Archive for the ‘research’ Category

New Study Supports Conclusion of Retracted 2020 Study Showing Unvaxxed Kids Healthier Than Vaxxed & the Vaxxed Had a 36% Higher Risk of Asthma

New Study Supports Conclusion of Retracted 2020 Study Showing Unvaxxed Kids Healthier Than Vaxxed

A new study by James Lyons-Weiler, Ph.D. and Dr. Russell Blaylock supports the conclusions of a study by Dr. Paul Thomas, published in November 2020 and later retracted after an anonymous reader expressed concerns.

In November 2020, a study that carefully examined 10 years’ worth of data from a pediatric practice in Oregon run by Dr. Paul Thomas was published. Five days following the publication of the study, Thomas’ license was suspended.

A month after that, the journal decided to inform the authors that an anonymous reader had expressed some concerns about the study.

This single reader’s comments that involved bad guesswork led ultimately to the journal’s decision to retract the paper, leaving the authors stunned.

The authors knew that the reader’s concerns had already been addressed during peer review, and expected the journal to rule in favor of not retracting the paper. The journal editorial board knew this, too.

The concern centered primarily on the question of whether the large differences in the number of medical visits required for attention to specific health conditions like anemia, gastroenteritis, asthma, ear infections and many others, were due to parents who did not vaccinate not showing up to their well-baby and well-child visits.

Because Thomas’ license was suspended, he had to focus on his case and try to keep his life from falling apart; the medical board kept postponing the hearing, and no hearing had occurred.

In fact, no hearing has been held to date.

James Lyons-Weiler, Ph.D. suggested to Thomas that perhaps the medical board had overreached by applying a penalty without due process, a fact that Thomas then shared with his lawyer.

When his lawyer wrote the medical board pointing out that Thomas had suffered a penalty without due process, they offered to reinstate his license, pending the outcome of a hearing, on the condition that he do no more research.

This clearly shows the agenda of the medical board was not to ensure that the children in the practice were receiving good pediatric medical care.

It is now clear that the singular priority of the medical board was to shut down Thomas’ practice of abiding by informed consent — as required by Oregon state law for all medical procedures — and to prevent him from sharing any additional findings from the 10 years of data that had been collected from his practice.

New study supports earlier conclusions by Thomas

Today, the study is revived by a second study, this time conducted by Lyons-Weiler and his medical collaborator, Dr. Russell Blaylock.

In this second study, the following questions were addressed:

  1. Which group of patients adhered to the regular well-child visit better, the vaccinated patients or those who had refused vaccines?
  2. In groups of patients matched for health check visitation usage, which adverse health outcomes following vaccination differed between vaccinated patients and those who refused vaccines?
  3. After adjusting for healthcare visitations and age, do vaccines still significantly affect overall adverse health conditions in a manner independent of their interaction with healthcare visitations and age?
  4. Did older patients in the practice who stopped vaccinating experience a decrease in the adverse health outcomes that have been attributed to vaccines?

The study results, which are found in the paper entitled “Revisiting Excess Diagnoses of Illnesses and Conditions in Children Whose Parents Provide Informed Permission to Vaccinate Them” show that the anonymous reader’s concerns were unfounded; the unvaccinated families made their well-child visits with greater frequency than the vaccinated families.

This study, funded by the public, answers the first questions.

The answer to the second question is “results vary,” but this may be due to smaller sample sizes reducing power (see the study for details).

The study split the patients into high, medium and low health care visitation usage blocks, and many of the adverse health effects are seen increased in the vaccinated group of patients within these blocks (blocks are groups of patients matched on health care visitation usage).

For the third question, the scientists found that after defining a model that included healthcare visit utilization and age, vaccines were still a significant factor that increased adverse health outcomes, many of which had previously been associated with vaccines.

Moreover, the authors also determined that vaccines were still significant following consideration of the interaction term between vaccination status and the other model factors.

Importantly, had the study authors not considered the interaction term, the results would have seemed to imply that vaccination was negatively predictive of adverse health outcomes.

In the model in which vaccines, health care visits per age and the interaction term was considered, the number of vaccines was a positive significant predictor of overall adverse health.

Interaction terms are usually ignored by studies that “adjust for” variables. Adding covariates into the model without considering the interaction term with the main effect — vaccines — can mask a significant effect on the rates of post-vaccination health issues, providing a misleading result.

It’s worth noting that breastfeeding — another correlate of lifestyle measures — had no significant singular or interaction effects.

Blaylock posed the final question to Lyons-Weiler, who conducted the data analysis.

When older children were studied, and those who had the most vaccines were compared to those to those of the same age who had fewer vaccines, a clear pattern emerged for most of the adverse health outcomes: the risk of having a higher adverse health outcome was higher in the most-vaccinated older children compared to the least-vaccinated older children to a degree that was larger than that expected given any variation between the two groups in healthcare visit utilization.

This reflects the positive health effects of vaccine cessation.

vaccine cessation
The age-matched effects of vaccine cessation. High Relative Risk values denote increased risk of a given health outcome in patients receiving more vaccines in the older age group (>1,500 days of age). The black bar shows the Relative Risk of HCV between these groups as a baseline.

The relative risk of adverse health outcomes in older children who continued to vaccinate compared to those who ceased vaccination in Thomas’ practice.

Combined, all of these results mean that the method developed by Lyons-Weiler to consider the number of office visits needed for adverse health outcomes represents a robust, reliable and rigorous advance in methodology for the study of adverse health outcomes following medical exposures, including vaccines.

The method, “Relative Incidence of Office Visits,” had already been shown to be more powerful.

Lyons-Weiler reports that this is necessarily so because the measure contains more information than mere rates of diagnosis.

The RIOV measure has a higher dynamic range than odds ratios and relative risks based on diagnosis only. Studies that focus on the rates of diagnosis are using a subset of RIOV but are only limiting their count of office visits to that for the initial diagnosis.

The authors estimated that vaccination increases the need for visits to the doctor for vaccine-related health outcomes at a rate of 2.56 to 4.98 new chronic-illness-related visits per unit increase in vaccination per year.

“That translates into far more chronic illness in vaccinating children than in those not vaccinating, a disease burden that is not considered in risk: benefit considerations when it comes to vaccine policies and laws,” said Lyons-Weiler.

The paper, which was subjected to blinded peer review, describes all of the details of the results, is open access, and is published in the peer-reviewed journal International Journal of Vaccine Theory, Research, and Practice.

The views and opinions expressed in this article are those of the authors and do not necessarily reflect the views of Children’s Health Defense

Overall, kids in the study who received 3 mg or more of vaccine-related aluminum had at least a 36% higher risk of developing persistent asthma than kids who got less than 3 the study’s lead author, Dr. Matthew F. Daley, told The Associated Press(See link for article)
It should be noted that the CDC has never done a study comparing the vaccinated with unvaccinated nor with children who weren’t exposed to aluminum at all in their vaccines.  To date, the CDC has never studied the cumulative effects of all the vaccines together.  Studies only look at one vaccine at a time typically compared to another vaccine (not a true placebo). 

Christopher Exley, Ph.D., an expert on aluminum toxicity, agreed the study will likely not result in altering the use of aluminum adjuvants in vaccines.

Exley added:

“The idea is to concede the smallest possible ground on aluminum toxicity and at the same time reinforcing what they want readers to think by citing multiple papers by stooges and those working directly for the aluminum industry — a classic example being where the authors look to reinforce that ingested aluminum is not a health issue in infants.”

Please read the entire article for all the details.

Also, please read James Lyons-Weiler’s take on it as well. Cox proportional hazard models were used to evaluate the
association between aluminum exposure and asthma inci-
dence, stratified by eczema

Weiler states:

Raise your hand if you’re a parent and you have noticed that your child with eczema seems at risk of autoimmune conditions, including autoimmune diseases of the airways like persistent asthma when exposed to vaccines.

The risk was associated with vaccine-associated aluminum dose – and the increased risk was HUGE. “aHz of 1.26 per 1 mg increase in aluminum” means a 26% increase in the risk of asthma per 1,000 mcg aluminum-containing vaccine received. Children on the CDC’s schedule receive 5,640 mcg of aluminum by age 13, so children with eczema have a 78% increase in their risk of developing asthma by age 13 compared to kids who receive no aluminum-containing vaccines.

Kids without eczema had a 19% increase in asthma risk per 1 mg increase in vaccine-sourced aluminum; by age 13, they have a 57% increased risk of asthma compared to kids who receive no aluminum-containing vaccines.

He then proceeds to wipe the floor with Dr. Paul Offit, who is calling for “extraordinary evidence” which Weiler states means randomized controlled trials (RCTs) which will never happen (just like in Lymeland).

Cancers Increasing Dramatically & Did the COVID Shot Worsen A Famous Doctor’s Cancer?

Cancers in Adults Under 50 Have Increased Dramatically Around The Globe

By Fiona MacDonald

Cancer has long been part of the human story. But a new review has shown that, recently, something has shifted.

Since 1990, the number of adults under the age of 50 developing cancer has increased dramatically around the world.

What’s concerning is that the increase in early-onset cancers doesn’t seem to be slowing down – and improvements in screening alone don’t seem to be able to fully explain the trend.

“We found that this risk is increasing with each generation,” says one of the researchers, Shuji Ogino, a pathologist and epidemiologist at Brigham and Women’s Hospital in Boston.

(See link for article)



  • The researchers looked at 14 cancer types:  breast, colorectal (CRC), endometrial, esophageal, extrahepatic bile duct, gallbladder, head and neck, kidney, liver, bone marrow, pancreas, prostate, stomach, and thyroid cancer – all of which are on the rise according to global cancer data.
  • Then they reviewed any available studies that could shed light on possible risk factors for these cancers by looking for clues in the literature describing any unique clinical and biological characteristics of tumors of early-onset cancers.
  • They found that early-onset cancer is an emerging global epidemic.
  • They found the following issues contributed to the uptick:
    • increased screening, however even countries that don’t have screening programs have increased cancer rates.
    • Diet
    • lifestyle
    • weight (obesity)
    • environmental exposures
    • microbiome
    • sedentary lifestyle
    • alcohol consumption
    • type 2 diabetes
  • Among the types of cancers studied 14 are related to the digestive system.
  • Regarding children, they are getting a lot less sleep than in the past.

The research has been published in Nature Reviews Clinical Oncology.

While the article doesn’t mention it specifically, radiation from wireless devices such as cell phones which have become prominent today may be adding to this cancer surge as well.  It is commonly known that EMFs wreak havoc in the body and many Lyme/MSIDS are particularly vulnerable.

Another little discussed topic is glyphosate, the major ingredient in Bayer-Monsanto’s Roundup which is the most widely used pesticide in the U.S. WHO and CA scientists both agree it is linked to cancer, yet the EPA concluded it was “safe” and “not likely” to cause cancer. The EPA has been forced to review this due to a federal judge finding the agency ignored human health studies, expert advice, and the agency’s guidelines for determining cancer risk. Source

And a 2021 study links lung cancer with mask usage.

Similarly to research regarding tick-borne illnesses, Alzheimer’s and cancer research have been controlled by a Cabal and researchers are currently accused of doctoring images, plagiarism, and faking data.

The article also doesn’t mention the link between the COVID mRNA shots and cancer:

  • the lipid nanoparticle mRNA COVID injection goes systemically into the entire body and doesn’t remain in the arm as thought.
  • It continues to produce the spike protein at least 60 days out if not longer and is being found 15 months later.
  • It also interferes with cancer blocking genes and they are seeing an uptick in cancers as well as other viruses now after the shots
  • there’s been a 40% increase in deaths those ages 18-64 years of age and an 84% increase in the 25-44 age group according to insurance companies.

The following story is a perfect example of the very real potential link:

Did a Famous Doctor’s COVID Shot Make His Cancer Worse?

A lifelong promoter of vaccines suspects he might be the rare, unfortunate exception.
Sept. 24, 2022
On September 22 of last year, Michel Goldman, a Belgian immunologist and one of Europe’s best-known champions of medical research, walked into a clinic near his house, rolled up his sleeve, and had a booster shot delivered to his arm.
Just a few weeks earlier, Michel, 67, had been to see his younger brother, Serge, the head of nuclear medicine at the hospital of the Université Libre de Bruxelles, where both men are professors. Michel was having night sweats, and he could feel swollen lymph nodes in his neck, so his brother brought him in for a full-body CT scan. When the images came through to Serge’s computer they revealed a smattering of inky spots, bunched near Michel’s left armpit and running up along his neck. It was cancer of the immune system—lymphoma.

Given his own area of expertise, Michel understood this meant he’d soon be immunocompromised by chemotherapy. With another winter on the way—and perhaps another wave of SARS-CoV-2 infections—that meant he had just a narrow window of opportunity in which his body would respond in full to COVID vaccination. Having received two doses of Pfizer the prior spring, Michel quickly went to get his third. If he was about to spend months absorbing poison as he tried to beat a deadly cancer, at least he’d have the most protection possible from the pandemic.

Within a few days, though, Michel was somehow feeling even worse. His night sweats got much more intense, and he found himself—quite out of character—taking afternoon naps. Most worryingly, his lymph nodes were even more swollen than before. He conferred with Serge again, and they set up another body scan for September 30, six days before Michel was scheduled to start his cancer treatment. Once again he sat in the radiology waiting room while his brother waited for the pictures to appear on his computer.

Serge’s bushy eyebrows furrowed when he spoke with Michel after having seen the scans. (“I will always remember his face, it was just incredible,” Michel told me.) The pictures showed a brand-new barrage of cancer lesions—so many spots that it looked like someone had set off fireworks inside Michel’s body. More than that, the lesions were now prominent on both sides of the body, with new clusters blooming in Michel’s right armpit in particular, and along the right side of his neck.  (See link for article)



  • It is unusual to see such a swift progression in just 3 weeks
  • He hand his brother had a gnawing feeling the booster made him sicker
  • The article erroneously states this is a very rare life-threatening side effect.  Doctors have been reporting this finding all over the world but are ignored.
  • An avid proponent of the shots, going to far as to reassure others about their safety, he’s definitely having a red pill experience.
  • Unfortunately he bought and propagated the lie that any chance of serious complications from the shots pale in comparison to the chance of complications from COVID.
  • Michael threw him into researching the mechanisms of action of the COVID shots and did find clues suggesting the the mRNA shots might be risky for a subset of the population as they are effective at generating a message and spurring its passage through helper T cells, which could give such a jolt to helper T cells that they go berserk.  Overstimulation on those prone to forming tumors in those already with cancer, overstimulation could make it worse.
  • He learned that body scans of some of those who get vaccines, including cancer patients, have shown heightened activity in the lymph nodes near the armpit on the side where the shot was received.
  • A mouse study also corroborated his experience.
  • Michael wrote a paper, about his experience titled “Rapid Progression of Angioimmunoblastic T Cell Lymphoma Following BNT162b2 mRNA Vaccine Booster Shot”
  • Worried his study would fuel vaccine skepticism he labored over every word, yet his paper follows earlier reports also suggesting a possible link between the COVID shot and lymphoma
  • Another doctor also worried that writing about five patients who had a relapse of kidney disease and eight patients who were newly diagnosed after getting the shot would also fuel vaccine skepticism.
  • Michael’s immunologist stated that the vaccine appeared to be related to the cancer’s behavior and then reneged by stating it’s just a case report – one patient.

Chronic Toxoplasmosis: Debilitating, Stealth, Underdiagnosed

By Dr. Mercola

Sept. 16, 2022

Story at-a-glance

  • At least one third of all people on Earth are infected with the parasite Toxoplasma gondii, averaging from 11-20% in the United States to 50% and higher in some Western European countries
  • The parasite has been implicated in ocular issues, schizophrenia, epilepsy, Alzheimer’s disease and various other neurological disorders, as well as in heart disease, pneumonia, recurrent headaches, even cancer; it is also known for causing psychological changes in its hosts
  • While the official word is that most toxoplasma infections are harmless and asymptomatic, the impact of the parasite could be much more devastating than the current mainstream medical convention presumes; it may also be cross-reacting with the spike protein and possibly contributing to the mystery of “long COVID”
  • According to recent research and clinical evidence, toxoplasma tissue cysts, previously considered harmless in immunocompetent patients, are capable of causing major health issues without converting to the cell-blasting form
  • Commonly used antibody tests can only detect antibodies for the “tachyzoite” (cell-blasting) form of the parasite but not the “bradyzoite” (tissue cyst) form
  • Dr Uwe Auf der Straße in Germany has done an important clinical investigation of the parasite, and his findings could shed light on “mystery” symptoms in many patients

Toxoplasma gondii, an intracellular protozoan organism, is a very “successful” parasite with extremely diverse host base and sophisticated, almost diabolical, methods of survival and proliferation.

It is found worldwide and is capable of infecting most warm-blooded animals as intermediate hosts, including people. It has also been found in some cold-blooded animals, such as fish. Its final hosts, inside which the parasites can sexually reproduce, are felines, including domestic cats. In the environment, toxoplasma can be found in soil, water, and other substances that have come in contact with the parasite, such as fertilizers.

At the moment, the predominant medical opinion is that at least one third of all people on Earth are in some way infected with this parasite1,2 averaging from 11-20% in the United States to 50% or higher in a number of Western European countries, for example in Germany.3 In Germany, the frequency of positive Toxoplasma detection increases from about 20% in the group of 18-29 year-olds, up to 77% in the group of 70-79 year-olds and for over 79 year-olds the frequency is 84%.4

The commonly known infection routes for people are eating uncooked meat, drinking contaminated water, or accidentally ingesting the parasite after cleaning a cat litter box.

While the official word is that most infections are harmless and asymptomatic, the impact of the parasite could be much more devastating than the current mainstream medical convention gives it credit for.

A few physicians and researchers who have been looking into Toxoplasma are challenging the conventional view on several counts. And while a number of “bombshell” scientific works on the topic have been published, the new discoveries have not yet made their way into the everyday medical practice.

It is extremely important that more doctors and researchers look into this right now — especially given the fact that an encounter with the spike protein has been shown to amplify latent or slow-developing biological malfunctions in people, and thus it is possible that “spike protein assisted” Toxoplasma may be wreaking havoc in many unsuspecting patients and contributing significantly to the mysterious “long COVID” or its injection-induced manifestation.

Toxoplasma Life Cycle

With a degree of oversimplification, there are three main forms in which this parasite exists during different phases of its life cycle. They are known as oocysts (eggs), tachyzoites (the actively proliferating adult form), and bradyzoites (tissue cysts).

The sexual reproduction of Toxoplasma gondii occurs within feline hosts. The cycle starts when the host ingests oocysts (“eggs”) or eats an animal infected with bradyzoites (tissue cysts).

Upon ingestion of the cysts, their protective wall is dissolved by proteolytic enzymes in the stomach and small intestine to release bradyzoites. The free bradyzoites then penetrate epithelial cells lining the small intestine where they proliferate to form new generations that can undergo sexual and asexual cycles. Following fertilization of the female gametes, a wall starts to form around the oocysts. The oocysts are then released to the environment along with feces.

Depending on the environment, it usually takes several days for the oocysts in feces to become infectious. Infectious oocysts can survive for up to several years in soil etc., until they are ingested by an intermediate host. Once they are ingested, their protective shield is also dissolved by proteolytic enzymes thus releasing the eggs into the intestine of the intermediate host.

They then penetrate epithelial cells lining the small intestine where they undergo a form of asexual reproduction to form tachyzoites. The newly formed tachyzoites then spread and actively penetrate other cells of the intermediate host where they are surrounded by a parasitophorous vacuole protecting them from the hosts’ immune system.5

The interesting thing about the parasitophorous vacuole is that the parasite uses a part of the membrane of the invaded host cell’s to form it, with the purpose of “hiding” from the host’s immune system.6

From there tachyzoites disseminate throughout the body and reach immunologically protected sites including brain, retina and fetus. In vitro studies revealed that tachyzoites can invade astrocytes, microglia and neurons of the mouse brain with subsequent formation of tissue cysts within these cells.7,8,9,10,11

As they continue dividing, tachyzoites ultimately cause the cell to break, releasing as many as 32 tachyzoites that then infect new cells. However, that activity usually attracts the attention of the immune system, which ultimately slows down tachyzoite multiplication. In response, the tachyzoites convert into bradyzoites (tissue cysts).12

In doing so, they change their surface structure nearly completely, which is a “major factor in the parasite’s strategy of survival” since the host’s immune system identifies microorganisms according to their surface structure, and by modifying its surface structure, toxoplasma increases its chance of successfully tricking the host’s immune system.13,14,15

The tissue cysts are common in a number of body tissues and organs including the eyes, cardiac muscle, neural tissue, and various visceral organs where they can last for the hosts’ entire lifetime.16

Houston, We Have a Problem

The general medical consensus (challenged by a small group of doctor and researchers) is that while the most active form of Toxoplasma, known as “tachyzoites” (the one that multiplies very fast and blasts host cells), can cause significant health issues, predominantly in immunocompromised hosts, the tissue cyst form (“bradyzoites”) is mostly innocuous, and, once the parasite succumbs to the attack by the host’s immune system and retreats into its tissue cysts, it just quietly sits inside those intracellular cysts and does very little.

Per mainstream medial convention, the vulnerable demographics are immunocompromised patients who can succumb to acute toxoplasmosis and develop potentially lethal inflammation of the brain (or become victims of a “reactivation,” where tissue cysts convert back to the fast-proliferating form, to the same effect), and newly infected pregnant women.

However, recent research has shown that bradyzoites, the toxoplasma tissue cysts, are not innocuous at all, and that they do reproduce inside the cysts and can cause inflammation and other issues without converting to tachyzoites, including in otherwise immunocompetent patients.

What complicates the issue even further is that nearly all commercially available tests (antibody blood tests and even PCR tests), are specific to the cell-blasting tachyzoite form of toxoplasma and do not detect the presence of tissue cysts.

And if that is the case — we are looking at a potentially large number of people ailing from “chronic active toxoplasma” that cannot be diagnosed by any of the commonly used methods. As a result — especially given that toxoplasma loves living in the brain — their very real and possibly exhausting physical disease may be classified or psychosomatic or straight out psychiatric.

They could be suffering from slow-developing brain inflammation, autism-like symptoms, dementia-like symptoms, or even pulmonary and heart issues — and the doctors might not even be looking in the direction of toxoplasma or ruling it out, based on negative antibodies.

(In its active form, the parasite has been implicated in ocular issues,17 heart disease,18 pneumonia,19 recurrent headaches,20,21 even cancer22 — as well as in addiction, schizophrenia, epilepsy, Alzheimer’s disease, and various neurological disorders.23,24,25 And even in its latent form, it is believed to cause psychological changes in its hosts, ranging from entrepreneurial26 to suicidal tendencies.27

Among the researchers doing groundbreaking research in Toxoplasma are Dr. Jaroslav Flegr in the Czech Republic, Dr. Robert Yolken and Dr. Vernon Carruthers in the U.S., Dr Uwe Auf der Straße in Germany, and others.

The Work of Dr. Uwe Auf der Straße in Germany

Dr Uwe Auf der Straße is a GP in Germany and the author of the book titled, “Shadow Disease Chronic Active Toxoplasmosis.”28 Here is what he has to say about the “limits of our current laboratory medicine”:

“The current medical opinion is still that a negative IgM excludes an active toxoplasmosis and thus the need of a therapy. Due to research having hinted repeatedly of a significant effect of bradyzoite activity, and due to my own observations, I definitely cannot agree with that.

Tests only react to tachyzoite-specific antibodies and the sensitivity of a standard test system in case of an initial infection is only 81.8%.29 Basic research has done further substantial work which questions the accuracy of Toxoplasma antibody assays.

‘The currently available solid phase immunoassays were developed in the 1970’s to detect strains which were circulating at that time and there are strong indications, that … standard assays may substantially underestimate the prevalence of Toxoplasma infection in a population and its effect on health and disease.’30

Further it has been proven that, in cases of a Toxoplasma infection, tachyzoite–specific IgG, IgM and even PCR can render negative results.31,32,33

In a chronic active course of the disease, reliability of our currently used lab methods has not been proven and these are most likely not suitable to detect Bradyzoites or their activity, let alone the cyst burden.

Research still focuses predominantly on acute rather than chronic toxoplasmosis and it is only a general assumption, that cases with chronically active courses of the disease as presented here could be diagnosed by using the usual antibody assays – to my knowledge this has never been proven.

The number of Toxoplasma carriers without detectable tachyzoites antibodies, who can potentially become ill from a chronic active toxoplasmosis based on an increased activity within the cysts is unclear.

From my observations it can be assumed that the number is significant, otherwise there would not appear so many younger patients in my case collection (about 40%), who suffer from a chronic active toxoplasmosis without any detectable tachyzoite antibodies.

If the immune system ceases to produce Tachyzoite antibodies after some years, the disease will no longer be detectable in the blood. This does not mean that the Toxoplasma in the cysts are inactive.

There are numerous indications that an increased activity in the cysts can trigger a symptomatic illness, since, contrary to older assumptions, bradyzoites do not rest, but can be active and can reproduce and cause illness. This refers to the findings of Fergusson et al. (1989), McLeod et al. (2008) and Watts et al (2015).”34,35,36

Dr. Uwe Auf der Straße has observed that there was no significant difference in observed symptoms between his patients with positive antibody tests and his patients with negative antibody tests in whom he suspected chronic active toxoplasmosis, based on the preliminary diagnostic method he developed and ruling out other illnesses that can produce similar symptoms. (He also took into consideration their positive reaction to toxoplasma therapy.)

“I am convinced that a significant activity within the cysts, predominantly of the bradyzoites, is the decisive reason for the illness in both groups of patients who all have inconspicuous IgM values with regards to tachyzoites, and whose IgG values, if indeed there are any, don’t show any direct correlation to the severity of the illness.

The clinical pictures in both groups are identical, and the toxoplasmosis therapy is even more effective in group B [negative antibody tests]. The bradyzoites, the activity of which we cannot measure, are currently underrated strongly, and our established laboratory values produce only a pretended security.

It is difficult to develop reliable bradyzoite-specific tests, since bradyzoites reveal themselves only rarely to the immune system and only lead to a limited antibody production.”37

“Thankfully, basic research has begun to address this problem, and there are new and promising methods being developed, with the aim of getting a grip on this problem and reveal hitherto not detectable toxoplasma presence and even to determine the cyst burden.”38

Toxoplasma and the Mind

According to Kathleen McAuliffe, author of the book “This Is Your Brain on Parasites,”39 researchers have noticed a strong correlation between toxoplasma infection and schizophrenia and other mental disorders in humans. She also notes studies where anti-psychotic drugs inhibited toxoplasma in vitro.

In fact, the mind-controlling ability is Toxoplasma’s “trademark.” I wrote about it earlier in the article titled, “Don’t Underestimate Mind-Controlling Parasites.”

To quote Dr. Uwe Auf der Straße again, whose book I can’t recommend enough, “symptoms comprise an increased risk for the occurrence of schizophrenia40,41,42,43 psychoses44 or aggressive behaviour, also a doubling of the risk for accidents in cases where Toxoplasma antibodies have been detected.45

Explanations for that may point to the mentioned behavioural changes and the decreasing psychomotor resilience46 due to Toxoplasma infections. It is scary that even an increase in the number of attempted suicides has been correlated with antibody detection in toxoplasmosis.”47,48

“It fits this bill that toxoplasmosis infected rats are known to lose all fear of cats. They literally seek them out in broad daylight, to be eaten in the end, a behaviour that is very advantageous for the spreading of Toxoplasma, but not so good for the rat. The consequence is clear.

When the host is ‘ripe’ and contains many bradyzoite – cysts, it is simply more useful for the parasite when dead instead of alive, particularly if the death is caused by a cat. Death by car accident or suicide are thus somehow “inappropriate”, but can be regarded as a somewhat macabre continuation of such behavioural disturbances in the present.”

Many Tricks of Toxoplasma

The tricks of this parasite are endless. For example, it knows how to hijack the host’s macrophages — and instead of being destroyed by a macrophage,49 take over it and use it as a temporary home to transform into the active form and then use it as a cab to travel around the host’s body!

According to the accepted view, the release of actual “eggs” from the ingested oocysts into the host’s system happens due to the processes in the host’s digestive system. However, a study came out showing that the process can happen in the absence of digestive factors, and that the parasite can not only survive but also transform into its most active form inside macrophages, after being “eaten”:

“Our results show that the oocyst internalization kinetics can vary among a given population of macrophages, but similar processes and dynamics could be observed. Most of the cells manipulate oocysts for ~15 min before internalizing them in typically 30 min … Liberated sporozoites within macrophages then differentiate into tachyzoites within 4-6 h following oocyst-macrophage contact.”

Another paper, titled, “Inhibition of nitric oxide production of activated mice peritoneal macrophages is independent of the Toxoplasma gondii strain,” shows that Toxoplasma is capable of inhibiting nitric oxide production. Nitric oxide, that plays an important role in immune response50,51 and is frequently mentioned in the context of COVID.

It’s an “enzyme that is expressed in activated macrophages, generates nitric oxide (NO) from the amino acid L-arginine, and thereby contributes to the control of replication or killing of intracellular microbial pathogens.”52

The overall mechanism that the parasite uses to invade host cells is beyond of the scope of this article but if you are curious, you can check out the paper titled, “How does Toxoplasma gondii invade host cells?” If you want to learn more about how it modulates host cell’s responses, there is another technical paper titled, “Toxoplasma gondii Modulates the Host Cell Responses: An Overview of Apoptosis Pathways.”

And if you want to learn more about Toxoplasma and brain blood barrier, you can read this paper, “Toxoplasma gondii and the blood-brain barrier.”

Toxoplasma and the Spike Protein: A Possible Connection?

According to Dr. Uwe Auf der Straße, a patient could be potentially simultaneously infected with Toxoplasma and with one or more other pathogens, some of the them kicking in as opportunistic infections.

In that case, the clinical picture may be even more confusing, and the condition of the patient may be more severe, even though Toxoplasma is capable of causing enough trouble if it manages to sufficiently proliferate — whether in its active form or inside the tissue cysts — all on its own.

Dr. Uwe Auf der Straße’s book was published in 2019, so there is nothing about COVID in the book — but it is not illogical to presume that when a person with latent or relatively slowly developing Toxoplasma encounters the spike protein, whether it’s from infection or from the COVID injection, it may create a “perfect storm” and kick Toxoplasma in high gear, creating debilitating and/or mysterious symptoms, resulting in vaccine injury or “long COVID.”53

If the percentage of people with chronic active Toxoplasma is as high as Dr. Uwe Auf der Straße suspects, it is also not illogical to assume that due to the deficiencies in the current diagnostic standards and tools, a lot of people suffering from chronic active Toxoplasma may not be properly diagnosed, and their maladies may be attributed to psychosomatic factors or remain a medical mystery.

An additional complication is that “atypical mixed forms of the known Toxoplasma strains, which are significantly more aggressive than the previously known Toxoplasma strains have been detected in Germany, and this might happen worldwide.”54,55,56

Anecdotally, per Dr. Uwe Auf der Straße, patients with chronic active toxoplasma could experience increased irritability where they “blow up” out of nowhere even though they realize that there is no good reason and don’t feel good about being so irritable — as well as anxiety or depression, with men more prone to irritability, and women more prone to anxiety and depression.

At the same time, also anecdotally, increased irritability has been observed in some recipients of the COVID injection. And while there can be lots of factors causing mood changes, it could be something to look into.

Curiously, there is an overlap between the list of natural remedies that have been studied as potentially treatments against toxoplasma and showed improvements — and the list of “alternative” COVID and “long COVID” treatments.57,58,59,60 Due to the tremendous complexity of the issue and the fact that myriads of factors impact our immune response and reactions to treatments, further investigation of the correlation by honest and curious is urgently needed.

Conventional toxoplasma treatments are considered effective in treating tachyzoites but there is no known conventional treatment for the tissue cyst form.61

Better Diagnostics: Some Hope

According to Dr Uwe Auf der Straße, in his practice, he found one particular testing method to be more reliable than the conventional ones:

“The Lymphocyte-Transformation-Test (LTT) has made my work on Toxoplasmosis easier in the last months, but is not (yet) used for the diagnose of toxoplasmosis on a wider scale. By means of this test, we can detect activity of our immune system’s T-lymphocytes, which react specifically towards certain pathogens.

While the immune system is dealing with certain pathogens, T-lymphocytes become specifically reactive to this pathogen, and the intensity of this reactivity can be measured pathogen-specifically.”

“This is measurable for about 4 weeks, and thus the LTT mirrors the current activity of pathogens. A more than threefold elevated stimulating index (SI) indicates, that specific T-cells are present in the blood and thus an active confrontation of the immune system and the tested germ takes place.

A validation concerning chronic active toxoplasmosis has not yet been performed, but according to both Dr. Hopf-Seidel and my own experiences with patients suffering from a chronic active toxoplasmosis, it is most likely more sensitive than the Toxoplasma IgM.”

Dr Uwe Auf der Straße also speaks highly of the work of Dr. Yolken’s team:

“I consider a new approach of the scientific group led by Professor Yolken in Baltimore to be promising. A paper on this approach has been published in June, 2018.62 The scientists used a known detection method (a Western blot test) for the detection of Toxoplasma proteins, which also give proof of the presence of Toxoplasma.

Of 25 patients, who were suffering from severe psychic disorders, 3 patients (8.2%) were diagnosed as positive with Toxoplasma IgG. Four times as many, 12 patients (35.3%) were then diagnosed with Toxoplasma by detection of Toxoplasma protein in their blood.

The detection of these proteins seems to offer a significantly more sensitive method to diagnose toxoplasmosis than the usual available antibody tests. Until this can be used as a routine procedure, the tests will have to be examined in further studies.”

“The same group of scientists is currently developing another highly-sensitive method, which can detect Toxoplasma cysts in every stage of the disease. This concerns the MAG1 antigen, which occurs in great numbers inside the bradyzoite cysts and in their outer membrane. Antibodies which are directed against this MAG1 antigen can be detected in the laboratory.

The scientist could prove in mice that the amount of MAG1 antibodies detectable in the blood showed a significant correlation to the amount of bradyzoite cysts inside the brain. It was also shown that in case of negative MAG1 antibody detection, no bradyzoite cysts were found.

This marker could possibly be used as a scale for a chronic infection and for the burden with bradyzoite cysts in the future. This would be a huge step for laboratory diagnostics and for affected patients even more so.”

“Another approach is that clues for a disturbed metabolism in patients with Chronic Fatigue Syndrome (CFS) are being investigated intensively. In 2016 it was proven that CFS patients share anomalies in 20 metabolic pathways of their mitochondria.63 One might picture mitochondria best as our cells’ power plants.

The intensity of the illness in CFS patients negatively impacts the activity of the metabolic pathways and the quantity of metabolites, which result from the mitochondria’s work. This “shutdown” of the metabolism has been interpreted as a shifting of the mitochondrial metabolism into “survival mode.”

Toxoplasma can also very severely affect the mitochondria,64 and the intensity of affliction is probably related to the strain of Toxoplasma which has infected the patient.65 It would be of utmost interest if the deviations in mitochondria metabolism during a chronic active toxoplasmosis might resemble those detected in ME/CFS patients, as there is strong overlap in the symptoms of both diseases. They might even be identical in some cases.”


It is possible that due to imperfect diagnostics and insufficient understanding of this parasite in the medical community, a lot of people with chronic active toxoplasma remain undiagnosed or diagnosed incorrectly, and suffer profoundly from the lack of proper treatment.

It is also possible that Toxoplasma is a significant factor, contributing to complications from spike protein toxicity. I believe that understanding this issue is important. It requires time and attention of researchers and doctors, and my prayer is for solid knowledge to come, and for the “mystery” suffering to end.

About the Author

To find more of Tessa Lena’s work, be sure to check out her bio, Tessa Fights Robots.


For more:

Toxoplasmosis causes many mental issues and psychiatrist E. Fuller Torry believes that 75% of schizophrenia is associated with infections, with Toxo a significant portion.

Skin Diseases After COVID Shot Appearing in Medical Journals, Including Genital Necrosis  Go here for photos

Worse than Monkeypox? Multiple Cases of Skin Diseases Following COVID-19 Vaccination Start Appearing in the Medical Journals

Comments by Brian Shilhavy
Editor, Health Impact News

More and more COVID-19 vaccine injuries are starting to make their way into the medical journals. Several recent published case studies focus on skin diseases, and we are publishing a few of them here today as a service to the public, since the corporate media will seldom, if ever, cover this news.

Since there is already a plan in place to promote a monkeypox outbreak as one of the next big “pandemics” (see: Plandemic II Launched to Keep Pandemic Funds Flowing to Big Pharma: MonkeyPox), it is important to document these existing skin diseases that are already occurring following the mass COVID-19 vaccination campaigns.

It would not surprise me one bit if the criminal government “health” agencies such as the FDA and CDC started publishing photos like you see in these studies and label them all as some new variant of “monkeypox” which would cause fear and panic in the public, when in fact these skin conditions are more likely COVID-19 vaccine side effects.

Five cases of new-onset pemphigus following vaccinations against coronavirus disease 2019

The Journal of Dermatology, August 2022


Pemphigus is a group of blistering disorders characterized by the formation of intraepithelial blisters in skin and mucous membranes induced by the binding of circulating autoantibodies to intercellular adhesion molecules. The pathogenesis is complex and not fully understood; however, genetic predisposition and various triggers are widely accepted as key factors in pemphigus development. A few cases of new-onset pemphigus following coronavirus disease 2019 (COVID-19) vaccination have already been published. The present paper reports a total of two cases of pemphigus foliaceous and three cases of pemphigus vulgaris that occurred following vaccinations against COVID-19, with anamnestic, clinical, and diagnostic data collection suggesting assumptions over a possible causal correlation.

Clinical pictures of our cases. Case 1: scattered superficial blisters and erythematous patches with scaly erosions involving the face and lower trunk (a–c). Case 2: diffuse erythematous-squamous patches with scaly and crusted erosions involving the face and the trunk, following a seborrheic distribution (d, e). Case 3: a few erosive lesions, diffuse scales, and crusted erosions involving the trunk (f, g). Case 4: painful erosions on the gums and soft palate (h). Case 5: erosive lesions on the oral cavity, nose, right cheek, and abdomen (i, j).

Read the full study here.

Pemphigus foliaceus triggered after inactivated SARS-CoV-2 vaccine: Coincidence or causal link?

Dermatological Therapy, August 2022

Pemphigus is a group of autoimmune blistering disorders associated with autoantibodies against the keratinocyte cell surface. Its exact cause is still unknown, but neoplasms, infections, medications, or vaccines are considered as possible triggering factors. Only one case of pemphigus vulgaris (PV) following vaccination with mRNA vaccine BNT162b2 has been reported.1 At the time of submission of the present report, this is the first case of Pemphigus foliaceus (PF) type triggered after inactivated SARS-CoV-2 vaccination.

(A) Clinical examination on admission showing scales and crusts with erythematous bases and superficial blistering all over the trunk. (B) Large erosions of the neck after extension of the superficial detachment. (C) Histopathology of lesion showing subcorneal loss of adhesion with acantholytic cells

Read the full study here.

New onset pemphigus foliaceus following AstraZeneca COVID-19 vaccination

Journal of the European Academy of Dermatology & Venereology – August 2022

Pemphigus vulgaris (PV) and pemphigus foliaceus (PF) are a rare group of immunobullous disorders that can lead to high morbidity and mortality.1 Analogous to the other autoimmune diseases, pemphigus is closely related to the immune response, which is impacted by several factors including; polymorphism of the genes, family history of other autoimmune disorders including pemphigus, gender, ethnicity, geographical area and environmental factors.2, 3 Several triggers including different drugs and treatments, diseases, vaccines, nutrients, micronutrients, pregnancy and stress have all been implicated in the aetiology of the disease.4 Here, we present a case with PF whose disease developed after administration of the first dose of ChAdOx1 nCoV-19 (AstraZeneca) vaccination and was exacerbated following the second dose of this vaccine.

(a) Large erosive annular erythematous plaques on the back. (b) Histology of the skin shows subcorneal acantholysis and blister filled with neutrophils. The epidermis is infiltrated by a large number of neutrophils. H and E stain ×100 magnification. (c) Direct immunofluorescent staining shows intercellular IgG in the epidermis in a chicken wire-like pattern. IMF stain ×200 magnification.

Read the full study here.

Pemphigus vulgaris following second dose of mRNA-(Pfizer-BioNTech) COVID-19 vaccine

Dermatological Therapy, August 2022

Pemphigus vulgaris (PV) is an autoimmune, blistering dermatosis caused by autoantibodies to desmoglein (Dsg) 1 and (Dsg) 3 targeting keratinocyte desmosomes. The etiology of the disease remains unknown. There are many inciting factors for PV development, including infections, medications, ultra-violets, radiations, trauma, burns, and underlying neoplasms. A limited number of patients developed PV following vaccination.1-3

Only a few cases of PV following the COVID-19 vaccine were reported since the beginning of the pandemic. We report a case of PV occurring 1 week after the second dose of mRNA-Pfizer-BioNTech COVID-19 vaccine.

(A) Flaccid blisters on healthy skin with post-bullous erosions. (B) Post-bullous erosions of the oral mucosa

Read the full study here.

Pemphigus vulgaris with advanced hypopharyngeal and gastric cancer following SARS-CoV-2 vaccination

The Journal of Dermatology, August 2022

Pemphigus vulgaris (PV) is an autoimmune blistering disease that causes intraepidermal blisters on the skin and mucous membranes. Autoantibodies targeting desmoglein 1 and desmoglein 3, which are cell adhesion molecules connecting epidermal cells, play an essential role in the development of PV.1 It has been suggested that one mechanism for the production of autoantibodies is molecular mimicry, i.e. a phenomenon in which different molecules have similar structures. For example, if antigens of pathogenic microorganisms such as bacteria and viruses are in a molecular mimicry relationship with self-antigens, immune responses to infections may cause autoimmune diseases.2, 3 Autoimmune diseases caused by molecular mimicry could also be caused by vaccines. However, cases of PV occurring after vaccination are rare.

The mechanism by which PV is triggered by vaccination is unknown. Here, we report a case of PV with latent hypopharyngeal and gastric cancer that manifested after SARS-CoV-2 vaccination. Skin symptoms, histopathological findings of skin blister lesions and gastrointestinal endoscopic findings. Flaccid bullae with erythema on the right lumbar region (a) and left upper arm (b) at the time of our hospital visit. The histopathological sections, stained with hematoxylin and eosin, revealed intraepidermal blisters (magnification × 200) (c). Direct immunofluorescent staining of tissue sections from blister lesions demonstrated IgG deposition on keratinocytes (magnification × 400) (d). Hypopharyngeal cancer (e) and gastric cancer (f). The white arrows indicate the margin of hypopharyngeal cancer.

Read the full study here.

Development of severe pemphigus vulgaris following ChAdOx1 nCoV-19 vaccination and review of literature

Journal of Cosmetic Dermatology – March 2022


Vaccines are indeed a boon for tackling the present COVID-19 pandemic. In India, ChAdOx1 nCoV-19 (Covishield) is the most commonly used vaccine in the government vaccination program for adults more than 18 years of age. It is a recombinant vaccine developed by Oxford-Astra Zeneca and manufactured in India by Serum Institute of India (SSI). Here, we report a case of severe pemphigus vulgaris following the second dose of ChAdOx1 nCoV-19 vaccination in an adult male. The patient developed septicemia during the course of hospital stay, and he was managed with systemic steroids, parenteral antibiotics, and intravenous immunoglobulins (IVIg) along with proper wound care. Patient started improving within 1 month of therapy. This case is being reported in view of the rarity of pemphigus vulgaris following ChAdOx1 nCoV-19 vaccine.(A) Multiple crusted erosions over face and left ear along with matted hairs over scalp; (B) multiple crusted erosions with overlying gentian violet seen on chest and abdomen; (C): extensive erosions with some islands of normal skin present over whole back

Read the full study here.


Genital necrosis with cutaneous thrombosis after COVID-19 mRNA vaccination

An 84‐year‐old Japanese woman presented to our department with a three‐day history of genital necrosis. She had received her first dose of Pfizer–BioNTech (New York, NY, USA; Mainz, Germany) BNT162b2 mRNA COVID‐19 vaccine 26 days before admission. Nine days after the vaccination, she developed increasing pain in her genital region. She denied any trauma or precipitating event. Her medical history was significant for deep vein thrombosis after orthopaedic surgery, for which she had been receiving edoxaban over the past three years. She had no other risk factors for thrombosis.


Skin necrosis at both COVID-19 vaccine injection sites

Delayed injection-site reactions (occurring 8 days or more after vaccination) are seen with the COVID-19 vaccine. Typically, the reaction is characterized by pain, erythema, and induration near the injection site that resolves within 5 days. 1 We report a case of necrotic eschars appearing one week after the second dose of the COVID-19 BNT162b2 (Pfizer) vaccine at the injection sites.



BTW: this is not new, it’s just taken this long to get these adverse events published in journals.

For more:

The Alarming Consequences of COVID-19 “Vaccines” For Children

Europe has suffered a horrifying 755% increase in excess deaths among children since EMA approved the experimental gene therapy shots for children.

The Alarming Consequences of COVID-19 Vaccines for Children

by Sep 19, 2022

Data from a recent study suggest that COVID-19 vaccines have a detrimental long-term effect on children’s immune systems regardless of prior infection.

The findings of a study reported in a letter to the editor of the New England Journal of Medicine (NEJM) on September 7 include alarming data that belies the authors’ conclusion that children between the ages of five and eleven should receive booster doses of a COVID‑19 vaccine in addition to the primary two-dose series. The authors expressed the view that even children who have already acquired natural immunity should get fully vaccinated plus boosted.“The rapid decline in protection against omicron infection that was conferred by vaccination and previous infection”, the study authors concluded, “provides support for booster vaccination.”

However, that conclusion does not follow logically from their study findings, which provide yet further evidence of the problem of “original antigenic sin”(See link for article)



At first the CDC lied by stating that natural immunity was weak and short-lived, and then lied again by claiming that it was inferior to the shots.  Natural immunity is robust, broad, and durable – unlike the mRNA gene therapy injections which have been linked with more adverse reactions and death than any other vaccine in the history of VAERS.

The key finding of the study actually showed that children with natural immunity who remained unvaccinated had much more durable protection than those who recovered from an infection and then got vaccinated – yet the authors declined to offer any comment on this fact.

Further, “vaccination” of previously infected children resulted in the children having an increased risk of infection in less than half a year since becoming vaccinated, demonstrating that far from conferring additional protective benefit, their naturally acquired immune protection is somehow being wiped out!

Important quote:

In sum, the accumulating data continue to point to the conclusion that vaccinating children confers a short-term protective benefit at the long-term cost of making them more susceptible to COVID‑19 throughout their lifetimes.

The result appears to be corroborated by a study the CDC refused to do on the vaccinated vs the unvaccinated

Scientists found no significant differences in rates of vaccine-preventable illnesses like hepatitis A or B, measles, mumps, rubella, influenza, meningitis or rotavirus.  As would be expected, vaccinated children did have lower likelihood of two vaccine-preventable illnesses compared to unvaccinated children: chicken pox (7.9% vs. 25.3%), and pertussis (2.5% vs. 8.4%).

The study suggests that fully vaccinated children may be trading the prevention of certain acute illnesses (chicken pox, pertussis) for more chronic illnesses and neurodevelopmental disorders like ADHD and Autism. The scientists also found that children born prematurely, who were vaccinated, were 6.6 times more likely to have a neurodevelopmental disorder.  Source