Archive for the ‘Viruses’ Category

Resolving Persistent Spike Protein Syndrome

https://townsendletter.com/resolving-persistent-spike-protein-syndrome/

Resolving Persistent Spike Protein Syndrome

Thomas E. Levy, MD, JD
Orthomolecular Medicine News Service

6/1/23

Article Excerpts:

As the acute cases of COVID have continued to decline, the prevalence of the Persistent Spike Protein (PSP) syndrome has continued to increase. The spike protein is that part of the COVID pathogen that attaches to ACE2 receptors throughout the body and permits the entry of the entire virus into the newly infected cell. There appear to be no cells, tissues, or organs in the body that are completely spared from this PSP attack once enough of it has been introduced into the body.

The persistent presence of the spike protein has been shown to be secondary to the inability to completely resolve a bout of COVID (chronic COVID or long-haul COVID) as well as the spike protein exposure from mRNA inoculation(s). And as more time has passed, the PSP syndrome following one or more mRNA shots has emerged as the most common reason for PSP, especially following a booster injection. Not surprisingly, the likelihood of developing a PSP syndrome relates directly to the total amount of spike protein exposure, and the amounts delivered by repeated inoculations substantially exceed the amounts that result from incompletely resolved cases of COVID.

The goal of any therapy designed to eliminate a chronic spike protein presence in the body needs to address its presence in the blood, its presence on the many ACE2 binding sites throughout the body, its presence inside the cells, and the mechanisms that allow it to replicate itself and keep it from being eliminated completely in the body. It has been shown that the sickest of PSP patients have intact spike protein circulating in the blood.1 

Multiple autopsy studies have revealed the presence of spike protein throughout the body, without any particular areas being spared.2,3

By itself, the spike protein is also toxic. As all toxins ultimately inflict damage by oxidizing biomolecules needed for normal metabolic function, any effective PSP protocol needs to include significant antioxidative capacity in order to repair damaged (oxidized) biomolecules. Spike protein has been shown to induce inflammation (acute oxidative stress) even without resulting in viral infection.4

Bio-Oxidative Therapies

While any therapy that can eradicate an infectious agent must involve its destruction via enhanced oxidation, the most prominent of these therapies involve the appropriate application of:

  • Vitamin C (multiple modalities)
  • Hydrogen peroxide (multiple modalities)
  • Ozone (multiple modalities)
  • Ultraviolet blood irradiation
  • Hyperbaric oxygen

While still not widely appreciated, these bio-oxidative therapies have been curing acute infectious diseases for very many years now.  (See link for article & references)

________________

**Comment**

Dr. Levvy states that a good protocol for PSP should include:

  • Ozone autohemotherapy, followed by
  • IV vitamin C (50 to 150 grams daily); or any oral form in the highest doses possible
  • IV or oral hydrocortisone (25 to 50 mg) in the IV or with the first oral dose of vitamin C
  • Ultraviolet blood irradiation if available
  • Hydrogen peroxide nebulization
  • Methylene blue, 25 to 50 mg two or three times daily
  • Proteolytic enzymes (bromelain, NAC, nattokinase)

Category:

Supplements, Treatment, Viruses

Benefits of Ozone on Mortality in Patients with COVID-19: A Systematic Review and Meta-analysis

https://petermcculloughmd.substack.com/p/benefits-of-ozone-on-mortality-in?

Benefits of Ozone on Mortality in Patients with COVID-19: A Systematic Review and Meta-analysis

Small Studies Found Benefit Yet Adjunctive Therapy Not Advanced to Large, Multicenter Trials

Sep 17, 2024

By Peter A. McCullough, MD, MPH

I attended an integrative medicine meeting recently and one of the speakers said that ozone was one of the most important tools in his practice. While this is a very broad topic with > 4000 papers listed in the National Library of Medicine with the MESH term “ozone therapy,” Hu et al briefly summarized:

“Ozone is a molecule composed of three oxygen atoms and a component of the atmosphere in nature, which has a strong oxidizing action. Ozone has a high-energy, variable molecular structure under normal temperature and is quickly and spontaneously decomposed into O2 and a single oxygen atom (O). It has strong activity in oxidation and a strong bactericidal effect on bacteria and viruses.10,11  Ozone therapy inactivates bacteria by disrupting their cell envelope through oxidation of phospholipids and lipoproteins, inhibits fungi growth, damages the capsid of viruses, and upsets the reproductive cycle by disrupting the virus-to-cell contact with peroxidation.12  Oxygen-ozone therapy causes an increase in the rate of red blood cell glycolysis, causing the stimulation of 2,3-diphosphoglycerate, which leads to an increase in oxygen released to the tissues.13”

Innovative physicians, from many parts of the world trialed ozone in hospitalized patients some of whom had 4-6 weeks in the hospital with acute COVID-19. Shang et al performed a meta-analysis of very small studies…. (See link for article)

_______________

https://pubmed.ncbi.nlm.nih.gov/36513208/

Benefits of ozone on mortality in patients with COVID-19: A systematic review and meta-analysis

Wenli Shang1, Yan Wang2, Guizuo Wang1, Dong Han3

Abstract

Background: The Coronavirus disease-2019 (COVID-19) pandemic continues, and the death toll continues to surge. Ozone therapy has long been used in the treatment of a variety of infectious diseases, probably through its antioxidant properties and the supply of oxygen to hypoxic tissues. This systematic review and meta-analysis aimed to determine the efficacy of ozone on mortality in patients with COVID-19.

Methods: A systematic search was made of PubMed, Embase, Cochrane Library, and clinicaltrials.gov, without language restrictions. Prospective controlled trials on treatment of COVID-19 with ozone, compared with placebo or blank, were reviewed. Studies were pooled to risk ratios (RRs) and weighted mean differences (WMDs), with 95% confidence intervals (CIs).

Results: Eight trials (enrolling 371 participants) met the inclusion criteria. Ozone therapy showed significant effects on mortality (RR 0.38, 95% CI 0.17-0.85; P = 0.02), length of hospital stay (WMD -1.63 days, 95% CI -3.05 to -0.22 days; P = 0.02), and polymerase chain reaction (PCR) positivity (RR 0.07, 95% CI 0.01-0.34; P = 0.001).

Conclusions: Ozone therapy significantly reduced mortality, PCR positivity, and length of stay in hospitalized patients with COVID-19. Ozone therapy should be considered for COVID-19 patients.

https://pubmed.ncbi.nlm.nih.gov/33169118/

No abstract available, but go here for the commentary:  https://www.cell.com/the-innovation/fulltext/S2666-6758(20)30063 1returnURL=https%3A%2F%2Flinkinghub.elsevier.com%2Fretrieve%2Fpii%2FS2666675820300631%3Fshowall%3Dtrue
All patients also were given antiviral agents and three were also treated with steroids.

Category:

research, Treatment, Viruses

Remdesivir: What You Should Know

Dr. Vernon Coleman: Remdesivir – What You Must Know

“Please share this article with everyone you know. And please send or show copies of this article to every doctor and nurse you can reach. “

Lioness of Judah Ministry
Aug 29, 2024

By Dr. Vernon Coleman

Remdesivir is described as a `broad spectrum antiviral drug’. It is a RNA polymerase inhibitor which disrupts the production of vital RNA. It is said to prevent the multiplication of SARS-CoV-2.

Remdesivir was introduced for the treatment of covid-19 patients who were in hospital suffering from covid-19, with or without pneumonia. It is still being widely used. I have been researching and writing about drugs since 1970 and I am appalled at the way that it now appears that in some countries some hospitals and doctors (and even nurses) are now routinely giving remdesivir to patients – particularly elderly patients – who do not have severe signs and/or symptoms of the flu or a flu-like viral infection. You can form your own opinion on whether remdesivir ever has a value by reading the following information.

1. Remdesivir is officially used to treat patients who have symptoms of covid-19 or who have covid-19 according to the discredited PCR test which no one with any functioning brain tissue should use. Anyone who uses a PCR test to diagnose covid-19 is a moron and you can tell them I said that. Please see my two recent articles (on http://www.vernoncoleman.com) entitled `PCR: How the PCR test has killed millions’ and `The PCR test can kill you’. A positive PCR test does NOT prove that you have covid-19, dandruff, chilblains, covid-19 or anything else.

2. Remdesivir seems to be very, very popular with very, very stupid doctors who seem to think it is a panacea for all illnesses. If their Mercedes or BMW breaks down they probably give the car a shot of remdesivir.

3. Remdesivir is given directly into a vein. Doctors who tell you that giving drugs via a vein is an entirely safe procedure are stupid. No medical procedure is entirely safe. Giving drugs by injection into a blood stream requires skill and experience to avoid dangers.

4. The brand name of remdesivir is Veklury. (Brand names always begin with an initial capital but generic names are all lower case.) If you are being given Veklury, you are being given remdesivir.

5. Remdesivir should be prescribed by a doctor and given under a doctor’s supervision. (Nurses may wear stethoscopes round their necks, but they are not doctors.)

6. Remdesivir must be given slowly over a period of between 30 minutes and 120 minutes.

7. Hospital patients are usually given remdesivir once a day for up to 10 days.

8. Patients not in hospital are usually given remdesivir once a day for three days.

9. Patients who are given remdesivir MUST have regular blood tests to check that their livers are functioning properly. If a doctor gives remdesivir without doing regular liver function tests he or she is dangerous and, in my opinion, should have their medical licence revoked.

10. Liver function tests MUST be done before remdesivir is prescribed. Any doctor who does not do liver tests before starting treatment should be sacked and have their medical licence revoked.

11. Severe renal toxicity has been noted in animal studies. (Some doctors claim that animal studies are irrelevant. I agree. But why do them if they are irrelevant?)

12. No one should be given remdesivir if they are allergic to it.

13. Anyone who has ever had liver disease or kidney disease should inform their doctor if he/she suggests prescribing remdesivir.

14. Anyone who is pregnant or breastfeeding should tell their doctor. The UK’s National Institute for Health and Care Excellence (NICE) says that the safety of covid-19 antiviral treatment during pregnancy has NOT been established.

15. Remdesivir may interact, to your disadvantage, with other prescription medicines, with over the counter medicines, with vitamins and with herbal products. Doctors who prescribe remdesivir must ask patients about all the medicines they are taking.

16. Remdesivir has received a number of reviews on drugs.com, and of the reviews listed on 24th August 2024, 19.38% or reviewers had a `positive experience’ but 47% had a `negative experience’.

17. According to the journal `Science’, in October 2020,`The World Health Organisation’s Solidarity Trial showed that remdesivir does not reduce mortality or the time covid-19 patients take to recover.’ And `A second, smaller placebo-controlled study of remdesivir on hospitalised covid-19 patients in China, published online by The Lancet on 29th April 2020, found no statistically significant benefit from the treatment – and the antiviral surprisingly had no impact on levels of the coronavirus’. I find it difficult to see why the FDA, the EU and the UK’s drug regulator all approved remdesivir, though they appear to have done so without worrying too much about the research showing that it was pretty useless.

18. Side effects which may occur when remdesivir is injected include: fast, pounding heartbeats; trouble in breathing; wheezing, shivering, itching, sweating, facial swelling, severe headache, a feeling of being about to pass out. Side effects subsequently may include nausea and abnormal liver function tests.

19. NICE reports that there are twelve drugs with which remdesivir inter-reacts. Any doctor prescribing remdesivir should know of these interactions – which are listed on the NICE website. So, for example, the manufacturers advise that patients avoid taking remdesivir with chlorquine, hydroxychloroquine and phenytoin. I cannot put a link to the NICE website because such links are not allowed. The list of side effects below was NOT taken from the NICE website. (Since it is a public sector body and paid for by taxpayers, you’d think that NICE would be delighted to share information about drug dangers wouldn’t you?)

20. Side effects which can occur in patients taking remdesivir may include:

Back pain
Bleeding
Blistering
Burning
Chest tightness
Chills
Coldness
Cough
Dark coloured urine (a possible sign of liver problems)
Difficulty in swallowing
Discolouration of skin
Fast heartbeat
Feeling of pressure
Fever

Flushing
Headache
Hives and itching
Infection
Inflammation
Light coloured stools (a possible sign of liver problems)
Lumps
Nausea and vomiting
Numbness
Pain
Puffiness or swelling of the eyelids or around the eyes, face, lips or tongue
Redness

Scarring
Seizures
Skin rash
Soreness
Stinging
Stomach pain, continuing
Swelling
Tenderness
Tingling
Trouble breathing
Ulceration
Unusual tiredness or weakness
Yellow eyes or skin (a possible sign of liver problems)

You will be relieved to know that not all patients would be expected to have all of these side effects, though a number of these side effects are classified as `common’.

Please share this article with everyone you know. And please send or show copies of this article to every doctor and nurse you can reach. Send copies to your GP and your local hospital.

Unlike NICE I like to share the information I obtain, and unlike YouTube and the mainstream media I believe that the truth should not be censored and must be shared as widely as possible. I suspect that this article will be suppressed and hidden by Google et al. And I am banned from all media (mainstream and online) so please help share this article.

Copyright Vernon Coleman August 2024

NOTE
My first two books `The Medicine Men’ and `Paper Doctors’ dealt with dishonesty and corruption in medicine. `The Medicine Men’ dealt with the relationship between doctors and the drug industry. `Paper Doctors’ dealt with medical research. Both were published in the 1970s and attracted much praise at the time (though not from the pharmaceutical industry or the medical establishment). You can purchase them both from the bookshop on www.vernoncoleman.com

The Defender just came out with two articles on how ‘huge’ financial incentives led hospitals to tragically use COVID treatments that killed patients, which included remdesivir and ventilators.

For more:

Category:

Treatment, Viruses

EBV & Lyme: A Patient’s Perspective

https://www.globallymealliance.org/blog/epstein-barr-virus-and-lyme-disease?

Epstein-Barr Virus and Lyme Disease: A Patient’s Perspective

by Jennifer Crystal on August 7, 2024

<span id="hs_cos_wrapper_name" class="hs_cos_wrapper hs_cos_wrapper_meta_field hs_cos_wrapper_type_text" style="" data-hs-cos-general-type="meta_field" data-hs-cos-type="text" >Epstein-Barr Virus and Lyme Disease: A Patient's Perspective</span>
Understand the intricate relationship between Epstein-Barr Virus (EBV) and Lyme Disease through the experience of Jennifer Crystal. Learn how they coexist and impact each other in chronic illness.

Whenever I write about my chronic tick-borne illnesses Lyme disease and babesiosis, I always add “as well as chronic active Epstein-Barr virus (EBV).” This is because for me—and for many Lyme disease patients—EBV and Lyme go hand in hand. Though one is a virus and one is a bacterial infection, the two wreak havoc on the body together, and a flare of one can lead to a flare of another.

I know this now, two decades into my chronic illness journey. When I got mononucleosis, which is caused by the Epstein-Barr virus, in 2003, I had no idea that I also had underlying tick-borne infections or that the two could impact each other. Since 1997, I’d had all sorts of strange symptoms, ranging from an on-and-off-again flu to migraine headaches, hypoglycemia, asthma, trembling hands, and hives. Each of these complaints had been treated individually—often brushed aside as stress or “just feeling run down”—and tick-borne illnesses were never considered. Ironically, every time the flu-like symptoms came back, I would worry that I had mono. I knew that virus could take a while to get over, and I didn’t have time to be sick. But the tests were always negative.

Until, days before I was to start a job as a Head Counselor at a summer camp in Maine, a sore throat and mounting fatigue led to a mono test that actually did come back positive. Now I really didn’t have time to be sick. I had just finished a strenuous year teaching high school English and Journalism in Colorado, had driven cross country by myself, and was supposed to spend only a few days with family in Connecticut before heading off to Maine.

The universe had other plans, but I didn’t agree with them. I knew that some people got over mono in a few weeks, while some cases took longer. I took a steroid to alleviate symptoms and speed up recovery. I rested in Connecticut for two weeks. And then, with my doctor’s permission, I set off for camp, where I so desperately wanted to be.

Camp needed me, too, as the oldest returning Head Counselor. But my being there that summer wasn’t fair to anyone—not to the campers, not to the directors, not to me. Camp needed me to give my all. I could for a short while, but then the fatigue of mono came raring back, and I found myself dragging my body to activities.

By the end of the summer, I was bedridden. By fall, I was no better. I slept twenty hours a day and developed new symptoms like burning extremities and hallucinogenic nightmares. Tests for mono remained positive, and tests for Epstein-Barr titers remained high. My doctor explained to me that people who have had mono will always test positive for Epstein-Barr virus, but that once the mono goes away, the Epstein-Barr virus titers should decrease (the same way those of us who had chicken pox as kids always carry that virus even though it’s not active). In my case, the titers remained high, which is known as chronic active Epstein-Barr virus, or chronic mono. The doctor told me it might take a couple years for me to get better.

I was devastated. I was supposed to work as a ski instructor in the Rocky Mountains that winter. That dream was shattered. Unable to support or care for myself, I had to surrender my independence and move back with my parents in Connecticut.

I blamed myself for the chronic active Epstein-Barr virus because I’d pushed my body to go to camp. While that choice probably didn’t help my recovery, there were much larger, physiological explanations for my immune system’s inability to fight off mono: Lyme disease, babesiosis, and ehrlichiosis, which my body had been harboring since an unknown tick bite while at camp in 1997.

For the first two years that I had chronic active EBV, I spent so much energy trying to convince people that I really was sick, not lazy; that EBV was a legitimate diagnosis; and that I wanted to be working, socializing, and exercising as I once had, but could barely move from bed—that I didn’t consider whether anything else might be going on in addition to EBV, rather than instead of it.

One day in 2005 while drying my hair, I noticed circular rashes on my elbows when I saw my raised arms in the mirror. I’d become so accustomed to writing symptoms off as nothing, but the naturopathic physician who’d been treating me for EBV said, “Those look suspiciously like the bullseye rashes of Lyme disease,” which he explained can show up months or years after a tick bite, in various places on the body. He said, “I’ve started to suspect you have something additional going on, like an underlying infection, because otherwise you would be getting better.” He sent me to a Lyme Literate Medical Doctor (LLMD) who ran tests that showed I indeed had three tick-borne diseases. The doctor traced these illnesses back to a red rash I’d had on my arm in 1997, just before all my symptoms began. He concluded that the tick-borne diseases had come out in full force when I got mono, because my immune system was so overtaxed that it couldn’t fight all the illnesses; instead, it was walloped by them.

Once I was adequately treated for tick-borne illness, my EBV symptoms quieted, but they still flare from time to time. Because Lyme and EBV symptoms can be so similar (flu-like symptoms, fatigue, achiness, low grade fever), sometimes it’s hard for me to tell which one is activated. Having spent so long in the throes of tick-borne disease treatment and recovery, I sometimes forget that I have EBV, too. But when symptoms flare, it’s important for me to take a step back and ask, is this Lyme, or could this be EBV? Sometimes it’s both and a slight change in a supplement can help either. Sometimes it’s EBV and I need to rest. Sometimes it’s babesiosis and I need a specific anti-malarial medication.

Scientists don’t yet know why there seems to be such a correlation between Lyme and EBV, though they have discovered EBV may be a leading cause of Multiple Sclerosis[i], a Lyme imitator. We know from emerging science and from patient experience that EBV seems to impact certain autoimmune disorders and infections, and vice-versa. Until the science catches up to the experience, the important thing is for patients with overlapping symptoms to keep both EBV and other medical conditions like Lyme disease in mind. If they’ve only been diagnosed with one, they may only be fighting half the battle.

[i] https://www.nih.gov/news-events/nih-research-matters/study-suggests-epstein-barr-virus-may-cause-multiple-sclerosis

Writer

Jennifer Crystal

Opinions expressed by contributors are their own. Jennifer Crystal is a writer and educator in Boston. Her work has appeared in local and national publications including Harvard Health Publishing and The Boston Globe. As a GLA columnist for over six years, her work on GLA.org has received mention in publications such as The New Yorker, weatherchannel.com, CQ Researcher, and ProHealth.com. Jennifer is a patient advocate who has dealt with chronic illness, including Lyme and other tick-borne infections. Her memoir, One Tick Stopped the Clock, is forthcoming from Legacy Book Press in September 2024. Ten percent of proceeds from the book will go to Global Lyme Alliance. Contact her via email below.

Email: lymewarriorjennifercrystal@gmail.com

Category:

Lyme, Viruses

Boy Gets Powassan Encephalitis After Camping Trip

https://danielcameronmd.com/powassan-encephalitis-camping-trip/

Young boy develops Powassan encephalitis after camping trip

powassan-encephalitis

Although the Powassan virus is considered to be a rare tick-borne illness, the number of cases is rising, and at an alarming rate. A recent survey found, a 4-fold rise in the number of Powassan virus cases in the US from 2014 to 2023 (compared with 2004 to 2013). [3]

By 

This summer, a 9-year-old boy in Canada developed Powassan virus encephalitis, a life-threatening condition. In Pennsylvania, another young child was hospitalized with the virus. And, in April, an older man from Massachusetts was infected. Meanwhile, last year, a Maryland resident died from Powassan encephalitis after contracting it in Canada.

Powassan virus – transmitted in 15 minutes

The Powassan virus, which is transmitted through the bite of an infected blacklegged tick, can be deadly. And, most concerning, as cases are rising, the infection can be contracted within 15 minutes of a tick attachment.

The virus can cause fever, headache, vomiting, loss of coordination and memory and speech problems. It can also cause encephalitis (inflammation of the brain) and meningitis (inflammation of the membranes surrounding the brain and spinal cord.)

However, it often does not present with any symptoms, according to the CDC.

POWV encephalitis can be deadly

From 2004 to 2022, the US reported 288 cases of Powassan virus infection. Of these cases, 72 (25%) occurred in children, 264 patients (92%) required hospitalization, and 36 patients (13%) died.1

Patients infected with the virus have a “10% risk of developing fatal encephalitis and up to 50% of infected patients have long-term neurologic damage.”2

Up to 50% of patients have long-term neurologic complications.

The prognosis for individuals with Powassan virus neuroinvasive disease is poor. The case fatality rate is 10%–15%, and survivors have about a 50% probability of persistent neurologic deficits, including headaches, altered mental status, and cognitive difficulties.1

There is no treatment for the Powassan virus.

Case Report: 9-year-old boy

In July, a 9-year-old boy, residing in Canada, was hospitalized with Powassan encephalitis, after returning from a camping trip in northern Ontario.

Blatman and colleagues describe the case in their article, “Powassan virus encephalitis in a 9-year-old.”1

The young boy was admitted to the hospital with a fever, neck stiffness and headache, which began 1 week after returning from his camping trip.

Initially, he was treated with ceftriaxone and vancomycin for suspected meningitis.

PCR testing of the CSF for viral causes of meningitis or encephalitis, however, was negative. Bacterial culture and Gram stain of the CSF sample was also negative. And, an MRI of the brain was unremarkable.

The patient had no known tick bites or rashes.

“Over the next 48 hours, blood cultures showed no growth,” the authors state. However, “The patient remained persistently febrile with ongoing severe headache.”

After 3 days in the hospital, the boy’s condition worsened and he was transferred to the ICU. At this point, he was nonverbal and nonresponsive to commands, according to the authors.

Testing for Lyme disease was negative.

“Tick-borne Powassan virus encephalitis is associated with high mortality and a risk of long-term neurologic sequelae in survivors.”

Repeat EEG showed generalized slowing of brain activity. Meanwhile, a repeat MRI of the patient’s brain and full spine showed subtle bilateral basal ganglia and substantia nigra.

However, CFS testing was negative for autoimmune encephalitis.

“Given concern for potential autoimmune encephalitis, the patient received intravenous immunoglobulin at a dosage of 1 g/ kg for 2 days, with notable improvement in his level of consciousness within 24–48 hour,” the authors state.

Within 2 months, the boy had made a complete recovery.

New research indicates that the Powassan virus may be more deadly in older patients. “… only minimal infectious doses of the virus were highly lethal in older mice and that lethality increased >10-fold with age,” states Mackow.2

“Increased awareness of Powassan virus among clinicians in Canada will likely lead to increased identification of Powassan virus and other arthropod-borne infections, which should always be reported,” the authors state.

Related Articles:4 cases of Powassan virus encephalitis
Powassan virus encephalitis contracted during winter months
Can Powassan virus cause encephalitis or other neurologic damage?
References:
  1. CMAJ 2024 August 26;196:E973-6. doi: 10.1503/cmaj.240227
  2. Megan C. Mladinich et al, Age-dependent Powassan virus lethality is linked to glial cell activation and divergent neuroinflammatory cytokine responses in a murine model, Journal of Virology (2024). DOI: 10.1128/jvi.00560-24
  3. Passive surveillance of Powassan virus in human-biting ticks and health outcomes of associated bite victims. Siegel, Eric et al. Clinical Microbiology and Infection, Volume 30, Issue 10, 1332 – 1334

________________

**Comment**

But like all things Lyme/MSIDS, Powassan can persist in humans as well as mice.

Powassan is NOT rare:

http://www.coppelabs.com/blog/why-is-powassan-virus-infection-still-described-as-rare-and-mysterious/  Excerpt:  

For the last two years, Coppe Laboratories has dedicated a significant amount of time and resources to dispelling the myth that infection with Powassan virus, a virus transmitted by tick bite, is rare. The Centers for Disease Prevention and Control (CDC) reports only 100 cases of Powassan virus infection in the United States in the last 10 years. Indeed, that statistic gives the illusion that Powassan infection is rare. However, did you know that the only infections reported to CDC are those that are life-threatening, particularly cases causing severe inflammation of the brain like the case reported in LiveScience?

Coppe has published three new papers in the last year that clearly show Powassan virus infection is not rare are at all, and until testing for this virus is included as part of tick-borne disease screening panels infections will continue to be underreported. Coppe’s Powassan Guide, which can be downloaded from the website, summarizes the findings from both tick and human Powassan prevalence studies, as well as defining the patient populations that would benefit most from Powassan testing.

Coppe Labs, a specialized CLIA-certified lab, right in Waukesha, Wisconsin tests for Powassan, West Nile, Anaplasma, Babesia, Human Herpes Viruses 6 & 7, COVID, and Lyme disease.

Wisconsin is a hot-spot for Powassan.

For more:

Regarding IVIG, it has been used successfully in many Lyme/MSIDS patients including this young autistic boy infected with borrelia, babesia, and bartonella.  After a rough week of treatment suddenly this boy could remember things, he became happy, social, and agreeable with fewer tics.  Disulfiram also played a key role as well as targeting bartonella.  IVIG has also been used in PANS and PANDAS with some success.

Susannah Cahalan, who wrote the book Brain on Fire: My Month of Madness, was given steroids, plasmapherisis, and IVIG for autoimmune encephalitis.

Dr. Frid uses IVIG for treating infections induced autoimmune encephalitis.

https://madisonarealymesupportgroup.com/2017/10/01/panspandas-steroids-autoimmune-disease-lymemsids-the-need-for-medical-collaboration/   Boy’s Lyme Disease Morphs into Autoimmune encephalopathy. It took 10 years and 20 doctors to find out 12-year-old Patrik had Lyme disease. Just 4 months later the doctors discovered he also has a condition where his immune system attacks his brain.

Category:

research, Ticks, Transmission, Treatment, Viruses