Wisconsin Representative Mark Pocan joined the Lyme Caucus yesterday.

The caucus is a bi-partisan group working together in Congress to take action on Lyme and other tick-borne diseases.  Under the leadership of Chris Smith (NJ) and Colin Peterson (MN), it has initiated letters and actions to benefit Lyme patients, such as the inclusion of the monies for Lyme and tick-borne diseases into the Congressionally Directed Medical Research Program (CDMRP) and language and Lyme monies into Appropriations over the years and has initiated favorable legislation.  Additionally, it has queried government agencies over policies not favorable to patients.  This has reminded the agencies that someone is looking over their shoulder.  Many meetings have been held and educational sessions in DC for Congress.

With nearly 400,000 new cases of Lyme Disease per year, and Wisconsin ranking 6th in the nation, this is an important issue to Wisconinites.  There are nearly 20 tick-borne diseases (and counting) being spread by at least 8 different ticks (and counting) with 30% of reported cases occurring in children between the ages of 0-19.  

In 2015, the caucus secured for the first time ever, $5 million in funding in the House Appropriations Committee annual military spending legislation, which was adopted in the Fiscal Year 2016 funding bill which was signed into law, and will provide resources for Lyme disease research through DOD’s innovative, high-risk, high reward program.

In December of 2016, the United States House of Representatives passed, and former President Obama signed, the 21st Century Cures Act.  The Cures Act included language, similar to a bill that Rep. Smith introduced previously, which created the Interagency Lyme and Tick-Borne Disease Working Group.  Specifically, the Working Group is comprised of federal and non-federal members tasked with reporting to Congress on scientific advances, research questions, surveillance activities and emerging strains in species of pathogenic organisms.

For more see:  https://madisonarealymesupportgroup.com/2016/12/14/21st-cca-signed-into-law/  Bill language is included at the bottom of the article.

Please contact your WI representative and ask them to join the Caucus.  Let’s represent Wisconsin well:  https://www.govtrack.us/congress/members/WI

Carl Tuttle’s eloquent rebuttal to The Clinical Relevance of Studies on Borrelia burgdorferi Persisters http://www.amjmed.com/article/S0002-9343(17)30480-1/fulltext, by Wormser and Baker:


MAY 21, 2017 — Once again we see the American Journal of Medicine giving carte blanche to Wormser/Baker who continues to deny persistent Borrilia infection despite all the evidence of chronic Lyme disease. This denial has left hundreds of thousands if not millions worldwide with no treatment options for an infection capable of ruining lives. It is the 21st Century do-it-yourself plague with faulty/misleading antibody tests, inadequate treatment, no medical training and absolutely no disease control.

This is certainly a crime against humanity on a scale not seen since the Holocaust.

Letter to the American Journal of Medicine:

The Clinical Relevance of Studies on Borrelia burgdorferi Persisters
Phillip J. Baker Ph.D., Gary P. Wormser, M.D. April 2017

May 21, 2017

The American Journal of Medicine
3615 N. Prince Village Place, Suite 181
Tucson, Arizona 85719
Attn: Editorial Board
Attn: Joseph Alpert, MD, Editor in Chief

Dear Dr. Alpert,

A worldwide community of physicians has been influenced by the ongoing disinformation campaign aimed at promoting the idea that Lyme is little more than a nuisance disease as health agencies across the globe are blindly following what has been deviously established here in the U.S. We are dealing with a life-altering/life-threatening infection with faulty/misleading antibody tests, inadequate treatment, no medical training and absolutely no disease control.

In 2001 Gerold Stanek, Medical University of Vienna clearly reported persistent Borrelia infection in a 64 year-old patient despite treatment with four courses of ceftriaxone. Stanek published in the British Journal of Dermatology in 2001 reporting that:

“Borrelia [the Lyme disease bacteria] may possibly be able to remain dormant in certain tissue compartments”

When publishing a paper in 2012 with co-author Dr. Gary Wormser of New York Medical College, Stanek claims “Most manifestations of Lyme borreliosis will resolve spontaneously without treatment.”

Dr. Gary Wormser of New York Medical College has had a global influence on the scientific community promoting his groupthink mentality concerning persistent Borrelia infection. (Wormser’s thirty year bias; “Chronic Lyme disease does not exist”)

In 2014 Stanek published the following paper endorsing antibiotic treatment in a male patient requiring a pacemaker from his Lyme disease infection; a pacemaker for a nuisance disease?

Lyme Borreliosis (Book Chapter)

There are hundreds of published articles referencing persistent infection but a handful of academics that control the narrative through editorial censorship continue to deny chronic Borrelia infection and have colluded to deny a life-altering/life-threatening disease as we see in the 2012 Stanek/Wormser collaboration.

Wormser’s focus over the past thirty years has always been on the acute stage of disease with treatment following the bulls-eye rash. The vast majority of patients disabled from Lyme never saw a bulls-eye rash and don’t recall a tick bite. (The state of Maine reported an average of 48.25% incidence of rash-related Lyme 2009-2012) These patients are ping-ponged through our misinformed medical community for months, years or decades as their infection progresses to a stage that is completely unaffected by the standard two to three weeks of antibiotics. It is this class of patient that Wormser refuses to acknowledge.

Post treatment symptoms after early detection and treatment are entirely different from untreated infection of months years or decades.

I ask two questions Dr. Alpert:

1. Is this scientific misconduct or criminal offense?
2. What role has journal editors played in this misinformation campaign?

A response to this inquiry is requested.

Carl Tuttle
Hudson, NH

P.S. You or a loved one is a single tick bite away from experiencing this travesty.

Cc: Iratxe Puebla, COPE Complaints Administrator
Thomas Reller, Vice President, Global Corporate Relations, Elsevier Inc.
Dr. Mihail Grecea, Ethics Expert, Elsevier


1. Br J Dermatol. 2001 Feb;144(2):387-92.
Isolation and polymerase chain reaction typing of Borrelia afzelii from a skin lesion in a seronegative patient with generalized ulcerating bullous lichen sclerosus et atrophicus.
Breier F1, Khanakah G, Stanek G, Kunz G, Aberer E, Schmidt B, Tappeiner G.

2. Lyme borreliosis.
Stanek G1, Wormser GP, Gray J, Strle F.

3. Peer Reviewed Evidence of Persistence of Lyme Disease Spirochete Borrelia burgdorferi and Tick-Borne Diseases

The following is a list of over 700 peer reviewed articles that support the evidence of persistence of Lyme and other tick-borne diseases. It is organized into different categories—general, psychiatric, dementia, autism and congenital transmission.

4. The state of Maine has an average of a 48.25% incidence of rash-related Lyme over the four year period 2009-2012 (they’ve only been making this report for 4 years). See page 3 of each document below:

http://www.maine.gov/dhhs/reports/lymereport.pdf — 2009, 59%
http://www.maine.gov/dhhs/mecdc/infectious-disease/epi/vector-borne/lyme/documents/lyme-legislature-2010.pdf — 2010, 43%
http://www.maine.gov/dhhs/mecdc/infectious-disease/epi/vector-borne/lyme/documents/2011-lyme-legislature.pdf — 2011, 42%
http://www.maine.gov/dhhs/mecdc/infectious-disease/epi/vector-borne/lyme/documents/2012-lyme-legislature.pdf — 2012, 49%

5. Scientific misconduct or criminal offense?

Dr. Gary Wormser has been fixated on the acute stage of disease after erythema migrans (Bulls-eye rash) and early treatment. His study design is then expected to apply to the entire Lyme patient population. Patients who had a prolonged exposure to the infection before diagnosis and initial treatment are almost always incapacitated. It is this class of patient that Wormser continuously avoids and ignores. His focus has been to discredit the disabled as opposed to finding a cure for late stage persistent disease.

For those of you who have not signed the petition, join the nearly 40,000 people calling for a Congressional investigation of the CDC, IDSA, and ALDF:  https://www.change.org/p/the-us-senate-calling-for-a-congressional-investigation-of-the-cdc-idsa-and-aldf?source_location=minibar

  Minding your mitochondria | Dr. Terry Wahls | TEDxIowaCity  Approx. 18 minutes

Diagnosed with MS, Dr. Terry Wahls received the best standard medicine had to offer.  After declining to the point of being in a wheel chair, she took matters into her own hands and learned how to properly fuel her body. Using the lessons she learned at the subcellular level, she used diet to cure her MS and get out of her wheelchair.

Lyme/MSIDS patients are often misdiagnosed with MS, Lupus, ALS, Fibromyalgia, Chronic Fatigue Syndrome, and other autoimmune diseases.  We also have issues with our mitochondria causing profound fatigue.

In this short, to the point video, Dr. Wahls gives vital information to all of us.

Bon appétit!

https://www.ncbi.nlm.nih.gov/m/pubmed/28490579/  Bartonella Species, an Emerging Cause of Blood-Culture-Negative Endocarditis.

Okaro U1, Addisu A2, Casanas B2, Anderson B3. Clin Microbiol Rev. 2017.

Since the reclassification of the genus Bartonella in 1993, the number of species has grown from 1 to 45 currently designated members. Likewise, the association of different Bartonella species with human disease continues to grow, as does the range of clinical presentations associated with these bacteria. Among these, blood-culture-negative endocarditis stands out as a common, often undiagnosed, clinical presentation of infection with several different Bartonella species. The limitations of laboratory tests resulting in this underdiagnosis of Bartonella endocarditis are discussed. The varied clinical picture of Bartonella infection and a review of clinical aspects of endocarditis caused by Bartonella are presented. We also summarize the current knowledge of the molecular basis of Bartonella pathogenesis, focusing on surface adhesins in the two Bartonella species that most commonly cause endocarditis, B. henselae and B. quintana. We discuss evidence that surface adhesins are important factors for autoaggregation and biofilm formation by Bartonella species. Finally, we propose that biofilm formation is a critical step in the formation of vegetative masses during Bartonella-mediated endocarditis and represents a potential reservoir for persistence by these bacteria.

https://www.ncbi.nlm.nih.gov/m/pubmed/28328183/  Seroprevalence of Bartonella species, Coxiella burnetii and Toxoplasma gondii among patients with hematological malignancies: A pilot study in Romania.

Messinger CJ1, Gurzau ES2,3, Breitschwerdt EB4,5, Tomuleasa CI6,7, Trufan SJ8, Flonta MM9, Maggi RG4,5, Berindan-Neagoe I6,10,11, Rabinowitz PM8. Zoonoses Public Health. 2017.

Patients receiving immunosuppressive cancer treatments in settings where there is a high degree of human-animal interaction may be at increased risk for opportunistic zoonotic infections or reactivation of latent infections. We sought to determine the seroprevalence of selected zoonotic pathogens among patients diagnosed with haematologic malignancies and undergoing chemotherapeutic treatments in Romania, where much of the general population lives and/or works in contact with livestock. A convenience sample of 51 patients with haematologic cancer undergoing chemotherapy at a referral clinic in Cluj-Napoca, Romania, was surveyed regarding animal exposures. Blood samples were obtained and tested for evidence of infection with Bartonella species, Coxiella burnetii and Toxoplasma gondii, which are important opportunistic zoonotic agents in immunocompromised individuals. 58.8% of participants reported living or working on a farm, and living or working on a farm was associated with contact with livestock and other animals. 37.5% of participants were IgG seroreactive against one or more of five Bartonella antigens, and seroreactivity was statistically associated with living on farms. Farm dwellers were 3.6 times more likely to test IgG seroreactive to Bartonella antibodies than non-farm dwellers. 47.1% of the participants tested T. gondii IgG positive and 13.7% tested C. burnetii IgG positive, indicating past or latent infection. C. burnetii IgM antibodies were detected in four participants (7.8%), indicating possible recent infection. These results indicate that a large proportion of patients with haematologic cancer in Romania may be at risk for zoonotic infections or for reactivation of latent zoonotic infections, particularly with respect to Bartonella species. Special attention should be paid to cancer patients’ exposure to livestock and companion animals in areas where much of the population lives in rural settings.

https://www.ncbi.nlm.nih.gov/m/pubmed/28405890/  Seroprevalence of Bartonella Species in Patients with Ocular Inflammation.

Brydak-Godowska J1, Kopacz D1, Borkowski PK2, Fiecek B3, Hevelke A4, Rabczenko D5, Tylewska-Wierzbanowska S3, Kęcik D1, Chmielewski T3. Adv Exp Med Biol. 2017.

Bartonella species, vector-borne etiologic agents of many systemic or self-limited infections, are responsible for a widening spectrum of diseases in humans, including inflammatory conditions of the eye. The aim of this study was to determine whether there is any relationship between uveitis and the evidence of Bartonella spp. infection in the serum, ocular fluid, and cataract mass in patients with intraocular inflammation. Polymerase chain reaction (PCR)-based tests and DNA sequencing were performed on surgery-extracted specimens of intraocular fluid and lens mass of 33 patients. Sera from 51 patients and 101 control subjects were tested for the presence of specific antibodies against Bartonella spp. Neither IgM-class antibodies against Bartonella spp. nor Bartonella spp. DNA were detected. A specific IgG-class antibody was found in 33.3% of the patients with uveitis. The rate of positive Bartonella serology was higher among the uveitis patients than that in control subjects. This high rate may in part result from unrecognized indirect mechanisms rather than the immediate presence and multiplication of Bartonella spp. in the eyeball. Nonetheless we believe that screening for Bartonella spp. should become part of the diagnostic workup in uveitis.

Another ocular study:  https://madisonarealymesupportgroup.com/2017/04/06/ocular-bartonellosis/

http://www.foxnews.com/health/2017/05/19/child-temporarily-paralyzed-after-undetected-tick-bite.html  Published May 19,2017

Fox News

Please see link above for the frightening video of a young girl from the state of Oregon struggling to stand and use her arms after an undetected tick bite.  Thankfully, an astute doctor had seen this in a few other children her age and upon a closer look, found a tick in the girl’s hair and diagnosed her with tick paralysis.

The tick they discovered was a dog tick and while it normally is not a tick known to carry Lyme Disease, they are keeping a close eye on her.  She has recovered from the paralysis.

http://www.columbia-lyme.org/patients/tbd_paralysis.html  Tick paralysis is caused by a neurotoxin in the salivary glands predominantly of female, egg-laden ticks, not by an infectious agent like Lyme Disease (borrelia).  Over 40 tick species are known to transmit it; however, in North America the most common culprits are Dermacentor variabilis (American dog tick) and Dermacentor andersoni (Rocky Mountain wood tick), but bites from Amblyomma (Lone Star) and Ixodes (deer tick) ticks can also cause tick paralysis. In the United States, tick paralysis is most common in the Pacific Northwest, Rocky Mountain states and southeastern part of the country. It occurs most frequently in the spring months, from April through June.

Lack of muscle control appears within 2-6 days of tick attachment followed by an ascending paralysis starting in the feet and legs. Numbness and tingling in the face and limbs, are frequently reported.

The apparent cure is simply removing the tick.  If the tick is not found and removed, paralysis can ascend to the trunk and affect respiratory muscles, which can be life-threatening.

Correct diagnosis is contingent upon awareness. Any case with loss of muscle control and ascending paralysis, especially in a patient who lives in a tick-endemic area should be considered suspicious for tick paralysis. Such patients should be searched immediately for ticks, particularly in body areas where the tick might not be immediately apparent, such as the scalp, hairline, ear canals or pubic region.

Tick paralysis is often confused with Guillain-Barré syndrome, and there are no tests for it, so be educated and educate others.

**Please note, often maps and predictions of infections based on tick type can be faulty.  All ticks have similar anatomy and can exchange body fluids with humans and other animals.  Even the CDC is stating that geographic ranges of ticks are expanding and the infections they carry are as well:  https://www.cdc.gov/cdcgrandrounds/archives/2017/March2017.htm

Please see this link for a wonderful tick identification chart.  It also shows them engorged and as you go over the tick it zooms in so you can really get a good look: http://www.tickencounter.org/tick_identification

  Published on May 17, 2017  

World Mercury Project

The first peer-reviewed study comparing health outcomes of vaccinated versus unvaccinated children implicates vaccines in a host of chronic illnesses.  http://oatext.com/pdf/JTS-3-186.pdf  Scientists found no significant differences in rates of vaccine-preventable illnesses like hepatitis A or B, measles, mumps, rubella, influenza, meningitis or rotavirus.  As would be expected, vaccinated children did have lower likelihood of two vaccine-preventable illnesses compared to unvaccinated children: chicken pox (7.9% vs. 25.3%), and pertussis (2.5% vs. 8.4%).

The study suggests that fully vaccinated children may be trading the prevention of certain acute illnesses (chicken pox, pertussis) for more chronic illnesses and neurodevelopmental disorders like ADHD and Autism. The scientists also found that children born prematurely, who were vaccinated, were 6.6 times more likely to have a neurodevelopmental disorder.

**My Comment**

This is important for Lyme/MSIDS patients as our immune systems are already fighting a war of epic proportions.  Dr. Garth Nicolson has found that vaccines are implicated with mycoplasma, Gulf War Syndrome, and Lyme/MSIDS, as well as the fact they contain immune suppressing things such as aluminum, thimerosal (50% mercury by molecular weight), squalene, human and animal DNA, and other damaging ingredients.  https://madisonarealymesupportgroup.com/2015/08/12/connecting-dots-mycoplasma/

For information on how vaccines are made as well as information on Lymerix, the Lyme Vaccine which was pulled off the market:  https://madisonarealymesupportgroup.com/2017/04/06/video-how-vaccines-are-made/

For an executive summary of Dr. Gentempo’s recent 9-part vaccine video series: https://madisonarealymesupportgroup.com/2017/03/30/ty-bollinger-the-truth-about-vaccines-series/ (Click on each separate link for each summary from everything from the horrific anthrax vaccine given to our service men and women to the blatant fraud, coverup and collusion of the CDC in regards to vaccines, information on Garasil & flu vaccines, mercury poisoning, and the relationship to Autism.) 

A relationship between Gardasil and Bartonella:  https://madisonarealymesupportgroup.com/2016/04/24/gardasil-and-bartonella/




Powassan/Deer Tick Virus and Borrelia Burgdorferi Infection in Wisconsin Tick Populations

Knox Konstance K., Thomm Angela M., Harrington Yvette A., Ketter Ellen, Patitucci Jacob M., and Carrigan Donald R. Vector-Borne and Zoonotic Diseases. May 2017 Online Ahead of Print

Powassan/Deer Tick Virus (POWV/DTV) is an emerging cause of arboviral neuroinvasive disease in the upper Midwest. These studies describe the prevalence and geographic distribution of Wisconsin ticks carrying POWV/DTV as well as the high frequency of Ixodes scapularis ticks coinfected with both POWV/DTV and Borrelia burgdorferi, the causative agent of Lyme disease. These findings suggest that concurrent transmission of POWV/DTV and B. Burgdorferi from coinfected ticks is likely to occur in humans.

Results (see link for maps and graphs of locations and results)

The distribution of I. scapularis and D. variabilis tick collection sites are categorized by geographic quadrant (QNW, QNE, QSW, & QSE) of the state (Fig. 1, Table 1). Nearly 80% of adult female I. scapularis ticks analyzed were collected from the northern half of the state (QNW and QNE) and accounted for 85% of POWV-positive ticks. While only 90 I. scapularis ticks were collected from the southern two quadrants, POWV-positive ticks were identified in both QSE and QSW. QNW I. scapularis ticks revealed the highest MLE of infection for both POWV and B. burgdorferi (4.67% and 23.42%, respectively). A separate analysis of I. scapularis collections from Harvest One endemic zone (Spooner/Hayward) QNW demonstrated a frequency of infection for both POWV (4.65%) and B. burgdorferi (27.91%) that is comparable to the total QNW (Fisher’s exact, p = 1.00 and p = 0.35, respectively). QSE contained the lowest MLE for POWV (1.53%), but B. burgdorferi-infected ticks were high with a MLE of 15.69%. Of the 295 D. variabilis ticks analyzed from both harvests, none (0%) had evidence of POWV infection; however, B. burgdorferi infection in D. variabilis ticks was seen in both QNW (3.1%) and QSW (2.86%), consistent with the high B. burgdorferi infection rate observed in I. scapularis ticks in these same quadrants.


Powassan Virus: An Emerging Arbovirus of Public Health Concern in North America

Hermance Meghan E. and Thangamani Saravanan. Vector-Borne and Zoonotic Diseases. May 2017 Online Ahead of Print

Powassan virus (POWV, Flaviviridae) is the only North American member of the tick-borne encephalitis serogroup of flaviviruses. It is transmitted to small- and medium-sized mammals by Ixodes scapularis, Ixodes cookei, and several other Ixodes tick species. Humans become infected with POWV during spillover transmission from the natural transmission cycles. In humans, POWV is the causative agent of a severe neuroinvasive illness with 50% of survivors displaying long-term neurological sequelae. POWV was recognized as a human pathogen in 1958 when a young boy died of severe encephalitis in Powassan, Ontario, and POWV was isolated from the brain autopsy of this case. Two distinct genetic lineages of POWV are now recognized: POWV (lineage I) and deer tick virus (lineage II). Since the index case in 1958, over 100 human cases of POWV have been reported, with an apparent rise in disease incidence in the past 16 years. This recent increase in cases may represent a true emergence of POWV in regions where the tick vector species are prevalent, or it could represent an increase in POWV surveillance and diagnosis. In the past 5 years, both basic and applied research for POWV disease has intensified, including phylogenetic studies, field surveillance, case studies, and animal model development. This review provides an overview of POWV, including the epidemiology, transmission, clinical disease, and diagnosis of POWV infection. Recent research developments and future priorities with regard to the disease are emphasized.

Early timeline of POWV transmission
The duration of I. scapularis attachment required for successful transmission of DTV to a host was found to be as little as 15 min (Ebel and Kramer 2004). This finding was particularly striking because unlike other tick-borne pathogens (Borrelia burgdorferi, Babesia microti, and Anaplasma phagocytophilum), very little time between tick attachment and virus transmission is needed for POWV. The reactivation period required for some nonviral tick-borne pathogens provides a grace period of approximately 24 h, where a minimal risk of transmission occurs if humans remove the attached tick within this timeline; however, there is no such grace period for POWV due to its very short timeline of transmission. These differences underscore why the timeline of POWV transmission must be carefully considered when analyzing the early immunomodulatory events that occur at the feeding site of the tick.

**My comment**
The idea of a “grace period” is ludicrous. Ticks do not understand grace, trust me. For accurate information about transmission times of Lyme see:
https://madisonarealymesupportgroup.com/2017/04/14/transmission-time-for-lymemsids-infection/.  In short, it can happen in hours for sure – not requiring the oft repeated dogma of 24-48 or more hours.  Ticks often feed partially and then drop off.  These partially fed ticks have spirochetes in their saliva and can transmit much more quickly.

Every single tick bite should be taken seriously!