Archive for the ‘Candida’ Category

Is MS an Infectious Disease?

The following link contains a collection of research on MS and infections including fungus, Lyme, parasites, and viruses.  Highly recommend.  The website is Pam Bartha’s who’s life was turned upside down at the age of 28 when she was diagnosed with MS.  She lost vision in one eye, was weak and had tingling in her legs, insomnia, and severe fatigue and headaches.  She was told by doctors there was nothing she could do but wait around and become disabled.  Her mother in law gave her a book that set her on a healing journey that continues today.  The book was “The Yeast Connection” by Dr. Crook.  In it he shared that he believed many diseases are actually caused by infections, especially the GI tract.  This doctor observed that when he treated the infections, his patients recovered. Go here to read Pam’s story as there are golden nuggets for all within and great recommendations.

Pam is a wellness researcher, educator and coach with a BS who is a certified teacher.  She offers a free consultation and personalized training on how to get your health back.  (I have no affiliation with this program and receive no monies)

https://livediseasefree.com/ms-infections/#multiple-sclerosis-and-lyme-disease

Multiple Sclerosis Infection – Is MS an Infectious Disease?

The following is an excerpt from Pam’s website.  Go to link for entire article

Multiple Sclerosis and Infection

Could Multiple Sclerosis be caused by infection? These studies and articles offer compelling evidence. Contact Us for more information.

Multiple Sclerosis and Infection
Multiple Sclerosis and Fungus
Multiple Sclerosis and Lyme Disease
Multiple Sclerosis and Parasites
Multiple Sclerosis and Viruses

1. Commensal microbiota and myelin autoantigen cooperate to trigger autoimmune demyelination.
Nature. 2011; 479(7374): 538-41. DOI: 10.1038/nature10554.

This study shows that microbes that live in the body (in particular the GI tract) are an essential factor in triggering the autoimmune response in MS and other diseases.

“Active multiple sclerosis lesions show inflammatory changes suggestive of a combined attack by autoreactive T and B lymphocytes against brain white matter. … The stimuli triggering this autoimmune conversion have been commonly attributed to environmental factors, in particular microbial infection. … We show that the commensal gut flora… is essential in triggering immune processes, leading to a relapsing-remitting autoimmune disease…“

2. Role of pathogens in multiple sclerosis.
International Reviews of Immunology. 2014; 33(4): 266-83. DOI: 10.3109/08830185.2013.823422.

This study states that “infectious pathogens (disease causing microbes) are the likely environmental factors involved in the development of MS.” It also identifies various microbes that are involved in the development of Multiple Sclerosis infection, which include various bacteria, parasites and viruses.

“Although the etiology of MS is unknown, genetic and environmental factors play a role. Infectious pathogens are the likely environmental factors involved in the development ofMultiple Sclerosis infection. Pathogens associated with the development or exacerbation of MS include bacteria, such as Mycoplasma pneumoniae and Chlamydia pneumoniae, the Staphylococcus aureus-produced enterotoxins that function as superantigens, viruses of the herpes virus (Epstein-Barr virus and human herpesvirus 6) and human endogenous retrovirus (HERV) families and the protozoa Acanthamoeba castellanii. Evidence, from studies with humans and animal models, supporting the association of these various pathogens with the development and/or exacerbation of MS will be discussed along with the potential mechanisms including molecular mimicry, epitope spreading and bystander activation. In contrast, infection with certain parasites such as helminthes (Schistosoma mansoni, Fasciola hepatica, Hymenolepis nana, Trichuris trichiura, Ascaris lumbricoides, Strongyloides stercolaris, Enterobius vermicularis) appears to protect against the development or exacerbation of MS… A complex interaction between the CNS (including the blood-brain barrier), multiple infections with various infectious agents (occurring in the periphery or within the CNS), and the immune response to those various infections may have to be deciphered before the etiology of MS can be fully understood.”

(See link for article)

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For more:

MS is another label that needs a better definition.  Since so many things can cause/exacerbate it, it’s a highly individual issue that needs to be approached as such.  There is no four-cornered box with this beast and you must be willing to go down many rabbit-holes to find answers.  This is a journey, not a destination.  There may be many issues that must be addressed in order to achieve health – just like with Lyme/MSIDS.

A Modern Holistic Protocol for Lyme Disease

**Comment**
Please read my review of this article at the end.

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**Comment**

My Review:

Red flags immediately go up when someone calls it “Lymes Disease,” because it announces the fact they are ignorant of the fact it all started in the town of Lyme, Connecticut with a cluster of cases in children who were misdiagnosed with juvenile arthritis (JA).  It’s Lyme disease, named after the town of Lyme.  Please go here for an excellent video by an experienced Lyme literate doctor on the history of Lyme disease, of which manifestations began long before this cluster of children.  Go here for a summary of the video and other important facts important to understand that not mentioned in Biomante’s  article that explain the sordid backstory, the reason Lyme/MSIDS research being used is fraudulent, and completely biased, the flagrant conflicts of interest within the agencies controlling the Lyme narrative, and The Cabal doing the only accepted research that does not take into account global research and independent research showing the organism persists despite treatment.

Regarding cases, this article is way off.  Reporting has been a problem from the beginning as the surveillance criteria has such a high bar that hardly anyone meets it.  Getting a positive on the 2-tiered CDC testing is akin to winning the lotteryThe world at large now knows that Lyme is woefully under-reported.  Nobody has a clue about coinfections.  To continue to regurgitate these unrealistically low numbers doesn’t help anyone and only demonstrates ignorance.  I also don’t appreciate the same mythology regarding where Lyme exists.  This has also been a problem and is a perfect example of bad science continuing to be used. Lyme is literally everywhere.  That’s all you need to know.  Don’t continue to downplay this.  It’s a plague of biblical proportions.

Regarding the research at the University of Connecticut finding only 53% had Bb and were misdiagnosed with Lyme arthritis, this too remains highly dubious.  All testing for this illusive organism is abysmal – plus current two-tiered CDC testing only tests for ONE strain when there are 100 strains and counting in the U.S. alone.  More are found on a regular basis.  Testing won’t pick of any of these other strains.  All parameters for case numbers are faulty.  

He announces that there is “hysteria” regarding the disease.  This immediately raises my blood pressure.  He truly is clueless.  This continued downplaying of a life-shattering, complex illness has been going on for over 40 years due to vested interests and faulty science needs to end.  The “untreatable form of Lyme disease could hit 2 million Americans,” and that isn’t even taking into account global numbers.  Lyme disease is more prevalent than AIDS, breast cancer, West Nile virus, H1N1, and Ebola.  He doesn’t mention that Lyme is congenitally transmitted and there is evidence being ignored that it is also sexually transmitted.

Biamonte’s description of the symptoms also shows his inexperience.  Lyme can virtually look like anything and mimic some 300-different diseases.  While some get the EM rash, many don’t.  The rash can also look quite differently on patients.  Strain diversity appears to make a difference regarding symptoms, with some strains causing more skin manifestations and some causing more joint manifestations – regardless, it is wrong to attempt to put this monster in a neat four-cornered box.  Further, ticks are migrating everywhere, intermingling, and nobody has a clue what that is going to do to strain diversity and symptomology.  Again, this hasn’t been studied in decades because according to The Cabal, it’s a done deal.  No further science required. 

Can you think of any other disease in which this attitude of ignorance is allowed and accepted?

I would also urge caution in blaming the black legged tick as the sole perp.  Since Bb and its many strains and all the coinfections are extremely fastidious organisms, early work as been done and then used again and again and again for decades.  Time for new, independently done science with new methods.  We desperately need transmission studies as the ones being used are decades old.  Ticks all bite, exchange fluids and have the potential to transmit diseases.  Don’t diminish the tick’s ability to side-line your life with things you never even knew existed!

The explanation of the 3 stages of the life cycles of ticks is also simplified.  It is known ticks can partially feed, drop off, and then transmit much more quickly  to the next victim.  We know ticks can parasitize each other. We know that ticks can survive harsh environments by burying under leaf litter and snow (or anything else they can find like wood chips in a playground). They also go through a hibernation period called diapause.  Ticks can also pass on infections to their offspring. There is much we don’t know – especially regarding transmission.

I would caution against using percentages of infected ticks to prove a point.  Remember, it only takes ONE tick, ONE bite, and your life could be changed forever.  Each tick is a potential bomb capable of infecting you with 19 and counting diseases.

The regurgitation that a tick must be attached for 36-48 hours to transmit infection is based on faulty science.  Minimum times for infection have never been determined.  It also does not take into account the fact pathogens have been found in the salivary glands, suggesting a much quicker transmission time, and that ticks often partially feed, drop off, and can infect you quicker.  Very old research is being used again, and again, and again, when reality has shown people getting infected within a few hours.  This mythology continues to downplay a modern-day scourge by using ancient data.  Some tick-borne infections can be transmitted within minutes.  Many of them look just like Lyme.  Another mistake is to focus solely on Lyme.  In my experience Babesia, Bartonella, and Mycoplasm are as bad if not worse than Lyme.  If you are infected with a few of these suckers at once, you are one sick dog.  And in my experience, this is the norm.

The section on “Lyme Disease Symptoms” again demonstrates this man’s inexperience.  Hardly anyone I know fits his limited list.  Again, research has shown the EM rash to be highly variable, and hardly ANYONE I work with has seen it.  Most also haven’t seen the tick.  Patients and their doctors often work completely in the dark, and what often happens is over time is bizarre unexplainable symptoms start cropping up more and more until life becomes unbearable.  At this point Bb and coinfections are virtually everywhere in the human bodyheavily entrenched and therefore, harder to treat.  This is reality. 

Also, people can jump from stage to stage in no particular order.  Some will experience psychiatric symptoms IMMEDIATELY and never have the rash, fever, joint pain, etc. 

In Stage II, Biamonte states about 10% will experience transient heart dysfunction.  Again, it’s very unwise to use percentages when testing misses a preponderance of cases and the organism is elusive. This study found an increasing burden of Lyme carditis in U.S. children’s hospitals.  Many are questioning if there could be subclinical cardiac involvement in early Lyme with children, and that’s another fly in the ointment.  Most testing won’t pick up problems with these patients because their symptoms are subclinical, yet they are severe to the patient. If I had a nickel for every time a patient told me the test didn’t find anything, I’d be a rich woman.  Just because testing didn’t reveal something, doesn’t mean something isn’t there.  This is truly the norm with tick-borne illness.  I didn’t start having heart issues until we started treating for Babesia and then all of a sudden, BOOM!  It felt like I was having a heart attack.  This is another reality.  Until you start utilizing anti-microbials, the immune system is confused and unable to deal with these infections because they fool the immune system by changing their outer surface proteins to look like the good guys.  Further, so many are misdiagnosed that percentages are meaningless.  Seriously.  Meaningless.  There are thousands out there who have Lyme carditis who have completely fallen through the cracks.  Thousands.

He states symptoms will decrease in weeks to months WITHOUT treatment.  It’s obvious he is reading Wormser and other Cabalist’s research as this is what they believe; however, in the real world symptoms wax and wane but never totally go away, and left untreated with only become more entrenched in the body.  Again, this illness often takes years to unravel.  Waxing and waning is a marquee symptom with tick-borne illness, but without treatment it will metastasize everywhere in the human body.  There is a connection with Lyme/MSIDS and cancer as well as brain diseases like ALS, dementia, Alzheimer’s, MS, etc.  Left untreated, the parasites will continue to live off the host weakening it year by year until they are a shell of themself. 

He states 10% will suffer chronic arthritis.  Let me be clear: nobody has a clue about the prevalence of arthritis in these poor patients.  Not a clue.  Putting this in a box, unless it’s Pandora’s is the biggest mistake being made. 

Regarding treatment, he omits to mention that even people diagnosed and treated early can require further treatment as symptoms return.  This is very common. 

He mentions direct testing being a “low-yield” procedure as so few organisms are found, but that “surely someone, somewhere is working to develop such an early test, probably based upon the DNA of the microorganism.”  This too shows the ignorance of the history of the suppression of direct detection techniques.  In fact a test has been found to be highly accurate but our corrupt public health “authorities” monopolize testing, and have done unethical things against competitors for decades.  Public health owns the patents on the organisms, the tests, the treatments, and the vaccines.  It’s a business, not a public health agency concerned with healthThis is imperative to understand.

He does mention the success of metronidazole or one of the other 5-nitroimidazoles in heavier does for a longer period of time.  I would agree, but never as a mono therapy.  Savvy Lyme literate doctors have learned from vast experience with thousands upon thousands of patients to layer treatment, never utilizing a mono therapy, to avoid antibiotic resistance.  Again, coinfections are common place and require different medications including anti-protozoan meds, anthelmintics, and more. The potential for candida should also be taken into account and dealt with.

Regarding the use of colloidal silver for Lyme, I completely disagree. This recent study shows stevia, Andrographis, Grapefruit seed extract, colloidal silver, monolaurin, and antimicrobial peptide LL37 didn’t do diddly.  Keep in mind this work is done in vitro – or in a lab, not the human body – although this follows my personal experience as well. This 2004 study shows that 3 samples of colloidal silver of 22 ppm and two samples of 403 and 413 ppm in an agar-well diffusion assay showed ZERO effect on the growth of test organisms but ALL were sensitive to ciprofloxacin.  Silver at 22ppm showed NO bactericidal activity in phenol coefficient tests.

The patients he mentions have already been treated with many antibiotics and have developed candida issues (not uncommon).  He doesn’t mention how long these patients were treated, which would be helpful to know.  Please know that a wise treatment would address candida along the way.  We took fluconazole twice a week throughout our treatment course along with a low or no sugar diet. 

I personally know patients that used silver and the result was they ended up wheel-chair bound.  They only worsened and worsened. 

He mentions research done in the 90’s showing that colloidal silver killed Bb after 24 hours of exposure.  The other research mentioned is from the 70’s.  If it was so effective, much more would have been done and trust me, desperate patients and the doctors who dare treat them would be using it, and they are not.  To claim that silver is virtually non-toxic is also premature.  Little has been done on it – particularly using it over long periods of time.  Again, metals are not harmless and accumulate in the body.  

I’m a huge proponent of using silver topically on wounds, etc.  Hospitals have shown the effectiveness of this substance for decades for cleaning and sterilizing objects topically.  Sometimes I will even use it to ward off a cold by spraying it on my throat for a few days.  Sometimes it appears to work and other times it doesn’t, which is only my personal observation.

Some claim that utilizing it along with antibiotics, potentiates the antibiotics.  My concern would be putting metals in a body already struggling.  Metals, after all, accumulate.  In fact, many Lyme/MSIDS and autism patients improve by using chelation which removes heavy metals. 

He states that artemisia has been used effectively for Lyme.  I would disagree.  This is an anti-malarial medicine that has action against Babesia, which is a cousin to malaria – a protozoan.  Due to the repeated mistakes in his article and the downplaying of the seriousness of this complex illness, I question his experience with not only being able to identify coinfections and their symptomology, but also the importance of treating each infection with specific antimicrobials that have action against it.

From clinical observation, Cat’s Claw is effective against Lyme; however, there is debate in the herbal world about the need for TOA free vs the whole herb.  Again, I’m not qualified to enter this debate, but Master herbalists write on it with conviction both ways.  In the end, we often are forced to experiment to determine the truth of the matter and even then patients often have different findings, reminding us of the complexity of the human body.  In the end, whatever works for you – USE IT! 

While it is wise is to rotate meds, savvy Lyme literate doctors have a method to their madness and pay close attention to the life-cycle of the organism as well as the plateaus patients experience.  Rotating, while important to guard against drug resistance, it is also important to layer treatments so they work synergistically together – also negating resistance and effectively dealing with coinfections and candida.

I have used Banderol and Biocidin with little effect.  I’m sure others have had a better experience, but one again – treatment should always be an individualized approach. 

Regarding length of treatment, one of the wisest, most experienced LLMD’s in Wisconsin (RIP) told me that in the 70’s when he treated this illness they labeled a “rickettsial” like illness –  as it wasn’t even named yet, he found that a few months to a year of treatment appeared to work.  He now states treating this takes YEARS – like 3-5 years.  So, according to this wise, experienced doctor, things have changed making this harder to treat.  Perhaps coinfection involvement has become more of a problem than in the past.

Please remember that according to the article, most of the patients Biamonte treats are seeing him for Candida AFTER they have already been treated for Lyme/MSIDS.  This would explain why he is perhaps seeing success after only one year.  They’ve already been treated, perhaps for years by someone else.  They have successfully beaten down and reduced the infection load and are now struggling with Candida, immunoconfusion, and the last vestiges of infections that have already been hit hard by antibiotics. 

Finally, it’s important to remember that this doctor is seeing patients that are suffering with significant blow-back.  His experience is going to be biased in this direction.  I wish he would stick with helping people recover from treatment that out of necessity is harsh (until something else is discovered) but not superimposing his beliefs that the treatments are wrong, or that colloidal silver is the answer to all our woes.

The fact that these patients are recovering in a year shows me that these patients are well on their way to health but need specialized help in dealing with damage caused by either the infections themselves, the harsh treatment required, or a combination of both. This problem is also quite common.

Hottest Lyme Disease Treatment Update 2022

https://www.treatlyme.net/guide/hottest-lyme-treatment-updates-2022  Video Here (Approx. 11 Min)

Hottest Lyme Disease Treatment Updates in 2022

Marty Ross MD on Top Lyme Treatment Updates of 2022

In the video in the link above Marty Ross MD describes the latest Lyme disease treatment updates in his integrative medicine practice. For more information about the topics discussed in the video article see the following resources:

Dr. Ross’s new book, Hacking Lyme Disease: An Action Guide to Wellness, will be released by early December 2022.

Disclaimer

The ideas and recommendations on this website and in this article are for informational purposes only. For more information about this, see the sitewide Terms & Conditions.

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**Comment**

Dr. Ross discusses peptides. These are not antimicrobials but help the body in fighting off infections and restoring the issues infections cause.  Ross as well as Dr. Holtdorf is evidently having a lot of success with them.

Ross also discusses biofilm busters such as lumbrokinase, which helps improve circulation, decrease muscle pain, and improve medicine delivery deep into tissues.  Those with hypercoagulation would also do well to check it out.  

A golden nugget he discussed was the finding that cryptolepis, a drug normally given for Babesia, also has action against Lyme (in vitro – or the lab, which may not transfer over to the human body or in vivo) This study also found that black walnut, Japanese knotweed, sweet wormwood, ccat’s claw, Cistus incanus, and Chinese skullcap at 1% extracts had good activity against Bb’s stationary phase compared to control antibiotics doxycycline and cefuroxime.

Important note: The minimum inibitory concentration (MIC) values of Artemisia annua, Juglans nigra, and Uncaria tomentosa were quite high for the growing phase of Bb, despite their strong activity against the non-growing stationary phase. On the other hand, the top two active herbs, Cryptolepis and Japanese Knotweed showed strong activity against both growing Bb and non-growing stationary phase.  In subculture studies, only 1% Cryptolepis extract caused complete eradication, while doxycycline and cefuroxime and other active herbs could not eradicate B. burgdorferi stationary phase cells as many spirochetes were visible after 21-day subculture.

Ross states both herbs are helpful for Bartonella as well as diflucan/fluconazole.  My LLMD had us pulse diflucan twice a week throughout our entire treatment (5 years).  I can say with experience we herxed on this drug, often.  It is a known anti-fungal; however, Dr. Hoffman (RIP) stated he believed it did far more than that, and I tend to agree having taken it.

In contrast, the study showed that Stevia rebaudiana, Andrographis paniculata, Grapefruit seed extract, colloidal silver, monolaurin, and antimicrobial peptide LL37 had little or no activity against stationary phase B. burgdorferi A few years ago all kinds of headlines came out that stevia cured Lyme. Nothing could be further from the truth.  Per usual, if something seems too good to be true, it usually is.

Dr. Klinghardt uses a sublingual form of Hyaluronic Acid to fool the cyst forms to open and become spirochetes so they can be killed by antimicrobials.  For more on Klinghardt’s treatment:  Klinghardt Lyme Protocol.

Hyaluronic Acid is a type of sugar molecule.  Many other Lyme literate doctors also use forms of sugar such as Stevia or Erythritol as “cyst busters,” in their treatment regimens.  Look for reputable sources of Erythritol as it is most commonly made with GMO cornstarch.

For more:

Minerals and Their Effect On The Immune System

https://soundcloud.com/user-467428748/minerals-and-their-effect-on-immune-system?in=user-467428748/sets/the-candida-chronicles-podcast  Approx. 30 Min.

Podcast with Michael Biamonte, CCN and others

This lecture explores how minerals like selenium, zinc & copper work with your immune system to help regulate it. It also explains the interactions between candida, viruses, and other infections with these minerals.

For more:

Effectiveness of Antibiotics Reduced When Multiple Bugs Present

https://phys.org/news/2022-03-effectiveness-antibiotics-significantly-multiple-bugs.html

Effectiveness of antibiotics significantly reduced when multiple bugs present

March 19, 2022

Gram-stained P. aeruginosa bacteria (pink-red rods) Credit: Wikipedia

A study has found that much higher doses of antibiotics are needed to eliminate a bacterial infection of the airways when other microbes are present. It helps explain why respiratory infections often persist in people with lung diseases such as cystic fibrosis despite treatment.

In the study, published today in The ISME Journal, researchers say that even a low level of one type of microbe in the airways can have a profound effect on the way other microbes respond to antibiotics.

The results highlight the need to consider the interaction between different species of microbe when treating infections with antibiotics—and to adjust dosage accordingly.

“People with often have co-infection with several pathogens, but the problem is we don’t take that into account in deciding how much of a particular antibiotic to treat them with. Our results might help explain why, in these people, the antibiotics just don’t work as well as they should,” said Thomas O’Brien, who carried out the research for his Ph.D. in the University of Cambridge’s Department of Biochemistry and is joint first author of the paper.

Chronic bacterial infections such as those in the human airways are very difficult to cure using antibiotics. Although these types of infection are often associated with a single pathogenic species, the infection site is frequently co-colonized by a number of other microbes, most of which are not usually pathogenic in their own right.

Treatment options usually revolve around targeting the pathogen, and take little account of the co-habiting species. However, these treatments often fail to resolve the infection. Until now scientists have had little insight into why this is.

To get their results the team developed a simplified model of the human airways, containing artificial sputum (‘phlegm’) designed to chemically resemble the real phlegm coughed up during an infection, packed with bacteria.

The model allowed them to grow a mixture of different microbes, including pathogens, in a stable way for weeks at a time. This is novel, because usually one pathogen will outgrow the others very quickly and spoil the experiment. It enabled the researchers to replicate and study infections with multiple species of microbe, called ‘poly-microbial infections’, in the laboratory.

The three microbes used in the experiment were the bacteria Pseudomonas aeruginosa and Staphylococcus aureus, and the fungus Candida albicans—a combination commonly present in the airways of people with cystic fibrosis.

The researchers treated this microbial mix with an antibiotic called colistin, which is very effective in killing Pseudomonas aeruginosa. But when the other pathogens were present alongside Pseudomonas aeruginosa, the antibiotic didn’t work.

“We were surprised to find that an antibiotic that we know should clear an infection of Pseudomonas effectively just didn’t work in our lab model when other bugs were present,” said Wendy Figueroa-Chavez in the University of Cambridge’s Department of Biochemistry, joint first author of the paper.

The same effect happened when the microbial mix was treated with fusidic acid—an antibiotic that specifically targets Staphylococcus aureus, and with fluconazole—an antibiotic that specifically targets Candida albicans.

The researchers found that significantly higher doses of each antibiotic were needed to kill bacteria when it was part of poly-microbial infection, compared to when no other pathogens were present.

“All three species-specific antibiotics were less effective against their target when three pathogens were present together,” said Martin Welch, Professor of Microbial Physiology and Metabolism in the University of Cambridge’s Department of Biochemistry and senior author of the paper.

At present antibiotics are usually only laboratory tested against the main pathogen they are designed to target, to determine the lowest effective dose. But when the same dose is used to treat infection in a person it often doesn’t work, and this study helps to explain why. The new model system will enable the effectiveness of potential new antibiotics to be tested against a mixture of microbe species together.

Poly-microbial infections are common in the airways of people with cystic fibrosis. Despite treatment with strong doses of antibiotics, these infections often persist long-term. Chronic infections of the airways in people with asthma and chronic obstructive pulmonary disorder (COPD) are also often poly-microbial.

By looking at the genetic code of the Pseudomonas bacteria in their lab-grown mix, the researchers were able to pinpoint specific mutations that give rise to this antibiotic resistance. The mutations were found to arise more frequently when other pathogens were also present.

Comparison with the genetic code of 800 samples of Pseudomonas from around the world revealed that these mutations have also occurred in human patients who had been infected with Pseudomonas and treated with colistin.

“The problem is that as soon as you use an antibiotic to treat a microbial , the microbe will start to evolve resistance to that antibiotic. That’s what has happened since colistin started to be used in the early 1990’s. This is another reminder of the vital need to find new antibiotics to treat human infections,” said Welch.

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**Comment**

Hopefully it’s clear why I would post this cystic fibrosis research on a Lyme/MSIDS website.  Lyme/MSIDS is also often a polymicrobial illness involving numerous pathogens. Logic would then follow that this complex illness would also be impossible to eradicate using a singular antibiotic against multiple infections that not only require different medications individually but that synergistically would also necessitate higher doses for a longer period of time. 

This is known, appreciated, and utilized by Lyme literate doctors when treating patients, and Lyme/MSIDS patients, researchers, and doctors alike have been screaming bloody murder about this topic for decades.  But they are laughed at and ridiculed, and written off as tin-foil hat wearing nut-jobs.

Mainstream research and medicine barely even acknowledge coinfection, a fact that is seen not only in ticks but daily in humans living in the real world.  They also deny pleomorphism – or the ability of borrelia, and other pathogens to shape-shift into different forms.  Research has shown borrelia shape-shifts when threatened so it can survive.

The current simplistic view of Lyme/MSIDS is killing people, and the sooner it is recognized and addressed the better.

One would hope that research showing the very real complexities and failure of standard treatment on patients with multiple infections simultaneously would cause even the most entrenched to consider the same possibility with Lyme/MSIDS.

One can only continue to hope.