https://unfiltered.doctorschierling.com/p/alpha-gal-part-ii-the-tick-was-always?

Alpha-Gal, Part II: The Tick Was Always a Needle

The one question I left standing in Part I — if it was the shots, why the tick at all? The scientific literature answers it in one word. Three questions that collapse the tick story from the inside.

Russell Schierling

Jul 10, 2026

For my children and grandchildren…

In Part I, I built a fence. On one side, I put everything solid — Richet’s Nobel Prize, the route problem, the Japanese gelatin admission, the billing codes, and my own Amish patient base, covered in ticks, completely unvaccinated, and, at least as far as I can ascertain, free of alpha-gal — with a single imported exception I’ll come back to shortly. On the other side, I put my suspicions about a declassed clandestine program, telling you where the record stopped, and my hunches began.

Today, there will be no discussion of Fort Detrick, Plum Island, or other biolabs / bioweapon labs. There’s no need for that when answering a question that several readers asked via the comment section. Before I had finished my morning coffee…

If it’s really the shots that cause alpha-gal, then why is the tick needed at all?

I’m going to answer this by asking and answering three very specific questions from the tick research community’s own filing cabinet — CDC-funded papers and the industry-disclosed authors. From the scientists who are certain the Lone Star tick is the primary cause.

I’m going to let people far smarter than me answer these questions using their own studies…

Question One: Why Is a Tick Needed At All?

But the condition has no historical precedent. It appears in the literature at a specific moment in the late 1980s, in multiple countries simultaneously, at a time when gelatin-containing vaccines were being introduced into childhood immunization schedules worldwide. Japanese researchers documented, confirmed through intervention, and published the direct causal relationship between gelatin-containing DTaP vaccines and alpha-gal sensitization in children who had never been exposed to ticks. That evidence is in the peer-reviewed record. It has been there since 1996. -From Dr Travis Johnson’s June 2 piece in Medium (The Allergy That Shouldn’t Exist: Vaccines, Gelatin, and the Strange Origins of Alpha-Gal Syndrome)

To answer question one, it isn’t — at least not as the origin. Vaccines alone can do it — the tick isn’t required for the mechanism. But historically, before the schedule ballooned, cases clustered in tick country. And the scientific literature already told you why.

Go pull the 2021 paper from Frontiers in Cellular and Infection Microbiology that tick researchers themselves titled Tick Saliva and the Alpha-Gal Syndrome: Finding a Needle in a Haystack. Read that title again. A needle in a haystack. They picked the metaphor, but pointed it at the park instead of at the vial.

Let’s watch what the tick actually does and why the needle in the haystack might be apropos, but maybe not in the way the authors intended…

Another 2021 study, this one on a tick enzyme, describes the alpha-gal sugar as being — their word, not mine — “injected into humans from the lone-star tick bite”. Injected. Not eaten. Not digested. Injected — straight through the skin, past the gut and its digestive processes, and into areas where food is not supposed to be. And as I stated in Part I, this is extremely similar to the phenomenon I have referred to for over two decades as “The Leakies”. Example: (Systemic Leaky Barrier Syndrome (SLBS): A Systems-Level Framework for Chronic Disease).

Earlier studies localized the sugar to the secretory vesicles of the tick’s salivary glands — meaning it’s built to be squirted into a bite. In other words, it’s not wrong to think of ticks as syringes with eight legs — light enough to cross your skin without tripping pressure receptors, and armed with saliva loaded with painkillers, anti-inflammatories, and anticoagulants that let them latch and drink for days without ever setting off the itch that would make you look down and flick them off.

What these devilish little creatures are loading into you is galactose-alpha-1,3-galactose — alpha-gal for short; the “ose” tells you it’s a sugar, like glucose or sucrose — a bit of mammalian sweetness squirted straight under your skin. And here’s the kicker nobody thus far has answered…

When it comes to the animals the tick commonly feeds on — deer, cattle, dogs, rodents, goats, hogs, etc — alpha-gal is “endogenous”. In other words, they build it, they carry it, and their immune systems recognize it as native (“self”) so that it’s not attacked as an invader. So the best answer for why it’s in the saliva at all isn’t that it works some mechanical trick like the anticoagulants and painkillers do — it’s camouflage. Dress your spit in the host’s own sugar, and it reads as ‘self,’ letting the tick guzzle until 100X normal size, all under the immune radar. Which means the sugar is doing exactly its job on a deer. (See link for article)

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**Comment**

Probably the most comprehensive article on the subject and is deserving of your time. I also highly recommend Part 1: Alpha-Gal: Why it Might Be the Shots, Not Just the Ticks. The Amish as a community due to defying government dictates, flourished during COVID as well.

The most important take-away is the fact that the Ozarks are full of deer, lone star ticks, and ‘vaccinated’ people, with the exception of the unvaccinated Amish whom are smack dab in the middle of the same area and free of AGS.

“Geography is an alibi the ‘forced vaccination community’ at large is hiding behind because ticks and the heavily vaccinated live side by side in so much of the country.” ~ Dr. Shierling

The good doctor also states he’s only come into ONE Amish person who got AGS, but that she grew up in Minnesota and received some vaccines as an infant…..

Another important take-away is that the younger the child and the larger the ‘vaccine’ dose(s), the more likely they are to develop sensitivity rather than tolerance. The Journal of the American Dental Association now quietly lists topical numbing gels, Gelfoam, prophy paste, catgut, bone graft materials — while noting manufacturers aren’t required to disclose animal-derived ingredients, as containing alpha-gal bearing excipients. This website has also posted the fact that graphene oxide was found in all three dental anesthetics tested.

For more on AGS:


https://imahealth.substack.com/p/the-hidden-drivers-of-inflammatory? Video Here

The Hidden Drivers of Inflammatory Bowel Disease

Crohn’s and colitis are called genetic, autoimmune, and idiopathic. What if all three labels are wrong? A new paper tests each against current evidence.

Independent Medical Alliance

Aug 02, 2026

Host: Dr. JP Saleeby | Guest: Josh Dech

What if Crohn’s disease and ulcerative colitis are caused by more than genetics alone?

Dr. Yusuf “JP” Saleeby, IMA Senior Fellow in Functional and Integrative Medicine, and gut health specialist Josh Dech take a closer look at what may contribute to inflammatory bowel disease, also known as IBD. The two recently co-authored a new paper published in the Journal of Independent Medicine. Their conversation traces how genetics, diet, gut health, and the environment may work together to shape both diseases.

Inflammatory bowel disease affects more than 7 million people worldwide and ranks among the fastest-growing chronic diseases globally. Nearly everyone diagnosed with Crohn’s disease or ulcerative colitis hears some version of the same three things: the disease is genetic, the immune system is attacking its own tissue, and the underlying cause is unknown. Those three explanations leave two treatments on the table, drugs and surgery, and they leave a patient nothing to investigate.

A new paper in the Journal of Independent Medicine argues that all three explanations fail against current evidence. Josh Dech and Dr. JP Saleeby, its co-authors, point out that each has been contested separately in the literature for two decades without anyone testing them as a set. Taken together, they conclude, the conventional model does not hold.

What replaces it is a disease that is partially heritable, environmentally activated, and immune-mediated, and the distinction is not academic for anyone living with one. If exposures determine whether susceptibility becomes disease, exposures can be found and changed. (See link for article, research paper and video)

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SUMMARY:

  • The authors found that genetics only explain about a quarter of disease risk for irritable bowel (IBD).
  • The authors argue that the autoimmune label, despite holding for decades, is based on the weakest evidence and that pathogenic transfer has never been demonstrated.
  • The serologic markers long cited as evidence for autoimmunity turn out to recognize microbial and fungal targets but are not autoantibodies in the classical sense.
  • A review of 53 meta-analyses across 71 risk factors shows the following exposures for IBD that are quantified and modifiable:
    • antibiotic exposure
    • oral contraceptives
    • breast-feeding was protective for Crohn’s and colitis
    • ultra-processed food
    • air pollution
    • psychological stress
    • mold
    • mycotoxins
  • The authors state current treatments complement but ignore the environmental exposures.
  • The authors give the following issues that should be questioned alongside a standard work-up:
    • Birth mode and feeding history
    • Early antibiotic courses, particularly before age 5
    • Water damage & mold exposure at home, work and in vehicles.
    • Adolescent diet, stressors, and infections

At this point in the article, they described a case report on a 14 year old whose Crohn’s progressed far enough that surgeons planned to remove most of his intestines & place a colostomy. Testing pointed to chronic Lyme disease and three months into treatment a repeated scope test found no lesions.

In short, they conclude the following answers for IBS: antibiotic stewardship, breastfeeding support, reducing ultra-processed food, and remediating indoor mold.

After reading the comments after the article, I would be remiss if I did not mention the ‘vaccine’ issue due to the fact they all introduce foreign substances the body recognizes as foe, priming it for later potential problems such as life-threatening allergies to many things including food, which many are also linking to Alpha Gal Syndrome (AGS), an allergy to animal products supposedly caused by ticks – with no solid proof, as well as the fact some get AGS without any known tick involvement. So while ticks play a part, they are obviously not the only ingredient required to get AGS.

Pathogenic priming was shown clearly with the COVID gene therapy injections.

For more:

https://rumble.com/v7cv61c-the-sleeper-agent-adam-finnegan.html? Video Here

The Sleeper Agent – Adam Finnegan. What We Still Don’t Understand About Tick-Borne Disease

DrSherriTenpenny

Streamed on:Jul 17, 8:00 pm

Most people do not fully understand the devastation that can follow a tick bite. This illness has stolen careers, destroyed families, robbed people of their memories, and left some wondering if they will ever get their lives back. For some, a single bite is followed by years of crushing fatigue, chronic pain, neurological problems, brain fog, and the frightening realization that they are no longer the person they used to be.

Adam Finnegan knows that reality personally. After developing chronic Lyme disease in 2016, he spent seven years trying to answer one question: Why do some people never seem to recover, and why are so many still searching for answers years after the bite itself has been forgotten?

That search became The Sleeper Agent: The Rise of Lyme Disease, Chronic Illness, and the Great Imitator Antigens of Biological Warfare.

In this episode, you’ll learn why Finnegan believes the story of Lyme disease may be much bigger than a tick bite. He explains the science of immune tolerance and why some chronically ill patients may look “normal” on paper while their health continues to deteriorate. He discusses why so many people with persistent symptoms feel abandoned by medicine and why the history of military research into ticks, animal pathogens, and biological warfare led him down a path he never expected to take.

You’ll hear why Erich Traub became one of the central figures in his investigation, why Operation Paperclip matters to this story, and why he believes some of the biggest questions surrounding chronic illness have not been fully answered.

The interview introduces the investigation. The book contains more than 1,000 citations and the deeper historical record.

This episode does not ask you to accept a conclusion. It asks you to look at the evidence, consider the questions, and decide for yourself whether the accepted story explains everything.

Because if one tiny tick can change a life forever, perhaps we still do not understand the full story of what happens next.

Purchase a copy of the book at: https://amzn.to/44Lc5jD

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For more:

Four years ago an article was posted asking if a chronic infection could be behind ME/CFS patients. The answer appears to be yes. A previous study found a link between Cytomegalovirus, EBS, and Human Herpesvirus-6 and ME/CFS. The following also connects Babesia and Bartonella to the condition.

https://www.lymedisease.org/bartonella-babesia-me-cfs/

Study uncovers hidden Bartonella and Babesia infections in ME/CFS patients

A new pilot study from North Carolina State University has found molecular evidence of Bartonella or Babesia infection in nearly half of 50 people diagnosed with myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS).

The findings suggest that vector‑borne pathogens may play a larger role in chronic illness than previously recognized.

Bartonella and Babesia are both transmitted primarily through arthropods—ticks, fleas, and lice—and have been linked to a range of persistent symptoms.

Improved testing has revealed that Bartonella, once thought to cause only short‑lived infections like cat scratch disease, can be associated with chronic and even neuropsychiatric symptoms. Babesia, best known as a tick‑borne parasite, has also been transmitted through blood transfusions and organ transplants.

For this study, researchers selected 50 participants from a larger group of chronically ill individuals with long‑term fatigue and neurological symptoms such as memory problems, tremors, disorientation, or anxiety.

Using quantitative PCR and DNA sequencing, the team detected:

  • Babesia in 10 participants
  • Bartonella in 11
  • Both pathogens in 2

That’s 23 out of 50 participants showing evidence of infection.

“ME/CFS diagnoses are primarily based on immunological biomarkers that can have numerous influences,” said study author Edward Breitschwerdt, Melanie S. Steele Distinguished Professor of Internal Medicine at NC State.

“Our goal was to detect the DNA of specific pathogenic microorganisms that might contribute to or cause a patient’s chronic illness.”

Breitschwerdt emphasized that the small sample size means the results can’t be generalized to all ME/CFS patients, but the unexpectedly high prevalence highlights the need for further research.

The study, supported in part by the Steven & Alexandra Cohen Foundation, appears in Pathogens.

SOURCE: North Carolina State University

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For more:

https://childrenshealthdefense.org/defender/sayer-ji-idaho-murder-indictment-institutional-pattern-refusing-admit-vaccines-can-cause-infant-death/?

Sayer Ji: Idaho Murder Indictment Highlights ‘Institutional Pattern’ of Refusing to Admit That Vaccines Can Lead to Infant Death

Idaho mother Andrea Shaw is facing murder charges in the deaths of 18-month-old twins because investigators failed to seriously consider a vaccine- and Tylenol-related cause of death, according to health writer Sayer Ji. On his Substack, Ji argued the Shaw case is evidence of a broader institutional refusal to recognize and investigate serious vaccine adverse events.

by Brenda Baletti, Ph.D.

July 23, 2026

This article was originally published by The Defender — Children’s Health Defense’s News & Views Website.

Tylenol bottles and vaccine bottle

Idaho mother Andrea Shaw is facing murder charges in the deaths of 18-month-old twins because investigators failed to seriously consider a vaccine and Tylenol-related cause of death, according to health writer Sayer Ji.

In a lengthy Substack essay, Ji argued the Shaw case is evidence of a broader institutional refusal to recognize and investigate serious vaccine adverse events.

Eighteen-month-old Dallas and Tyson Shaw received multiple routine childhood vaccinations — including DTaP, flu and hepatitis A vaccines — during an April 2025 pediatric visit.

According to court documents, Andrea and her mother-in-law told the pediatrician that the children’s father had previously experienced an adverse reaction to a flu vaccine. But the pediatrician dismissed their concerns, and a nurse administered the shots.

Within a day, the twins developed fevers, lethargy, difficulty walking, blue lips, sunken eyes and diarrhea. They didn’t want to eat. Emergency department physicians documented a “post-immunization reaction” before discharging the children with instructions to treat their symptoms with children’s Tylenol and popsicles.

Eight days later, both toddlers were found dead. One year later, prosecutors charged Shaw with first-degree murder, a capital offense, and she is being held without bail.

Prosecutors said investigators ruled out other possible causes of death — including heat exposure, carbon monoxide poisoning, poisoning and vaccines. They concluded the only “conceivable explanation” was that the twins were suffocated.

But the unusual nature of the deaths should have prompted a more thorough examination of potential medical causes rather than an assumption of criminal conduct, Ji said.

So far, prosecutors, doctors and other experts have refused to examine all the evidence, according to Ji, who wrote:

“What the state of Idaho refuses to see is that the etiology of these deaths is not obscure, not speculative, and not novel. It is, in fact, the most extensively documented — and most systematically suppressed — pediatric toxicology pattern of the past eighty years. …

“This is not a whodunit. It is a why-won’t-we-look. And it is the inevitable, engineered consequence of a public-health apparatus that has spent forty years insisting — against a mountain of its own peer-reviewed evidence — that all childhood vaccines are ‘safe and effective’ for all children.

“When the medicine kills, the mother must have.”

Other recently vaccinated twins died from SIDS

The prosecutors’ assertion that murder can be the only explanation for why “both would die at the same time, on the same night, in the same room, in the same bed,” is “exactly backwards,” according to Ji.

Case reports dating back to the mid-20th century include eight other cases involving healthy twins who became ill shortly after immunization and died within hours to 10 days.

In a 1946 case, the U.S. Institute of Medicine concluded that the vaccines led to anaphylaxis in one set of twins. In another case, the Institute of Forensic Medicine in Istanbul, Turkey, ruled that twin infants who received vaccines, then became ill and were treated with Tylenol and then suffered simultaneous deaths “consistent with SIDS,” sudden infant death syndrome.

The Shaw twins’ deaths fit a previously observed clinical pattern rather than representing an unprecedented event, he argued.

Possible biological mechanism connecting vaccines and infant death

Vaccines can directly cause infant deaths, including SIDS, through a specific, traceable biological chain, not simply through correlation, Ji said. He laid out three types of evidence:

  • The immune/nervous system pathway: Vaccines trigger an immune response that releases inflammatory molecules known as cytokines. In infants, these molecules can cross into the brain and disrupt a part of the brainstem that controls breathing and the ability to wake up and gasp during oxygen deprivation — the same disruption researchers have found in autopsies of SIDS cases.
  • Specific autopsy findings: Pathologists at the University of Milan examined infants who died shortly after vaccination and found underdeveloped brainstem structures and heart-conduction abnormalities. They argue this shows a physical mechanism related to vaccines, not just developmental abnormalities.
  • Statistical/database signals: Several analyses, including one by von Kries in Germany, an Italian case study series, and Vaccine Adverse Event Reporting System (VAERS)-based studies by Goldman and Miller and Miller, report elevated death rates in windows shortly after vaccination and a strong correlation between the number of vaccine doses a country requires and its infant mortality rate.

This evidence offers a solid explanation for vaccine-induced death and would be consistent with the eight-day gap between the Shaw twins’ vaccination and their deaths, Ji said.

“The picture that emerges is not ambiguous. It is a coherent, cross-decade, cross-methodology body of work spanning pathology, epidemiology, VAERS analysis, ecological correlation, and pharmacovigilance signal detection,” he wrote.

Tylenol depletes body’s principal antioxidant defenses

While some researchers have for decades suggested there is a link between vaccines and infant deaths, Ji presents new evidence — not widely discussed until recently — that acetaminophen, or Tylenol, acts as an “amplifier” to the vaccine mechanism that can be deadly for some infants.

The Shaw twins, like many children who experience post-vaccination fevers, were treated with Tylenol on the advice of their doctor.

Drawing on recent work by William Parker, Ph.D., Ji argued that acetaminophen depletes glutathione, one of the body’s principal antioxidant defenses, at the same time that vaccines increase oxidative stress through immune activation.

The combination of vaccination-induced inflammation and acetaminophen-related glutathione depletion created what Ji described as a synergistic toxic reaction capable of damaging vulnerable regions of the brainstem.

He pointed to evidence from a 2008 study, which found that children given acetaminophen after the measles-mumps-rubella vaccine were about eight times more likely to develop autism compared to children given ibuprofen or no analgesic.

National Institutes of Health-funded study that measured acetaminophen in umbilical cord blood also found infants with higher levels had a 3.6 times greater risk of being diagnosed with autism.

Since the 1980s, physicians have routinely recommended acetaminophen following childhood vaccinations without recognizing what Ji argues are potentially dangerous interactions between the drug and vaccine-induced immune responses.

‘The institutional pattern is not accidental’

The possibility that vaccines could be linked to infant death has been systematically minimized and ignored by public health agencies and the medical establishment, according to Ji.

Decades of published research documenting adverse neurological and inflammatory reactions following vaccination have received little attention because to acknowledge even rare fatal complications would undermine public confidence in vaccination programs, Ji said.

That’s why he believes investigators usually begin with the assumption that vaccines cannot be responsible — which leaves homicide as the only remaining explanation when no obvious other cause is identified.

“The institutional pattern is not accidental,” he wrote. “It is the same governance-by-classification that has characterized every major pharmaceutical harm suppression of the past century — from thalidomide to Vioxx to opioids.”

The elimination in 1979 of medical classification codes that medical examiners can use to classify a death as related to vaccines has institutionalized this practice by effectively eliminating vaccines as a recordable cause of death — even though the National Vaccine Injury Compensation Program has continued to recognize the link between vaccines and infant deaths.

It remains unknown whether the coroner in the Shaw case looked for signs of vaccine-induced death in the post-mortem examination. Did the coroner examine each section of the brainstem, look for cytokine expression, examine the cardiac conduction system or measure cerebral glutathione?

The finding of “pulmonary vascular congestion” that prosecutors cited “is consistent with mechanical suffocation — but it is equally consistent with central respiratory arrest from any cause, including vaccine + acetaminophen-induced brainstem failure,” Ji wrote.

‘Entire edifice of universal childhood vaccination as a moral imperative collapses’

Ji compared the Shaw case to several high-profile cases in the United Kingdom, including that of Angela Cannings, whose conviction for murdering her two infant sons in the 1990s was overturned after experts concluded that natural causes couldn’t be ruled out.

The Shaw prosecution similarly relies on statistical assumptions about the improbability of two children dying naturally rather than on definitive forensic evidence demonstrating homicide, Ji said.

Ultimately, Ji frames the Shaw prosecution as more than an isolated criminal case.

He wrote that Andrea “is the predictable output of a public-health information architecture that requires — as a matter of institutional survival — that vaccine deaths not exist.”

Were that fact to be taken seriously, “the entire edifice of universal childhood vaccination as a moral imperative collapses.”

Instead, risk-benefit decisions about whether to receive vaccines would have to be made on a case-by-case basis with full disclosure of the different types of risks. Blanket safety claims would have to be abandoned.

Ji called for a new forensic review to look at evidence of vaccine- and Tylenol-induced death by a pathologist with appropriate expertise. He called for disclosure of the lot numbers of the vaccines the twins received, and a dismissal of Shaw’s murder indictment.

This article was originally published by The Defender — Children’s Health Defense’s News & Views Website under Creative Commons license CC BY-NC-ND 4.0. Please consider subscribing to The Defender or donating to Children’s Health Defense.

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