house. She made an appointment to see a local allergy specialist right away.

A Mystified Allergist

The specialist spent nearly two hours going over everything the woman had been exposed to — food, plants, toxins, anything that might have triggered this nearly fatal allergic reaction. There were no new exposures that day, nothing she hadn’t eaten or touched many times before and after this latest attack. The most common cause of severe allergic reactions in adults is food, but the allergist couldn’t identify any likely suspects. He was mystified. He asked her to share her diagnosis when she got one.

For months after returning to her home on Long Island, the woman was anxious about everything she ate, and she worried every night when she went to bed. She always kept a bottle of Benadryl and an EpiPen with her, but still she was terrified about what might happen if she was too far from a hospital the next time.

‘I Need a Nurse!’

When her next attack happened — just 10 months later — she was already in Brookhaven Memorial Hospital in East Patchogue, N.Y. She was being treated with antibiotics for a devastating case of gastroenteritis due to salmonella. Her first meal, after days of nothing but clear liquids, was beef brisket with potatoes and carrots. It smelled good, but she had no appetite. She made herself eat a few bites anyway, knowing it was her first step toward going home.

A couple of hours later, she felt a strange itch on the top of her head. She scratched reflexively. Then the recognition hit her like a slap: Not now, she thought. She grabbed the IV pole, still dripping fluids into her system, and ran out into the hallway. “I need a nurse,” she shouted. Her heart was pounding, and she knew what was coming next. Hospital staffers in scrubs descended on her. Was she having a panic attack? No, an allergy attack, she told them.

They helped her back into bed and gave her oxygen, Benadryl and steroids. “What happened?” someone asked. She told the whole story, plus something she now realized — every one of her attacks seemed to come a few hours after she ate beef. She didn’t go through this every time she had a hamburger or steak; meat was a regular and much-loved part of her diet. But she was pretty sure that she had steak — or beef brisket, this time — before each episode.

Her doctors were dubious. New food allergies — especially severe ones like hers — are uncommon in adults. This was much more likely to be an allergic response to one of the antibiotics they were giving her. The patient, though, found that theory hard to swallow. It might explain this episode, but what about the earlier ones? She hadn’t been on antibiotics then. The doctors had no answer.

Was a Tick to Blame?

A nurse had a different theory about what happened, one the patient had heard before but never believed. There was some kind of tick, the nurse told her, whose bite could make you allergic to meat. She didn’t know much about it. But, the nurse suggested, she should check it out.

The woman had been bitten by ticks before — who on Long Island hasn’t? But was it really possible for a bite to produce this crazy reaction? Indeed it was, she discovered, when she got home and began doing some research. The bite of the lone star tick — named for a white spot shaped like Texas on the arachnid’s back — could cause an allergic reaction to mammalian meat. The trigger was a sugar, identified as galactose-α-1,3-galactose and more casually known as alpha-gal, a carbohydrate found in the flesh of all nonprimate mammals.

How the tick bite triggers this allergy is not yet known. The link between the tick — whose range extends from southern Florida to Maine and as far west as Iowa — and the resulting alpha-gal allergy was first described in 2009 by Thomas Platts-Mills, a professor at the University of Virginia, who himself developed the disorder. Unlike most food allergies, in which symptoms occur within minutes of consuming the allergen, the alpha-gal reaction is delayed. The symptoms — ranging from a rash to nausea to shortness of breath and even anaphylaxis — can appear four to six hours after a meal containing meat. Stranger still, the reaction doesn’t occur after every exposure.

A Diet Changed Forever

The diagnosis of mammalian meat allergy (M.M.A.) can be confirmed with a blood test that identifies antibodies to alpha-gal. The patient contacted Diane Cymerman, an allergist she had seen years earlier for seasonal allergies. Cymerman asked her to list all the foods she consumed before her last episode in the hospital and had her blood tested for antibodies to everything on the list, down to the black pepper and parsley seasoning. And to alpha-gal.

The first results came back the following week: She had a moderate allergy to beef, but everything else was normal. The following month, the test results for alpha-gal antibodies came back. She was wildly allergic to galactose-α-1,3-galactose. Cymerman called the patient with the news. She had to avoid eating meat from mammals — and everything derived from them, including Jell-O and other foods and medications made from gelatin. Even safe foods cooked on a grill that has also been used for meat can be contaminated with enough alpha-gal to trigger a reaction.

The patient contacted the allergist back in Iowa and told him what she had. He was amazed. He had only recently heard a lecture on this phenomenon. He had never seen it before her case.

It hasn’t been easy for this Iowa transplant to give up beef and other meat that comes from mammals. Some days, she tells me, just thinking about a juicy hamburger or steak makes her stomach growl. But she remembers her terror and that long drive to the Iowa hospital and sticks to chicken, fish and vegetables.

Lisa Sanders, M.D., is a contributing writer for the magazine and the author of “Every Patient Tells a Story: Medical Mysteries and the Art of Diagnosis.” If you have a solved case to share with Dr. Sanders, write her at



For more on Alpha-Gal:

Approx. 5 Min.

Great video by Dr. Greger at on tick bites, meat allergies, and chronic urticaria  Australian allergy specialist Sheryl van Nunen got her red meat allergy mystery solved when there was a surge in allergic reactions in the U.S. to a drug, Cetuximab, used to treat colorectal cancer developed using a mouse cell line, also containing alpha-gal.


If you haven’t read the articles on vaccines, please do. and
Some vaccines used to be run through mouse brains. This is important to know as mice are one of the biggest reservoirs for borrelia, the causative agent known to cause Lyme Disease.

More damning evidence of the mouse/vaccine connection:







Mechanisms of Vaccine Injury – Part 2 Nov, 2017 by James Lyons Weiler

THIS IS THE SECOND PART OF A SERIES OF ARTICLES ON THE BIOLOGICAL MECHANISMS OF VACCINE INJURY. The first of the series focused on the scientific evidence for (1) vaccine-induced mitopathy, (2) vaccine-induced persistent gliosis, (3) vaccine-induced endoplasmic reticulum failures (with both damage to detoxification pathways and to compromised immune systems via downregulation of ERAP1).  Part 1:

This article presents the scientific basis of autoimmunity from vaccines.

WHEN MOST OF THE KNOWLEDGE from basic science (cell culture and animal studies) seemingly falls apart in clinical trials, something is terribly wrong. Translational research falls apart when the basic science has it wrong, or when the clinical studies are flawed, fraudulent, or non-existent. Our governments have, in the past, relied heavily on the absence of evidence to assure the public on vaccine safety. “There are no studies” does not mean that the studies have been conducted… sometimes, it simply means “We don’t yet know”. They also rely on consistently over-cooked results that would raise the ire of any bona fide and ethical statistician.

Which is why it was most unscientific of the IOM to call for an end to science – after they ignored all of the basic studies on the question of a link between vaccines and autism – even though not all vaccines had been studied.

First, Immunology 101.

The mammalian adaptive immune system is a marvel of natural selection. Foreign proteins, or part of proteins (antigens) are taken up by antigen-presenting cells (APCs) including dendritic cells (DCs), where they processed into peptides and then loaded onto major histocompatibility complex (MHC) molecules for presentation to CD8 T cells via clonotypic T cell receptors (TCRs). (Stick with me, all of this is relevant, I assure you).

One type of T cell, the cytolytic T cells (Tc) can directly lyse a target pathogen cell. Tc’s are activated by MHC Class I on APCs. Another type of T cell, T helper cells (Th’s) are activated by MHC class II and release cytokines that have direct cellular effects themselves. Cytokines also activate macrophages, monocytes and B cells. B cells in particular express surface receptors that bind to surface antigens. When Th cells signal B cells, the B cells secrete antibodies that are supposed to be uniquely specific for the antigen responsible for the Th signal. Those antibodies can bind their specific targets alone, or they can also bind to and activate macrophages simultaneously, via the Fc receptor.

‘Priming’ of the immune system toward autoimmunity has been postulated. Vaccination, of course, often involves multiple exposures due to boosters. There are four mechanisms by which host infection by a pathogen can lead to autoimmunity. These are:

Molecular Mimicry. The pathogen carries elements that are similar enough in amino acid sequence or structure to self-antigen that the pathogen acts as a self-‘mimic’. In molecular mimicry, T or B cells activated in response to the pathogen also happen to be cross-reactive to self proteins. This can lead to direct autoimmune damage and false-positive “friendly-fire” activation of the immune system (via, for example, cytokine signalling due to the release of cytokines as result of autoimmune attacks on self proteins, cells, and tissues).

Epitope Spreading. A pathogen can also cause autoimmune disease via epitope spreading. In epitope spreading, damage to self-tissue occurs due either to the immune response to tissue infected by a persisting pathogen, or direct lysis of healthy cells by the pathogen. APCs take up antigens released from damaged tissue, initiating a self-specific immune response.

Bystander Activation. In this model, an indirect or non-specific activation of autoimmune cells is caused by the generally inflammatory environment that results from infection. The non-specific activation of one part of the immune system leads to the activation of other parts.

Cryptic Antigens. Foreign antigens can lead to autoimmunity via the activation of immunity to antigens that are not usually dominant – they are instead normally invisible to the immune system. It is generally described as an increase in “subdominant” antigens, and is usually attributed, like bystander activation, to the inflammatory environment that arises after infection. The involvement of cryptic antigens is considered likely when one observes increased protease production, and differential processing of released self-epitopes by APCs.

In the first article of this series, I reviewed the role of thimerosal as a specific inhibitor of ERAP1, a critically important protein for healthy immune systems. Read this description of the role of ERAP1 in APCs (Rock et al. 2010):

“ERAP1-deficient cells have reduced surface levels of MHC class I molecules and the peptide-MHC complexes that are made are less stable than on wild type cells… These results suggest that ERAP1 makes an important contribution both to the quantity and quality of peptides available for antigen presentation.”

Peptides presented by healthy cells come from normal autologous (self-generated) genes and are ignored because the immune system is tolerant to them. Aberrant peptide presentation due to low ERAP1 will lead to confusion of the immune system. When CD8 T-cells detect what appear to be foreign proteins because they are mis-processed, any tissue expressing the incorrectly trimmed peptides, either on their surface of during cell death is at risk of autoimmune attack.

The process of thimerosal-compromised ERAP1 failure will match the cryptic peptide model because peptides that do not normally initiate an immune response will appear to be highly immunogenic.


When a total breakdown of normal controls against attacks against the self occurs, widespread autoimmune attacks lead to recurrent cycles of cytokine release and cell death – and any, or all of the processes described can be unleashed simultaneously. Organ damage, damage to blood vessels, and high fever can result in death.

A study of the rates of anaphylaxis reported to the Vaccine Safety Datalink (VSD) by McNeil et al. (2016) resulted in a finding of 33 confirmed cases of anaphylaxis from January 2009 through December 2011. Using the rate of 33 confirmed cases after 25,173,965 doses, they estimated the risk of anaphylaxis to be 1.31/million doses. It is interesting to note that 85% of the case of anaphylaxis had prior evidence of atopy (3 with prior anaphylaxis, 16 with asthma, and 9 with specific prior allergies).

The rate of 1.31 per million doses may seem small. However, a search of the VAERS (Vaccine Adverse Event Reporting System) database over the same time period (January 2009 through December 2011) reveals that 550 cases of anaphylaxis were reported to VAERS. VAERS is a passive reporting system, and users must acknowledge that the data cannot be used to attribute causality. Individuals who like to claim that VAERS is a sufficient and adequate system point to the positive finding of problems with a rotavirus vaccine. But unless causality can be established using data from VAERS, negative association results of no increase in risk of adverse events are suspect due to under-reporting.

Under-reporting of vaccine injuries is a serious issue for VAERS; estimates range from only 1% to 10% of adverse events captured. Using that range, the actual number of cases of anaphylaxis nationwide would be anywhere from 550 (20.1 per 1,000,000 doses), to 55,000 (2,000 per 1,000,000 doses). Although they are required to report all vaccine adverse events, there is no penalty to doctors who fail to report. Clearly, the post-market surveillance systems that are supposed to allow our scientists to detect upticks in vaccine injury (pharmacovigilence) do not work.

Guillan-Barré Syndrome (GBS)

The National Vaccine Compensation Program recently added GBS as a vaccine injury for which plaintiffs can be awarded compensation. GBS occurs when vaccines (or viruses) cause an autoimmune reaction against myelin proteins. These proteins act as insulation around nerves, and are essential for proper transmission of nerve impulses. GBS is not the only demyelination disease; in fact, most of the conditions that involve demyelination mostly differ in which tissue the syndrome represents (e.g., transverse myelitis (TM), multiple sclerosis (MS), amyotrophic lateral sclerosis (ALS) and neuromyelitis optica (NMO).

Molecular mimicry was seen as a likely culprit way back in 1989:

“Thus, it is biologically plausible that injection of an inactivated virus, bacterium, or live attenuated virus might induce in the susceptible host an autoimmune response by deregulation of the immune response, by nonspecific activation of the T cells directed against myelin proteins, or by autoimmunity triggered by sequence similarities of proteins in the vaccine to host proteins such as those of myelin. The latter mechanism might evoke a response to a self-antigen, so-called molecular mimicry (Fujinami and Oldstone, 1989).”

Molecular mimicry due to exposure of individuals to proteins via infection by pathogens is non-controversial and widely accepted. Some examples of sequence-level similarity include the measles P3 protein and the human mylein basic protein (MBP), which shares at least 78% sequence identity:



The likely effect of this level of sequence similarity is autoimmunity against the MBP, leading to change in the behavior of a host from mobile, and upright, to supine and sedentary – behaviors sure to elicit a social response by the host’s conspecifics, perhaps increasing the likelihood of transmission of the virus.

Some individuals are likely to more susceptible to autoimmunity due to exposure to proteins from pathogens owing to genetic variation that changes one or two amino acids in their self antigen sequence, making it more similar in sequence or in structure to the pathogen antigen.

The principles of autoimmunity from both viral infection and from vaccination have been absolutely demonstrated both in animals and in humans. McCoy et al. (2006) found that mice that were first exposed to a vaccinia virus encoding mylein protein, and then vaccinated against murine cytomegalovirus developed multiple sclerosis. In humans, cross-reactivity between MBP and a herpesvirus-6 protein has been reported (Tejada-Simon, et al. 2003). At the population level, a small increase in risk of MS and other demyelinating disorders due to vaccination (any vaccine) has been detected (Langer-Gould et al., 2014), but the authors concluded that the increased risk was real, but small.

A natural experiment in France occurred when the government brought Hepatitis B vaccination to the population. The uptake in HepB vaccine was followed by an increase in reported cases of MS to insurers (Figure 1). The government stopped purchasing the HepB vaccine, and as the remaining lots worked their way through the healthcare systems, HepB vaccine uptake declined, followed by a decline in the reported cases of MS (Figure 1). Other than a large randomized clinical trial that did not “correct for” or exclude individuals at risk of MS, this is the closest demonstration of causality between MS and vaccination that we can expect to see.

francemsFigure 1. See Le Houézec, D. 2014. Evolution of multiple sclerosis in France since the beginning of hepatitis B vaccination. Immunol Res. 2014; 60(2-3): 219–225.

This outcome is not surprising, given the large amount of similarity shared by proteins in the HepB proteome and human neurological proteins (Ricco and Kanduc, 2010). Ample evidence exists in the form of cross-reactivity between antibodies against HepB myelin mimics and human myelin-related proteins (Bogdanos et al., 2005).

Individuals might experience varying degrees of severity of demyelination after vaccination due to genetic variation. Half of their myelin proteins and myelin oligodendrocyte glycoproteins might show low cross-reactivity because one of their parents had the canonical amino acid sequence, while the other half of their proteins might be more reactive due to genetic variation that alters the amino acid sequence in a way that increases the similarity to the pathogen’s matching peptide sequences. Further, over time, the progression and severity of demyelination could occur due to increased autoimmunity to the other version of the protein due to a process similar to bystander activation.

Aluminum Hydroxide and Autoimmunity

Vaccine risk denialists would have the public believe that the amount of aluminum in vaccines is not a threat to public health. With the exclusion of thimerosal in most pediatric vaccines came an expansion of the pediatric schedule, adding many vaccines that include aluminum hydroxide – with numerous doses.

It may surprise some, therefore, that aluminum hydroxide is routinely used in animal studies designed to model allergic rhinitis, (seasonal allergies), asthma (Elsakkar et al, 2016), food allergies (Ahren et al., 2014), autoimmunity, demyelination syndromes, and many other conditions (e.g., chronic prostatitis/chronic pelvic pain syndrome). Many of these studies use aluminum hydroxide to induce conditions identical to mysterious diseases in humans to show the efficacy of novel treatments.

Vaccine-Induced Asthma. Some of these studies take the additional step and use (all but in name) vaccine mimics – aluminum hydroxide combined with ovalbumin. Elsakkar et al. (2016), for example, used both aluminum hydroxide and ovalalbumin to induce asthma in mice to study the effect of adalimumab (aka Humira(R)). Other studies (e.g., Brandt et al., 2006) had previously demonstrated non-specific airway allergic reactions after dietary exposures to ovalbumin following exposure to aluminum hydroxide.

Vaccine-Induce Food Allergies. Up to 200 children die from anaphylaxis due to peanut allergies each year in the US, and the cost of caring for children with food allergies is estimated to be between $25 and $31 Billion – annually. In spite of this cost, no standard medical procedures exist advising parents against feeding their children peanuts or peanut butter after vaccinations.

In fact, the opposite approach is being tried. With limited scientific studies backing, the National Institutes of Health’s has, in 2017, recommended that parents feed infants as young as 4 to 6 months peanut puree to reduce the risk of the development of peanut allergies. This is based on one clinical study – with questionable results. While the study cites a marked reduction in the rate of evidence of peanut allergy in children with other allergies, only 82.9% of the SPT-positive group who were fed peanuts made it to the outcome determination, compared to 98% of the SPT positive arm who were not fed peanuts. The difference in patient attrition (98%-82.9%) in the SPT-positive group is a whopping 15% – much, much higher than the incidence of peanut allergy in the population (estimates range from 0.4%-1.4%). The drop-out rate difference would easily swamp the actual risk due to genotype.

Studies of treatments to reduce anaphylatic reaction (e.g., Shishehbor et al., 2010) use aluminum hydroxide. It known that that the simultaneous exposure of rats and mice to aluminum hydroxide and to foods such as egg. wheat, dairy, and nuts will likely cause the development of vaccine-induced food allergies. It would be remarkable if somehow humans did not also develop autoimmunity due to the combined effects of thimerosal ERAP1 suppression and the adjuvantive effects of aluminum hydroxide.

Rheumatoid Arthritis

A case report of three cases of RA after HepB vaccination (Gross et al., 1995) provides a clue that it is likely to be caused by vaccines in some people. Other similar reports exist, but population-level studies have failed to detect an association. Mouse models of arthritis exist that employ – you guessed it – injected aluminum hydroxide. So why would epidemiologic studies of samples of patients from the general population fail to detect association of RA and vaccines?

In humans, RA risk due to variation in the HLA genotypes should be a factor considered when designing studies of risk of RA due to vaccination. No study of the effect of vaccination on RA risk on individuals with HLA genotypes of known risk to RA has been conducted. These individuals are at highest risk. Like autism, RA risk has a genetic component, but certainly involves environmental factors. Karlson et al. (2013) found that the best model that could predict risk of RA includes both genes and environment. Unfortunately, vaccination status was not studied. If RA risk is enhanced via HLA genotype, only studies of that consider HLA RA risk (high and low) and vaccination status (vaccinated vs. unvaccinated) could answer the question of association whether HLA x vaccine interaction exists.

Molecular mimicry to pathogen proteins is suspected in RA (Singh and Karrar, 2014). Cross-reactivity is observed against mycobacteria protein and human cartilage (Holoshitz et al., 1986). Antibodies reactive to bacterial enolase have also been found in patients with RA (Lundberg et al., 2008). The specific human protein to which cross-reactivity was found is the human enolase protein (aka. CEP1, Enolase 1 (ENO1) alpha enolase 5-21). Rashid et al., (2007) found elevated antibodies that are cross-reactive to Proteus microbes – as well as to antigens that are not cross-reactive – in patients with RA.

Many patients with RA show cross-reactivity with citrullinated proteins. High among the likely candidates of self-antigen sources is the human protein vimentin. It is highly plausible that prior, or simultaneous exposure to vaccines and to non-pathogenic, or mildly pathogenic bacteria would be sufficient to induce (trigger) RA in genetically susceptible individuals. Studies focused on thorough searches for cross-reactive antibodies matching both human targets in tissues – and in pathways – are required.

It is worth noting that HLA genotypes confer risk of a diversity of autoimmune conditions, and thus studies of familial risk and environmental risk would be made more powerful by combining conditions influenced by similar HLA genotypes. It is plausible that the reason why whole population studies fail to detect association may well be that the association exists for an genetically identifiable minority of individuals.

It is also worth noting that some genetic variants in the ERAP1 gene, which is down-regulated by thimerosal, are associated with ankylosing spondylitis (AS) – an autoimmune form of arthritis in which the vertebrae of the spine can become fused. Individuals with RA are removed from studies of AS (e.g., Wang et al. 2012). Variants in ERAP1 are also associated with juvenile idiopathic arthritis (Hinks e al., 2011) and psoriasis (Strange et al., 2010).


One of the best examples of vaccine-induced autoimmunity is the sporadic occurrence of narcolepsy among families in Europe following H1N1 swine flu vaccination. Narcolepsy is a debilitating disorder in which sufferers suddenly fall asleep without warning. Researchers at Stanford University suspected the target protein was orexin (aka hypocretin), a protein secreted by cells in the brain that regulate the sleep/wake cycles. Their initial successful attempts to identify cross-reactive antibodies could not be reproduced, but then a study found strong cross-reactivity to influenza nucleoprotein and a receptor of hypocretin, hypocretin receptor 2 (Ahmed et al., 2015).

The vaccine that caused narcolepsy across Europe was Pandemrix, manufactured by GlaxoSmithKline. Other H1N1 flu vaccines with lower amounts of influenza nucleoprotein did not lead to narcolepsy. Incredibly, ministers in DPW in Britain fought to prevent a dozen children who developed narcolepsy due to vaccination with Pandemrix from being compensated for injury from the Vaccine Damage Payment Act. Fortunately for those individuals injured by Pandremix vaccination, the courts sided with the families.


Anti-brain protein antibodies are known to be found in children with autism (e.g., Zimmerman et al., 2007; Singer et al., 2008; Heuer et al., 2011). In 1988, a major report on pertussis and pertussis immunization (Cherry et al., 1988) reported:

“For more than 25 years, it has been known that pertussis vaccine is a reliable adjuvant for the production of experimental allergic encephalitis… This experimental allergic encephalomyelitis is mediated by sensitized lymphocytes rather than serum antibody mechanisms. Pertussis vaccine has also been used as an adjuvant in the following experimental autoimmune diseases: thyroiditis, myocarditis, glomerulonephritis, uveoretinitis, and hemolytic anemia.”

They then cited a lack of population-level association studies (i.e., no evidence) that pertussis vaccination results in these conditions in children. Gallup (2004) reminded the research community of this long-standing knowledge of the autoimmune-induced powers of DPT, citing his son’s case as an example. His son has “regressive autism and tested positive for myelin basic protein antibodies, has elevated measles antibody titers, T-cell abnormalities and colitis“.

Unless and until we study either “vaccine injured” vs. “not injured” to find biomarker signatures of moderate and serious adverse events, as was conducted with success for mild adverse events by Christian et al. (2015), or “likely susceptible” vs. “not likely susceptible” based on genetics, the study of the role of vaccines in inducing autoimmunity in humans will remain forever broken.

For now, the evidence that exists is simply overwhelming: vaccines can cause autoimmunity in some people, and we need (a) genetic screens to keep people out of harm’s way; (b) state bans on the use of unsafe peptides and epitopes in vaccines; (c) a test of Th1/Th2/Th17 balance prior to vaccination; (d) aluminum patch testing for aluminum sensitivity prior to vaccination. Consideration of HLA genotype for RA risk, or at least family history of autoimmune disorder, should be a matter of routine to reduce the likelihood of any vaccine-induced autoimmunity. The HLA-DR4 genotype is higher in frequency in autism (Lee et al., 2006).

We also need to start holding pediatricians accountable to treat all moderate and serious adverse events due to vaccination with appropriate follow-up testing, and emergent medical intervention. Failure to attend to their vaccine-injured patients should be reported as malpractice. The public does not need CDC, nor NIH, nor the AMA or AAP to acknowledge that vaccines induce autoimmunity in humans occurs to file complaints to the medical board or to their State Legislature.

NB: While completing this article, news of the publication of findings of record measurements of aluminum in the post-mortem autopsied brains of children with autism by Dr. Chris Exley’s team make a rather stunning bookend. The time has arrived for every person to let every member of the medical community know that aluminum is making us sick, and that one way or the other, they will be held responsible if they persist in their silence on the use of aluminum in vaccines. With all due respect, pediatricians, please join the Vaccine Risk Aware community in our calls for a reduction in the amount of aluminum in vaccines, a ban on thimerosal, and a ban on the use of unsafe peptides that match human proteins in vaccines.


Ahmed SS et al., 2015. Antibodies to influenza nucleoprotein cross-react with human hypocretin receptor 2. Sci Transl Med;7294):294ra105. doi: 10.1126/scitranslmed.aab2354.

Bogdanos et al., 2005. A study of molecular mimicry and immunological cross-reactivity between hepatitis B surface antigen and myelin mimics. Clinical and Developmental Immunology 12:217-224.

Cherry, J.D. 1988. Report of the task force on pertussis and pertussis immunization, Pediatrics 81:6 Part 11 (June 1988) Supplement pp 936-984.

Christian, LM et al. 2015. Proinflammatory cytokine responses correspond with subjective side effects after influenza virus vaccination. Vaccine. 33(29): 3360–3366.

Gallup, R. 2004. The new MMR? No! The Old DPT

Gross et al., 1995. Arthritis after hepatitis B vaccination. Report of three cases. Scand J Rheumatol. 24(1):50-2.

Heuer L et al., 2011. Association of a MET genetic variant with autism-associated maternal autoantibodies to fetal brain proteins and cytokine expression.Transl Psychiatry. 1:e48. doi: 10.1038/tp.2011.48.

Hinks A, Martin P, Flynn E, et al. 2011. Subtype specific genetic associations for juvenile idiopathic arthritis: ERAP1 with the enthesitis related arthritis subtype and IL23R with juvenile psoriatic arthritis. Arthritis Res Ther 13:R12.

Holoshitz J, 1986. T lymphocytes of rheumatoid arthritis patients show augmented reactivity to a fraction of mycobacteria cross-reactive with cartilage. Lancet. 2(8502):305-9.

Karlson, E et al., 2013. Association of Environmental and Genetic Factors and Gene-Environment Interactions with Risk of Developing Rheumatoid Arthritis. Arthritis Care Res (Hoboken). 65(7): 1147–1156.

Langer-Gould A 2014. Vaccines and the risk of multiple sclerosis and other central nervous system demyelinating diseases. JAMA Neurol. 71(12):1506-13. doi: 10.1001/jamaneurol.2014.2633.

Lee et al., 2006. HLA-DR4 in families with autism. Pediatr Neurol. 35(5):303-7.

Lundberg, K et al., 2008. Antibodies to Citrullinated a-Enolase Peptide 1 Are Specific for Rheumatoid Arthritis and Cross-React With Bacterial Enolase. Arthritis & Rheumatism 58:3009–3019

McCoy L et al., 2006. Multiple sclerosis and virus induced immune responses: autoimmunity can be primed by molecular mimicry and augmented by bystander activation. Autoimmunity. 39(1):9-19.

Rashid et al, 2007. Rheumatoid arthritis patients have elevated antibodies to cross-reactive and non cross-reactive antigens from Proteus microbes. Clin Exp Rheumatol. 25(2):259-67.

Ricco, R and D. Kanduc. 2010. Hepatitis B virus and Homo sapiens proteome-wide analysis: A profusion of viral peptide overlaps in neuron-specific human proteins. Biologics. 4: 75–81.

Rock KL et al., 2010. Proteases in MHC class I presentation and cross-presentation. J Immunol. 184(1):9-15. doi: 10.4049/jimmunol.0903399.

Singer HS et al., 2008. Antibodies against fetal brain in sera of mothers with autistic children. J Neuroimmunol. 194:165–172.

Singh A, Karrar S. 2014. The role of intracellular organisms in the pathogenesis of inflammatory arthritis. Int J Inflam. 2014:158793. doi: 10.1155/2014/158793.

Strange A, Capon F, Spencer CC, et al. 2010. A genome-wide association study identifies new psoriasis susceptibility loci and an interaction between HLA-C and ERAP1. Nat Genet 2010;42:985–90.

Tejada-Simon MV et al., 2003. Cross-reactivity with myelin basic protein and human herpesvirus-6 in multiple sclerosis. Ann Neurol. 53(2):189-97.

Wang, C-M et al., 2012. ERAP1 genetic variations associated with HLA-B27 interaction and disease severity of syndesmophytes formation in Taiwanese ankylosing spondylitis Arthritis Research & Therapy 201214:R125

Zimmerman AW et al., 2007. Maternal antibrain antibodies in autism. Brain Behav Immun. 21(3):351-7. Epub 2006 Oct 6.

Dr. Lyons-Weiler is a research scientist and author of three books, the latest of which is “The Environmental and Genetic Causes of Autism”. He is available for speaking engagements and book signing events at your location. To contact, follow on twitter @lifebiomedguru, email ebolapromo[at], and connect via LinkedIn

Bought – Documentary on Pharma, Vaccines, & GMOs

The truth about vaccines and Big Pharma, and info on GMO foods

Bought, according to Dr. Mercola, reveals: Forced vaccinations are part and parcel of this larger scheme where industries write the rules and profit from public health policies, such as recommendations for universal use of all federally recommended vaccines and state mandatory vaccination laws that restrict or eliminate vaccine exemptions.”

People opting out of the flu vaccine are losing their jobs.  What’s behind this draconian measure?  Why, it’s Money of course.

“The goal of officials at the U.S. Centers for Disease Control and Prevention (CDC) is to achieve a 90 percent health care worker vaccination rate by 2020,22 and a key strategy for meeting this goal is to tie a health care facility’s employee flu vaccination rate to the facility’s Medicare and Medicaid reimbursements from the federal government.23

In other words, health care facilities participating in the Centers for Medicare and Medicaid Services Inpatient Prospective Payment System Hospital Inpatient Quality Reporting Program that fail to meet a 90 percent employee flu vaccination rate now get reimbursed 2 percent LESS from Medicare and Medicaid.

This is a drop in funding that can translate into hundreds of thousands of dollars each year.24 This loss of federal funding, far more so than any concern for patient welfare, is a more likely explanation for why hospitals are now choosing to fire essential medical personnel refusing a flu shot rather than allow them to simply wear a mask during flu season, as was done in the past.

So, who came up with this strategy? A key ‘mastermind’ behind the Patient Protection and Affordable Care Act, abbreviated as ACA, but colloquially known as Obamacare, was Elizabeth Fowler, chief health policy counsel to the Democratic chairman of the Senate Finance Committee, Max Baucus. Evidence suggests Fowler drafted the entire legislation.25,26

As reported by The Guardian in 2012, before joining Baucus’ office, Fowler was vice president for public policy and external affairs at WellPoint, the largest health insurance provider in the U.S. ‘Watch the five-minute Bill Moyers report from 2009 …  on the key role played in all of this by Liz Fowler and the ‘revolving door’ between the health insurance/lobbying industry and government officials at the time this bill was written and passed,’ The Guardian wrote.27”


Being informed about vaccines is everyone’s right; however, those infected with Lyme/MSIDS need to be even more vigilant, informed, and proactive as their bodies are in a war of epic proportions which translates to a messed up immune system.  Since the mechanism of vaccines is to illicit an immune response, this helps explain how in an infected person, vaccines themselves can trigger latent infections as discussed by this Scottish doctor: and here as well:  Asymptomatic girls after receiving Gardasil activated dormant Bartonella which was confirmed by testing.

Microbiologist Judy Mikovitz also blew the whistle on retroviruses contaminating vaccines:

For the basics on how vaccines work:

How vaccines are made:

For highlighted notes on a 9-part documentary on vaccines which includes many types of vaccines:

Don’t be fooled.  The push for vaccines has far more to do with money than science.