Lyme Disease Protocol
By Michael Biamonte, C.C.N.
Discovery of Lyme Disease
There are about 1,200 cases of Lymes Disease reported across the United States each year; there were 1,282 cases reported in 1934. The disease remains concentrated along the coastal plain of the Northeast and Mid-Atlantic region, in the upper Midwest, and along the Pacific coast, although the disease has been reported in 32 states.
In a Science News report, researchers at the University of Connecticut Health Center in Farmington and the Yale-New Haven Hospital examined 70 children diagnosed with Lymes Arthritis Disease and found that only 53% actually harbored the Lymes-causing bacterium Borrelia burgdorferi. The remaining 47% had been misdiagnosed.
There is some hysteria regarding the incidence of this disease, possibly due to extensive adverse publicity. One thousand two hundred and eighty-two cases out of perhaps several hundred thousand with tick bites is not exactly a national emergency, although for a percentage of those afflicted, the disease can be rather significant and even catastrophic.
Lyme disease has the following symptoms: begins with reddened area that doesn’t itch, resulting from tick bite, but expands over time, measuring several inches across; clearing of bite area begins in center resembling a bull’s-eye; flu-like symptoms: chills, fever, fatigue, joint and muscle pain; may develop a rash which disappears in a few days; may have tingling and numbness; non-symmetrical joint problems; other symptoms may also occur; sometimes sensitivity to light, stiff neck, headache, sleepiness, mood changes and memory loss; swelling and aching joints for months or years at a time; and vague, migrating musculature pains.
The characteristics of Lyme Disease were first laid out in 1975 when two mothers were told that their children had Juvenile Rheumatoid Arthritis. The name “Juvenile” does not distinguish its clinical pattern from that of “Adult” Rheumatoid Arthritis, but merely tells the parent that this horrible, crippling disease occurred in their child, a fact that most parents already know. What is new is the diagnoses of “Rheumatoid Arthritis.”
These two mothers soon learned that many other children and adults in their geographical region were afflicted with the same symptoms, and since Rheumatoid Arthritis does not seem to cluster in a regional geography (with some exceptions), Dr. Stephen E. Malawista of Yale University, among others, began to look for a source of this apparently new disease. Dr. Malawista discovered that many of his patients suffered from a range of symptoms, among which, might be those that resembled Rheumatoid Arthritis.
The cause of Lyme Disease was determined to be a microbe transmitted by a tick, in this first instance, from the species Ixodes capularis. Since this tick was common in the grasses and woods near Lymes, Connecticut, the cluster of symptoms obtained the name “Lymes Disease.” As Dr. Willy Burgdorferi, who worked for Rocky Mountain Laboratories in Hamilton, MT, identified the damaging microbe. The bacteria was named Borrelia burgdorferi, which is a spiral-shaped bacterium similar in shape to the spirochete, Treponema pallidum, which causes syphilis. Since this initial set of discoveries, it’s clear that similar diseases have existed in Australia, Africa, Europe and Asia. It also appears in every one of the states in the United States, but seems to be particularly common in northern California, Minnesota and the Northeast. Infection by Borrelia burgdorferi occurs chiefly in the spring, summer or early fall, because of the life cycle of the Ixodes scapularis tick.
Three Stages of Borrelia Burgdorferi
There are three stages to the life cycle of the tick, and at each stage they have a favorite host, although they will attach themselves to a range of animals, including the human species.
The larva from Ixodes scapularis emerges in the summer from eggs deposited in the spring, and attaches itself to a small vertebrate such as a white-footed mouse, where it imbibes its first meal. If this mouse is infected with Borrelia burgdorferi spirochetes, the larva feeding on the mouse’s blood will also become infected.
Later, the larva molts into a nymph, and during the spring and summer (usually mid-May through July) this nymph takes a second meal. If the larva was infected, it may very well pass Borrelia burgdorferi onto its second host. This nymph is now about the size of a small seed, say, a poppy seed, and is responsible for most human infections.
The nymph molts again, and by October is the size of a larger seed, like an apple’s. Again this tick feeds, at least by winter or spring, and they also mate to produce eggs that begin the cycle all over again. Usually ticks do their mating on white-tailed deer, which is why they are referred to as a “deer tick”.
In some regions of the United States, between 15 and 30 percent of the Ixodes scapularis nymph and adult ticks are infected with Borrelia burgdorferi — some 50 percent of adult ticks are infected. The adult ticks are more likely to infect humans because they have had more opportunity, throughout their life-cycle, to do so.
About 1 to 3 percent of adults who are bitten by the infected tick contract Lymes disease, meaning that a high percentage of those infected are able to master the infection.
The tick attaches itself to the skin of its host, where it takes its meal of blood. At this time Borrelia burgdorferi begins to multiply in the gut of the tick, whence it crosses into the tick’s circulation system, migrating to the salivary glands and passing with the tick’s saliva through the host’s skin.
A tick must be attached to its host for 36 to 48 hours before an infectious dose of Borrelia burgdorferi is transmitted. This is fortunate, because most folks who are bitten by a tick will find it prior to the infectious event.
Lyme Disease Symptoms
Those infected by the bacteria Borrelia burgdorferi usually have a set of characteristic symptoms.
Stage I Symptoms:
1. About 60% will notice a round rash called an erythema chronicum migrans (ECM), as doctors like to have a nice, neat name for everything they observe.
2. Three days to a month later there will be a redness at or near the site of the tick bite.
3. The reddened area does not itch or hurt, but it will expand over time until it may measure several inches across.
4. There is a clearing that begins in the center, as the rash enlarges, resembling a bull-eye. Some may acquire the rash, but fail to see these characteristics because of the bite’s location.
5. The rash may disappear within weeks or even days.
6. Days or weeks later, a variety of other early symptoms affecting many areas of the body appears, and these symptoms are thought to be from the spread of the spirochete to many different tissues through the blood stream. The symptoms will include flu like symptoms, such as chills, fever, fatigue, joint and muscle pains, and loss of appetite.
Stage II Symptoms:
Weeks to months later, about 10% of those afflicted will experience transient heart dysfunction. There will be varying degrees of heart blockage. Neurological abnormalities include headaches, profound fatigue, meningitis, cranial nerve problems (neuropathies), including fascia palsies, and sensory and motor nerve problems.
Cardiac problems occur with 5 to 10 percent of those infected, if they have been untreated. Usually this condition is not noticed by the infected person, but can be detected by a physician. The heart irregularities persist for but a week to 10 days and probably will not require the use of a pacemaker.
Early symptoms may also include mild musculoskeletal disturbances, where patients complain of vague, migrating pain without swelling in muscles, tendons or joints. The jaw, the temporomandibular joint, may be affected. These symptoms, too, will decrease in weeks to months. However, in about a half a year after the initial infection, 50% of those infected (without treatment) will suffer episodes of obvious arthritis, including the symptoms of swelling and discomfort in one or more joints, but often the knee.
Stage III Symptoms:
Ten percent of those who reach the “arthritic” point will go on to suffer chronic Lyme Arthritis. These patients will find joints swelling for months at a time, or certain joints will become enlarged and achy for a year or more. In these latter stages, joints, the central nervous system and the skin may be involved. Arthritis can develop from a few weeks to several years after Stage I. Sixty percent suffer at least one episode of arthritis if untreated. Usually the joint arthritis is but one-sided, and migration of the joint pain may prefer the larger joints, especially the knees.
Attacks may last for weeks or months, although they may also become less frequent over time and eventually disappear, leaving about 10% with damaged joints.
Sometimes neurological problems also appear, in about 20 percent of untreated patients, including Bell’s palsy. Bell’s palsy is one of our listed “Rheumatoid Diseases,” a collagen tissue disease, and so there must be more than one causation for that affliction. Other neurological afflictions include meningitis (sensitivity to light, stiff neck, headache), encephalitis (sleepiness, mood changes, memory loss), and radiculoneuropathy, where the roots of nerves that stem from the spinal cord to the periphery of some level of the body becomes irritated. These regions may be painful, tingle, or even go numb.
In traditional Rheumatoid Arthritis, a joint affected on one side of the body will also have a matching joint affected on the other side of the body. This is not true for Lyme Arthritis where only one joint may be affected on one side of the body.
Although the skin, heart, joints and nervous system are usually targeted, as the Borrelia burgdorferi bacteria can invade any system in the body, every organ or system in the body can also produce its own variation of symptoms. This ability to invade all human systems, too, is a similarity to the syphilis spirochete.
Traditional and Untraditional Treatments
Thankfully, if diagnosed early enough, antibiotics can easily wipe out the invading population, and bring about swift remission. (The antibiotics, of course, should be heavily accompanied with Lactobacillus acididophilus so that while treating the Lyme Disease, we do not also unwittingly bring about a fungal infection of candidiasis.)
The key to solving Lyme Arthritis is early diagnoses and antibiotic treatment. Early diagnoses can be difficult, especially when the characteristic rash is not present. Since flu like symptoms can arise from many different sources, as described in Dr. Paul Pybus’ The Bernheimer Effect, it becomes most difficult for a physician to make early diagnoses. The patient’s history, especially their recent exposure to woods, ticks, and bites, and especially noting the characteristic bulls-eye lesions on the skin, all are most important for early diagnosis.
Although a definitive test for Borrelia burgdorferi bacteria is possible, the test is presently a low-yield procedure. A direct examination of body fluids and tissues is not recommended because there will be so few organisms found. There are no blood tests that can make early diagnoses of Lyme Arthritis within the time length required for an early diagnosis, although surely someone somewhere is working to develop such an early test, probably based on DNA of the microorganism. The study of body serums, serology, using indirect immunofluorescent assay or enzyme-linked immunoabsorbant assay has a slow antibody response and is positive in but 50% of Stage I infections, and should antibiotics be used, the test is often aborted.
Since Lyme Arthritis is potentially disabling, extreme vigilance must be taken by those who traverse woods and grasses, but overall, it may not cause serious problems for more than 10 percent of those who have received Borrelia burgdorferi through a tick bite. Many who think they have Lyme Arthritis actually suffer from other forms of disease states, but among those who are found among the 10 percent seriously affected, there seems to be no good solution to the problem, because, after the early stage of the disease, antibiotics seem to be ineffective.
The primary problems with traditional treatments consists of the following:
1. Inability to diagnose the disease early without specific noting of the bulls-eye lesions, or having at hand an accurate, clear, case history. This delay affects treatment response by use of antibiotics, and often also causes over-extended usage of antibiotics;
2. Over-extended usage of antibiotics increases overgrowth of organisms-of-opportunity in the intestinal tract, such as Candida albicans, which condition also creates additional disease states, including some that mimic various arthritides forms, and also increases food allergies over time;
3. Many treated cases linger with pain, increasing systemic damage, and lessened vigor over many years, often ending up damaged organs and joints. There is hope, however.
Anti-Amoebic (Anti-Microbial) Treatment
Gus J. Prosch, M.D., Jr. of Birmingham, Alabama suggests a trial of the Rheumatoid Arthritis or Rheumatoid Disease treatment protocol as recommended by The Arthritis Fund/The Rheumatoid Disease Foundation: “I’ve seen Lymes Arthritis Disease clear up after using a course of anti-micro-organism drugs as recommended by Professor Roger Wyburn-Mason for Rheumatoid Diseases.
“Although Lymes Arthritis Disease and other diseases such as Gout, Carpal Tunnel Syndrome, and Tendonitis are not supposed to be the same kind of diseases as Rheumatoid Arthritis, I’ve seen them all respond one time or another to the same treatment we use for Rheumatoid Arthritis.”19
In the case of Lymes Arthritis Disease, Dr. Prosch will give metronidazole (or one of the other 5-nitroimidazoles described) in heavier doses, for a longer period of time than recommended for Rheumatoid Disease.
Artificial Fever and Herbs
Agatha Thrash, M.D. and Calvin Thrash, M.D. write that “About one-third of patients with chronic infectious arthritis derive substantial benefits from fever treatments, one-third derive only moderate benefits, and one-third little or no help.
“In gonococcal arthritis, swelling and pain is often astonishingly helped. Patients suffering from [Osteoarthritis] receive temporary benefit, and the fever treatments may be used along with general arthritis treatment of diet and physical conditioning.”24
The Biamonte Protocol
The protocol that I have developed comes from several years of research. I have worked with a many Lymes patients. Many of them came to me due to their antibiotic treatments causing Candida.
I have found Mild Silver protein to be a very effective alternative to antibiotics in the treatment of Lyme disease. Department of Health & Human Services (NIH) and cancer center laboratory test results, regarding Lymes disease.
Borrelia burgdorferi & b hermsti, organisms associated with the causing the symptoms of Lyme Disease, were tested at the Department of Heath and Human Services, Rocky Mountain Laboratories, & Fox Chase Cancer Center, respectively in 1995, colloidal silver in concentrations of 15 PPM And 150 PPM demonstrated that no live spirochetes of either borrellia burgdorferi (B310 or b hermsti (HS-1) survived after 24 hours of exposure, in the Rocky Mountain Labs study. Ref. (11, 4-D)
The Fox chase Cancer Center, Philadelphia, Pennsylvania, demonstrated growth inhibition of borrelia burgdorferi using colloidal silver in concentrations as low as two to ten PPM. Much more rapid effects demonstrated in higher concentrations e.g. fifteen to seventy-five PPM. Ref. (12, 4-F) This offers great hope to Lymes victims as the draw back of antibiotics, aside from causing yeast infections and antibiotic resistance is that antibiotic treatments in many cases have been observed to cause susceptibility to relapse of Lyme.
Silver protein has a long history as an effective antibiotic. In recent years it has been found effective against not only bacteria but virus as well. Silver has gone through a “rebirth” in the alternative medical industry, due to its broad spectrum applications. It is found effective against very resistive bacteria. Science Digest” March 1978 issue reported in the article “Our mightiest Germ fighter,” “Thanks to eye-opening research, silver is emerging as a wonder of modern medicine. An antibiotic kills perhaps a half-dozen different disease organisms, but silver kills some 650. Resistant strains fail to develop. Moreover, silver is virtually non-toxic.” Dr. Harry Margraf, a biochemist and professional researcher and associate of the late Carl Moyer, M.D., chairman of Washington University’s Department of Surgery in the 1970’s concluded the article, with the following “Silver is the best all-around germ fighter we have.” Ref. (6.)
Anti-malaria medications also play an important role in the elimination of Lymes. The herb Artemisia has been used by the US Army as a Malaria “drug”. This herb traditionally has been used as an anti-parasitic. When combined with silver protein it has been found to be very effective in the elimination of Lymes with low incident of replace. I feel that Silver and Artemisia must be used together! TAO free cats claw and Borrelogen have also been found effective in eliminating lyme and are often used with the silver as a phase 0.
Just as in our candida protocols we rotate anti-lyme microbials to guard against the possibility of drug resistance or antibiotic resistance occurring.
While the rotation is underway we also treat the secondary infections that accompany the lyme. A new version of Biocidin has been developed for lyme. Banderol lomatium and Houttuynia can also be rotated to destroy Lyme.
Anecdotal observations by practitioners who have used this protocol conclude that the course of treatment should last approximately 1 year. The incident of relapse has been very low in those who have completed a year’s course. Apparently the “tail” of the spirochete will tend to survive antibiotic therapy and release more of the disease causing eventual relapse. I have seen excellent results with this protocol. It is also compatible with Candida treatments that are so often necessary in the Lymes patient due to their previous antibiotic use.
Michael Biamonte, CCN
William Hammesfahr, MD
The Biamonte Center
For Clinical Nutrition
Red flags immediately go up when someone calls it “Lymes Disease,” because it announces the fact they are ignorant of the fact it all started in the town of Lyme, Connecticut with a cluster of cases in children who were misdiagnosed with juvenile arthritis (JA). It’s Lyme disease, named after the town of Lyme. Please go here for an excellent video by an experienced Lyme literate doctor on the history of Lyme disease, of which manifestations began long before this cluster of children. Go here for a summary of the video and other important facts important to understand that not mentioned in Biomante’s article that explain the sordid backstory, the reason Lyme/MSIDS research being used is fraudulent, and completely biased, the flagrant conflicts of interest within the agencies controlling the Lyme narrative, and The Cabal doing the only accepted research that does not take into account global research and independent research showing the organism persists despite treatment.
Regarding cases, this article is way off. Reporting has been a problem from the beginning as the surveillance criteria has such a high bar that hardly anyone meets it. Getting a positive on the 2-tiered CDC testing is akin to winning the lottery. The world at large now knows that Lyme is woefully under-reported. Nobody has a clue about coinfections. To continue to regurgitate these unrealistically low numbers doesn’t help anyone and only demonstrates ignorance. I also don’t appreciate the same mythology regarding where Lyme exists. This has also been a problem and is a perfect example of bad science continuing to be used. Lyme is literally everywhere. That’s all you need to know. Don’t continue to downplay this. It’s a plague of biblical proportions.
Regarding the research at the University of Connecticut finding only 53% had Bb and were misdiagnosed with Lyme arthritis, this too remains highly dubious. All testing for this illusive organism is abysmal – plus current two-tiered CDC testing only tests for ONE strain when there are 100 strains and counting in the U.S. alone. More are found on a regular basis. Testing won’t pick of any of these other strains. All parameters for case numbers are faulty.
He announces that there is “hysteria” regarding the disease. This immediately raises my blood pressure. He truly is clueless. This continued downplaying of a life-shattering, complex illness has been going on for over 40 years due to vested interests and faulty science needs to end. The “untreatable form of Lyme disease could hit 2 million Americans,” and that isn’t even taking into account global numbers. Lyme disease is more prevalent than AIDS, breast cancer, West Nile virus, H1N1, and Ebola. He doesn’t mention that Lyme is congenitally transmitted and there is evidence being ignored that it is also sexually transmitted.
Biamonte’s description of the symptoms also shows his inexperience. Lyme can virtually look like anything and mimic some 300-different diseases. While some get the EM rash, many don’t. The rash can also look quite differently on patients. Strain diversity appears to make a difference regarding symptoms, with some strains causing more skin manifestations and some causing more joint manifestations – regardless, it is wrong to attempt to put this monster in a neat four-cornered box. Further, ticks are migrating everywhere, intermingling, and nobody has a clue what that is going to do to strain diversity and symptomology. Again, this hasn’t been studied in decades because according to The Cabal, it’s a done deal. No further science required.
Can you think of any other disease in which this attitude of ignorance is allowed and accepted?
I would also urge caution in blaming the black legged tick as the sole perp. Since Bb and its many strains and all the coinfections are extremely fastidious organisms, early work as been done and then used again and again and again for decades. Time for new, independently done science with new methods. We desperately need transmission studies as the ones being used are decades old. Ticks all bite, exchange fluids and have the potential to transmit diseases. Don’t diminish the tick’s ability to side-line your life with things you never even knew existed!
The explanation of the 3 stages of the life cycles of ticks is also simplified. It is known ticks can partially feed, drop off, and then transmit much more quickly to the next victim. We know ticks can parasitize each other. We know that ticks can survive harsh environments by burying under leaf litter and snow (or anything else they can find like wood chips in a playground). They also go through a hibernation period called diapause. Ticks can also pass on infections to their offspring. There is much we don’t know – especially regarding transmission.
I would caution against using percentages of infected ticks to prove a point. Remember, it only takes ONE tick, ONE bite, and your life could be changed forever. Each tick is a potential bomb capable of infecting you with 19 and counting diseases.
The regurgitation that a tick must be attached for 36-48 hours to transmit infection is based on faulty science. Minimum times for infection have never been determined. It also does not take into account the fact pathogens have been found in the salivary glands, suggesting a much quicker transmission time, and that ticks often partially feed, drop off, and can infect you quicker. Very old research is being used again, and again, and again, when reality has shown people getting infected within a few hours. This mythology continues to downplay a modern-day scourge by using ancient data. Some tick-borne infections can be transmitted within minutes. Many of them look just like Lyme. Another mistake is to focus solely on Lyme. In my experience Babesia, Bartonella, and Mycoplasm are as bad if not worse than Lyme. If you are infected with a few of these suckers at once, you are one sick dog. And in my experience, this is the norm.
The section on “Lyme Disease Symptoms” again demonstrates this man’s inexperience. Hardly anyone I know fits his limited list. Again, research has shown the EM rash to be highly variable, and hardly ANYONE I work with has seen it. Most also haven’t seen the tick. Patients and their doctors often work completely in the dark, and what often happens is over time is bizarre unexplainable symptoms start cropping up more and more until life becomes unbearable. At this point Bb and coinfections are virtually everywhere in the human body – heavily entrenched and therefore, harder to treat. This is reality.
Also, people can jump from stage to stage in no particular order. Some will experience psychiatric symptoms IMMEDIATELY and never have the rash, fever, joint pain, etc.
In Stage II, Biamonte states about 10% will experience transient heart dysfunction. Again, it’s very unwise to use percentages when testing misses a preponderance of cases and the organism is elusive. This study found an increasing burden of Lyme carditis in U.S. children’s hospitals. Many are questioning if there could be subclinical cardiac involvement in early Lyme with children, and that’s another fly in the ointment. Most testing won’t pick up problems with these patients because their symptoms are subclinical, yet they are severe to the patient. If I had a nickel for every time a patient told me the test didn’t find anything, I’d be a rich woman. Just because testing didn’t reveal something, doesn’t mean something isn’t there. This is truly the norm with tick-borne illness. I didn’t start having heart issues until we started treating for Babesia and then all of a sudden, BOOM! It felt like I was having a heart attack. This is another reality. Until you start utilizing anti-microbials, the immune system is confused and unable to deal with these infections because they fool the immune system by changing their outer surface proteins to look like the good guys. Further, so many are misdiagnosed that percentages are meaningless. Seriously. Meaningless. There are thousands out there who have Lyme carditis who have completely fallen through the cracks. Thousands.
He states symptoms will decrease in weeks to months WITHOUT treatment. It’s obvious he is reading Wormser and other Cabalist’s research as this is what they believe; however, in the real world symptoms wax and wane but never totally go away, and left untreated with only become more entrenched in the body. Again, this illness often takes years to unravel. Waxing and waning is a marquee symptom with tick-borne illness, but without treatment it will metastasize everywhere in the human body. There is a connection with Lyme/MSIDS and cancer as well as brain diseases like ALS, dementia, Alzheimer’s, MS, etc. Left untreated, the parasites will continue to live off the host weakening it year by year until they are a shell of themself.
He states 10% will suffer chronic arthritis. Let me be clear: nobody has a clue about the prevalence of arthritis in these poor patients. Not a clue. Putting this in a box, unless it’s Pandora’s is the biggest mistake being made.
Regarding treatment, he omits to mention that even people diagnosed and treated early can require further treatment as symptoms return. This is very common.
He mentions direct testing being a “low-yield” procedure as so few organisms are found, but that “surely someone, somewhere is working to develop such an early test, probably based upon the DNA of the microorganism.” This too shows the ignorance of the history of the suppression of direct detection techniques. In fact a test has been found to be highly accurate but our corrupt public health “authorities” monopolize testing, and have done unethical things against competitors for decades. Public health owns the patents on the organisms, the tests, the treatments, and the vaccines. It’s a business, not a public health agency concerned with health. This is imperative to understand.
He does mention the success of metronidazole or one of the other 5-nitroimidazoles in heavier does for a longer period of time. I would agree, but never as a mono therapy. Savvy Lyme literate doctors have learned from vast experience with thousands upon thousands of patients to layer treatment, never utilizing a mono therapy, to avoid antibiotic resistance. Again, coinfections are common place and require different medications including anti-protozoan meds, anthelmintics, and more. The potential for candida should also be taken into account and dealt with.
Regarding the use of colloidal silver for Lyme, I completely disagree. This recent study shows stevia, Andrographis, Grapefruit seed extract, colloidal silver, monolaurin, and antimicrobial peptide LL37 didn’t do diddly. Keep in mind this work is done in vitro – or in a lab, not the human body – although this follows my personal experience as well. This 2004 study shows that 3 samples of colloidal silver of 22 ppm and two samples of 403 and 413 ppm in an agar-well diffusion assay showed ZERO effect on the growth of test organisms but ALL were sensitive to ciprofloxacin. Silver at 22ppm showed NO bactericidal activity in phenol coefficient tests.
The patients he mentions have already been treated with many antibiotics and have developed candida issues (not uncommon). He doesn’t mention how long these patients were treated, which would be helpful to know. Please know that a wise treatment would address candida along the way. We took fluconazole twice a week throughout our treatment course along with a low or no sugar diet.
I personally know patients that used silver and the result was they ended up wheel-chair bound. They only worsened and worsened.
He mentions research done in the 90’s showing that colloidal silver killed Bb after 24 hours of exposure. The other research mentioned is from the 70’s. If it was so effective, much more would have been done and trust me, desperate patients and the doctors who dare treat them would be using it, and they are not. To claim that silver is virtually non-toxic is also premature. Little has been done on it – particularly using it over long periods of time. Again, metals are not harmless and accumulate in the body.
I’m a huge proponent of using silver topically on wounds, etc. Hospitals have shown the effectiveness of this substance for decades for cleaning and sterilizing objects topically. Sometimes I will even use it to ward off a cold by spraying it on my throat for a few days. Sometimes it appears to work and other times it doesn’t, which is only my personal observation.
Some claim that utilizing it along with antibiotics, potentiates the antibiotics. My concern would be putting metals in a body already struggling. Metals, after all, accumulate. In fact, many Lyme/MSIDS and autism patients improve by using chelation which removes heavy metals.
He states that artemisia has been used effectively for Lyme. I would disagree. This is an anti-malarial medicine that has action against Babesia, which is a cousin to malaria – a protozoan. Due to the repeated mistakes in his article and the downplaying of the seriousness of this complex illness, I question his experience with not only being able to identify coinfections and their symptomology, but also the importance of treating each infection with specific antimicrobials that have action against it.
From clinical observation, Cat’s Claw is effective against Lyme; however, there is debate in the herbal world about the need for TOA free vs the whole herb. Again, I’m not qualified to enter this debate, but Master herbalists write on it with conviction both ways. In the end, we often are forced to experiment to determine the truth of the matter and even then patients often have different findings, reminding us of the complexity of the human body. In the end, whatever works for you – USE IT!
While it is wise is to rotate meds, savvy Lyme literate doctors have a method to their madness and pay close attention to the life-cycle of the organism as well as the plateaus patients experience. Rotating, while important to guard against drug resistance, it is also important to layer treatments so they work synergistically together – also negating resistance and effectively dealing with coinfections and candida.
I have used Banderol and Biocidin with little effect. I’m sure others have had a better experience, but one again – treatment should always be an individualized approach.
Regarding length of treatment, one of the wisest, most experienced LLMD’s in Wisconsin (RIP) told me that in the 70’s when he treated this illness they labeled a “rickettsial” like illness – as it wasn’t even named yet, he found that a few months to a year of treatment appeared to work. He now states treating this takes YEARS – like 3-5 years. So, according to this wise, experienced doctor, things have changed making this harder to treat. Perhaps coinfection involvement has become more of a problem than in the past.
Please remember that according to the article, most of the patients Biamonte treats are seeing him for Candida AFTER they have already been treated for Lyme/MSIDS. This would explain why he is perhaps seeing success after only one year. They’ve already been treated, perhaps for years by someone else. They have successfully beaten down and reduced the infection load and are now struggling with Candida, immunoconfusion, and the last vestiges of infections that have already been hit hard by antibiotics.
Finally, it’s important to remember that this doctor is seeing patients that are suffering with significant blow-back. His experience is going to be biased in this direction. I wish he would stick with helping people recover from treatment that out of necessity is harsh (until something else is discovered) but not superimposing his beliefs that the treatments are wrong, or that colloidal silver is the answer to all our woes.
The fact that these patients are recovering in a year shows me that these patients are well on their way to health but need specialized help in dealing with damage caused by either the infections themselves, the harsh treatment required, or a combination of both. This problem is also quite common.