Archive for the ‘Autism’ Category

Injecting Aluminum Documentary – FREE – But Hurry!  Approx. 90 Min. Click here to see FREE VIDEO

Courtesy of Stop Mandatory Vaccination

About Injecting Aluminum

In the early 90s, a mysterious muscular disease began to surface among multiple patients in France. A team of doctors in Paris discovered that these patients had developed a new disease called Macrophagic Myofascitis, or MMF, which occurs when the aluminum hydroxide adjuvant from a vaccine remains embedded in the muscle tissue.

Note: This film was released in France under the title L’Aluminum, les vaccins and les deux lapins (translated: “Aluminum, vaccines and two rabbits”). The aluminum adjuvant was only tested for 28 days, on two rabbits, and their remains have mysteriously disappeared.

Injecting Aluminum features groundbreaking interviews with leading aluminum specialists such as:


Dr. Romain Gherardi, the Director of the French National Institute of Health and Medical Research


Dr. Yehuda Shoenfeld, founder of the leading Centre for Autoimmune Diseases at the Sheba Medical Center


“Mr. Aluminum”, Dr. Christopher Exley, biologist at the University of Stirling with a PhD in the Ecotoxicology of Aluminum (Aluminum in brain tissue in Autism)


Dr. Jérôme Authier, neurologist and head of the Center of Reference of neuromuscular diseases of the Henri Mondor Hospital in Créteil, France.

Frequently Asked Questions

Available worldwide in 250+ territories (excluding France)

To watch the film, just click on link above.

After viewing online for free, you can also purchase DVDs.

To receive 40% off the Injecting Aluminum DVDs until March 8th, enter code SMV during check out from our store (code must be entered to receive the discount).


Note: This film was released in France under the title L’Aluminum, les vaccins and les deux lapins (translated: “Aluminum, vaccines and two rabbits”). The aluminum adjuvant was only tested for 28 days, on two rabbits, and their remains have mysteriously disappeared.

CMSRI (Children’s Medical Safety Research Institute) helped fund the grants for several scientists featured in Injecting Aluminum, to do research, specifically Dr. Christopher Exley as well as Dr. Romain Gherardi, Dr. Jerome Authier. This nonprofit organization is dedicated to funding independent research at leading universities and medical facilities worldwide to identify the causal factors behind today’s epidemic of chronic illness and disabilities.

More on Vaccines:  I highlight a 9 part series on vaccines.


Dr. Frid: Children & Lyme

 Approx. 40 Min

Dr. Elena Frid:  Children & Lyme

Published on Mar 9, 2018

Dr. Elena Frid, a Board Certified New York City Neurologist, with clinical interest in Autoimmune and Neuro-Lyme discusses Children who have been effected by Lyme disease at an annual Tick Talk in Pennsylvania.
She goes into depth about Autoimmune Encephalitis (AE), Neuro-developmental issues with Lyme and co-infections, PANS/PANDAS, illustrates case studies, discusses blood work protocols, and protective methods all must follow in order to prevent and protect themselves against Lyme disease.


For More:


The Importance of Vitamin D, K, and Magnesium for Lyme/MSIDS Patients

Without Magnesium, Vitamin D Supplementation May Backfire

vitamin d

Story at-a-glance

  • When taking high-dose vitamin D3, it’s important to also take extra vitamin K2 and magnesium to avoid complications associated with excessive calcification
  • Magnesium, the fourth most abundant mineral in your body, is a component necessary for the activation of vitamin D, and without sufficient amounts of it, your body cannot properly utilize the vitamin D you’re taking
  • As many as 50 percent of Americans taking vitamin D supplements may not get significant benefit, as the vitamin D simply gets stored in its inactive form due to magnesium deficiency
  • The only way to determine how much sun exposure is enough and/or how much vitamin D3 you need to take is to measure your vitamin D level, ideally twice a year
  • To assess your magnesium level, check your RBC magnesium level and track signs and symptoms of magnesium insufficiency to determine how much magnesium you need. Low potassium and calcium are also common laboratory signs indicating magnesium deficiency

By Dr. Mercola

I’ve previously written about the importance of taking vitamin K2 when you’re taking high-dose supplemental vitamin D to avoid complications associated with excessive calcification in your arteries. Now, research highlights the vital importance of taking magnesium in combination with vitamin D as well.

Magnesium, the fourth most abundant mineral in your body, is a component necessary for the activation of vitamin D, and without sufficient amounts of it, your body cannot properly utilize the vitamin D you’re taking.1,2,3,4

This may actually help explain why many need rather high doses of vitamin D to optimize their levels — it could be that they simply have insufficient amounts of magnesium in their system to activate the vitamin D. As noted by co-author Mohammed Razzaque, professor of pathology at Lake Erie College of Osteopathic Medicine in Pennsylvania:5

“People are taking vitamin D supplements but don’t realize how it gets metabolized. Without magnesium, vitamin D is not really useful. By consuming an optimal amount of magnesium, one may be able to lower the risks of vitamin D deficiency, and reduce the dependency on vitamin D supplements.”

Without Additional Magnesium, Vitamin D Supplementation May Be Ineffective

According to this scientific review,6 as many as 50 percent of Americans taking vitamin D supplements may not get significant benefit, as the vitamin D simply gets stored in its inactive form. As reported in the press release by the American Osteopathic Association:7

“… [C]onsumption of vitamin D supplements can increase a person’s calcium and phosphate levels even if they remain vitamin D deficient. The problem is people may suffer from vascular calcification if their magnesium levels aren’t high enough to prevent the complication. Patients with optimum magnesium levels require less vitamin D supplementation to achieve sufficient vitamin D levels …

Deficiency in either of these nutrients is reported to be associated with various disorders, including skeletal deformities, cardiovascular diseases, and metabolic syndrome. While the recommended daily allowance for magnesium is 420 mg for males and 320 mg for females, the standard diet in the United States contains only about 50 percent of that amount. As much as half of the total population is estimated to be consuming a magnesium-deficient diet.”

Higher Magnesium Intake Lowers Risk of Vitamin D Deficiency

Indeed, previous research has indicated that higher magnesium intake helps reduce your risk of vitamin D deficiency — likely by activating more of it. As noted in one 2013 study:8

“Magnesium plays an essential role in the synthesis and metabolism of vitamin D and magnesium supplementation substantially reversed the resistance to vitamin D treatment in patients with magnesium-dependent vitamin-D-resistant rickets … High intake of total, dietary or supplemental magnesium was independently associated with significantly reduced risks of vitamin D deficiency and insufficiency respectively.

Intake of magnesium significantly interacted with intake of vitamin D in relation to risk of both vitamin D deficiency and insufficiency. Additionally, the inverse association between total magnesium intake and vitamin D insufficiency primarily appeared among populations at high risk of vitamin D insufficiency.

Furthermore, the associations of serum 25(OH)D with mortality, particularly due to cardiovascular disease and colorectal cancer, were modified by magnesium intake, and the inverse associations were primarily present among those with magnesium intake above the median. Our preliminary findings indicate it is possible that magnesium intake alone or its interaction with vitamin D intake may contribute to vitamin D status.”

Vitamin D Lowers Mortality Risk Associated With Heart Disease

Vitamin D, a steroid hormone, is vital for the prevention of many diseases, including but not limited to Type 2 diabetes, age-related macular degeneration (the leading cause of blindness), Alzheimer’s disease, heart disease and well over a dozen different types of cancer (including skin cancer). Vitamin D also exhibits its infection-fighting abilities in the treatment of tuberculosis, pneumonia, colds and flu.

Studies have also linked higher vitamin D levels with lowered mortality from all causes.9,10,11 Most recently, a Norwegian study12 published in the Journal of Clinical Endocrinology and Metabolism found “a normal intake of vitamin D” significantly reduces your risk of death if you have cardiovascular disease.13

About 4,000 patients diagnosed with stable angina pectoris (chest pain caused by coronary heart disease) were followed for 12 years. The average age at the outset of the study was 62. Overall, those with vitamin D blood levels between 16.8 and 40 ng/mL (42 to 100 nmol/L) had the lowest mortality risk.

Evidence Suggests Higher Vitamin D Levels Are Better

Interestingly, these findings are actually at odds with mounting research showing 40 ng/mL is at the low end of optimal. The new target is 60 to 80 ng/mL, but even a level upward of 100 ng/mL appears safe and beneficial for certain conditions, especially cancer. For example:

  • Having a serum vitamin D level of 40 ng/mL has been shown to reduce your risk for cancer by 67 percent, compared to having a level of 20 ng/ml or less; most cancers were found to occur in people with a vitamin D blood level between 10 and 40 ng/mL14,15
  • Research published in 2005 showed women with vitamin D levels above 60 ng/mL had an 83 percent lower risk of breast cancer than those with levels below 20 ng/mL16
  • A 2007 study found women over 55 who raised their average serum level to 38 ng/mL lowered their risk of all invasive cancers, including breast cancer, by 77 percent17

Other recent research18 has also highlighted the importance of vitamin D for the prevention and treatment of heart disease, showing it plays a vital role in protecting and repairing damage to your endothelium. The findings also suggest vitamin D3:

  • Helps trigger production of nitric oxide — a molecule known to play an important signaling role in controlling blood flow and preventing blood clot formation in your blood vessels
  • Significantly reduces oxidative stress in your vascular system, which is important to help prevent the development and/or progression of cardiovascular disease

According to vitamin D researcher Dr. Michael Holick, author of “The Vitamin D Solution: A Three-Step Strategy to Cure Our Most Common Health Problem,” vitamin D deficiency — defined as a level below 20 ng/mL — can raise your risk of heart attack by 50 percent. What’s worse, if you have a heart attack while vitamin D deficient, your risk of dying is nearly guaranteed.

Vitamin D Levels Below 20 ng/mL Are Inadequate for Optimal Health

Vitamin D levels below 20 ng/mL have repeatedly been shown to raise your risk for a number of other problems as well, including depression19 and Type 2 diabetes. Research suggests 20 ng/mL is not even adequate for the prevention of osteomalacia (softening of your bones).

In the case of depression, having a vitamin D level below 20 ng/mL will raise your risk by as much as 85 percent, compared to having a level above 30 ng/mL,20 and evidence suggests vitamin D deficiency may be a significant driver of the rise we see in both mood and mental disorders such as psychotic21 and obsessive compulsive22disorders.

As for diabetes, an analysis23 by GrassrootsHealth reveals people with a median vitamin D level of 41 ng/mL have a diabetes rate of 3.7 per 1,000, whereas those with a median serum level of just 22 ng/mL have a diabetes rate of 9.3 per 1,000. In other words, raising your level above 40 ng/mL can lower your risk of Type 2 diabetes by nearly 60 percent.

All in all, there’s very little reason to believe that a level as low as 16 ng/mL would be protective against death if you have heart disease.

Research also shows higher vitamin D levels can help prevent and/or treat:

Dry eye syndromes24,25 and macular degeneration26,27
Autoimmune diseases, including psoriasis
Gastrointestinal diseases28
Infectious diseases, including influenza and HIV29,30
Inflammatory rheumatic diseases31 such as rheumatoid arthritis
Osteoporosis and hip fractures
Neurological diseases such as Alzheimer’s disease32,33 and epilepsy. In one study,34 epileptics given a one-time megadose of vitamin D3, ranging from 40,000 IUs all the way up to 200,000 IUs, followed by a daily dose of 2,000 to 2,600 IUs a day for three months to bring each individual’s vitamin D status to at least 30 ng/mL, resulted in significant improvements.

Ten out of 13 had a decrease in the number of seizures, five of which experienced more than a 50 percent reduction. Overall, the group had a 40 percent reduction in the number of seizures.

Lupus. According to researchers in Cairo,35 most patients with systemic lupus erythematosus have some level of vitamin D deficiency, defined as a level of 10 ng/mL or less, or insufficiency, a level between 10 and 30 ng/mL.
Obstructive sleep apnea. In one study, 98 percent of patients with sleep apnea had vitamin D deficiency, and the more severe the sleep apnea, the more severe the deficiency.36
Falls, fractures, dental health and more. A 2006 review37 looking at vitamin D intakes and health outcomes such as bone mineral density, dental health, risk of falls, fractures and colorectal cancer, found “the most advantageous serum concentrations of 25(OH)D begin at 30 ng/mL, and the best are between 36 to 40 ng/mL.”
Obesity. Research38 has shown vitamin D supplementation (4,000 IUs/day) combined with resistance training helps decrease your waist-to-hip ratio — a measurement that is far better at determining your risk for Type 2 diabetes and heart disease than body mass index.
Neurodegenerative diseases, including multiple sclerosis (MS).39,40,41 Research shows MS patients with higher levels of vitamin D tend to experience less disabling symptoms.42

Assess Your Vitamin D and Magnesium Levels

The best way to optimize your vitamin D level is through sensible sun exposure. Unfortunately, this can be difficult for many, especially during the winter season and/or if you live in northern regions. If you cannot obtain sufficient amounts of vitamin D through sun exposure, taking a supplement is recommended. Remember that the only way to determine how much sun exposure is enough and/or how much vitamin D3 you need to take is to measure your vitamin D level, ideally twice a year.

The D*Action Project by GrassrootsHealth is a cost-effective way to do this, while simultaneously progressing valuable research. To participate, simply purchase the D*Action Measurement Kit and follow the registration instructions included. To assess your magnesium level, check your RBC magnesium level and track signs and symptoms of magnesium insufficiency to determine how much magnesium you need. Low potassium and calcium are also common laboratory signs indicating magnesium deficiency.

The Interplay of Vitamin D, Calcium, Magnesium and Vitamin K2

When supplementing, also remember to take synergistic effects with other nutrients into account. If you take high-dose vitamin D, you may also need to increase your intake of:

  • Magnesium
  • Vitamin K2
  • Calcium

These four nutrients — vitamins D and K2, calcium and magnesium — all work in tandem, and rely on sufficient amounts of each to work optimally.Lack of balance between these four nutrients is why calcium supplements have become associated with increased risk of heart attack and stroke, and why some experience symptoms of vitamin D toxicity. Here’s a summary of some of the most important correlations between these nutrients:

Excessive vitamin D in combination with lack of vitamin K2 may cause overabsorption of calcium, which in turn may result in calcium deposits in your heart and kidneys. Part of the explanation for these adverse side effects is that vitamin K2 keeps calcium in its appropriate place — in your teeth and bones and out of soft tissues and arteries.

While the optimal ratios between vitamin D and vitamin K2 have yet to be established, taking somewhere between 100 to 200 micrograms (mcg) of K2 is beneficial. Telltale signs of vitamin K2 insufficiency include osteoporosis, heart disease and diabetes. You’re also more likely to be deficient if you rarely eat vitamin K2-rich foods (see listing below).

Vascular calcification is also a side effect of low magnesium, so when taking vitamin D3, you need both vitamin K2 and magnesium to make sure everything is working properly.

Maintaining an appropriate calcium-to-magnesium ratio is also important, as magnesium helps keep calcium in your cells so they can function better. Historically, mankind ate a diet with a calcium-magnesium ratio of 1-to-1,43 which is likely close to ideal.

Magnesium and vitamin K2 also complement each other, as magnesium helps lower blood pressure, which is an important component of heart disease.

Eat a Varied Diet and Get Sensible Sun Exposure

The take-home message is that anytime you’re taking supplemental magnesium, calcium, vitamin D3 or vitamin K2, you must take all the others into consideration as well. While supplements can be helpful in some instances, your best and safest bet is to simply eat more calcium-, magnesium- and vitamin K2-rich foods, along with sensible sun exposure.

Food-derived nutrition is typically going to be the most beneficial, and this is particularly true for calcium. When you take a biologically foreign form of calcium, or when your body’s ability to direct calcium to the right places becomes impaired (as when you are deficient in vitamin K2 and/or magnesium), calcium is deposited where it shouldn’t be, such as in your arteries. It’s more likely your body can use calcium correctly if it’s plant-derived calcium. Here’s a quick summary of foods known to be high in these important nutrients:

Vitamin K2

Grass fed organic animal products such as eggs and butter, fermented foods such as natto, goose liver pâté and vitamin K2-rich cheeses such as Brie and Gouda


Almonds and cashews, bananas, broccoli and Brussels sprouts, brown rice, free-range pastured egg yolk, flaxseed, grass fed raw milk, mushrooms, pumpkin seeds, sesame seeds, sunflower seeds and leafy green vegetables, especially spinach, Swiss chard, turnip greens, beet greens, collard greens, kale, Bok Choy and romaine lettuce


Raw milk from grass fed cows (who eat plants), leafy green vegetables, the pith of citrus fruits, carob and wheatgrass

The Role of Vitamin D in Disease Prevention

A growing body of evidence shows that vitamin D plays a crucial role in disease prevention and maintaining optimal health. There are about 30,000 genes in your body, and vitamin D affects nearly 3,000 of them, as well as vitamin D receptors located throughout your body.

According to one large-scale study, optimal Vitamin D levels can slash your risk of cancer by as much as 60 percent. Keeping your levels optimized can help prevent at least 16 different types of cancer, including pancreatic, lung, ovarian, prostate and skin cancers.

How Vitamin D Performance Testing Can Help Optimize Your Health

Is it any wonder then that no matter what disease or condition is investigated, vitamin D appears to play a crucial role? This is why I am so excited about the D*Action Project by GrassrootsHealth. Dr. Robert Heaney is the research director of GrassrootsHealth and is part of the design of the D*action Project as well as analysis of the research findings.

GrassrootsHealth shows how you can take action today on known science with a consensus of experts without waiting for institutional lethargy. It has shown how by combining the science of measurement (of vitamin D levels) with the personal choice of taking action and, the value of education about individual measures that one can truly be in charge of their own health.

In order to spread this health movement to more communities, the project needs your involvement. To participate, simply purchase the D*Action Measurement Kit and follow the registration instructions included. (Please note that 100 percent of the proceeds from the kits go to fund the research project. I do not charge a single dime as a distributor of the test kits.)

As a participant, you agree to test your vitamin D levels twice a year during a five-year study, and share your health status to demonstrate the public health impact of this nutrient. There is a $65 fee every six months for your sponsorship of this research project, which includes a test kit to be used at home, and electronic reports on your ongoing progress. You will get a follow up email every six months reminding you “it’s time for your next test and health survey.”

To order:



Great article showing how vitamins interact and often depend upon one another, which is why obtaining them naturally through food-choices is always safest; however, as this article points out, vitamin D is obtained by direct sunlight hitting the skin.  We Northerners don’t get enough making supplementation with D almost a necessity.  Please have your levels tested as D is a fat soluble vitamin that can build up in the body and become toxic.

It is my experience that Lyme/MSIDS patients here in Wisconsin are low in Magnesium as well as D and supplementation always helps them.

Interestingly, besides the plethora of things listed above, vitamin D supplementation has been shown to help Autism:   The scientists reported significant improvements in autism symptoms and social maturity among the exposed group and no reported gains among the placebo group. Some of the improvements reported among the exposed children included reductions in irritability, hyperactivity, social withdrawal, and inappropriate speech. ATEC scoring among the exposed group was significantly improved notably in the scores relating to sociability, cognitive awareness, and behavior.

Vitamin D has also been found to reduce risk of cancer:  “These findings support the hypothesis that vitamin D has protective effects against cancers at many sites,” the authors write.

More on Magnesium:

More on Depression:








Vaccines Could Contribute to Disease Epidemics Due to Retrovirus Contamination

The vaccine manufacturing process could be contributing to disease epidemics due to retrovirus contamination

Image: The vaccine manufacturing process could be contributing to disease epidemics due to retrovirus contamination

(Natural News) Vaccines are highly controversial, and much of the debate rightly surrounds their connection to autism and their inclusion of mercury and other heavy metals. However, there is another aspect of vaccines that doesn’t get as much attention even though it could have grave repercussions for life on this planet: Retrovirus contamination.

A type of virus whose genes are encoded into RNA rather than DNA, retroviruses function differently than other types of viruses but can be just as devastating. Human retroviruses include HIV and human T-cell lymphotropic virus 1, which has been associated with some leukemias and lymphomas.

All animals have some retroviruses integrated into their genomes, with as much as 15 percent of the human genome believed to be made up of ERV human retroviruses. They contribute to diseases such as cancer, autoimmune diseases, and neuroimmune diseases.

The manufacturing process involved in human and veterinary vaccines uses live animal and human cells to serve as master seed stock. The process was developed before the mechanisms of retroviruses were fully understood, which means that no one was aware of the risks at the time. While many retroviruses are harmless and remain essentially dormant, they become harmful when they are exposed to cells from other species, which is what can happen during the vaccine manufacturing process itself as well as during the administration of vaccines that contain cell components from other species. Incidentally, this is similar to what has been creating problems when researchers try to transplant pig tissues and organs into humans.

As new vaccines were developed in the 1970s, exposure to retroviruses intensified. These retroviruses have now been associated with many of the chronic illnesses modern humans are facing, from cancer and autism to Alzheimer’s and Chronic Fatigue Syndrome. It is believed that as many as 20 million Americans could be infected with retroviruses, but not all of them will go on to develop a serious illness. They can be thought of as sleeping giants, who are only awakened when there is an immune deficiency.

A recent study published in the journal Biologicals outlined how endogenous retroviruses (ERVs) are infecting cats and how the feline retrovirus known as RD-114 has contaminated vaccines for other pets. There are at least two ERVs in cat genomes, and the RD-114 virus is found in a wide range of vaccines for dogs and other animals. In fact, a different study showed that RD-114-related viruses contaminated around a third of the 25 commercial vaccines for dogs and cats tested.

Retrovirus contamination in vaccines widespread

Research scientist Judy A. Mikovits even goes so far as to say that nearly all human vaccines are contaminated with at least one retrovirus, usually from an animal thanks to the animal byproducts used in vaccine creation. Six percent of Americans now have mouse-related retroviruses, for example, and that almost certainly comes from vaccines.

Take the example of the rotavirus vaccines RotaTeq and Rotarix, which made headlines a few years ago when they were found to be contaminated with pig viruses. The problem becomes even worse when multiple vaccines are given in combination, as is often the case with children and infants. Dr. Mikovits points out that one pig retrovirus can cause symptoms similar to AIDS when there is a simultaneous immune system activation, such as concurrent vaccination, creating a “high-risk scenario for disease in humans.”

Vaccines are risky enough when you take retroviruses out of the equation, but this vastly underreported problem could eventually prove to be the biggest danger of vaccines yet.

Sources for this article include:



Lyme/MSIDS can activate the immune system and be that “sleeping giant,” the author points out.  Dr. Lambert has treated a number of young women who fell ill after their HPV vaccination, which seems to have stimulated a latent Lyme infection to reactivate.  Asymptomatic girls after receiving Gardasil activated dormant Bartonella which was confirmed by testing.

More on retroviruses:  What did microbiologist Judy Mikovitz get for exposing retroviruses in vaccines?  She got jailed without a search warrant and publicly demolished by those who can’t stand exposure to the truth.



Transplacental Transmission & Fetal Damage With Lyme Disease


The two sides of the coin in pregnancy and LYME DISEASE

Hello friends of the network DERMAGIC EXPRESS, I bring you today another topic for what I call the “SAGA” on LYME DISEASE, in this case the controversial issue of TRANSPLACENTAL TRANSMISSION AND FETAL DAMAGE AND DEATH in pregnant women and infected with the feared BORRELIA BURGDORFERI.

I have found numerous references; most claim that BORRELIA in pregnant women with LYME DISEASE traverses the placenta and reaches the fetus and can cause multi organic damage, including the death of the same, intrauterine or a few hours or days after birth. Other authors say that this is false.

The CDC (Center of Control of Infectious Diseases) affirms that if the pregnant woman with LYME does her treatment, the child will be born healthy and recommends the use of the antibiotic AMOXICILLIN, because DOXYCYCLINE can cause damage to the developing fetus. The question here is what would happen if the BORRELIA species is resistant to AMOXICILLIN? Or the antibiotic to which BORRELIA is sensitive cannot be indicated because it would harm the fetus?

BORRELIA BURGDORFERI was discovered in 1982 by the aforementioned Willy Burgdorfer, the causal agent of the ERYTHEMA MIGRANS or LYME DISEASE, and only 1 year later the first study in 1983 described that it is suspected that this ESPIROCHETE can cross the placenta and infect the fetus, study published by Shirts SR, Brown MS, and Bobitt JR. under the name of “Listeriosis and borreliosis as causes of antepartum fever”. (8)

Later in the year 1985 Schlesinger PA, Duray PH, Burke BA, Steere AC, Stillman MT published a paper called “Maternal-fetal transmission of the spirochete of Lyme disease, Borrelia burgdorferi” where they report a case of a woman who developed LYME DISEASE and did not receive treatment with antibiotics. The child was born at 35 weeks of pregnancy and died of congenital heart disease the first week of life. The autopsy revealed the LYME ESPIROCHETE in the SPLEEN, KIDNEYS AND BONE MARROW. (2)

Later, the same WILLY BURGDORFER the discoverer and “father” of the ESPIROCHETE BORRELIA, who along with Dr. Alan Mc Donald and Jorge Benach PhD, published in the year 1987 (31 years ago) a work they called “stillbirth following maternal LYME DISEASE.” and I quote from the conclusions of these scientists: (24.)

“… Two cases of transplacental transmission of the BORRELIA BURGDORFERI were found associated with fetal death and congenital malformations, different anomalies were detected in each case …”

“… We recommend that pathologists study the tissues of stillborn fetuses in search of BORRELIA BURGDORFERI especially those with cardiac anomalies, and clinical doctors investigate the exposure during the first trimester of pregnancy to BORRELIA BURGDORFERI and in these cases determine if cardiac organogenesis is complete by the end of the first trimester of pregnancy”

“… We believe that there is enough evidence to alert women living in endemic areas of LYME DISEASE and doctors to recognize the early signs and symptoms of the disease and to start treatment with PENICILLIN at the same dose of SYPHILIS as used in pregnant women in the first trimester, regardless of the results of the laboratory tests … “

Another study that is worth noting is the one made by the MEDLINE database updated for the year of July 2012, the last revision of November 2012 of 88 journal articles from the PUBMED database, which I summarize as follows:

Maternal-Fetal Transmission of Lyme Disease


1.) Mothers with active Lyme Disease,Treated: 14.6% of the pregnancies with sequelae,
2.) Untreated: 66.7% of the pregnancies with sequelae,
3.) Unknown as to treatment: 30.3% with sequelae.
4.) Specific adverse outcomes included: cardiac 22.7%, neurologic 15.2%, orthopedic 12.1%, ophthalmic 4.5%, genitourinary 10.6%, miscellaneous anomalies 12.1%, 2nd trimester demise 12.1%. Highest rate of adverse outcome (72.7%) in women with infection acquired prior to or during first trimester.)

Now I will put a summary of the most frequent clinical manifestations described in the studies of children born to mothers with LYME disease, LYME positive


1.) LOW GRADE FEVER: 59% -60%
5.) ABDOMINAL PAIN: 20-29%
7.) NAUSEA: 23%
15.) VERTIGO: 30%
20.) ANXIETY: 21%
21.) ANGER OR RAGE: 23%
23.) IRRITABILITY: 54% -80%
25.) DEPRESSION: 13%
27.) PHOTOPHOBIA: 40-43%
46.) AUTISM.

There are numerous studies showing a clear EVIDENCE that the BORRELIA BURGDORGFERI in pregnant women is able to cross the placenta and infect the fetus. I could get tired here of giving you the description of each of them. But I will give you ALL the BIBLIOGRAPHIC REFERENCES that I found from the year 1983 until the year 2017, first the ones I found and then a chronology of ALL of them.

I close this issue which is HIGHLY DISCUSSED TODAY, with a post by Angélica Johansson, a great fighter against THIS PLAGUE that I found in my LINKEDIN network about the future of the planet and the LYME DISEASE … I quote:

“…”1 million people are predicted to get infected with Lyme disease in the USA in 2018. Given the same incidence rate of Lyme disease in Europe as in the USA, then 2.4 million people will get infected with Lyme disease in Europe in 2018. In the USA by 2050, 55.7 million people (12% of the population) will have been infected with Lyme disease. In Europe by 2050, 134.9 million people (17% of the population) will have been infected with Lyme disease. Most of these infections will, unfortunately, become chronic.  

The estimated treatment cost for acute and chronic Lyme disease for 2018 for the USA is somewhere between 4.8 billion USD and 9.6 billion USD and for Europe somewhere between 10.1 billion EUR and 20.1 billion EUR. If governments do not finance IV treatment with antibiotics for chronic Lyme disease, then the estimated government cost for chronic Lyme disease for 2018 for the USA is 10.1 billion USD and in Europe 20.1 billion EUR.

If governments in the USA and Europe want to minimize future costs and maximize future revenues, then they should pay for IV antibiotic treatment up to a year even if the estimated cure rate is as low as 25%. The cost for governments of having chronic Lyme patients sick in perpetuity is very large….”

But what you see every day is a fight between IDSA and ILADS, CDC and others on the subject of whether it is a simple tick bite and you take an antibiotic and you cure or that it is a disease of difficult diagnosis and high cost of treatment. Between believers and non-believers to summarize. The truth is that it is spreading all over the world in leaps and bounds.

And if you have doubts that this ESPIROCHETE may or may not harm the fetus of pregnant women, cause birth defects, and many other consequences including, stillborn babies, read this “MOUNTAIN” of references that I leave here.

   Approx. 25 Min.

Sue Faber, RN and Co-Founder of LymeHope speaks to pregnancy and Gestational Lyme at the LymeHope Education Event, Oakville, Ontario on November 3, 2017.  Published on Jan 18, 2017

Dr. Elena Frid, a board-certified NYC neurologist and specialist in Lyme disease & other vector-borne diseases, discusses congenital Lyme disease.  

**Comment**  In reference to Dr. Frid’s comment that congenital Lyme is rare, I would disagree.  We have not been keeping track of numbers and there are probably way more than is being acknowledged.

CONCLUSION: BORRELIA BURGDORFERI, not only transmitted by the tick bite, is TRANSMITTED by sexual contact, fluids and can also colonize the fetus of pregnant women if there is no effective treatment able to eradicate it during the same. And it is not exclusive to the Northern Hemisphere. The BORRELIA is also in the Southern Hemisphere.

Greetings to all.

Dr. José Lapenta.

1.) Lyme disease during pregnancy. Infect Dis Clin North Am. 1997 Mar;11(1):93-7. ]PUBMED] Silver HM1.

2.) Maternal-fetal transmission of the Lyme disease spirochete, Borrelia burgdorferi. Ann Intern Med. 1985 Jul;103(1):67-8. [PUBMED]. Schlesinger PA, Duray PH, Burke BA, Steere AC, Stillman MT.

3.) Borrelia burgdorferi in a newborn despite oral penicillin for Lyme borreliosis during pregnancy. Pediatr Infect Dis J. 1988 Apr;7(4):286-9. [PUBMED]. Weber K1, Bratzke HJ, Neubert U, Wilske B, Duray PH.

4.) Neonatal skin lesions due to a spirochetal infection: a case of congenital Lyme borreliosis? Int J Dermatol. 1997 Sep;36(9):677-80. [PUBMED]. Trevisan G1, Stinco G, Cinco M.

5.) Confirmation of Borrelia burgdorferi spirochetes by polymerase chain reaction in placentas of women with reactive serology for Lyme antibodies. Gynecol Obstet Invest. 1996;41(4):240-3. [PUBMED]. Figueroa R1, Bracero LA, Aguero-Rosenfeld M, Beneck D, Coleman J, Schwartz I.

6.) Detection of Borrelia burgdorferi DNA in urine of patients with ocular Lyme borreliosis.
Pleyer U1, Priem S, Bergmann L, Burmester G, Hartmann C, Krause A. Br J Ophthalmol. 2001 May;85(5):552-5. [PUBMED]

7.) Culture and identification of Borrelia spirochetes in human vaginal and seminal secretions [version 1; referees: 1 not approved]. Marianne J. Middelveen1, Jennie Burke2, Eva Sapi3, Cheryl Bandoski3, Katherine R. Filush3, Yean Wang2, Agustin Franco2, Arun Timmaraju3, Hilary A. Schlinger1, Peter J. Mayne1, Raphael B. Stricker1
Source: F1000 RESEARCH

8.) Listeriosis and borreliosis as causes of antepartum fever. Obstet Gynecol. 1983 Aug;62(2):256-61. [PUBMED]. Shirts SR, Brown MS, Bobitt JR.

9.) Maternal-fetal transmission of the Lyme disease spirochete, Borrelia burgdorferi.
Schlesinger PA, Duray PH, Burke BA, Steere AC, Stillman MT.

10.) Gestational Lyme borreliosis. Implications for the fetus. Rheum Dis Clin North Am 1989 Nov 15:657-77. MacDonald AB. Source: . Rheum Dis Clin North Am 1989 Nov 15:657-77

11.) transplacental Lyme borreliosis infant mortality. Arthritis Rheum 1987; Volume 30, Number 4, 3(Suppl):S50. Lavoie PE;Lattner BP;Duray PH; Barbour AG; Johnson HC.

12) Lyme Borrelia positive serology associated with spontaneous abortion in an endemic Italian area.) Acta Eur Fertil. 1988 Sep-Oct;19(5):279-81. [PUBMED]. Carlomagno G1, Luksa V, Candussi G, Rizzi GM, Trevisan G.

13.) Infection with Borrelia: Implications for Pregnancy. James M O’Brien 1. and 2 Odessa P Hamidi. Division of Maternal Fetal Medicine, Pennsylvania College of Medicine, USA. Department of Obstetrics and Gynecology, Pennsylvania College of Medicine, USA.

14.) MEDLINE results for: borrelia pregnancy AND human. 88 journal articles in the PubMed
database BDH, July 2012, Latest Revision Novemb
er 2012,

15.) Infants born to mothers with antibodies against Borrelia burgdorferi at delivery. Eur J Pediatr. 1989 Feb;148(5):426-7. [PUBMED]. Nadal D1, Hunziker UA, Bucher HU, Hitzig WH, Duc G.

16.) Human fetal borreliosis, toxemia of pregnancy, and fetal death..Amanda B Macdonald
Published .1986 in Zentralblatt fur Bakteriologie, Mikrobiologie…Hyg A.1986 Dec;263(1-2):189-200

17.) Congenital relapsing fever (Borrelia hermsii).Blood, 15 November 2000, Vol. 96, No. 10, pp. 3333-3333William A. Dittman. Sr, Sacred Heart Medical Center, Spokane, WA.

18.) Lyme Disease and Pregnancy. James M. Alexander and Susan M. Cox. Department of Obstetrics and Gynecology, University of Texas Southwaestern Medical Center, Dallas, TX

19.) Teratogen update: Lyme disease. Teratology. 2001 Nov;64(5):276-81. [PUBMED]. Elliott DJ1, Eppes SC, Klein JD.

20.) Borreliosis during pregnancy: a risk for the unborn child?. Vector Borne Zoonotic Dis. 2011 Jul;11(7):891-8. doi: 10.1089/vbz.2010.0102. Epub 2010 Oct 6. [PUBMED]. Mylonas I1.

21.) Intrauterine transmission of Borrelia burgdorferi in dogs. Am J Vet Res. 1993 Jun;54(6):882-90. [PUBMED]. Gustafson JM1, Burgess EC, Wachal MD, Steinberg H.

22.) Fetal outcome in murine Lyme disease. Infect Immun. 1995 Jan;63(1):66-72. [PUBMED] Silver RM1, Yang L, Daynes RA, Branch DW, Salafia CM, Weis JJ.

23.) The association between tick-borne infections, Lyme borreliosis and autism spectrum disorders. Med Hypotheses. 2008;70(5):967-74. Epub 2007 Nov 5. [PUBMED]. Bransfield RC1, Wulfman JS, Harvey WT, Usman AI. The full text here:

24.) Gestational Lyme Disease Case Studies of 102 Live Births. by Charles Ray Jones, M.D., Harold Smith, M.D., Edina Gibb,. and Lorraine Johnson, JD, MBA

25.) Stillbirth following maternal LYME DISEASE. N Y State J Med. 1987 Nov;87(11):615-6.
[PUBMED] MacDonald AB, Benach JL, Burgdorfer W. Source: full text:

26. ) The Enlarging Spectrum of Tick Borne Spirochetoses; R.R. Parker Memorial address. Reviews of Infectious Diseases, vol.8, no.6 (Nov-Dec 1986), pp.932940 Source fulle text:

27.) Teratogenic effects of the bacteria Borrelia sp. on the fetuses of pregnant women with Lyme disease. Sliwa, Leopold. Nowa Medycyna 04/2011. (Translation of above article)

28.) Lyme disease in pregnancy: case report and review of the literature. Obstet Gynecol Surv. 2007 Jan;62(1):41-50. [PUBMED] Walsh CA1, Mayer EW, Baxi LV.

29.) Borreliosis During Pregnancy: A Risk for the Unborn Child? VECTOR-BORNE AND ZOONOTIC DISEASES. Volume 11, Number 7, 2011. Mary Ann Liebert, Inc..DOI: 10.1089/vbz.2010.0102. Ioannis Mylonas. Source full text:

Lyme Disease and Pregnancy, Maternal Fetal Transmission of Lyme Disease:

1983 Shirts SR, Brown MS, Bobitt Jr. Listeriosis and borreliosis as causes of antepartum fever. Obstet Gynecol 1983;62:256.

1985 Schlesinger PA, Duray PH, Burke BA, Steere AC, Stillman MT. Maternal fetal transmission of the Lyme disease spirochete, Borrelia burgdorferi. (1985) Ann Intern Med, 103, 67-8.

1985 MMWR. Update: Lyme Disease and Cases Occurring during Pregnancy—United States. Vol. 34, No. 25 (June 28, 1985), pp. 376- 378, 383-384

1986 MacDonald A. Human fetal borreliosis, toxemia of pregnancy, and fetal death. Zentralbl Bakteriol Mikrobiol Hyg A. 1986 Dec;263(1-2):189-200.

1986 Burgdorfer, W., The Enlarging Spectrum of Tick Borne Spirochetoses; R.R. Parker Memorial address. Reviews of Infectious Diseases, vol.8, no.6 (Nov-Dec 1986), pp.932940

1986 Markowitz LE, Steere AC, Benach JL, et al. Lyme disease during pregnancy. JAMA.(1986); 255(24), 3394-6.

1987 MacDonald AB, Benach JL, Burgdorfer W. Stillbirth following maternal Lyme disease. N Y State J Med. 1987 Nov;87(11):615-6.

1987. Lavoie PE, Lattner BP, Duray PH, Barbour AG, Johnson HC. Culture positive seronegative transplacental Lyme borreliosis infant mortality. (1987) Arthritis Rheum, 30(4), 3(Suppl):S50.

1988 Weber K; Bratzke HJ, Neubert U, Wilske B, Duray PH. (1988) Borrelia burgdorferi in a newborn despite oral penicillin for Lyme borreliosis during pregnancy. Pediatr Infect Dis J, 7:286-9.

1988 Carlomagno G, Luksa V, Candussi G, et al. (1988) Lyme Borrelia positive serology associated with spontaneous abortion in an endemic Italian area. Acta Eur Fertil 19(5), 279-81.

1988 Medici F, Benach J, Williams C. Lyme Disease during Pregnancy A Cord Blood Serosurvey. Annals New York Academy of Sciences. Volume 539, Lyme Disease and Related Disorders Pages 504–506.

1988 Health and Welfare Canada. Canada Diseases Weekly Report, June 4, 1988. Lyme disease in Canada.
1988 Lyme disease in Canada. Epidemiologic Report. CMAJ Vol. 139, Aug 1, 1988
1989 MacDonald A. Gestational Lyme borreliosis. Implications for the fetus. Rheum Dis Clin North Am. 1989 Nov;15(4):657-77.

1989 Halperin JJ., Dattwyler R., et al. A Perspective on the treatment of Lyme Borreliosis. Reviews of infectious diseases. Vol. 11 Supp 6. Sept/Oct 1989. S1518-1525

1989 Nadal D, Hunziker UA, Bucher HU, et al. (1989) Infants born to mothers with antibodies against Borrelia burgdorferi at delivery. Eur J Pediatr 148(5), 426-7.

1989 Steere et al. Lyme Seropositivity and pregnancy outcome in the absence of symptoms of Lyme disease. Scientific Abstracts June 12-17, 1989. 53 Annual Meeting of American College of Rheumatology.

1991 Lakos A. Lyme Borreliosis in Hungary in the years 1984 through 1989. Parasit hung., 24;5-51, 1991

1992 ACOG Committee Opinion. Lyme disease during pregnancy. Int J Gynecol Obstet 1992, 39; 59-60.

1992. Bracero LA, Wormser GP, Leikin E. Tejani N. Prevalence of seropositivity to the Lyme disease spirochetes during pregnancy in an epidemic area: A preliminary report. J Matern Fetal Investig. 1992(2): 265-268

1993 Hercogova J, Tomankova M, Frosslova D, Janovska D. Early-stage lyme borreliosis during pregnancy: treatment in 15 women with erythema migrans. Ceska Gynekol 58(5):229-232.

1993 Strobino BA, Williams CL, Abid S, et al. (1993) Lyme disease and pregnancy outcome: a prospective study of two thousand prenatal patients. Am J Obstet Gynecol 169(2 Pt 1), 367-74.

1994 Gasser R. et al. A Most Unusual case of a whole family suffering from late Lyme Borreliosis for Over 20 years. Angiology Vol. 45, No. 1: 85-86.

1994 Trevisan G. Lyme Borreliosis; A general survey. Acta dermatovenerologica A.P.S. Vol 3, 94, No. 1/2 4-12

1994 Elsukova LV, Korenberg EI, Kozin GA., [Pathology of pregnancy and the fetus in Lyme disease] [Article in Russian]. Med Parazitol (Mosk). 1994 Oct-Dec;(4):59-62

1995 Gardner T. Infectious Diseases of the Fetus and Newborn, 4th edition, New York, NY. W.B. Saunders Company (1995) Chapter 11, Lyme Disease. page 447 – 528.

1995 Williams CL, Strobino B, Weinstein A, et al. (1995) Maternal Lyme disease and congenital malformations: a cord blood serosurvey in endemic and control areas. Paediatr Perinat Epidemiol 9(3), 320-30.

1995 Schmidt, B. et al. Detection of Borrelia burgdorferi DNA by Polymerase Chain Reaction in the Urine and Breast Milk of Patients with Lyme Borreliosis. DIAGN MICROBIOL INFECT DIS 1995;21:121-128.

1995 Alexander, J. Cox, S. Lyme disease and Pregnancy. Infectious diseases in Obstetrics and Gynecology 3?256-261 (1995)

1996. Figueroa R. et al. Confirmation of Borrelia burgdorferi Spirochetes by Polymerase Chain Reaction in Placentas of Women with Reactive Serology for Lyme Antibodies. Gynecol Obstet Invest 1996; 41?240-243

1996. Maraspin V, Cimperman J. Treatment of Erythema Migrans during Pregnancy. Clinical Infectious Diseases 1996; 22?788-93

1997 Silver H. (1997) Lyme Disease During Pregnancy. Inf Dis Clinics of N. Amer. Vol 11, No 1.

1997 Trevisan G, Stinco G, Cinco M. Neonatal skin lesions due to a spirochetal infection; a case of congenital lyme borreliosis? International Journal of Dermatology 36; 677-99

1999 Norris C., Gardner T. Aseptic Meningitis in the Newborn and Young Infant. Am Fam Physician 1999 May 15;59(10):2761-2770

2001 Elliot D, Eppes S., Klein J. Terratogen Update; Lyme disease. TERATOLOGY 64?276 – 281 (2001)

2001 Gardner T. Chapter 11, Lyme Disease. Remington and Klein: Infectious diseases of the Fetus and Newborn, Fifth edition. New York, NY. W.B. Saunders Company 2001 pgs. 519-641

2001 Gardner T. Lyme disease in pregnancy. Program and abstracts of the 14th International Scientific Conference on Lyme Disease and Other Tick-Borne Disorders; April 21-23, 2001; Hartford, Connecticut.

2003 Goldenberg, R. L and C. Thompson (2003). “The infectious origins of stillbirth.” Am J Obstet Gynecol 189(3): 861-73.

2003 Salvato, WT, Salvato P. Lyme disease: ancient engine of an unrecognized borreliosis pandemic? Medical Hypotheses 60(5): 742-759.

2005 Onk G, Acun C, Kalayci M, Cagavi F, et al. (2005) Gestational Lyme disease as a rare cause of congenital hydrocephalus. J Turkish German Gynecology Association Artemis, 6(2), 156-157.

2005 Jones CR, Smith H, Gibb E, Johnson L (2005) Gestational Lyme Disease: Case Studies of 102 Live Births. Lyme Times. Gestational Lyme Studies 34-36

2005 Goldenberg et al. Maternal Infection and Adverse Fetal and Neonatal Outcomes. Clin Perinatol 32 (2005) 523–559.

2006 Walsh et al. Lyme disease in pregnancy. Obstetrical and Gynecological Survey. CME Review Volume 62, Number 1.

2007 Bransfield, Robert C. et al. The association between tick borne infection, lyme borreliosis and autism spectrum disorder. Medical hypotheses (2007)

2008 Hercogova J, Vanousova D. Syphilis and borreliosis during pregnancy. Dermatol Ther 21(3), 205-9.

2008 Theiler RN, Rasmussen, S. et al. Emerging and Zoonotic infections in women. Infect Dis Clin North Am 2008 December ; 22(4): 755–viii

2009 Lakos et al. Maternal Lyme borreliosis and pregnancy outcome. International Journal of Infectious Diseases 14 (2010) e494–e498

2009. Hulinska D, Votypka J, Vanousova D, Hercogova J, Hulinsky V, Drevova H, Kurzova K, Uherkova L. Identification of Anaplasma phagocytophilum and Borrelia Burgdorferi sensu lato in Patients with Erythema Migrans. Folia Microbiol. 54(3), 246-256 (2009)

2011 Mylonas I. Borreliosis During Pregnancy: A Risk for the Unborn Child? Vector Borne Zoonotic Dis. 11?891-8.

2011 Sliwa, Leopold. Teratogenny wplyw bakterii Borelli sp. Na ploy matek chorujacych na borelioze z Lyme. Zaklad Biologi Rozwoju Czlowieka. Instytus Pielegniarstwa.

2011 Sliwa, Leopold. Teratogenic effects of the bacteria Borrelia sp. on the fetuses of pregnant women with Lyme disease. Nowa Medycyna 04/2011. (Translation of above article)

2012 Relic, Milijana, Relic, Goran. Lyme borreliosis and pregnancy. Vojnosanit Pregl 2012; 69(11): 994–998.

2014 Onyett, H . Lyme disease in Canada: Focus on Children. Paediatr Child Health 2014;19(7):379-83

2014 OʼBrien, JM. Martens MG. Lyme disease in pregnancy; a New Jersey medical advisory. MD advisory, Winter 2014, pgs 24-27

2015 Krysztof PJ et al. Congenital tick borne Diseases: Is this an alternative route of transmission of tick borne pathogens in Mammals? Vector-Borne and Zoonotic Diseases Vol 15, Number 11, 2015.

2015 Hu LT, Tsibris AM, Branda JA. Case Records of the Massachusetts General Hospital: Case 24-2015; A 28 year-old pregnant woman with fever, chills, headache and fatigue. N Engl J Med. 2015 Jul 30;373(5):468-75.

2016 Maldonato, Y, Nizet, V, Klein, J, Remington, J, Wilson, C. Current concepts of Infections of the Fetus and Newborn Infant. Chapter 1. page 6. Infectious Diseases of the Fetus and Newborn Infant. 8th Edition. 2016

2017 OʼBrien, JM. Baum JD. Case Report. The Journal of Family Practice. August 2017; 66(8) pg E9-10 Updated and printed by JC on November 2, 2017

2017 March of Dimes. Lyme disease and Pregnancy. Retrieved from:

2017 Centers for Disease Control,USA. Pregnancy and Lyme Disease. Retrieved from:

Compiled Dec 6, 2017 – by JC and Sue Faber RN

Produced by Dr. Jose Lapenta R. Dermatologist
Maracay Estado Aragua Venezuela 2.018
Telf: 02432327287-02432328571




Fundraiser for Autism in April


Every April, which is Autism Awareness Month, Sound Choice scientists gather to celebrate and share our work with parents, doctors, leaders and organizations who believe in practicing science with a conscience.

This year’s event will focus on ethical stem cell treatments and their ability to offer REAL HOPE for families with autistic children. Dr Deisher and her staff of scientists will share the recent news on their support of a non-toxic drug that can optimize stem cell treatment outcomes for those affected by autism.

Recently published clinical studies have confirmed Sound Choice’s research that there is an ecological factor involved in onset illnesses such as autism and cancer.  With these new data we are planning a strategic study on vaccine safety delving further into the dangers of vaccines manufactured in human cell lines. Studies such as these are expensive and time-consuming. Please come learn how you can partner with us to be a part of a cure.

Guest Speaker: Matthew “Del” Bigtree

This year we are excited to welcome our guest speaker, Matthew “Del” Bigtree, an Emmy Award-winning producer and investigative journalist. Del was a co-producer of the controversial documentary, Vaxxed: From Cover-Up To Catastrophe. His investigational journalism is conducted by efforts to “demand the formation of a genuine independent research [bodies] conducting legitimate science.”

We genuinely need your involvement to provide the world with this critical research. Please join us this year. There will be open bar with food and fund-raising activities, such as a raffle, silent auction and dessert dash. To register, please follow the link below or contact us at or call us at (206) 906-9922.

Buy Tickets Here:

Sound Choice Pharmaceutical Institute, 1749 Dexter Ave North, Seattle, WA, 98109


Holistic MD Awarded Millions From the Regulators Who Humiliated Him

Holistic MD/PhD/vaccine skeptic awarded millions from regulators who humiliated him


Mark Geier MD, Ph.D., is a world-renowned expert who spoke out against the dangers of mercury in vaccines and helped get thimerosal out of the shots like the DPT (now the DTaP). At one time he even had a successful medical practice in Rockville. However, seven years ago the Maryland Board of Physicians suspended his license because he was treating autistic children with a drug called Lupron.

But now, the regulators who stripped his credentials are in serious trouble, “ordered to each personally pay tens of thousands of dollars in damages by a judge who says the board abused its power in an attempt to humiliate the doctor and his family.” 1

In 2012, the board posted a cease-and-desist order on their website alleging “Geier had improperly prescribed medication for himself, his wife and his son while his license was suspended.”2 However, the order also somehow named the drugs in question (a serious breach) which led to a field day for online critics of Geier who began to openly mock him and his family in blogs and on comment threads.

While the family says the state publicized those details for vengeance, to punish a holistic doctor with unconventional ideas, state officials deny that, saying it was an “honest mistake.”(Inside sources tell us the state is 100% lying and unethical and was on an outright witch hunt.)

But, regardless of whether or not it was an accident, Montgomery County Circuit Court Judge Ronald B. Rubin sided with the Geiers and awarded them $2.5 million in damages, calling the order a “significant breach of medical privacy.” He went on to accuse the board and its staff for failing to preserve emails related to the case and pleading ignorance about the order on the witness stand.3

He said,

“If their testimony were to be believed, which the court does not, it is the worst case of collective amnesia in the history of Maryland government and on par with the collective memory failure on display at the Watergate hearings.”4

He ordered 14 board appointees, the board’s lead attorney and the lead investigator on the Geier case to pay half of the damages out of their own pockets, between $10,000 and $200,000 apiece, depending on their net worth.5

The defendants plan to appeal the decision and the spokeswoman for the Maryland Attorney General’s Office, Raquel Coombs said, “We believe there are serious errors in both the facts and the law and will vigorously pursue those on appeal.”6 Sure you will Raquel, do you even have a soul? I don’t think so. #SatansLittleHelper

The Washington Post had the following propaganda drivel to say,

“Public health experts consider “anti-vaxxers” as a grave threat to one of the most significant medical developments in human history. Some Facebook users share Geier’s videos to urge against flu shots, even amid the worst flu outbreak in nearly a decade.”7(Wash Post, you forgot that many died right after their flu shots and some didn’t even have the flu).

While many of the case records remain under seal, the order shows that board staff continued to track blogs and news articles, chronicled his downfall, mocked him and his child in emails, and totally reveled “in their humiliation.”8 Super professional.

In fact, board attorney Victoria Pepper, who drafted the cease-and-desist order, was known to refer to Geiers as “Daddy G” and “Baby G”9 in emails to board investigator Josh Shafer (he led the probe of the Geiers). In one email he responded, “Maybe we can help make it a bad month.”

“At trial, Pepper said she knew the Geiers’ private medical information would be online as a result of the order, but didn’t think it would be embarrassing. She said she named the drugs to clarify that they weren’t dangerous controlled substances and named the recipients to clarify that Geier wasn’t prescribing the medication or juveniles.

The judge called those reasons ‘fabrications,’ adding that ‘Pepper viewed Dr. Geier and his practice to be so abhorrent that she was willing to do ‘whatever it took’ to tarnish his reputation.’”10

In his ruling, Judge Rubin said, “The Board of Physicians is not an ornamental office. It is a serious public trust. It was breached horribly in this case.”11 We couldn’t agree more.

We are delighted that justice has been served. As the defendants are appealing, we will update you as more information becomes available.