Archive for the ‘Malaria’ Category

Artesunate on Short Term Memory in Lyme Borreliosis

http://www.medical-hypotheses.com/article/S0306-9877(17)30288-8/fulltext

Lyme borreliosis is associated with memory deficits. While this may be related to cerebral infection by Borrelia bacteria, it may also be caused by concomitant co-infection by Babesia protozoa. The anti-malarial artemisinin-derivative artesunate has been shown to be effective against a number of Babesia species and to have efficacy against human cerebral malaria. We hypothesised that concomitant administration of artesunate in Lyme borreliosis patients would help alleviate the severity of self-reported short-term memory impairment. This hypothesis was tested in a small pilot study in which patients were treated with both an intravenous antibiotic and oral artesunate (20 mg four times per day); treatment was associated with a reduction in the severity of short-term memory difficulties (P ≃ 0.08). In light of these findings, we recommend that a formal randomised, placebo-controlled study be carried out.

 

For more on Babesia:  https://madisonarealymesupportgroup.com/2016/01/16/babesia-treatment/

More on Lyme:  https://madisonarealymesupportgroup.com/2016/02/13/lyme-disease-treatment/

Wolbachia – The Next Frankenstein?

Transmission electron micrograph of Wolachia within an insect cell

Credit:  Public Library of Science/Scott O’Neill

The latest in the effort for world domination over bugs and the diseases they carry is Wolbachia, a Gram-negative bacterium of the family Rickettsiales first found in 1924 and in 60% of all the insects, including some mosquitoes, crustaceans, and nematodes (worms). For those that like numbers, that’s over 1 million species of insects and other invertebrates. It is one of the most infectious bacterial genera on earth and was largely unknown until the 90’s due to its evasion tactics. It’s favorite hosts are filarial nematodes and arthropods.

Wolachia obtains nutrients through symbiotic relationships with its host. In arthropods it affects reproductive abilities by male killing, parthenogenesis, cytoplasmic incompatibility and feminization. However, if Wolbachia is removed from nematodes, the worms become infertile or die. These abilities are what make it so appealing for insect controlcytoplasmic incompatibility, which essentially means it results in sperm and eggs being unable to form viable offering.

http://www.slideserve.com/babu/wolbachia  (Nifty slide show here)

It also makes it appealing for use in human diseases such as elephantiasis and River Blindness caused by filarial nematodes, which are treated with antibiotics (doxycycline) targeting Wolbachia which in turn negatively impacts the worms. Traditional treatment for lymphatic Filariasis is Ivermectin but they also use chemotherapy to disrupt the interactions between Wolbachia and nematodes. This anti-Wolbachia strategy is a game-changer for treating onchocerciasis and lymphatic filariasis.  https://www.sciencedaily.com/releases/2017/03/170316120451.htm

Lyme/MSIDS patients often have nematode involvement.

https://microbewiki.kenyon.edu/index.php/Wolbachiahttps://www.psychologytoday.com/blog/emerging-diseases/200902/tick-menagerie-lyme-isnt-the-only-disease-you-can-get-tick  Both Willy Burgdorfer, the discoverer of the Lyme bacterium, as well as Richard Ostfeld, an animal ecologist found nematode worms in ticks. Since then, some provocative research involving nematodes, Lyme/MSIDS, dementia, and Alzheimer’s has been done.

https://madisonarealymesupportgroup.com/2016/06/03/borrelia-hiding-in-worms-causing-chronic-brain-diseases/https://madisonarealymesupportgroup.com/2016/08/09/dr-paul-duray-research-fellowship-foundation-some-great-research-being-done-on-lyme-disease/https://madisonarealymesupportgroup.com/2016/07/10/greg-lee-excellent-article-on-strategies-for-neurological-lyme/https://madisonarealymesupportgroup.com/2015/10/18/psychiatric-lymemsids/

https://www.scientificamerican.com/article/how-a-tiny-bacterium-called-wolbachia-could-defeat-dengue/  Yet, according to many, Wolbachia is the next eradicator of Dengue Fever and possibly Malaria, chikungunya, and yellow fever because it stops the virus from replicating inside mosquitoes that transmit the diseases. The approach is also believed to have potential for other vector-borne diseases like sleeping sickness transmitted by the tsetse fly.  Evidently, Wolbachia does not infect the Aedes aegypti mosquito naturally, so researchers have been infecting mosquitoes in the lab and releasing them into the wild since 2011. The article states it hopes that the method works and expects infection rates in people to drop and hopes that the mosquitoes will pass the bacterium to their offspring, despite it disappearing after a generation or two of breeding and needing to “condition” the microbes to get them used to living in mosquitoes before injecting them. They also state Wolbachia is “largely benign for mosquitoes and the environment,” and “To humans, Wolbachia poses no apparent threat.” Their work has shown that the bacterium resides only within the cells of insects and other arthropods. They also state that tests on spiders and geckos that have eaten Wolbachia mosquitoes are just fine and show no symptoms. An independent risk assessment by the Commonwealth Scientific and Industrial Research Organizatioin (CSIRO), Australia’s national science agency, concluded that, “Release of Wolbachia mosquitoes would have negligible risk to people and the environment.”

Interestingly, trials are underway in Vietnam, Indonesia, and now Brazil.

They state that scaling up operations to rear enough Wolbachia mosquitoes is too labor-intensive and in Cairns they are going to put Wolbachia mosquito eggs right into the environment. Evidently, other researchers are wanting to release genetically modified (GMO) mosquitoes that carry a lethal gene, and they’ve done it, and it’s causing an uproar:   http://america.aljazeera.com/articles/2013/11/9/genetically-modifiedmosquitoessetoffuproarinfloridakeys.html

http://www.naturalnews.com/2017-07-25-googles-sister-company-releasing-20-million-mosquitoes-infected-with-fertility-destroying-bacteria-depopulation-experiment.html  As of July 14, 2017, Google’s bio-lab, Verily Life Sciences,  started releasing Wolbachia laced mosquitoes in California as part of project, Debug Fresno to reduce the mosquito population.

http://www.greenmedinfo.com/blog/research-exposes-new-health-risks-genetically-modified-mosquitoes-and-salmon  Numerous studies show unexpected insertions and deletions which can translate into possible toxins, allergens, carcinogens, and other changes.  Science can not predict the real-life consequences on global pattens of gene function.

So, why question the use of Wolbachia as a bio-control?

For Lyme/MSIDS patients, 3 words: worms and inflammation.

Dogs treated for heart worm (D. immitis) have trouble due to the heart worm medication causing Wolbachia to be released into the blood and tissues causing severe Inflammation in pulmonary artery endothelium which may form thrombi and interstitial inflammation. Wolbachia also activates pro inflammatory cytokines. Pets treated with tetracycline a month prior to heart worm treatment will kill some D. immitis as well as suppress worm production. When given after heart worm medication, it may decrease the inflammation from Wolbachia kill off.
http://www.critterology.com/articles/wolbachia-and-their-role-heartworm-disease-and-treatment

The words worms and inflammation should cause every Lyme/MSIDS patient to pause. Many of us are put on expensive anthelmintics like albendazole, ivermectin, Pin X, and praziquantel to get rid of worms and are told to avoid anything causing inflammation due to the fact we have enough of it already. We go on special anti-inflammatory diets and take systemic enzymes and herbs to try and lower inflammation.   https://madisonarealymesupportgroup.com/2016/04/22/systemic-enzymes/

Seems to me, many MSIDS/LYME patients when treated with anthelmintics, will have Wolbachia released into their blood and tissues causing wide spread inflammation, similarly to dogs.

And that’s not all.

According to a study by Penn State, mosquitoes infected with Wolbachia are more likely to become infected with West Nile – which will then be transmitted to humans.“This is the first study to demonstrate that Wolbachia can enhance a human pathogen in a mosquito, one researcher said. “The results suggest that caution should be used when releasing Wolbachia-infected mosquitoes into nature to control vector-borne diseases of humans.” “Multiple studies suggest that Wolbachia may enhance some Plasmodium parasites in mosquitoes, thus increasing the frequency of malaria transmission to rodents and birds,” he said.  The study states that caution should be used when releasing Wolbachia-infected mosquitoes into nature. https://www.sciencedaily.com/releases/2014/07/140710141628.htm

So besides very probable wide spread inflammation, and that other diseases may become more prevalent due to Wolbachia laced mosquitoes, studies show Wolbachia enhances Malaria in mosquitos. Lyme/MSIDS patients are often co-infected with Babesia, a malarial-like parasite that requires similar treatment and has been found to make Lyme (borrelia) much worse. It is my contention that the reason many are not getting well is they are not being treated for the numerous co-infections.  Some Lyme/MSIDS patients have Malaria and Lyme.

Regardless of what the CDC states, all the doxycycline in the world is not going to cure this complicated and complex illness.

Lastly, with Brazil’s recent explosion of microcephaly, the introduction of yet another man-made intervention (Wolbachia laced mosquitos) should be considered in evaluating potential causes and cofactors. And while the CDC is bound and determined to blame the benign virus, Zika, there are numerous other factors that few are considering – as well as the synergistic effect of all the variables combined. Microcephaly could very well be a perfect storm of events.
https://madisonarealymesupportgroup.com/2016/12/21/how-zika-got-the-blame/https://madisonarealymesupportgroup.com/2016/03/04/health-policy-recap/https://madisonarealymesupportgroup.com/2016/03/08/fixation-on-zikapolio/

I hate bugs as much as the next person, but careful long-term studies of Wolbachia are required here.

https://www.ncbi.nlm.nih.gov/pubmed/20394659  “Despite the intimate association of B. burgdorferi and I. scapularis, the population structure, evolutionary history, and historical biogeography of the pathogen are all contrary to its arthropod vector.

In short, borrelia (as well as numerous pathogens associated with Lyme/MSIDS), is a smart survivor.

While borrelia have been around forever with 300 strains and counting worldwide, epidemics, such as what happened with Lyme Disease in Connecticut are not caused by genetics but by environmental toxins – in this case, bacteria, viruses, funguses, and stuff not even named yet.

Circling back to Wolbachia.

Hopefully it is evident that many man-made interventions have been introduced into the environment causing important health ramifications: Wolbachia laced mosquitoes and eggs, GMO mosquitoes including CRISPR, and in the case of Zika in Brazil, whole-cell pertussis vaccinations (DTap) for pregnant women up to 20 days prior to expected date of birth, a pyriproxyfen based pesticide applied by the State in Brazil on drinking water, as well as aerial sprays of the insect growth regulators Altosid and VectoBac (Aquabac, Teknar, and LarvX, along with 25 other Bti products registered for use in the U.S.) in New York (Brooklyn, Queens, Staten Island, and The Bronx) to combat Zika. “We feel it’s critical that the scientific community consider the potential hazards of all off-target mutations caused by CRISPR, including single nucleotide mutations and mutations in non-coding regions of the genome … Researchers who aren’t using whole genome sequencing to find off-target effects may be missing potentially important mutations. Even a single nucleotide change can have a huge impact.”  http://articles.mercola.com/sites/articles/archive/2017/06/13/crispr-gene-editing-dangers.aspx?utm_source=dnl&utm_medium=email&utm_content=art3&utm_campaign=20170613Z1_UCM&et_cid=DM147520&et_rid=2042753642

All of this is big, BIG business.

Is the introduction of Wolbachia another puzzle piece in the perfect storm of events causing or exacerbating human health issues?

The jury’s still out, but it’s not looking good – particularly for the chronically ill.

The Kite Mosquito Patch

Approximately 2 min.

http://www.kitepatch.com/kite-patch

Dr. Anandasankar Ray and his team from the University of California, Riverside, has developed the Kite Patch, a small patch worn on clothing to protect against mosquitos.  The patch blocks the mosquitoes’ ability to track and detect humans for up to 48 hours.  It should be for sale in 2017.  http://magazine.ucr.edu/44

All Kite products are free from DEET or any other potentially harmful or toxic chemicals.  The company is committed to replacing outdated and potentially unhealthy repellents that have dominated the market for decades.

The patch could help protect against malaria, West Nile, Dengue fever, and other mosquito-borne diseases.

http://www.nature.com/nature/journal/v474/n7349/full/nature10081.html  Journal article.

http://well.blogs.nytimes.com/2015/08/11/high-tech-hope-for-repelling-mosquitoes/?_r=0  One person’s review.

 

Babesia Cure?

http://news.yale.edu/2016/06/06/combination-therapy-cures-tick-borne-illness-mice

Yale researchers have found that combining atovaquone and ELQ-334, at low doses, cleared Babesia in mice and prevented recurrence up to 122 days.

ELQ stands for Endochin like quinolone and is a preclinical candidate that targets the liver and blood stages of malarial organisms.

http://malariajournal.biomedcentral.com/articles/10.1186/1475-2875-13-339  It’s been known since 1948 that Endochin has anti-malarial properties; however, it has proven to be ineffective in vivo against human malaria.  Recent advances have suggested revisiting previously abandoned lead molecules to be possible viable anti-malarial drug candidates.

http://www.ncbi.nlm.nih.gov/pubmed/23019377  ELQ-271 and ELQ-316 are effective against acute and latent toxoplasmosis.

When I asked my pharmacist about the ELQ’s, he said he couldn’t find anything about the manufacturing process, and that as far as side effects, there won’t be a much information available until ELQ-334 proceeds further in the approval process. Sometimes side effects don’t show up until well after drugs have been on the market.  He also stated that there were only 800 cases of neuropathy from 1998-2013 reported to the FDA for quinolines.  

While I could be wrong, Endochin like quinolone could possibly mean it is made with fluoride.

http://articles.mercola.com/sites/articles/archive/2009/07/18/antibiotics-to-avoid–the-plague-due-to-fdas-oversight-failure.aspx  Quinolones are made with fluoride, which enables them to penetrate into tissue, including your brain.  This ability is what makes them valuable against tick borne infections.

Omniflox, Raxar, Trovan, Zagam, and Tequin have all been banned due to their side effects; however, Cipro, Levaquin, Avelox, and Floxin continue to be prescribed.

In Dr. Cohen’s 2001 study, the following side effects were documented:

*Nervous system symptoms occurred in 91 percent of patients (pain, tingling and numbness, dizziness, malaise, weakness, headaches, anxiety and panic, loss of memory, psychosis)
*Musculoskeletal symptoms in 73 percent of patients (tendon ruptures, tendonitis, weakness, joint swelling)
*Sensory symptoms in 42 percent of patients (tinnitus, altered visual, olfactory, and auditory function)
*Cardiovascular symptoms in 36 percent of patients (tachycardia, shortness of breath, chest pain, palpitations)
*Skin reactions in 29 percent of patients (rashes, hair loss, sweating, intolerance to heat or cold)
*Gastrointestinal symptoms in 18 percent of patients (nausea, vomiting, diarrhea, abdominal pain)
A comprehensive list of reactions can be found at Dr. Cohen’s site Medication Sense.

According to Dr. Mercola, quinolones are too often prescribed for minor problems such as sinus, bladder, and prostate infections. He feels these super-antibiotics should be used as a last line of defense.

 

Be armed with facts to make an informed decision about these antibiotics with your LLMD (Lyme literate doctor).  One of the most experienced LLMD’s in Wisconsin states that he has used quinolones for over 20 years without tendon rupture.  It’s important to notify your doctor immediately if you notice symptoms such as tendon pain or anything else that doesn’t seem right.

 

 

 

 

 

 

 

 

 

 

 

Toxoplasmosis

Toxoplasma-gondii-660x380

Toxoplasma gondii,

by AJ Cann http://phenomena.nationalgeographic.com/2013/04/26/mind-bending-parasite-permanently-quells-cat-fear-in-mice/

http://www.cdc.gov/parasites/toxoplasmosis/epi.html Toxoplasmosis is caused by a common protozoan parasite, Toxoplasma gondii, and is the leading cause of death attributed to food borne illness in the U.S. More than 60 million carry it but are asymptomatic. It is also on the of the CDC’s “Neglected Parasitic Infections,” and has been targeted for public health action.

Transmission:  food (undercooked contaminated meat, or knives, utensils, cutting boards, or other foods that had contact with contaminated meat), congenitally (mother to infant), and in blood transfusions, and organ transplants. Sexual transmission is theorized.  However, cats, the only known hosts, play an important role, by eating infected rodents, birds, or other small animals and shedding oocysts in their feces up to 3 weeks after infection. An infected cat contaminates the litter box and/or the soil or water if it goes outside. Transmission to humans occurs after accidental ingestion. In the human host, the parasites form tissue cysts in skeletal muscle, myocardium, brain, and eyes, and may remain for the life of the host, and can reactivate when the immune system is compromised.

http://www.ncbi.nlm.nih.gov/pubmed/16457490  There is evidence of coinfection of Toxoplasmosis with Lyme Disease. This particular patient was initially diagnosed with MS and had symptoms of clumsiness and weakness of the right extremities, and years later was also diagnosed with LD (borrelia). Toxoplasmosis is significant in people who are immuno-suppressed, and Lyme Disease will trigger a previous asymptomatic case.

http://chronic-lyme-disease-solutions.com/Toxoplasmosis.html  http://www.mayoclinic.org/diseases-conditions/toxoplasmosis/basics/symptoms/con-20025859

Symptoms: body aches, swollen lymph nodes, fatigue, headache, confusion, seizures, coordination problems, fever, lung problems, blurred vision, encephalitis, mental illnesses such as schizophrenia, depression, and bipolar disorder. It has been linked with anti-social, aggressive, and jealous behavior in men, and promiscuity in women. Children born with it may develop hearing loss, mental disability, blindness, and even death.

http://www.theatlantic.com/magazine/archive/2012/03/how-your-cat-is-making-you-crazy/308873/  Stanford’s Robert Sapolsky and British groups say that Toxoplasmosis in lab rats changes the wiring in their brains which can take away their fear response, drawing them to their number one predator — cats. Years ago Czech scientist, Jarosav Flegr, noticed reckless traits in his own behavior which included crossing the street in the middle of dense traffic and openly scorning the Communists who ruled his native Czechoslovakia. He accidentally discovered he had the parasite when he was asked to donate blood to test a diagnostic kit for Toxo. He discovered that the French have infection rates as high as 55%, due to their desire for steak prepared saignant, which literally means, “bleeding,” while Americans have a 10-20% infection rate. Neurobiologist Ajai Vyas found Toxo cysts in rat testicles and semen and that the protozoan then moves into the female womb, typically infecting 60% of pups, then heads to her brain to affect her behaviors eventually getting back to the cat. This leads to the possibility of sexual transmission in humans. The research also found that 75% of the females preferred the infected males. Psychiatrist E. Fuller Torry points out that schizophrenia rose in prevalence in the latter half of the 18th century just when people in London and Paris started keeping cats as pets. He believes that 75% of schizophrenia is associated with infections, with Toxo a significant portion.

Once a human becomes infected the parasite needs to get back into the cat, the only place where it sexually reproduces. Due to the impoverished Soviet economy, Flegr gave personality tests and computer-based tests to assess reaction times to infected and non infected Czech students. His findings were so strange he tested then civilian and military populations. He found: infected men wore rumpled old clothes, had fewer friends, and were more hesitant, while infected women wore expensive, designer brands, had more friends, and were extremely trusting – doing what they were told. Both had slower reaction times, less attentiveness, an abnormal fear response, and were two and a half more times more likely to be in traffic accidents. Two Turkish studies have replicated the traffic accident finding. He also also found that 12 of 44 schizophrenia patients had reduced gray matter, with the decrease occurring almost exclusively in those who tested positive for Toxoplasmosis.

http://www.medicalnewstoday.com/articles/295012.php?trendmd-shared=0  Medical News Today reported on a study claiming the parasite is responsible for around a fifth of schizophrenia cases. Now, new research by Johns Hopkins provides further evidence of this association after reviewing two previous studies which identified a link between cat ownership in childhood and development of schizophrenia and other mental disorders later in life and then  comparing them with the results of a 1982 National Alliance for the Mentally Ill (NAMI) questionnaire.  The questionnaire revealed that around 50% of individuals who had a cat as a family pet during childhood were diagnosed with schizophrenia or other mental illnesses later in life, compared with 42% who did not have a cat during childhood. 

T. gondii may be the culprit. 

Researchers at the Academic Medical Centre in Amsterdam, the Netherlands conducted a meta-analysis of more than 50 studies that established a link between T. gondii and increased risk of schizophrenia.

http://www.medicalnewstoday.com/articles/247346.php   Women carrying IgG antibodies to Toxo when giving birth have a higher risk of self-harm or suicide later on, especially if antibody levels are high.

Diagnosis:  http://www.mayoclinic.org/diseases-conditions/toxoplasmosis/basics/tests-diagnosis/con-20025859 Serology to check for antibodies to the parasite, although tissue cysts may be observed through stained biopsy. The CDC recommends all positive results be confirmed by a specialty lab for Toxoplasmosis. In some cases if testing is done too soon, there will be a false negative, and it would be wise to consider retesting later to give the body a chance to produce antibodies. A positive means you are actively infected or that you are asymptomatic. Congenital cases are found using molecular methods such as PCR or with an ultrasound scan that reveals hydrocephalus (fluid in the brain). A negative ultrasound does NOT rule out infection.

Please see your health practitioner for Treatment

Treatment: Healthy people keep the organism in check and do not require treatment; however, if you are also fighting MSIDS, you should consider this organism in your treatment picture.
http://www.mayoclinic.org/diseasesconditions/toxoplasmosis/basics/treatment/con-20025859
Pyrimethamine (Daraprim), a malarial drug is the typical drug of choice, which may prevent your ability to absorb the B vitamin, folate, necessitating supplementation. In conjunction, Sulfadiazine is used, with Clindamycin (Cleocin) as an alternative. Those with HIV/AIDS may need to take these medications for life or until the CD4 remains high for 3-6 months. Spiramycin, an experimental drug in the U.S., is used in Europe to reduce a baby’s risk of neurological problems and may be obtained from the FDA.

Similarly to borrelia, the causative agent of Lyme Disease, once the parasite is in brain cells; however, antibiotics cannot kill off the thick-walled cysts.

Prevention: http://www.mayoclinic.org/diseases-conditions/toxoplasmosis/basics/prevention/con-20025859

*Wear gloves when you garden or handle soil and wash your hands thoroughly with soap and water afterward.
*Don’t eat raw or undercooked meat.
*Wash kitchen utensils thoroughly. After preparing raw meat, wash cutting boards, knives and other utensils in hot, soapy water to prevent cross contamination of other foods. Wash your hands after handling raw meat.
*Wash all fruits and vegetables. Scrub fresh fruits and vegetables, especially if you plan to eat them raw. Remove peels when possible, but only after washing.
*Don’t drink unpasteurized milk. Unpasteurized milk and other dairy products may contain toxoplasma parasites.
*Cover children’s sandboxes. If you have a sandbox, cover it when your children aren’t playing in it to keep cats from using it as a litter box.

If you’re pregnant or otherwise at risk of toxoplasmosis or its complications, take these steps to protect yourself:
*Help your cat stay healthy. Keep your cat indoors and feed it dry or canned cat food, not raw meat. Cats can become infected after eating infected prey or undercooked meat that contains the parasite.
*Avoid stray cats or kittens. Although all stray animals need good homes, it’s best to let someone else adopt them. Most cats don’t show signs of T. gondii infection, and although they can be tested for toxoplasmosis, it may take up to a month to get the results.
*Have someone else clean your cat’s litter box. If that’s not possible, wear gloves and a face mask to change the litter. Then wash your hands well. Change the litter daily so that excreted cysts don’t have time to become infectious.

Hyperthermia and MSIDS

Sitting in my doctor’s office, I read an article that intrigued me but made me shudder simultaneously.  In the November 8, 2013 issue of Science pp. 684-687, I read of Plasmodium vivax, the long considered “benign” malaria parasite which threatens billions of people, but more interestingly to me as an MSIDS patient, was it’s historical usage as a cure for tertiary syphilis.  Physicians in the late 19th century believed that high fever could help cure many mental illnesses.  These poor patients were institutionalized with a dismally gruesome future of increasingly neurotic behavior and paralyzation.  They had no hope.

Austrian psychiatrist Julius Wagner-Jauregg initially used tuberculin and salmonella toxins but his fever experiments failed.  He reasoned this was due to too low of a fever, so in 1917 when a soldier fighting in the Balkans was admitted to his ward with Malaria, he tried again using his blood to inoculate nine neurosyphilis patients.  Six recovered.

Thus started the wave of malariotherapy which became the treatment for tertiary syphilis.  No one is sure how it worked but the resulting high fevers appeared to help the patients’ immune systems.  About half resumed to normal activities; many resumed independent lives.

According to Kevin Baird of the Eiikman Oxford Clinical Research Unit in Jakarta, this medicinal use of P. vivax is in part to blame for the neglect of the disease it causes as people assumed it must be harmless even though it killed as many as 15% of patients who had the treatment.

This background paves the way for what is to follow:

https://www.youtube.com/watch?v=WLYZcju9RGM&sns=em

The above youtube is not only an excellent expose on MSIDS in Australia, but also on the current usage of hyperthermia.   Australian patients, who appear to have MSIDS are ignored and told it’s all in their heads.  The video shows patients getting worse, having to quit work, and breaking down in front of the camera.

Same story, different country.  

Kudos to Dr. Schloeffel who is one Australian doctor who refuses to accept patient abuse and neglect and treats his patients clinically not basing all of his decisions on faulty testing.

Due to the lack of acceptance and treatment, many Australian MSIDS patients are heading to Germany to receive the old fashioned hyperthermia treatment at St. George Clinic.  Dr. Frederich Douwes, stumbled upon Hyperthermia as a possible cure for MSIDS while treating cancer patients.  Again, hyperthermia gives the body an artificial fever.  For over 6 hours a patient’s body is heated to 41.7 degrees.

Dauwes says he has treated over 18,000 whole body hyperthermia patients with no negative side-effects.  Other modalities for MSIDS patients are included as well such as ozone, Reiki, acupuncture, foot spa detox, magnetic and laser therapy and IV antibiotics.  It costs anywhere from $30,000 – $55,000 for treatment.

The video is approximately 23 minutes long and worth every minute of it.  Very well done.  Although published in 2014, nothing much has changed in regards to general physician knowledge either in Australia or the United States.

Lastly, this raises a question:  supposedly “between 1917 and the rise of penicillin in the 1940’s, tens of thousands of syphilis patients were infected with malaria.” p. 686.  We know for sure syphilis is spread through sexual contact.  They not only had syphilis but malaria.  What happened to those people and their off-spring?  Is there a connection between the malaria experiment on syphilis patients and MSIDS today?

And hyperthermia?  I’m just thankful they aren’t using Malaria!