Archive for the ‘research’ Category

Actor Dennis Quaid Joins Secretary Kennedy to Discuss Lyme Disease

Kennedy and Quaid do not discuss treatments for Lyme. The title below is misleading. What they do discuss is that HHS is supposedly throwing a lot of money at research which up until now has done ZERO to help sick patients. I don’t expect this latest PR stunt to do much either. I pray I’m wrong. Go here for the reasons why.

https://abc6onyourside.com/news/nation-world/actor-dennis-quaid-joins-secretary-kennedy-to-discuss-treatments-for-lyme-disease-ai-hhs

Actor Dennis Quaid joins Secretary Kennedy to discuss treatments for Lyme disease


by CHARLOTTE HAZARD | The National News Desk Mon, August 17, 2026

WASHINGTON (TNND) — Health and Human Services Secretary Robert F. Kennedy Jr. and actor Dennis Quaid teamed up Monday to make an announcement about furthering research about Lyme disease.

“At HHS, we are taking bold new actions against Lyme disease, alpha-gal syndrome, and other tick-borne diseases—accelerating research, improving care, and driving new treatments,” Kennedy wrote on the social media platform, X. “Millions of Americans need faster diagnoses, better care, and real answers.”
https://platform.twitter.com/embed/Tweet.html?dnt=false&embedId=twitter-widget-0&features=eyJ0ZndfdGltZWxpbmVfbGlzdCI6eyJidWNrZXQiOltdLCJ2ZXJzaW9uIjpudWxsfSwidGZ3X2ZvbGxvd2VyX2NvdW50X3N1bnNldCI6eyJidWNrZXQiOnRydWUsInZlcnNpb24iOm51bGx9LCJ0ZndfdHdlZXRfZWRpdF9iYWNrZW5kIjp7ImJ1Y2tldCI6Im9uIiwidmVyc2lvbiI6bnVsbH0sInRmd19yZWZzcmNfc2Vzc2lvbiI6eyJidWNrZXQiOiJvbiIsInZlcnNpb24iOm51bGx9LCJ0ZndfZm9zbnJfc29mdF9pbnRlcnZlbnRpb25zX2VuYWJsZWQiOnsiYnVja2V0Ijoib24iLCJ2ZXJzaW9uIjpudWxsfSwidGZ3X21peGVkX21lZGlhXzE1ODk3Ijp7ImJ1Y2tldCI6InRyZWF0bWVudCIsInZlcnNpb24iOm51bGx9LCJ0ZndfZXhwZXJpbWVudHNfY29va2llX2V4cGlyYXRpb24iOnsiYnVja2V0IjoxMjA5NjAwLCJ2ZXJzaW9uIjpudWxsfSwidGZ3X3Nob3dfYmlyZHdhdGNoX3Bpdm90c19lbmFibGVkIjp7ImJ1Y2tldCI6Im9uIiwidmVyc2lvbiI6bnVsbH0sInRmd19kdXBsaWNhdGVfc2NyaWJlc190b19zZXR0aW5ncyI6eyJidWNrZXQiOiJvbiIsInZlcnNpb24iOm51bGx9LCJ0ZndfdXNlX3Byb2ZpbGVfaW1hZ2Vfc2hhcGVfZW5hYmxlZCI6eyJidWNrZXQiOiJvbiIsInZlcnNpb24iOm51bGx9LCJ0ZndfdmlkZW9faGxzX2R5bmFtaWNfbWFuaWZlc3RzXzE1MDgyIjp7ImJ1Y2tldCI6InRydWVfYml0cmF0ZSIsInZlcnNpb24iOm51bGx9LCJ0ZndfbGVnYWN5X3RpbWVsaW5lX3N1bnNldCI6eyJidWNrZXQiOnRydWUsInZlcnNpb24iOm51bGx9LCJ0ZndfdHdlZXRfZWRpdF9mcm9udGVuZCI6eyJidWNrZXQiOiJvbiIsInZlcnNpb24iOm51bGx9fQ%3D%3D&frame=false&hideCard=false&hideThread=true&id=2089341520142860328&lang=en&origin=https%3A%2F%2Fthenationaldesk.com%2Fnews%2Famericas-news-now%2Factor-dennis-quaid-joins-secretary-kennedy-to-discuss-treatments-for-lyme-disease-ai-hhs&sessionId=51f13d675c0a47329476626caf9665dc0679d3ef&theme=light&widgetsVersion=6a3ad42b224df%3A1778106238597&width=550px

In a video attached to the post, Quaid said that his friend, the late Kris Kristofferson spent years living with Alzheimer’s.

“But Alzheimer’s wasn’t the problem,” Quaid said. “He actually had Lyme disease and when the doctors finally diagnosed him correctly in 2016 and started the right treatment, his symptoms did improve.”

Kennedy said that HHS is investing millions of dollars in innovation to develop AI-powered tools to help improve patient care. (See link for article and videos)

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**Comment**

This would be a tad more believable if it didn’t include a Hollywood actor, but here we are.

I don’t trust AI powered tools as far as I can throw them. Throwing millions of dollars into research won’t do a thing either as long as that research continues to skirt truth of persistent/relapsing infection, pleomorphism (shape shifting organism), and polymicrobialism (the involvement of other organisms).

The physician locator on the HHS website takes you directly to the ILADS (International Lyme and Associated Diseases Syndrome) website. Further, when I typed in Wisconsin, a bunch of doctors popped up I do not know at all, which is not a good sign. They might have taken a ‘fundamentals’ course but that does not make them experienced in treating this beast.

Further, this is a pretty tough thing to treat via telemedicine, which many of the doctors on the list offer.

For more:

Another Take on Dapsone Protocols: Dr. Marty Ross

Dapsone for Lyme Persisters. A Miracle Antibiotic?

https://treatlyme.com/guide/dapsone-lyme-persisters/

Updated: July 31, 2026

Horowitz Dapsone Protocols: Risks vs Benefits Explained

About Dapsone & Persisters

Dapsone, a Leprosy drug, can help some with treatment-resistant Lyme and Bartonella due to persisters plus treatment resistant Babesia. In 2016, Richard Horowitz, MD, and Phyllis Freeman, PhD, published research on 100 patients. The study showed 59 percent of people had improvements with dapsone of 100 mg or less.

More recently, Dr. Horowitz has continued refining a higher-dose approach, publishing a more detailed double-dose/high-dose protocol in 2023.

However, my experience with dapsone is mixed. While it helps some, it is also a very difficult medicine for many to take.

In this article, I review:

  • my experience with this novel medicine,
  • a specific antibiotic protocol that includes dapsone as a normal-dose regimen,
  • how to decrease side effects and Herxheimer reactions on dapsone,
  • the risks of using dapsone,
  • the chances for treatment success using a dapsone regimen, and
  • a detailed look at, and critique of, Dr. Horowitz’s newer double-dose/high-dose protocol and why I don’t support it.

(See link for video and article)

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**Comment**

I always appreciate doctors who share their clinical experience. It’s sometimes the only way we can gather intel on whether or not we should try a certain treatment. Case in point – I wrote about my severe psychosis after taking disulfiram, followed by another practitioner’s experiences in ameliorating symptoms as well as an update on a few points of consideration.

So, while Dapsone may be a perfect fit for some, it may not for others.

This is another point that never makes the news. Reactions to treatment varies widely in Lymeland, probably because some have been infected for years and their bodies are simply overwhelmed, making another drug appear to be a toxin by the body.

Dr. Ross points out the following issues:

  • You shouldn’t take Dapsone if your G6PD levels are low.
  • Dapsone blocks folate metabolism which can lead to anemia
  • Dapsone suppressed bone marrow which can also lead to anemia
  • Dapsone is a harsh drug that causes severe herxes, allergic rash, methemoglobinemia, and many side effects

The article contains Horowitz’s 2016 Dapsone regimen. Ross states he does not recommend the Dought or high dose Dapsone protocol and doesn’t offer it in his practice. He also points out that Horowitz’s 2023 paper which offered HDDCT to 50 patients, but only 25 are considered in the results leaving one to ask how many dropped out due to side effects. Further, Horowitz is the treating physician, the sole study author, and one of three others from his practice who performed the validation and analysis. There was no control group, no blinding, no independent review, and no oversight.

Ross does feel; however, that some sho failed other treatments could consider the normal-dose dapsone 100mg protocol if carefully monitored.

For more:

The Hidden Drivers of Inflammatory Bowel Disease (Lyme Disease is One)

https://imahealth.substack.com/p/the-hidden-drivers-of-inflammatory? Video Here

The Hidden Drivers of Inflammatory Bowel Disease

Crohn’s and colitis are called genetic, autoimmune, and idiopathic. What if all three labels are wrong? A new paper tests each against current evidence.

Independent Medical Alliance

Aug 02, 2026

Host: Dr. JP Saleeby | Guest: Josh Dech

What if Crohn’s disease and ulcerative colitis are caused by more than genetics alone?

Dr. Yusuf “JP” Saleeby, IMA Senior Fellow in Functional and Integrative Medicine, and gut health specialist Josh Dech take a closer look at what may contribute to inflammatory bowel disease, also known as IBD. The two recently co-authored a new paper published in the Journal of Independent Medicine. Their conversation traces how genetics, diet, gut health, and the environment may work together to shape both diseases.

Inflammatory bowel disease affects more than 7 million people worldwide and ranks among the fastest-growing chronic diseases globally. Nearly everyone diagnosed with Crohn’s disease or ulcerative colitis hears some version of the same three things: the disease is genetic, the immune system is attacking its own tissue, and the underlying cause is unknown. Those three explanations leave two treatments on the table, drugs and surgery, and they leave a patient nothing to investigate.

A new paper in the Journal of Independent Medicine argues that all three explanations fail against current evidence. Josh Dech and Dr. JP Saleeby, its co-authors, point out that each has been contested separately in the literature for two decades without anyone testing them as a set. Taken together, they conclude, the conventional model does not hold.

What replaces it is a disease that is partially heritable, environmentally activated, and immune-mediated, and the distinction is not academic for anyone living with one. If exposures determine whether susceptibility becomes disease, exposures can be found and changed. (See link for article, research paper and video)

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SUMMARY:

  • The authors found that genetics only explain about a quarter of disease risk for irritable bowel (IBD).
  • The authors argue that the autoimmune label, despite holding for decades, is based on the weakest evidence and that pathogenic transfer has never been demonstrated.
  • The serologic markers long cited as evidence for autoimmunity turn out to recognize microbial and fungal targets but are not autoantibodies in the classical sense.
  • A review of 53 meta-analyses across 71 risk factors shows the following exposures for IBD that are quantified and modifiable:
    • antibiotic exposure
    • oral contraceptives
    • breast-feeding was protective for Crohn’s and colitis
    • ultra-processed food
    • air pollution
    • psychological stress
    • mold
    • mycotoxins
  • The authors state current treatments complement but ignore the environmental exposures.
  • The authors give the following issues that should be questioned alongside a standard work-up:
    • Birth mode and feeding history
    • Early antibiotic courses, particularly before age 5
    • Water damage & mold exposure at home, work and in vehicles.
    • Adolescent diet, stressors, and infections

At this point in the article, they described a case report on a 14 year old whose Crohn’s progressed far enough that surgeons planned to remove most of his intestines & place a colostomy. Testing pointed to chronic Lyme disease and three months into treatment a repeated scope test found no lesions.

In short, they conclude the following answers for IBS: antibiotic stewardship, breastfeeding support, reducing ultra-processed food, and remediating indoor mold.

After reading the comments after the article, I would be remiss if I did not mention the ‘vaccine’ issue due to the fact they all introduce foreign substances the body recognizes as foe, priming it for later potential problems such as life-threatening allergies to many things including food, which many are also linking to Alpha Gal Syndrome (AGS), an allergy to animal products supposedly caused by ticks – with no solid proof, as well as the fact some get AGS without any known tick involvement. So while ticks play a part, they are obviously not the only ingredient required to get AGS.

Pathogenic priming was shown clearly with the COVID gene therapy injections.

For more:

Study Uncovers Hidden Bartonella and Babesia Infections in ME/CFS Patients

Four years ago an article was posted asking if a chronic infection could be behind ME/CFS patients. The answer appears to be yes. A previous study found a link between Cytomegalovirus, EBS, and Human Herpesvirus-6 and ME/CFS. The following also connects Babesia and Bartonella to the condition.

https://www.lymedisease.org/bartonella-babesia-me-cfs/

Study uncovers hidden Bartonella and Babesia infections in ME/CFS patients

A new pilot study from North Carolina State University has found molecular evidence of Bartonella or Babesia infection in nearly half of 50 people diagnosed with myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS).

The findings suggest that vector‑borne pathogens may play a larger role in chronic illness than previously recognized.

Bartonella and Babesia are both transmitted primarily through arthropods—ticks, fleas, and lice—and have been linked to a range of persistent symptoms.

Improved testing has revealed that Bartonella, once thought to cause only short‑lived infections like cat scratch disease, can be associated with chronic and even neuropsychiatric symptoms. Babesia, best known as a tick‑borne parasite, has also been transmitted through blood transfusions and organ transplants.

For this study, researchers selected 50 participants from a larger group of chronically ill individuals with long‑term fatigue and neurological symptoms such as memory problems, tremors, disorientation, or anxiety.

Using quantitative PCR and DNA sequencing, the team detected:

  • Babesia in 10 participants
  • Bartonella in 11
  • Both pathogens in 2

That’s 23 out of 50 participants showing evidence of infection.

“ME/CFS diagnoses are primarily based on immunological biomarkers that can have numerous influences,” said study author Edward Breitschwerdt, Melanie S. Steele Distinguished Professor of Internal Medicine at NC State.

“Our goal was to detect the DNA of specific pathogenic microorganisms that might contribute to or cause a patient’s chronic illness.”

Breitschwerdt emphasized that the small sample size means the results can’t be generalized to all ME/CFS patients, but the unexpectedly high prevalence highlights the need for further research.

The study, supported in part by the Steven & Alexandra Cohen Foundation, appears in Pathogens.

SOURCE: North Carolina State University

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For more:

Self-Reported Observations of Unusual White Fibrous Structures in Embalmed Corpses: Multi-Year Survey Results

https://ijirms.in/index.php/ijirms/article/view/2201

Self-Reported Observations of Unusual White Fibrous Structures in Embalmed Corpses: Multi-Year Survey Results from Embalmers in Five Countries, 2022–2025

Thomas F. Haviland*·Laura Kasner·Daniel SantiagoiD

DOI:10.23958/ijirms/vol11-i07/2201· Pages: 204 – 208· Vol. 11, No. 07, (2026)· Published: July 1, 2026

PDFCitationShare

Views: 22,752 PDF downloads: 6,044

Abstract

Background: Beginning in 2020–2021, embalmers in multiple countries reported observing large, tough, rubbery white or off-white fibrous structures in the veins and arteries of embalmed corpses, which they described as distinct from classic postmortem clots.

Methods: We conducted four annual cross-sectional surveys (2022–2025) of active embalmers in the United States, Canada, United Kingdom, Australia, and New Zealand using SurveyMonkey. A dual distribution strategy (professional associations and direct emails to funeral homes) was used. Core questions assessed observation of unusual white fibrous structures and estimated percentage of corpses affected.

Results: Across 808 total responses, the proportion of embalmers reporting observation of these structures ranged from 66% to 83%. Weighted average prevalence in affected corpses ranged from 19% to 27%. The 2022 survey showed a marked increase in first observations beginning in 2020 and accelerating in 2021.

Conclusions: Multiple years of surveys document consistent self-reported observations by experienced embalmers of unusual white fibrous structures in a substantial fraction of corpses, with a clear increase noted around 2020–2021. These findings constitute a potential safety signal that warrants independent investigation by forensic pathologists and biomedical researchers to characterize the structures and determine their etiology.

(Click on top link for full article and pictures)

New White Fibrous Clots Publication

Dr. John Campbell

July 23, 2026

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**Comment**

For the ‘science’ skeptics out there, this is what research looks like when no ‘external’ funding or grants are received. Hence the use of SurveyMonkey.

Researchers know that when they study a topic that doesn’t fit the narrative, they are entirely on their own. No large NIH grants should be expected because our government is completely in bed with Big Pharma, who literally couldn’t care less if you live or die.

Lymeland has been in this lovely parallel universe for nigh on 40 years. Any research that moves the needle forward at all is independently funded and since research has become so incredibly expensive, cheap tools like SurveyMonkey are utilized.

In the case of Lyme/MSIDS, it is often called “Big Data,’ and relies on patient surveys. If you haven’t done it already, make sure to fill out the MyLymeData Project which allows patients to pool their health information through a secure website. “Big data” projects use advanced technology to gather and analyze huge amounts of patient data, which can assist researchers in studying disease patterns and answering important questions such as why do some people recover from Lyme disease, while others remain ill?

For more on the clots found in the COVID injected: