Archive for the ‘vaccines’ Category

Comparative Diets to Address Chronic Inflammation

Comparative Diets to Address Chronic Inflammation


The following is the first half of a two-part article on nutrition that addresses chronic inflammation.

One of the hallmarks of many chronic diseases and disorders is unresolved inflammation in the body. Chronic inflammation can develop when the immune system’s normal inflammatory response to an implied threat continues unabated rather than turning off once the threat is gone.1

Chronic inflammation is a common link among autoimmune disorders like rheumatoid arthritis, lupus and multiple sclerosis; in cardiovascular disorders that lead to heart attacks and strokes; in neurological disorders such as Alzheimer’s, Parkinson’s and epilepsy; and in mental disorders such as depression and schizophrenia.1 Vaccination has been reported to trigger the development of autoimmune disorders associated with chronic inflammation. 2

Infections and Vaccination: Two Different Kinds of Inflammatory Responses and Immunity

Infections and vaccines stimulate different kinds of inflammatory responses in the body to produce antibodies that confer two different kinds of immunity. Naturally acquired active immunity is attained after a person experiences a viral or bacterial infection and the body mounts an inflammatory response to stimulate the production of antibodies and confers long lasting natural immunity. Artificially acquired immunity, which is not identical to naturally acquired immunity, is attained when a person receives a vaccine and the body mounts an inflammatory response to produce antibodies and confers temporary immunity. Booster doses of vaccines to re-stimulate inflammatory responses are often given to lengthen artificial vaccine acquired immunity. 3

Depending upon various genetic, biological and environmental risk factors, some people do not resolve inflammation either after an infection or vaccination and can develop chronic inflammation in the body that leads to chronic health problems.45 In addition to lab altered viruses and bacteria, there are many recognized toxins in childhood vaccines that either singly or in combination cause inflammation in the brain and other parts of the body, including mercury, aluminum, formaldehyde, MSG, antibiotics, polyethylene glycol (antifreeze), squalene, virus like particles and adventitious agents.67

Acute inflammation is easy to recognize: heat, swelling, pain and redness at the site of injury or infection. Chronic inflammation is not quite so obvious, but there are common symptoms that indicate its presence. Some of the most frequently reported include headaches and brain fog, bloating and other digestive problems, joint pain, rashes, fatigue, weight gain, gum disease and mood issues8—many signs familiar to parents of autistic and/or vaccine-injured children.9

Diets Address Chronic Inflammation in Vaccine-Injured Children

The childhood vaccine schedule used in the U.S. has been questioned as a potential factor in the development of inflammatory chronic brain and immune system disorders in children.10

It is an unfortunate fact that those who question the safety of vaccines often “come to the table” following a firsthand experience with a vaccine reaction… in other words, too late to avoid the potentially devastating impact such a reaction can have on their own life or the life of their child. Since conventional medicine rarely acknowledges the connection between vaccination and chronic brain and immune disorders in children, it can be difficult to know where to turn after a vaccine reaction has occurred and there is often lag time before parents find a supportive network. In the search for healing, one of the first avenues explored by parents and doctors specializing in biomedical and holistic health interventions involves nutrition therapy.

Diet is among the most basic of approaches to addressing chronic inflammation. The connection between diet and the risk for developing inflammatory disorders has been recognized for at least 50 years, though studies have been inconclusive about the role played by specific foods and nutrients.11 Nevertheless, harnessing the power of food often can help counteract a chronic inflammatory process and improve some of the related symptoms.

Dietary Fundamentals for Reducing Inflammation

With all the “named” diets available, it can be daunting to decide which direction to turn. Most anti-inflammatory diets share certain basic tenets: avoid sugar and processed foods; stay away from refined flour, wheat, white foods like pasta, rice and bread; and eliminate unhealthy fats. Foods that are often recommended to reduce inflammation in the body are fresh fruits, dark green leafy vegetables, high-quality proteins like cold-water fish, and healthy fats. Some nutritionists suggest that the so-called nightshade foods, which include tomatoes, eggplant, peppers, goji berries and white potatoes, may trigger inflammation in some people,9 and commercial milk products may also cause inflammation in people who are sensitive to lactose or milk proteins.11

Food additives, including dyes, preservatives and artificial flavorings and sweeteners, and high-fructose corn syrup have been pinpointed as problematic for many children with autism spectrum disorders (ASD)12 and some nutritionists suggest avoiding them when trying to reduce systematic inflammation through dietary changes.

The Difference Is in the Details

Some of the most well known diets that surface in an online search for foods that fight inflammation include: the Gut and Psychology Syndrome (GAPS), Paleo, Mediterranean, Atkins, DASH, TLC, Mayo Clinic, Weight Watchers, Raw Food, Keto, The Zone, Whole30, Autoimmune Protocol, Dr. Hyman’s Detox and Dukan…to name just a few.  The annual U.S. News & World Report review of dietary rankings13 and other reviews14of current diet trends can provide an overview for understanding different dietary approaches.

What Do the Experts Say?

The choice of an “anti-inflammatory” diet that limits foods, which have been identified as “pro-inflammatory,” depends on consideration of individual factors, such as specific food sensitivities, personal taste preferences, or the simple desire to try a dietary regimen that sounds interesting.

According to Harvard University’s HealthWatch, “Choose the right foods, and you may be able to reduce your risk of illness. Consistently pick the wrong ones, and you could accelerate the inflammatory disease process.”15 Included in the HealthWatch list of pro-inflammatory foods that should be avoided to reduce inflammation include:

  • Refined carbohydrates, such as white bread and pastries
  • French fries and other fried foods
  • Soda and other sugar-sweetened beverages
  • Red meat (burgers, steaks) and processed meat (hot dogs, sausage)
  • Margarine, shortening, and lard

Anti-inflammatory foods include:

  • Tomatoes
  • Olive oil
  • Green leafy vegetables, such as spinach, kale, and collards
  • Nuts like almonds and walnuts
  • Fatty fish like salmon, mackerel, tuna, and sardines
  • Fruits such as strawberries, blueberries, cherries, and oranges


For More:  In this talk Cyndi O’Meara discusses challenges with wheat.  Diagnosed with MS, Dr. Terry Wahls received the best standard medicine had to offer. After declining to the point of being in a wheel chair, she took matters into her own hands and learned how to properly fuel her body. Using the lessons she learned at the subcellular level, she used diet to cure her MS and get out of her wheelchair.

VL15 Lyme Vaccine – Another Fraud?  by Dr. Jose Lapenta

Hello friends of the DERMAGIC EXPRESS network brings you another hot topic today with respect to LYME DISEASE or CHRONIC ERYTHEMA MIGRANS ….NEW VACCINE VL15 FOR LYME DISEASE … ANOTHER FRAUD? 
The first thing I will remind you, once again, is that in the year 1998 the FDA approved the LYMErix VACCINE to be used against this disease, based on”TARGET” or “DIANA” the proteins of Surface of the BORRELIA BURGDORFERI denominated OspA. The laboratory responsible for marketing the vaccine was GlaxoSmithKline (GSK).
1,400,000 doses were released, and the adverse effects reported by VAERS (ADVERSE EFFECTS REPORTING SYSTEM), which I published in the article STORYS OF VICTIMS OF THE VACCINE FOR THE LYME DISEASE here you can read them.
The vaccine resulted in the death of at least 229 people, of whom 43 were SUICIDATED 7 months after receiving the 2nd dose. 

The main side effect was ARTHRITIS, especially the patients with HISTOCOMPATIBILITY ANTIGEN CLASS II, HLA DR4, the laboratory omitted this data in the vaccination, ALSO THE VACCINE DETERIORATED the health of the carriers of the DISEASE. The laboratory also omitted this fact. 

At the end, the LYMErix vaccine was discontinued in 2002 for three reasons: 
And again you’ll be wondering why I’m telling you this HISTORY THAT HAS 15 YEARS OF EVOLUTION. I’ll give you the answer clear and precise !!!!
All this I am explaining to you because throughout these 15 years, THE LYME DISEASE has spread widely in the United States and Europe. In 2008, 440,000 new cases were reported in the United States and 85,000 in Europe. Currently, in 2017, 300,000 new cases are reported annually in the United States according to the CDC (Center for Disease Control and Prevention).
On the other hand, as I already explained, LYME DISEASE, due to the biological characteristics of BORRELIA (SPIROCHETA), which becomes “undetectable” to diagnostic tests, has become a health problem in these countries. An antibiotic treatment costs between $20 and $1000, and INSURANCE COMPANIES DO NOT WANT TO KNOW ABOUT CHRONIC DISEASES, BECAUSE THE COST IS VERY HIGH. Crude reality.
Here comes the GHOST of the LIMErix vaccine and a new FRENCH BIOTECHNOLOGY company, named VALNEVA, who proudly presented on April 11, 2017 at the World Congress on Vaccines, the project of a “NEW” vaccine for LYME DISEASE, called With CODE VL15-101. 

The FDA approved on 9 December 2016, the preliminary tests, leaving pending FINAL APPROVAL based on SUCCESS or failure thereof. The study began in 2016 in Europe, Belgium and will be performed this year 2017 in the United States, in 180 healthy people over 40 years (See attached).
HERE I PUT YOU WHAT VALNEVA SAYS ABOUT THE VL15 PROJECT (See attached)===============================================================
“… Valneva’s vaccine candidate is based on OspA, one of the most dominant surface proteins expressed by the bacteria when present in a tick. The target indication for Valneva’s vaccine candidate is the prophylactic active immunization against Lyme disease in children and adults. Valneva’s program is the only active vaccined evelopment program for Lyme disease in the pharmaceutical industry. Valneva intends to initiate a Phase I trial in the US and Europe in 2016 with the primary objectives of evaluating safety and tolerability. Immunogenicity for six OspA serotypes will also be monitored for different dose groups and formulations. Pre-clinical results indicated that Valneva’s vaccine candidate can provide protection against the majority of Borrelia species…”

Excellent project hopefully ALL what they propose and the vaccine be a SUCCESS, but NOW…

1.) WHEN LYMErix was withdrawn the FDA PANEL concluded that the LIMErix vaccine was not associated with the production of ARTHRITIS. This is false. VAERS REPORT 322 cases of ARTHRALGIA associated with the vaccine. And the development of VACCINE-INDUCED ARTHRITIS in HLA patients HLA DR-4, now called LYME ARTHRITIS. 
2.) LYMErix was a SCIENTIFIC “SUCCESS” says valneva in his exposition. This is also FALSE. WHY DOES THE POPULATION REJECT IT? Because of the great side effects !!! Why did the patients wrote to the FDA PRAYING that LYMErix was removed from the market. ? SIMPLY, BECAUSE IT WAS A FAILURE. 
3.) WHY DID VALNEVA not mention the patients who were DISABLED FOR EVER by the LYMErix vaccine and the 229 DEATHS I REPORT THE SAME FDA. ? 
I’m going to tell you why VALNEVA “OMITTED” these data: THE ANSWER IS SIMPLE AND UNIQUE: 
The new vaccine VL15-101 that is being proposed for LYME DISEASE is based on the same immunological concept of LYMErix VACCINE OspA. Surface proteins of BORRELIA BURGORFERI. The rest is history. (see attached)

On the other hand, it is interesting that you know that in your immune system there is the Major Histocompatibility Complex (MHC) and its HLA (Histocompatibility Antigens) Molecules Class I A, B, and C, and CLASS II DR and DQ MOLECULES. This have been studied by geneticists For more than 30 years and numerous DERMATOLOGICAL and NON-DERMATOLOGICAL diseases have been associated with these antigens.
In the case of LYME DISEASE, HLA DR-4 antigen and HLA B-27 have been associated with ARTHRITIS, while other infectious diseases present in a certain person can “detonate” an HLA antigen to express itself and thereby Worsen a PRE-EXISTING illness, or express a NEW ILLNESS. 
The big question is this – Are HLA studies in the population affected by LYME DISEASE being done?  These studies were done before placing the LYMErix vaccine? They are currently being done with volunteers for the “NEW VACCINE VL15?”. 
As a tip I tell you that Australia did a study on 555 dogs looking for BORRELIA BURGDORFERI, and I came to the conclusion that in this area of ​​the planet there is no LYME disease. In Canada they did it in 2013-2014 and it was concluded that BORRELIOSIS is endemic in that country.
On the other hand, it is well known that LYME DISEASE that does not respond well to treatment, becomes CHRONIC and costs are high. AND ALL OF YOU KNOW THAT INSURANCE COMPANIES DO NOT WANT TO PAY. 
Then it is easier, to create a new expectation, A NEW VACCINE 15 YEARS AFTER THE FAILURE OF LYMErix, for two important facts:
Hopefully VALNEVA will do the proper science and not mislead anyone, and the FDA WILL NOT APPROVE THE VACCINE if the proper science is not done.  If it passes the tests WELCOME the new one.
Finally I say the following, VACCINE VL15 is in PHASE I, which means that are testing the SAFETY in people, if it overcomes this obstacle, it will come to Phase II, to prove how well it works PREVENTING LYME disease. Then Phase III, to test it in a broader population RANGE and at different doses.
Cost: about $3 billion. As a note, I tell you that 86% DO NOT PASS the TWO FINAL STAGES, AND 94% of drugs tested on ANIMALS do NOT pass HUMAN tests. So VL15 of valneva has a LONG ROAD to go.
And now I ask you? Are you willing to try a VACCINE while being totally healthy that could trigger in you ANY ILLNESS you would never otherwise suffer? 
Did you know that in your immunological composition are HISTOCOMPATIBILITY ANTIGENS, (HLA) that by a simple incidence can “TRIGGER” unknow disease?
We will be looking forward to this new project and as always I wish the BEST TO THIS COMPANY THAT TRIES TO DEVELOP a new LYMErix or VL15 for LYME disease.
In the references and attachments the facts …
1.) Immunogenetic Markers Definition in Latvian Patients with Lyme Borreliosis and Lyme Neuroborreliosis.
2.) Associations of HLA DR and DQ molecules with Lyme borreliosis in Latvian patients.
3.) The genospecies B. burgdorferi s.l., isolated from ticks and from neurological patients with suspected Lyme borreliosis.
4.) [Critical analysis of reference studies on aluminium-based adjuvants toxicokinetics].
5.) Human-leukocyte antigen class II genes in early-onset obsessive-compulsive disorder.
6.) Differential diagnoses of suspected Lyme borreliosis or post-Lyme-disease syndrome.
7.) HLA-B27-associated reactive arthritis: pathogenetic and clinical considerations.
8.) Searching for Lyme borreliosis in Australia: results of a canine sentinel study.
9.) Canine infection with Borrelia burgdorferi, Dirofilaria immitis, Anaplasma spp. and Ehrlichia spp. In Canada, 2013-2014.
10.) Aluminum vaccine adjuvants: are they safe?
11.) Lyme Disease Testing by Large Commercial Laboratories in the United States 
1.) Immunogenetic Markers Definition in Latvian Patients with Lyme Borreliosis and Lyme Neuroborreliosis.
Kovalchuka L1, Cvetkova S2, Trofimova J3, Eglite J4, Gintere S5, Lucenko I6, Oczko-Grzesik B7, Viksna L8, Krumina A9,10.
Author information 
Institute of Food Safety, Animal Health and Environment BIOR, Riga LV-1076, Latvia.
Institute of Food Safety, Animal Health and Environment BIOR, Riga LV-1076, Latvia.
Institute of Food Safety, Animal Health and Environment BIOR, Riga LV-1076, Latvia.
Laboratory of Clinical Immunology and Immunogenetic, Riga Stradiņš University, Riga LV-1067, Latvia.
Department of Family Medicine, Riga Stradiņš University, Riga LV-1067, Latvia.
Centre for Disease Prevention and Control of Latvia, Riga LV-1005, Latvia.
Department of Infectious Diseases, Medical University of Silesia, 40-055 Katowice, Poland.
Department of Infectology and Dermatology, Riga Stradiņš University, Riga LV-1006, Latvia.
Institute of Food Safety, Animal Health and Environment BIOR, Riga LV-1076, Latvia.
Department of Infectology and Dermatology, Riga Stradiņš University, Riga LV-1006, Latvia. 
The aim of this study was to determine the human leukocyte antigen (HLA)-DRB1 alleles in two groups of patients in Latvia: patients with Lyme borreliosis and patients with Lyme neuroborreliosis. The study included 216 patients with Lyme borreliosis, 29 patients with Lyme neuroborreliosis and 282 control persons. All surveyed persons were residents of Latvia. The HLA-DR genotyping was performed by polymerase chain reaction- sequence specific primer (PCR-SSP). The predisposition to the Lyme borreliosis is associated with the HLA-DRB1*07, -DRB1*17(03), -DRB1*04, -DRB1*15(02) alleles. The allele -DRB1*11(05), -DRB1*14(06) and -DRB1*13(06) were significantly more frequent in controls. In-group with Lyme neuroborreliosis differences were found for the -DRB1*07 and -DRB1*04 alleles, but only HLA-DRB1*07 allele was statistically significant after Bonferroni correction and associated with Lyme neuroborreliosis in Latvian patients.
2.) Associations of HLA DR and DQ molecules with Lyme borreliosis in Latvian patients.
Kovalchuka L1, Eglite J, Lucenko I, Zalite M, Viksna L, Krumiņa A.
Author information 
Riga Stradiņš University, Clinical Immunology and Immunogenetic laboratory, Kronvalda Str 9, Riga, Latvia. 
Many autoimmune diseases are associated with variants of HLA genes such as those encoding the MHC complex. This correlation is not absolute, but may help in understanding of the molecular mechanism of disease. The purpose of this study was to determine HLA-DR,-DQ alleles in Latvian patients with Lyme borreliosis and control (healthy) persons. Case patients and control subjects were similar in age, gender and ethnic heritage and differed only as regards the presence of Borrelia burgdorferi infection. The study included 25 patients with clinical stage – erythema migrans and 30 control (healthy) persons. HLA genotyping was performed by PCR with sequence-specific primers.
The results show difference in HLA-DRB1 alleles distribution between patients and control subjects. The frequencies of HLA-DRB1 *04 (OR 11.24; p < 0.007) and HLA-DRB1 *17 (03) (OR 8.05; p < 0.033) were increased in the Lyme disease patients. And the frequency of allele DRB1*13 (OR 0.12; p < 0.017) was lower in Borreliosis patients and higher in control group. But, significant differences in frequencies of HLA-DQ alleles we did not detect.
HLA predisposition to Lyme borreliosis appears not to be limited to HLA molecules, but some HLA-DR alleles also have a significant influence, and, may have implications in our understanding of pathogenesis of this disease. In particular, HLA-DRB1*04 and DRB1 *17 (03) may contribute to the Lyme borreliosis development in Latvian population.
3.) The genospecies B. burgdorferi s.l., isolated from ticks and from neurological patients with suspected Lyme borreliosis.
Neuro Endocrinol Lett. 2011;32(4):491-5. 
Bazovska S1, Durovska J, Derdakova M, Taragelova V, Pancak J, Zaborska M, Traubner P.
Author information 
Institute of Epidemiology, Comenius University, Bratislava, Slovakia. 
Lyme borreliosis (LB) is the most disseminated tick-borne disease in the Northern hemisphere, and infestation with ticks is one of the essential factors influencing transmission of the disease to humans. This work intends to compare the occurrence of borrelia circulating in indigenous ticks and in patients suffering from neurological diseases.
The total of 660 nymphs and 567 adult ticks from the Bratislava and Košice areas was examined over the years 2001-2004, and the cerebrospinal fluid (CSF) of 82 neurological patients suffering from suspected Lyme borreliosis infection was investigated in the 2007-2009 period, using the polymerase chain reaction method (PCR).
PCR investigation proved presence of borrelia in 23.3% of the total 1227 ticks; of these, co-infection was found in 2.7% of all ticks. Borrelia garinii (9.9%) and B. valaisaina (9.2%) were the prevalent types. PCR investigation of the CSF samples of 32 patients with clinically diagnosed Lyme borreliosis showed the presence of B. burgdorferi s.l. in 17 cases. Positive results were found also in patients with unclear or different diagnoses. In cases where the genospecies could be identified, B. garinii was most frequently found (8x), followed with B. burgdorferi s.s. (4×) and B. afzelii (3×).
The high infestation level of ticks with borrelia, mainly with B. garinii which is the most-often documented borrelia species identified in neurological patients, is indicative of a high risk of this contamination in Slovakia. B. garinii were found also in our neuroborreliosis patients, whereas their proof in the CSF of patients with suspected neuroborreliosis or with a different clinical diagnosis pointed upon their persistence after an infectious experience. However, knowledge of not only the genospecies but also of the genotypes capable of eliciting an invasive disorder would be necessary for better clarification of the relationship between borrelia and their peccant capacity. Identification of the invasive borrelia types circulating in nature, and clarification of the vector vs. human infection incidence relationship is of importance from the aspect of detailed knowledge of the epidemiology of this disease.
4.) [Critical analysis of reference studies on aluminium-based adjuvants toxicokinetics].
Ann Pharm Fr. 2017 May 30. pii: S0003-4509(17)30033-0. doi: 10.1016/j.pharma.2017.04.004. [Epub ahead of print] 
[Article in French]
Masson JD1, Crépeaux G2, Authier FJ1, Exley C3, Gherardi RK4.
Author information 
Inserm U955 E10, centre expert de pathologie neuromusculaire, « Biologie du système neuromusculaire », hôpital Henri-Mondor, faculté de médecine, université Paris-Est-Créteil, 94010 Créteil, France.
Inserm U955 E10, centre expert de pathologie neuromusculaire, « Biologie du système neuromusculaire », hôpital Henri-Mondor, faculté de médecine, université Paris-Est-Créteil, 94010 Créteil, France; École nationale vétérinaire d’Alfort, 7, avenue du Général-de-Gaulle, 94700 Maisons-Alfort, France.
Aluminium and Silicon Research Group, The Birchall Centre, Lennard-Jones Laboratories, Keele University, ST5 5BG, Staffordshire, Royaume-Uni.
Inserm U955 E10, centre expert de pathologie neuromusculaire, « Biologie du système neuromusculaire », hôpital Henri-Mondor, faculté de médecine, université Paris-Est-Créteil, 94010 Créteil, France. Electronic address: 
We reviewed the three reference toxicokinetic studies commonly used to suggest innocuity of aluminum (Al)-based adjuvants. A single experimental study was carried out using isotopic 26Al (Flarend et al., 1997). This study ignored adjuvant cell capture. It was conducted over a short period of time (28 days) and used only two rabbits per adjuvant. At the endpoint, Al retention was 78% for aluminum phosphate and 94% for aluminum hydroxide, both results being incompatible with quick elimination of vaccine-derived Al in urines. Tissue distribution analysis omitted three important retention sites: the injected muscle, the draining lymph node and bone. Two theoretical studies have evaluated the potential risk of vaccine Al in infants, by reference to the oral Minimal Risk Level (MRL) extrapolated from animal studies. Keith et al., 2002 used a too high MRL (2mg/kg/d), an erroneous model of 100% immediate absorption of vaccine Al, and did not consider renal and blood-brain barrier immaturity. Mitkus et al. (2011) only considered absorbed Al, with erroneous calculations of absorption duration. They ignored particulate Al captured by immune cells, which play a role in systemic diffusion and the neuro-inflammatory potential of the adjuvant. MRL they used was both inappropriate (oral Al vs injected adjuvant) and far too high (1mg/kg/d) with regard to experimental studies of Al-induced memory and behavioral changes. Both paucity and serious weaknesses of these studies strongly suggest that novel experimental studies of Al adjuvants toxicokinetics should be performed on the long-term, including post-natal and adult exposures, to ensure innocuity and restore population confidence in Al-containing vaccines.
5.) Human-leukocyte antigen class II genes in early-onset obsessive-compulsive disorder.
Rodriguez N1,2,3, Morer A2,3,4, González-Navarro EA3,5, Gassó P1,3, Boloc D1, Serra-Pagès C3,5,6, Lafuente A1,2,3, Lazaro L2,3,4,7, Mas S1,2,3.
Author information 
a Dept. Anatomic Pathology, Pharmacology and Microbiology , University of Barcelona , Barcelona , Spain.
b Centro de Investigación Biomédica en Red de Salud Mental (CIBERSAM) , Barcelona , Spain.
c Institut d’Investigacions Biomèdiques August Pi i Sunyer (IDIBAPS) , Barcelona , Spain.
d Department of Child and Adolescent Psychiatry and Psychology, Institute of Neurosciences , Hospital Clinic de Barcelona , Barcelona , Spain.
e Immunology Service , Centre de Diagnostic Biomèdic, Hospital Clínic Dept , Barcelona , Spain.
f Dept. Biomedicine , University of Barcelona , Barcelona , Spain.
g Psychiatry and Clinical Psychobiology , University of Barcelona , Barcelona , Spain. 
The exact aetiology of obsessive-compulsive disorder (OCD) is unknown, although there is evidence to suggest a gene-environment interaction model. Several lines of evidence support a possible role of the immune system in this model.
The present study explores the allele variability in HLA genes of class II (HLA-DRB1, HLA-DQB1) in a sample of 144 early-onset OCD compared with reference samples of general population in the same geographical area.
None of the 39 alleles identified (allele frequency >1%) showed significant differences between OCD and reference populations. Pooling the different alleles that comprised HLA-DR4 (including DRB1*04:01, DRB1*04:04 and DRB1*04:05 alleles) we observed a significantly higher frequency (X21 = 5.53, P = 0.018; OR = 1.64, 95% CI 1.08-2.48) of these alleles in the early-onset OCD sample (10.8%) than in the reference population (6.8%).
Taking into account the role of HLA class II genes in the central nervous system, the results presented here support a role of the immune system in the pathophysiological model of OCD.
6.) Differential diagnoses of suspected Lyme borreliosis or post-Lyme-disease syndrome.
Eur J Clin Microbiol Infect Dis. 2007 Sep;26(9):611-7. 
Seidel MF1, Domene AB, Vetter H.
Author information
The symptoms of Lyme borreliosis are similar to those of a variety of autoimmune musculoskeletal diseases. Persistence of complaints is frequently interpreted as unsuccessful antibiotic treatment of Borrelia-associated infections. However, such refractory cases are rare, and re-evaluation of differential diagnoses helps to avoid the substantial risk of long-term antibiotic therapy. In this study, we analyzed patients who presented to our rheumatology unit with previous suspected or diagnosed Lyme borreliosis. Eighty-six patients from a 3.5-year period were evaluated. The mean age of patients was 49.2 +/- 17.2 years; 60% (n = 52) reported a tick bite and 33% (n = 28) an erythema. Forty-seven percent (n = 39) had positive enzyme-linked immunoassay results and Western blots (Mikrogen, Martinsried, Germany). All but 12 patients had already received antibiotic treatment previously. Nine percent (n = 8) had ongoing or recent Lyme borreliosis. Twenty-nine percent (n = 25) showed clinical symptoms and radiographic changes compatible with degenerative disorders of the cervical and/or lumbar spine. These patients were significantly older when compared to the other patients (59.3 +/- 13.7 years vs 46.1 +/- 17.2 years, p = 0.001). Seventeen percent (n = 16) had arthropathies related to psoriasis or rheumatoid arthritis. Twelve percent (n = 10) were positive for the HLA B27 antigen. Other diseases were less frequent. Six patients (7%) could not be diagnosed conclusively, and four of these patients had negative Borrelia immunoassay results. In conclusion, Borrelia-associated diseases were rare in this study. Differential diagnoses helped to initiate a successful disease-specific therapeutic strategy.
7.) HLA-B27-associated reactive arthritis: pathogenetic and clinical considerations.
Clin Microbiol Rev. 2004 Apr;17(2):348-69. 
Colmegna I1, Cuchacovich R, Espinoza LR.
Author information 
Section of Rheumatology, Department of Medicine, LSU Health Science Center, New Orleans, Louisiana 70112, USA. 
Current evidence supports the concept that reactive arthritis (ReA) is an immune-mediated synovitis resulting from slow bacterial infections and showing intra-articular persistence of viable, non-culturable bacteria and/or immunogenetic bacterial antigens synthesized by metabolically active bacteria residing in the joint and/or elsewhere in the body. The mechanisms that lead to the development of ReA are complex and basically involve an interaction between an arthritogenic agent and a predisposed host. The way in which a host accommodates to invasive facultative intracellular bacteria is the key to the development of ReA. The details of the molecular pathways that explain the articular and extra-articular manifestations of the disease are still under investigation. Several studies have been done to gain a better understanding of the pathogenesis of ReA; these constitute the basis for a more rational therapeutic approach to this disease. 
Reactive arthritis (ReA) is defined as a sterile synovitis developing after a distant infection, usually in the genitourinary or gastrointestinal tract. The detection of microbial components (microbial DNA and RNA) in the joints of patients with ReA has led to the reconsideration of this definition (59). Currently, ReA is better defined as an immune-mediated synovitis resulting from slow bacterial infections and showing intra-articular persistence of viable nonculturable bacteria and/or immunogenetic bacterial antigens synthesized by metabolically active bacteria residing in the joint and/or elsewhere in the body. A classification into HLA-B27-associated and nonassociated forms has also been proposed. Post-streptococcal, Lyme, and viral arthritis are HLA-B27 nonassociated and should be described as distinct entities under the general heading of “infection-related arthritides.” 
This article focuses on HLA-B27-associated ReA. This form of ReA belongs to the group of spondyloarthropathies (SpA), is triggered by bacteria (which enter the body through the mucosal surfaces) from the genera Campylobacter, Chlamydia, Salmonella, Shigella, and Yersinia, and is clinically associated with oligoarthritis of the lower limbs and sometimes with urethritis and conjunctivitis.
8.) Searching for Lyme borreliosis in Australia: results of a canine sentinel study.
Parasit Vectors. 2017 Mar 13;10(1):114. doi: 10.1186/s13071-017-2058-z. 
Irwin PJ1,2, Robertson ID3, Westman ME4, Perkins M5, Straubinger RK6.
Author information 
Vector and Water-Borne Pathogen Research Group, School of Veterinary and Life Sciences, Murdoch University, Murdoch, Western Australia, 6150, Australia.
College of Veterinary Medicine, School of Veterinary and Life Sciences, Murdoch University, Murdoch, Western Australia, 6150, Australia.
College of Veterinary Medicine, School of Veterinary and Life Sciences, Murdoch University, Murdoch, Western Australia, 6150, Australia.
Sydney School of Veterinary Science, University of Sydney, Sydney, New South Wales, 2006, Australia.
Pymble Veterinary Clinic, Philip Mall, Kendall Street, West Pymble, New South Wales, 2073, Australia.
Department of Infectious Diseases and Zoonoses, Bacteriology and Mycology, Ludwig-Maximilians-University Munich, 80539, Munich, Germany. 
Lyme borreliosis is a common tick-borne disease of the northern hemisphere that is caused by bacterial spirochaetes of the Borrelia burgdorferi (sensu lato) (Bbsl) complex. To date, there has been no convincing evidence for locally-acquired Lyme borreliosis on the Australian continent and there is currently a national debate concerning the nature and distributions of zoonotic tick-transmitted infectious disease in Australia. In studies conducted in Europe and the United States, dogs have been used as sentinels for tick-associated illness in people since they readily contact ticks that may harbour zoonotic pathogens. Applying this principle, we used a combination of serological assays to test dogs living in tick ‘hot spots’ and exposed to the Australian paralysis tick, Ixodes holocyclus, for evidence of exposure to B. burgdorferi (s.l.) antigens and other vector-borne pathogens.
Altogether, 555 dogs from four demographic groups were recruited into this study. One dog had evidence of exposure to Anaplasma spp. but no other dog was positive in screening tests. A total of 122 dogs (22.0%) had a kinetic ELISA (KELA) unit value > 100, and one dog with a high titre (399.9 KELA units) had been vaccinated against B. burgdorferi (sensu stricto) before travelling to Australia. Older dogs and those with a history of tick paralysis were significantly more likely to have a KELA unit value > 100. Line immunoassay analysis revealed moderate-to-weak (equivocal) bands in 27 (4.9%) dogs.
Except for a single dog presumed to have been exposed to Anaplasma platys, infection with Anaplasma spp. B. burgdorferi (s.l.), Ehrlichia spp., and Dirofilaria immitis, was not detected in the cohort of Australian dogs evaluated in this study. These results provide further evidence that Lyme borreliosis does not exist in Australia but that cross-reacting antibodies (false positive results) are common and may be caused by the transmission of other tick-associated organisms.
9.) Canine infection with Borrelia burgdorferi, Dirofilaria immitis, Anaplasma spp. and Ehrlichia spp. In Canada, 2013-2014.
Parasit Vectors. 2017 May 19;10(1):244. doi: 10.1186/s13071-017-2184-7. 
Herrin BH1, Peregrine AS2, Goring J3, Beall MJ3, Little SE4.
Author information 
Department of Veterinary Pathobiology, Center for Veterinary Health Sciences, Oklahoma State University, Stillwater, OK, USA.
Department of Pathobiology, Ontario Veterinary College, University of Guelph, Guelph, ON, Canada.
IDEXX Laboratories, Inc, Westbrook, ME, USA.
Department of Veterinary Pathobiology, Center for Veterinary Health Sciences, Oklahoma State University, Stillwater, OK, USA. 
Canine test results generated by veterinarians throughout Canada from 2013-2014 were evaluated to assess the geographical distribution of canine infection with Borrelia burgdorferi, Dirofilaria immitis, Ehrlichia spp., and Anaplasma spp.
The percent positive test results of 115,636 SNAP® 4Dx® Plus tests from dogs tested were collated by province and municipality to determine the distribution of these vector-borne infections in Canada.
A total of 2,844/115,636 (2.5%) dogs tested positive for antibody to B. burgdorferi. In contrast, positive test results for D. immitis antigen and antibodies to Ehrlichia spp. and Anaplasma spp. were low, with less than 0.5% of dogs testing positive for any one of these three agents nationwide. Provincial seroprevalence for antibodies to B. burgdorferi ranged from 0.5% (Saskatchewan)-15.7% (Nova Scotia); the areas of highest percent positive test results were in proximity to regions in the USA considered endemic for Lyme borreliosis, including Nova Scotia (15.7%) and Eastern Ontario (5.1%). These high endemic foci, which had significantly higher percent positive test results than the rest of the nation (P < 0.0001), were surrounded by areas of moderate to low seroprevalence in New Brunswick (3.7%), Quebec (2.8%), and the rest of Ontario (0.9%), as well as northward and westward through Manitoba (2.4%) and Saskatchewan (0.5%). Insufficient results were available from the westernmost provinces, including Alberta and British Columbia, to allow analysis.
10.) Aluminum vaccine adjuvants: are they safe?
Curr Med Chem. 2011;18(17):2630-7. 
Tomljenovic L1, Shaw CA.
Author information 
Neural Dynamics Research Group, Department of Ophthalmology and Visual Sciences, University of British Columbia, Vancouver, BC, V5Z 1L8, Canada. 
Aluminum is an experimentally demonstrated neurotoxin and the most commonly used vaccine adjuvant. Despite almost 90 years of widespread use of aluminum adjuvants, medical science’s understanding about their mechanisms of action is still remarkably poor. There is also a concerning scarcity of data on toxicology and pharmacokinetics of these compounds. In spite of this, the notion that aluminum in vaccines is safe appears to be widely accepted. Experimental research, however, clearly shows that aluminum adjuvants have a potential to induce serious immunological disorders in humans. In particular, aluminum in adjuvant form carries a risk for autoimmunity, long-term brain inflammation and associated neurological complications and may thus have profound and widespread adverse health consequences. In our opinion, the possibility that vaccine benefits may have been overrated and the risk of potential adverse effects underestimated, has not been rigorously evaluated in the medical and scientific community. We hope that the present paper will provide a framework for a much needed and long overdue assessment of this highly contentious medical issue.
11.) Lyme Disease Testing by Large Commercial Laboratories in the United States 
Alison F. Hinckley Neeta P. Connally James I. Meek Barbara J. Johnson Melissa M. Kemperman Katherine A. Feldman Jennifer L. White Paul S. Mead
Clin Infect Dis (2014) 59 (5): 676-681. 
Published: 30 May 2014 
Large commercial laboratories in the United States were surveyed to determine Lyme disease testing frequency, practices, and results. Approximately 3.4 million tests were conducted in 2008; 62% in accordance with recommendations. We estimate that 288 000 infections occurred among 2.4 million patients from whom samples were submitted.
Background. Laboratory testing is helpful when evaluating patients with suspected Lyme disease (LD). A 2-tiered antibody testing approach is recommended, but single-tier and nonvalidated tests are also used. We conducted a survey of large commercial laboratories in the United States to assess laboratory practices. We used these data to estimate the cost of testing and number of infections among patients from whom specimens were submitted.
Methods. Large commercial laboratories were asked to report the type and volume of testing conducted nationwide in 2008, as well as the percentage of positive tests for 4 LD-endemic states. The total direct cost of testing was calculated for each test type. These data and test-specific performance parameters available in published literature were used to estimate the number of infections among source patients.
Results. Seven participating laboratories performed approximately 3.4 million LD tests on approximately 2.4 million specimens nationwide at an estimated cost of $492 million. Two-tiered testing accounted for at least 62% of assays performed; alternative testing accounted for <3% of assays. The estimated frequency of infection among patients from whom specimens were submitted ranged from 10% to 18.5%. Applied to the total numbers of specimens, this yielded an estimated 240 000 to 444 000 infected source patients in 2008.
Discussion. LD testing is common and costly, with most testing in accordance with diagnostic recommendations. These results highlight the importance of considering clinical and exposure history when interpreting laboratory results for diagnostic and surveillance purposes.


For more:   So …in 1994, the CDC hosted a consensus conference in Dearborn, MI, along with a dozen labs across the country and together they falsified the very definition of Lyme disease by eliminating the neurological, immunosuppressive type which account for 85% of the cases.

If they conveniently ‘determined’ that the 85% group – those with an immunosuppression neurological outcome––simply did NOT exist, then they could claim that their vaccine was 85% effective. With no immunosuppressive, neurological disease to find, then the vaccine would be a hit with the remaining 15% who presented with an arthritic, bad-knee type only. A brilliant marketing scheme at the expense of millions worldwide for the years to come.
You see, you CANNOT create a vaccine for an OspA fungal antigen — the TRUE definition of chronic Lyme. It can’t be done. And the OspA vaccination known as LYMErix caused the same disease from a syringe as it does when you get Lyme disease from a tick bite. LYMErix victim’s immune systems were destroyed by the vaccine because OspA is an endotoxin that causes immunosuppression and subsequent severe neurologic multi-system disease.
So by narrowing the definition and by claiming that only one type of the disease (the bad knee arthritic type) exists, then they could sell a vaccine. Not only that. They were also able to profit by limiting the number of labs that were sanctioned and thereby cornering the market on the patents of a variety of tick borne diseases.
THE FIRST LYME VACCINE WAS A COMPLETE BUST.  In a vile cesspool of conflicts of interest are university patent holders, drug companies, and the FDA itself as another patent holder. It generated 40 million dollars before it was yanked. (2008, Drymon) One doctor stated that 21 patients developed severe arthritis after receiving the LYMERIX vaccine. The biological mechanism hypothesis was that the outer surface protein A (OspA), which was the antigenic component of the LYMErix vaccine, induced autoimmunity in genetically susceptible individuals, including high levels of autoantibody to OspA in their synovial fluid.

Dr. Stricker states:

Another Lyme OspA Vaccine Whitewash
The meta-analysis by Zhao and colleagues comes to the conclusion that “the OspA vaccine against Lyme disease is safe and its immunogenicity and efficacy have been verified.” The authors arrive at this sunny conclusion by excluding 99.6% of published articles that demonstrate potential problems with the OspA vaccine.Furthermore, the authors ignore peer-reviewed studies, FDA regulatory meetings and legal proceedings that point to major problems with OspA vaccine safety (1-3). This whitewash bodes ill for future Lyme vaccine candidates because it fosters disregard for vaccine safety among Lyme vaccine manufacturers and mistrust among potential Lyme vaccinees.

Also, I find it highly intriguing that the push here, and always, is for a vaccine; not a cure, not helping patients with pain, not making care accessible and affordable. Just in creating a new cash-cow for a select few.  Patients be damned.


A Tale of 3 Metals, the Fate of Western Civilization, & What We Can Do About it

A Tale of Three Metals and The Fate of Western Civilization

by jameslyonsweiler



The Romans drank beverages prepared in lead vessels, and brought spring water into their homes through lead pipes. Lead poisoning undoubtedly hastened the fall of the Roman empire. So when we think about the evidence that we are harming ourselves, and our children, with lead in the water, mercury in the air, mercury in flu vaccines, and aluminum in many other vaccines, one has to wonder: what are the likely effects on society?

  1. African Americans will suffer the most. Due to Vitamin D deficiency, African Americans at northern latitudes can be expected to be most sensitive to toxins because they rely on dietary Vitamin D to drive their cellular detoxification systems. The fix? Measure blood Vitamin D levels, and absent any mutation that would preclude increased doses of Vitamin D, improve brain health via addition Vitamin D supplementation.
  2. Young adults (millenials) will have different sociality, and higher rates of early-age psychological disorders such as schizophrenia. They may also experience higher rates of early age of onset Parkison’s disease, Alzheimer’s disease, and other neurodegenerative diseases. The fix? Filter aluminum out of the water, try silica-rich mineral waters, silica drops, with a preference for sources with the more biologically available silicic acid. In short, detoxify their food, water, and everything in their environment, and more (see below).
  3. Young children with special education needs will tend to be more violent and brain studies will show increased presence of amyloid precursor protein, the kind responsible for Alzheimer’s disease.
  4. There will be a population-wide downward shift in IQ.
  5. There will be a plague of multiple chemical sensitivity.
  6. Academics will be stretched thin and the curriculum dumbed down to the point where schools will have to stop giving grades. When 20% of the class can no longer function academically to take and exam, the rest will be asked to “help” their classmates learn.
  7. Families will become increasingly stressful social units. Divorce rates will skyrocket.
  8. People will become increasingly dependent on the State (Nanny State).
  9. Those most able to withstand the toxic effects of accumulating neurotoxins will become increasingly taxed because their income and property will have to sustain an increasingly demanding medico-government empire.
  10. When they, too, begin to fall apart, the tax base will falter.
  11. Violence will become increasingly common. Those most damaged will tend to kill and injure those who are capable.
  12. America will tear itself apart from within.

This doomsday scenario is not inevitable. So what can we do to prevent this?

  1. Listen to the mothers. They have experience in what works. NIH has avoided real research on neurodevelopment disorders that address neurotoxic metal exposure since the CDC worked so hard to defraud the public on the vaccine/autism link. They gambled, lost, and we now pay the cost.
  2. These solutions must be tested in combinations in clinical studies to insure safety, and also to validate them (if they do help). They must be studied NOW, before it’s too late.

Option 1. Environmental Detoxification. Remove all neotoxins from your home. Use reverse osmosis water filters, and use filtered water for everything – even cooking – because aluminum is used to condition the water coming from the tap. Fluoride is another issue, and your filtration should also remove fluoride. Eat organic foods and nothing out of aluminum containers. Certainly never cook in aluminum pots.

Option 2. Get the Aluminum Out. Consider using high silicic acid mineral water, or adding silicic acid drops to your filtered water to bind any aluminum from food. Other possibilities include malic acid, magnesium, and acetoacetic acid:

Principles of Orthomolecularism. R.A.S. Hemat: “Aluminum can be effectively complexed and excreted with silicon, a complex of malic acid and mg, and acetoacetic acid.”

Precise combinations that work best and are safe are not yet determined. That’s why we need studies.

Doctor Toni Bark, MD informs me that ketogenic diet can also help reduce brain inflammation and reduce the effects of toxic metals from the body and the brain – including the reduction of brain amyloid. And Dr. Richard Frey’s research on intranasal insulin and intranasal deferoxamine seems very promising for the actual removal of iron and aluminum from the brain. Care should be taken to conduct any such research under the direct care of a physician.

Option 3. Up the Vitamin D3, watch the A, Avoid Folic Acid. Dr. Keith Baggerly, MD, has determined that the FDA flubbed in it recommended daily Vit D intake:–+K+Baggerly+2016-2017 As a result, most Americans are Vit D deficient. Increased Vitamin D3 can be expected to improve many aspects of health by helping our cells properly fold proteins. Vits A and D are antagonistic, and so watch all sources of Vit A and make sure you and your child are not taking in too much Vitamin A. Read The Big Vitamin D Mistake: and Grant Genereux’s resources on Vit A toxicity [1] [2].

Much of our population has MTHFR mutations that cause problems with Folic Acid. Moms taking prenatal vitamins should seek methyl folate or folinic acid: instead of folic acid. Children’s vitamins with methyl folate are also available.

Option 5. Reduce Brain Inflammation. Chronic low-grade inflammation is a hallmark of autism. Powerful brain antioxidants include N-acetylcysteine and glutathione. It seems likely that everyone with a brain could benefit from less brain inflammation.

Option 6. Improve the Gut. The commensal (helpful) bacteria in the large intestine can become significantly altered after antibiotic use to treat ear infections, most likely caused by harm from to the immune system from thimerosal. Pro-biotics may help, as will eating organic.

Option 7. Keep This Handy for Bad Head Days. Brain dysfunction from metal-induce excitotoxicity involves high glutmate levels in the brain. Oxaloacetate can reduce blood glutamate levels, allowing the excess glutamate in the brain to spill into the blood. Oxaloacetate is used after stroke to reduce the exitotoxic brain injury. Research is needed to determine if it should be used after vaccination to reduce the incidence of vaccine-induced excitotoxicity. And aluminum should be removed from vaccines because the schedule results in toxic doses in infants.

Option 8. HBOT is HOT. Consider Hyperbaric Oxygen Therapy (HBOT). HBOT can increase de novo neurogenesis. If the brain has suffered a loss of neurons due to toxic exposure, increased neurogenesis – at the right time in development – could ultimately be shown to increase IQ.

Option 9. Avoid Thimerosal. If you choose to use a flu vaccination, ask the doctor for the type of flu shot that does not contain thimerosal.

Option 10. Tell Congress You Want Research Reform.

No studies of the synergistic toxicity of aluminum, lead and mercury have been conducted at doses reflecting the vaccine schedule and daily exposure due to leaching of lead from pipes into homes.

We know which homes have lead pipes. Departments of Health should consider telling parents of children in those homes to avoid exposures to mercury and to aluminum – in other words, to skip vaccines that contain these neurotoxic metals. The children will become more educated, better behaved, make better decisions, commit fewer crimes, and overall have better lives. Toxicity of lead, aluminum and mercury is synergistic.

No studies of the options and combinations of options listed above have been conducted to determine if we could improve overall brain health in children and adults. This research is badly needed. YOU can make it happen.

Make an appointment with your Congressional Representative and ask them to create the Brain Health 2030 initiative designed to reverse the ill effects of the past 30 years of industry and medicine on brains, and on our childrens’ brains. These interventions are not intrusive. Studies could be done also with the Department of Education to determine whether reports of violence decrease, grades increase, drop-out rates decrease if entire SCHOOLS – including administrators – are enrolled in Healthy Brain programs, which could incorporate aspects of mindfulness.

You can join the Neurodevelopment Research Reform group on Facebook where ideas on the Brain Health 2030 initiative are shared. And you can support our efforts to compile the most promising evidence-based approaches to improving brain health by supporting IPAK’s Neurodevelopment Research Reform Initiative.

This article is a call for research reform. These statements have not been evaluated by the Food and Drug Administration. This product is not intended to diagnose, treat, cure, or prevent any disease. Check with your physician before changing any mode of medical treatment for your child, or yourself.

Further Reading:

Lyons-Weiler, J and R. Ricketson. 2018. Reconsideration of the Immunotherapeutic Pediatric Safe Dose Levels of Aluminum. Journal and Trace Elements in Medicine and Biology 48:67 73.

Thanks to Tim Lundeen for the Vit A information and of course to the outstanding medical doctor, Dr. Toni Bark, MD for permission to cite her expertise.

Dr. Lyons-Weiler is a research scientist and author of three books, the latest of which is “The Environmental and Genetic Causes of Autism”. He is available for speaking engagements and book signing events at your location. To contact, follow on twitter @lifebiomedguru, email ebolapromo[at], and connect via LinkedIn


For more:





CDC’s Troubling Lack of Research Ethics

MARCH 29, 2018

A Matter of Right and Wrong: The CDC’s Troubling Lack of Research Ethics

By Brian S. Hooker, Science Advisor, Focus for Health and Board Member, World Mercury Project

Documents obtained by the Freedom of Information Act indicate that Centers for Disease Control (CDC) officials gave preferential treatment to the Danish grantees (including Dr. Poul Thorsen) at Aarhus University, and took no apparent action to evaluate the veracity of any of the study data when theft of over $1 million of grant money by Thorsen was made known. Furthermore, in 2009, when CDC officials including Dr. Coleen Boyle (Director of the National Center for Birth Defects and Development Disabilities [NCBDDD]), Marshalyn Yeargin-Allsopp (Chief of the Developmental Disabilities Branch), Joanne Wojcik (Public Health Analyst, Developmental Disabilities Branch), and Diana Schendel (former CDC NCBDDD senior epidemiologist) became aware that Thorsen failed to obtain legally required ethics permissions for the autism bio and genetic data projects, these CDC employees worked with the Danish grantees to hide this fact.

…studies were conducted and results published without legally-required ethics clearances.

These officials have never made public that both the validity of childhood autism in the Danish Psychiatric Central Register (Lauritsen et al. 2010 Journal of Autism and Developmental Disorders) and the population-based study of measles, mumps, and rubella vaccination and autism (Madsen et al. 2002 New England Journal of Medicine) studies were conducted and results published without legally-required ethics clearances. They knew that the psychiatric registry records were reviewed without required permissions, a serious ethical violation.

On the 30th of November 2009, Coleen Boyle, Diana Schendel, Joanne Wojcik, and Marshalyn Yeargin-Allsopp, along with Danish grantees, failed to report this serious ethical violation in previously published research, as well as a study near publication. They sought to cover their tracks on their failure to ensure Poul Thorsen had obtained all the needed ethical approvals for autism bio and genetic studies. Two studies were published in which legally required ethical permissions were apparently never applied for and granted, according to their notes. When repeatedly asked to provide them, Thorsen did not.

These acts were a clear violation of both ethical and legal standards.

It appears that the biggest concern the CDC-Danish collaborators had at that point was getting ethical coverage for Diana Schendel’s paper on autism and inflammatory markers that was about to be published. They decided to extend the permissions obtained by another researcher (Dr. Rikke Maimburg from Aarhus) to Schendel’s work because Maimburg had been approved in 2000 for an unrelated study entitled ‘Obstetric factors and autism’. Their thinking was that since Diana’s study used mothers’ obstetric files, the 2000 Ethics Committee approval could be extended to her study (even though the committee never reviewed Schendel’s study design.) However, permissions for child records, which were also reviewed, were never obtained. These acts were a clear violation of both ethical and legal standards.

Thorsen, the principle investigator for the granted CDC funds, failed to provide evidence after the fact that he had obtained ethical committee permissions for his research on bio and genetic markers and autism. The team eventually determined no requests had ever been submitted. By all appearances, they found that studies related to the two papers, one previously published, were done without legally-required ethical approval. The two papers in question are:

  1. A population-based study of measles, mumps, and rubella vaccination and autism. Madsen KM, Hviid A, Vestergaard M, Schendel D, Wohlfahrt J, Thorsen P, Olsen J, Melbye M., 2002, New England Journal of Medicine 347:1477
  2. Validity of childhood autism in the Danish Psychiatric Central Register: findings from a cohort sample born 1990-1999. Lauritsen MB, Jørgensen M, Madsen KM, Lemcke S, Toft S, Grove J, Schendel DE, Thorsen P., 2010, Journal of Autism and Developmental Disorders 40:139
the seminal Denmark study on the MMR and autism, used to “debunk” the vaccine-autism hypothesis both in the Institute of Medicine’s 2004 report “Vaccines and Autism” as well as in the National Vaccine Injury Compensation Program’s Omnibus Autism Proceedings, is an illegal study.

Notes from the Nov. 30, 2009 phone call between the CDC officials and Danish grantees include:

We have not been able to find a permission from ethical committee that covers both bio and genetic markers and autism. As far as we understand only one paper has been published using these data and Diana will soon be submiting (sic) another. Apart from the letter from the EC in our region (attached) we have not been able to locate the permission for abstraction of the psychiatric records, and it is likely that it does not exist.

Dr. Coleen Boyle, current Director of NCBDDD at CDC, should have shut this grant down immediately upon being informed that ethical clearances were not in place. She should have immediately contacted the Department of Health and Human Services Office of Research Integrity as well as the Office of Inspector General. Funding should have been discontinued. She should have led the charge to have these papers retracted. There should have been, at a minimum, a press release from the CDC to inform the public. Instead, Dr. Boyle along with Dr. Marshalyn Yeargin-Allsopp, Joanne Wojcik and Dr. Diana Schendel worked with their Danish Grantees to ‘fix it’ and retrospectively apply ethics approvals. Their next call on 14 December 2009 closed the case on Poul providing information to Aarhus.

Poul Thorsen’s reply from the Dec. 14, 2009 conference call was:

“As I have stated before to Carsten and Erik, I have not been able to locate the ethical approval for the autism pilot study. I recommend that new approvals are requested just as stated in the notes from the last conference call.”

Not only is this egregious behavior for government officials, but it also points to poor grant management by CDC staff. The CDC staff responsible for this multimillion dollar grant, starting with Diana Schendel, should have had copies of all the legally required ethics permissions in hand before the Danish grantees were allowed to get started, and before the first US taxpayer dollar was sent to Denmark.

Also, most notable is the fact that the seminal Denmark study on the MMR and autism, used to “debunk” the vaccine-autism hypothesis both in the Institute of Medicine’s 2004 report “Vaccines and Autism” as well as in the National Vaccine Injury Compensation Program’s Omnibus Autism Proceedings, is an illegal study. Without proper ethics approval, it should have never been published and indeed should be retracted from the New England Journal of Medicine.

This is one of two studies that the CDC has completed regarding the MMR vaccine and autism. Of course, the other study was completed by DeStefano et al. (2004 Pediatrics 113:259) and showed a statistically significant relationship between MMR timing and the incidence of autism, specifically in boys, despite the fact that the authors erroneously dismissed the relationship as evidence that individuals with autism had to get early vaccines to enroll in special education programs.

CDC officials in the Immunization Safety Office and the Developmental Disabilities Branch have repeatedly omitted data and manipulated statistical results to deny any relationship between vaccines and neurodevelopmental disorders.


How can the CDC be trusted with stewarding vaccine safety for the U.S. if this is an example of how ethics are regarded within their ranks? Unfortunately, this is just one of many instances of research fraud and malfeasance within the CDC. CDC officials in the Immunization Safety Office and the Developmental Disabilities Branch have repeatedly omitted data and manipulated statistical results to deny any relationship between vaccines and neurodevelopmental disorders. These same officials have covered up the embezzlement of Federal funds by CDC grantee, Dr. Poul Thorsen as well as the romantic relationship between Thorsen and his CDC grant supervisor, Dr. Diana Schendel. As described by Robert F. Kennedy, Jr.,

this agency is truly “a cesspool of corruption” and should be disqualified from protecting the health of our children.



And this institution has led the charge on all things Lyme/MSIDS related.

For more on the CDC and corruption:

CDC Corruption with Lyme:   An anti-trust law suit of $57 million is being prepared against the CDC. They are accused of deliberately suppressing the use of an accurate DNA direct diagnostics for Lyme disease.  After publishing the 2013 article ‘A simple method for the detection of live Borrelia spirochetes in human blood using classical microscopy techniques’, professor Laane was invited to give a lecture at the 2014 Norvect conference in Oslo. I was present at that conference and still remember how nervous he was. The reason was that several medical professors complained to his university. He was threatened with losing his job, if he would speak at the conference. In fact, he did not literally speak – as you can see in the movie below – but used performing arts to show the slides of the spirochetes. Professor Laane was fired anyway and his laboratory was closed down. Masters went over the charts with a fine tooth comb and found huge CDC errors. Another contentious point was the arbitrary cut off date imposed by the CDC which did not pick up serious late-stage symptoms. Another was the CDC’s rejection of many positive blood tests performed in its own lab, as well as other lab work showing “motile spirochetes” in nearly 5% of lone star nymphs. Long story short, after numerous revisions, Masters could never sign onto the bastardized study. The CDC had purposely tossed out data and manipulated the results.
The CDC essentially tried blackmailing Masters into signing off on the study before they would let him see the final draft. Refusing the bait, he published a letter of objection in the Journal of Infectious Diseases as well as an article of his own in Missouri Medicine which showed Missouri patients met the CDC surveillance definition for LD and growing evidence that lone star ticks were infected with an unidentified spirochete causing identical symptoms of LD in patients. When the CDC study came out they unbelievably attributed the rashes they labeled STARI to an allergy to tick saliva! In the acknowledgment section of the paper where dozens of folks were thanked, they completely omitted the man who made it all possible – Dr. Ed Masters.  RIP – we will never forget you. The racketeering scheme to downplay the severity of Lyme disease as identified in the RICO lawsuit should be addressed and documented by the TBD Working Group and not ignored as if this crime does not exist otherwise it will be business as usual at the Centers for Disease Control and we’ll have another thirty years of failure to properly diagnose, treat and control this life-altering infection. 10 points – Suing the Architects of Lyme Policy as Task Force Meets to Review it  (complete convoluted history about Lymerix found in this link)  In 2012, when the CDC revised the official case number of new Lyme disease infections per year from 30,000 to over 300,000 new Lyme––overnight, those of us in the know understood that they were getting the public ready to accept this upcoming vaccine with open arms and a sense of relief. In other words, scare the public just enough to make them open to wanting this product without causing mass panic nationwide or worldwide. And without having to explain their denial to date.  Did you know that the LYMERIX vaccine caused 640 emergency room visits, 34 life threatening reactions, 77 hospitalizations, 198 disabilities, and 6 deaths? In a vile cesspool of conflicts of interest are university patent holders, drug companies, and the FDA itself as another patent holder. It generated 40 million dollars before it was yanked. (2008, Drymon)


The Truth Never Stood in the Way of a Good Story – How to Be Wakefielded

A lot of stuff comes across my desk.  The following is one such item that does a great job explaining how large establishments with deep pockets and financial ties to powerful sources can and do ruin anyone they deem a threat.  The media disseminates and propagates it, and before long lies are accepted as fact.  The public is caught in the cross hairs and suffers due to this bullying which has effectively hijacked Science.  Used By Permission From Wellness Journeys

The Truth Never Stood in the Way of a Good Story

Whenever I read that Wakefield’s research has been thoroughly debunked and that he is considered a fraud and a quack, it saddens me because that is just not what happened, and Wakefield has never been proven to be a quack.

That is just the story everyone wants us to believe.

A lie can travel halfway around the world while the truth is putting on its shoes.

Perhaps one or two of you don’t know about Dr Andrew Wakefield, who was attacked and maligned for his study that discovered problems with the MMR vaccination. Whenever someone says vaccination and autism in the same sentence, a deluge of screams echoing from the far ends of the earth is heard, saying, “That has been so debunked!”


To debunk a scientific study, one needs to debunk the science of that study. The man responsible for this so-called debunking is Brian Deer, who is not a scientist but a professional skeptic (and journalist), who was hired to debunk Wakefield’s study and write up the story. Coincidentally, Deer belongs to a group of professional skeptics that Fiona Godlee addressed during one of their meetings.  Fiona Godlee is the Chief Editor of the British Medical Journal.

According to Tim Bolen, an investigative reporter, it was Godlee who hired Deer to debunk Wakefield, while other sources say it was the Sunday Times who hired Deer. Both sources are linked here:…/brian-deer-hired-to-find-somet…

The thing is, if you start to investigate this entire imbroglio, you’ll find that certain details have been ignored by a media more interested in sensationalism.

A simple point of fact is that when the British Medical Journal published Deer’s story, the article looked nothing like anything the journal had published in its esteemed history. It was, just like the rest of the media, a bit sensational, and lacking any real science. To a scientist, it looked like a libelous, ad hominemattack (an attack on the person of Andrew Wakefield).

We’ve been told that since Wakefield’s research, there have been further studies that showed the exact opposite, but we have to ask, were those studies fraudulent? while Wakefield’s study was not? I mean, who is to say which side is science and which side is fraud?

There are no objective arbiters in a field whose bottom line is the almighty dollar (or pound or euro). And when these cases go to court, the judges are not familiar with the science so both sides can bullshit their way through any court hearing using terminology that goes way over the court’s collective mind.

Since it seems that everyone in the world has come out against Wakefield (in fact there’s a new verb in the lexicon, “to be wakefielded”) and most doctors are afraid to go against the tidal wave of criticism, one must ask why have the actual findings in Wakefield’s study been ignored by everyone and the media and why was something debunked that actually never happened.

For instance, the study never claimed that MMR vaccine caused autism. Did you know that? Well here is the actual statement from the paper itself:

We did not prove an association between measles, mumps, and rubella vaccine and the syndrome described [autism]. Virological studies are underway that may help to resolve this issue.

Next, it is suggested in the paper that the three in one vaccinations be broken up and administered at different times because there has never been a problem when that was exactly how they were handled, whereas they did find a significant problem when the three were given at once. 

That is really the only suggestion of the paper, and after the hullabaloo died down, Britain’s General Medical Council took that option away from the people. Three-in-one was it, and that’s that.

And finally, in the end, because the sampling was so small, the researchers did exactly what every study does that is small like this, it said that more research must be done before we really know anything.

The idea that vaccinations should be broken up and administered over an extended period of time is not a new one. But there really have never been any studies on how often, how many, and the timing of all these vaccines being administered to infants. Never. Doctors rely on adverse reactions being reported, but in the Vaccine Adverse Event Reporting System database there have been reports of children turning into The Incredible Hulk. There are no real studies and the data cannot be trusted.

It’s just assumed, as Dr Paul Offit states, that “each infant would have the theoretical capacity to respond to about 10,000 vaccines at any one time.”

Well, I’ve got a few things for you to peruse. First off, here is Wakefield’s original paper published in The Lancet (which has been since been retracted):…/ar…/PIIS0140673697110960/fulltext

Next, there is a recently released movie on Amazon (if you have Amazon Prime it’s free) that I think everyone must see, which give’s Wakefield’s side. You’ll see he’s not a monster:

I would love to hear your comments. I’m not an anti-vaxer. I’m just interested in the truth and in good science; not science that is backed by billions of dollars, but science that is backed by sound methodology.

Wait A Minute! There’s More . . . And You Have To Hear This!

Because of Brian Deer’s so-called “debunking” of what will henceforth be known as the “Wakefield Study,” The Lancet’s retraction of the published results, and all the uproar and hullabaloo, the United Kingdom’s General Medical Council regulatory board ruled against the Wakefield team, denouncing them as frauds and they all lost their licenses to practice medicine.

Having moved to Texas where he still finds it hard to practice because of the defamation that began in England, Wakefield filed a defamation lawsuit against the BMJ and Brian Deer. It cost him millions to do so, millions being donated by his supporters. The courts threw it out on the grounds of jurisdiction, meaning, Texas was not the place to file this lawsuit. Wakefield would have to go to England to do that. And it only took the Texas courts (if I’m not mistaken) over three years to decide this.

But this isn’t the news you just have to hear. 

This did not make headlines. You didn’t hear about it. I didn’t hear about it. How could we hear anything when the moment the MMR is questioned, reporters who wouldn’t know real science if they tripped and fell into it just started echoing the continued screed of the BMJ, “That’s been thoroughly debunked! That’s been thoroughly debunked! That’s been thoroughly debunked!”

It’s hard to hear the truth when the lie is so loud. 

But, in 2012, just 6 years ago to this month (March), one of the doctors on Wakefield’s team, won his High Court appeal, with the Judge in the case quashing the General Medical Council’s findings. Though retired, Professor John Walker-Smith got his license to practice medicine returned to him and a third doctor, Professor Simon Murch was also cleared. 

The justice in this case ruled that there was “inadequate reasoning” to pronounce these researchers quacks and frauds and anything else the British press wanted to call them. That they performed their study and published it in good faith with no intent to do harm or defraud the public. But the good justice just had to reiterate that, “There is now no respectable body of opinion which supports (Dr Wakefield’s) hypothesis, that MMR vaccine and autism/enterocolitis are causally linked.”

Once again, that was never Wakefield’s hypothesis, and no, nobody has published a study linking autism to the MMR. The judge is correct on this last point.

However, there is a whistle blower from the CDC who has quite a bit to say on the subject.

In August 2014, Dr. Thompson revealed that the data underlying CDC’s principle vaccine safety studies demonstrated a causal link between vaccines and autism or autism symptoms, despite CDC’s claims to the contrary. According to Thompson, based upon interpretation of the data, “There is biologic plausibility right now to say that thimerosal causes autism-like features.” Dr. Thompson invoked federal whistleblower protection in August 2014. [Ref]

No, you won’t find any studies about thimerosal because the drug companies have removed it from their infant vaccines. 

So, as you can see, we’re cleared up nothing here. The media still screams, “That’s been thoroughly debunked!” thimerosal is still linked to autism like symptoms, and the MMR is given all at once, though in the US you may request to have them given separately, a year apart.

And Andrew Wakefield is still a quack, at least according to conventional, profit based medicine.

You can call me an antivaxxer or a quack all you want, but as a person truly interested in the Science, I am more than aware that anyone saying all vaccines are great or all vaccines are dangerous is not being scientific at all. When science actually focuses on healing with no conflicts of interest to worry about, then we’ll start to find the truth about all of medicine; and then we will start to focus on healing our brothers and sisters.

Copyright © 2018 Minnesota Wellness Publications, All rights reserved.


For More:  (Many more links following the article in this link)  The Lyme vaccine has caused the same symptoms as the disease and yet they keep peddling it without a care.  This article has the harrowing details of cases affected negatively by the Lyme vaccine.  Seriously heartbreaking stuff.

HHS Proposes Rule Expanding Religious & Conscience Protections in Health Care – Including Vaccinations: Please Provide Comments

HHS Proposes Rule Expanding Religious and Conscience Protections in Health Care


The Office of Civil Rights (OCR) in the federal Department of Health and Human Services (HHS) recently released a wide-ranging proposed rule that focused on increasing protections for the rights of conscience and religious liberties in the area of health care services. Several provisions of the rule pertain to the issue of parental rights.

Interested people can provide comments until Tuesday, March 27.

The proposed rule, Protecting Statutory Conscience Rights in Health Care; Delegations of Authority, (83 FR 3880) states that:

HHS proposes this rule to enhance the awareness and enforcement of federal health care conscience and associated anti-discrimination laws, to further conscience and religious freedom, and to protect the rights of individuals and entities to abstain from certain activities related to health care services without discrimination or retaliation.

In addition to laying down some new regulations, it also revises old regulations to make certain that the correct practices and policies are in place that will help promote the rights of individuals and entities in the area of individual health care decisions. Toward this end, the proposed rule states the following:

Through this rulemaking, the Department proposes to grant overall responsibility to its Office of Civil Rights (OCR) for ensuring that the Department, its components, HHS programs and activities, and those who participate in HHS programs or activities comply with federal laws protecting the rights of conscience and prohibiting associated discriminatory policies and practices in such programs and activities.

One provision affecting parental rights addresses the issue of vaccinations. While does not take any official position on the use of vaccinations, the provision in this proposed rule does guarantee greater protection for parents to make their own informed medical decisions for their children. Section XII C (1) (i) specifically makes reference to ensuring that persons and entities are not “being required to administer or receive certain vaccinations derived from aborted fetal tissues as a condition of work or receipt of educational services.” This rule also includes broader “provisions [to] protect parents who conscientiously object to their children being forced to receive certain treatments or health interventions.” (Section III (I)) is excited to see the steps that HHS is taking to ensure that parents have the right to make their own choices regarding the health care of their children.

If you would like to comment and voice your opinions regarding the proposed rule, formal comments may be submitted here:

Finally, if you would like to learn more about the new Conscience and Religious Freedom Division within the OCR whose jurisdiction the provisions of this rule fall under, visit

Other issues the proposed rule concerns:

  • Conscience protections related to abortion, sterilization, and certain other health services to participants in programs—and their personnel—funded by the Department (the Church Amendments, 42 U.S.C. 300a-7);
  • Conscience protections for health care entities related to abortion provision or training, referral for such abortion or training, or accreditation standards related to abortion (the Coats-Snowe Amendment, 42 U.S.C. 238n);
  • Protections from discrimination for health care entities and individuals who object to furthering or participating in abortion under programs funded by the Department’s yearly appropriations acts (e.g., Consolidated Appropriations Act, 2017, Pub. L. 115-31, Div. H, Tit. V, sec. 507(d) (the Weldon Amendment) and at Div. H, Tit. II, sec. 209);
  • Conscience protections under the Patient Protection and Affordable Care Act (ACA) related to assisted suicide (42 U.S.C. 18113), the ACA individual mandate (26 U.S.C. 5000A(d)(2)), and other matters of conscience (42 U.S.C. 18023(c)(2)(A)(i)-(iii), (b)(1)(A) and (b)(4));
  • Conscience protections for objections to counseling and referral for certain services in Medicaid or Medicare Advantage (42 U.S.C. 1395w-22(j)(3)(B) and 1396u-2(b)(3)(B));
  • Conscience protections related to the performance of advanced directives (42 U.S.C. 1395cc(f), 1396a(w)(3), and 14406);
  • Conscience protections related to Global Health Programs to the extent administered by the Secretary (22 U.S.C. 7631(d); Consolidated Appropriations Act, 2017, Pub. L. 115-31, Div. J, Tit. VII, sec. 7018 (Helms Amendment));
  • Exemptions from compulsory health care or services generally (42 U.S.C. 1396f & 5106i(a)(1)), and under specific programs for hearing screening (42 U.S.C. 280g-1(d)), occupational illness testing (29 U.S.C. 669(a)(5)); vaccination (42 U.S.C. 1396s(c)(2)(B)(ii)), and mental health treatment (42 U.S.C. 290bb-36(f)); and
  • Protections for religious nonmedical health care (e.g., 42 U.S.C. 1320a-1, 1320c-11, 1395i-5 and 1397j-1(b)).

(These laws will be collectively referred to as “Federal health care conscience and associated anti-discrimination laws” for purposes of this Notice of Proposed Rulemaking.)

Congress Receives Vaccine Safety Project Details Since the CDC & FDA Ignore Their Own Data and Proclaim Vaccines Do Not Cause Autism

Congress Receives Vaccine Safety Project Details Including Actions Needed for Sound Science and Transparency


 Approx. 9 Min.

Vaccine Safety Project Trailer

Since the FDA considers vaccines to be “biologics” not drugs, they have the capacity to fast track them without mandating rigorous studies as drugs must go through.

This short informative video shows exactly how vaccines are not rigorously studied with any sort of sincere science.

According to HHS’s own calculations, if they were able to capture ALL adverse events, they state that nearly 6 MILLION Americans would be adversely affected every year by vaccines if all of them were reported to the current Vaccine Adverse Events Reporting System (VAERS).

The committee overseeing vaccine safety should be absolutely free of any conflicts of interest, particularly the pharmaceutical industry, yet the opposite is true.

A new investigation in 2009 found that:

  • CDC has a systemic lack of oversight of the ethics program.
  • 97% of committee members’ conflict disclosures had omissions.
  • 58% had at least one unidentified potential conflict of interest.
  • CDC & FDA scientists receive royalties of $150K per year on vaccines they develop.

Looking at the IOM 700 page report on DTap, the conclusion reads,

“The evidence is inadequate to accept or reject a causal relationship between diphtheria toxoid-, tetanus toxoid-, or acellular pertussis-containing vaccine and autism.”

What this means is they claim they couldn’t find any study on the relationship between Dtap and autism.  But, in fact they acknowledge in the first paragraph that there is one study that does show a causal relationship between Dtap and autism, but they reject it because it provides data from passive surveillance (VAERS) that lacked a comparison of an unvaccinated population.

So while the CDC website cites the 2011 Institute of Medicine study that “vaccines do not cause autism,” they seem to be counting on people not reading the 700+ page report. Anyone who does can find that the researchers behind this study ignored existing research showing a correlation between vaccines and autism.

Rather than doing their due diligence (scientific studies) they just proclaim vaccines do not cause autism.

For more:

Vaccines have been found to activate latent Lyme/MSIDS infections: (Please read comment section after article for more links) Asymptomatic girls after receiving Gardasil activated dormant Bartonella which was confirmed by testing.