Author Archive

Bachelorette’s Ryan Sutter Meets 16-Year-Old ‘Inspiration’ Amid His Own Lyme Disease Battle: She’s a ‘Survivor’

https://www.usmagazine.com/celebrity-news/news/bachelorettes-ryan-sutter-meets-an-inspiration-amid-health-battle/

Bachelorette’s Ryan Sutter Returns to the Gym Amid Mystery Illness Battle: ‘Doing My Best to Get Back to My Best'
Ryan Sutter John Nacion/Startraks

Staying positive! As Ryan Sutter continues to deal with several health issues this year, the Bachelorette alum took to social media to reflect on his journey following a meeting with a Lyme disease survivor.

Stars Who’ve Battled Lyme Disease: Justin Bieber, Bella Hadid, More

“Two things crossed my mind when I saw this picture. The first, how lucky I am to have met Olivia Goodreau. Olivia is the 16 year old founder of @livlymefoundation She is a ten year Lyme survivor, a true inspiration and has done more in her short life so far than most ever do in a lifetime,” Sutter, 46, wrote in a lengthy post via Instagram on Sunday, August 29. “Proud to have accepted a position on the board at LiveLyme.”

The former reality star then reflected on his appearance after having to deal with mystery symptoms for over a year before officially receiving a proper diagnosis.

(See link for article)

26 of 27 Scientists Denying COVID Wuhan Lab Origin Have Direct Ties to Lab

https://www.newstarget.com/2021-09-13-conspiracy-lancet-scientists-denied-covid-theory-wuhan-ties

TRUE CONSPIRACY: More than 96% of Lancet “scientists” who denied covid lab origin theory have direct ties to Wuhan

09/13/2021 / By Ethan Huff
TRUE CONSPIRACY: More than 96% of Lancet “scientists” who denied covid lab origin theory have direct ties to Wuhan

As you may recall from the early days of the plandemic, 27 “scientists” penned a letter published in The Lancet that claimed the Wuhan coronavirus (Covid-19) did not originate in a Wuhan laboratory. Well, it turns out that 26 of these scientists have direct ties to the Chinese lab in question, making their claims entirely untrustworthy.

That letter, which was published on March 7, 2020, “strongly condemned conspiracy theories” related to the Chinese Virus that suggest it did not come about by chance from bat soup at a wet market. Chinese Germs, they insist, are a product of nature, not of genetic tampering.

This declaration was intended to be the end of the conversation, except for the fact that inquiring minds decided to look into the matter further. What they found is that almost all of the scientists in question have major conflicts of interest when it comes to telling the truth.  (See link for article)

__________________

**Comment**

Highlights:

  • One author of the paper, Peter Daszak, president of EcoHealth Alliance, funneled money directly to the WIV to conduct illegal gain-of-function research on coronaviruses.  He declared no conflicts of interest, but The Lancet had to publish an addendum acknowledging his connection.
  • Several more authors hail from Britain’s Wellcome Trust, which also sent money to the WIV to develop genetically modified (GMO) coronaviruses capable of infecting humans.
  • The letter suppressed all inquiry into the true origins of COVID, which is still being debated today.
  • Go to this link if you are interested in learning more about what the investigation uncovered and for each researcher’s ties to the Wuhan lab.

The following quote shows the depths of this heinous conspiracy:

“Conflicts of interest were not reported for any of the other 26 signers of the letter – not even those with obviously material undisclosed conflicts such as EcoHealth employees and Predict contractors,” says Rutgers University molecular biology Prof. Richard Ebright

For more on the COVID bioweapon:

Nobel Prize Winner Dr Luc Montagnier on the origin of COVID-19 states:

  • SARS-CoV-2 appears to be a benign bat coronavirus modified to integrate spike proteins that allows the virus to enter human cells by attaching to ACE-2 receptors
  • The virus also appears to have been modified to integrate an envelope protein from HIV called GP141, which tends to impair the immune system. A third modification appears to involve nanotechnology, which allows the virus to remain airborne longer

Cellular and molecular microbiologist, Dr. Judy Mikoviz also believes COVID-19 is a product of human manipulation:  https://madisonarealymesupportgroup.com/2020/05/04/retroviral-lyme-msids-role-in-covid-19-facemasks-are-immunosuppressive/  She states:

  • COVID-19 — the disease — is not caused by SARS-CoV-2 alone, but rather that it’s the result of a combination of SARS-CoV-2 and XMRVs (human gammaretroviruses)

  • SARS-CoV-2 also appears to have been manipulated to include components of HIV that destroys immune function along

Expert cardiologist warns the spike protein in the COVID shots represent a man-made genetic sequence from a man-made “virus” that causes inflammation, blood clotting, could cause Mad Cow Disease.

FOI Documents Reveal Senior Chinese Scientist Infected With COVID From Lab in Early 2020

https://usrtk.org/biohazards/senior-chinese-scientist-acquired-sars-cov-2-in-lab-infection-accident/

Senior Chinese scientist acquired SARS-CoV-2 in lab infection accident, virologist says

In what may be the first known case of a lab-acquired infection with the novel coronavirus that causes Covid-19, a senior scientist was infected with SARS-CoV-2 in a prestigious laboratory in Beijing in early 2020, according to virologists’ emails obtained by U.S. Right to Know.

The National Institute for Viral Disease Control and Prevention (NIVDC), where the infection is said to have occurred, is a part of the Chinese Center for Disease Control and Prevention. In 2004, a SARS virus outbreak was traced to a lab–acquired infection from the NIVDC.

The revelation that an experienced scientist was infected with SARS-CoV-2 while working in a premier virology lab in Beijing underscores concerns about the health risks posed by biolabs researching pandemic pathogens, and in particular, facilities operated by the Chinese government.  (See link for article)

_____________________

Summary:

  • In an exchange of emails in preparation to refute that COVID came from a lab, a Chinese virologist working at Ohio State University states his former NIVDC director was infected with COVID from the lab.
  • This virologist was also concerned about laboratory COVID infections as it is “much more contagious than SARS-1.”
  • There does not appear to be any public disclosure or reporting of this lab-acquired infection of SARS-CoV-2 from the NIVDC.

Laboratory acquired infections are very real.  In fact, Willy Burgdorfer, the “discoverer” of Lyme, got infected with Lyme in the lab when infected rabbit urine got into his eye. “Bitten: The Secret History of Lyme Disease and Biological Weapons,” also details other incidents, including another form of transmission under Operation Mongoose, where a CIA operative who pushed large boxes of infected ticks out of airplanes infected his baby boy when he returned home.  The baby had a serious inflammatory brain disease with grim prospects and only survived due to a Spanish surnamed resident who recognized the disease and treated it appropriately.  When the CIA operative questioned his commander if the episode had anything to do with his work, he was told to burn all the clothes he took to Cuba.  “Burn everything.”

Regarding COVID, US intelligence has uncovered that Wuhan lab researchers were hospitalized with COVID in 2019.

For more:

Also see: 

COVID-19 mRNA Shots Leading to CNS Inflammation: A Case Series on MS Patients

https://link.springer.com/article/10.1007/s00415-021-10780-7

COVID-19 mRNA vaccination leading to CNS inflammation: a case series

Abstract

The availability of vaccines against the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), provides hope towards mitigation of the coronavirus disease 2019 (COVID-19) pandemic. Vaccine safety and efficacy has not been established in individuals with chronic autoimmune diseases such as multiple sclerosis (MS). Anecdotal reports suggest that the vaccines may be associated with brain, spinal cord, peripheral nervous system, and cardiac inflammation. Based on the high morbidity and unpredictable course of COVID-19, and the need to achieve herd immunity, vaccination has been recommended for patients with MS. We report clinical and MRI features of seven individuals who received the Moderna (n = 3) or Pfizer (n = 4) SARS-CoV-2 mRNA vaccines. Within one to 21 days of either the first (n = 2) or second (n = 5) vaccine dose, these patients developed:

  • neurologic symptoms and MRI findings consistent with active CNS demyelination of the optic nerve, brain, and/or spinal cord.
  • symptoms included visual loss, dysmetria, gait instability, paresthesias, sphincter disturbance, and limb weakness.

Age ranged from 24 to 64 (mean 39.1) years; five were woman (71.4%). The final diagnosis was:

  • exacerbation of known stable MS (n = 4, two were receiving disease-modifying therapy at the time of vaccination),
  • new onset MS (n = 2), or
  • new onset neuromyelitis optica (n = 1)

All responded to corticosteroid (n = 7) or plasma exchange (n = 1) therapy, with five returning to baseline and two approaching baseline. Large prospective studies are required to further investigate any possible relationship between COVID-19 vaccines and acute CNS demyelination.

___________________

**Comment**

Lyme/MSIDS is often misdiagnosed as MS. 

For more on the Lyme/MS connection:

New study:  https://madisonarealymesupportgroup.com/2021/09/15/cellular-blueprint-of-ms-lesions/

For more on the COVID injections, which aren’t vaccines:

Pivotal clinical trial data from the 3 marketed COVID-19 vaccines was reanalyzed using “all cause severe morbidity”, a scientific measure of health, as the primary endpoint. “All cause severe morbidity” in the treatment group and control group was calculated by adding all severe eventsreported in the clinical trials. Severe events included both severe infections with COVID-19 and all other severe adverse events in the treatment arm and control arm respectively. This analysis gives reduction in severe COVID-19 infections the same weight as adverse events of equivalent severity.

Results prove that none of the vaccines provide a health benefit and all pivotal trials show a statically significant increase in “all cause severe morbidity” in the “vaccinated” group compared to the placebo group.

Corrupt public health ‘authorities’, researchers, and a bought out media play games with statistics as pointed out in this astute article:  https://madisonarealymesupportgroup.com/2021/09/01/lies-damned-lies-vaccine-statistics/

These groups are making “acceptable catastrophic errors” – the kind of error one is allowed to make when they are perceived to have the correct opinion.  Arguments and statistics used towards the goal of getting every single person “vaccinated” for COVID are given far less scrutiny and are accepted as true more readily, than any arguments or statistics that might be perceived as counterproductive towards that goal.

Important quote:

When “former Director of the CDC” Dr. Tom and others use the non-infection efficacy numbers to discuss the vaccines, they are, intentionally or not, misleading the public. It is something that should end immediately.

With the exception of infection rates, the efficacy numbers convey no useful information to citizens about their risks once they have been vaccinated. Instead, it may cause the vaccinated to place themselves and others at greater risk if they operate on this misinformation.

When you are finally counting things and dividing things counted which matter, such as how many infected people went on to die in each group, no remnant of the 90% (vaccine efficacy) numbers remain. In the graph above6, there is no information available to suggest the death rate per infection is any different in the vaccinated group compared to the unvaccinated group. You can see why by revisiting the number infected and the number who died in each group.

Cellular Blueprint of MS Lesions

https://www.nih.gov/news-events/news-releases/nih-scientists-build-cellular-blueprint-multiple-sclerosis-lesions

Friday, September 10, 2021

NIH scientists build a cellular blueprint of multiple sclerosis lesions

Study lays the groundwork for potential new therapies for progressive multiple sclerosis.

Chronic lesions with inflamed rims, or “smoldering” plaques, in the brains of people with multiple sclerosis (MS) have been linked to more aggressive and disabling forms of the disease. Using brain tissue from humans, researchers at the National Institutes of Health’s National Institute of Neurological Disorders and Stroke (NINDS) built a detailed cellular map of chronic MS lesions, identifying genes that play a critical role in lesion repair and revealing potential new therapeutic targets for progressive MS. The study was published in Nature.

“We identified a set of cells that appear to be driving some of the chronic inflammation seen in progressive MS,” said Daniel Reich, M.D., Ph.D., senior investigator at NINDS. “These results give us a way to test new therapies that might speed up the brain’s healing process and prevent brain damage that occurs over time.”

Chronic active lesions are characterized by a slow, expanding rim of immune cells called microglia. Microglia normally help protect the brain, but in MS and other neurodegenerative diseases, they can become overactive and secrete toxic molecules that damage nerve cells. Other cells found at the edge of the lesions, such as astrocytes and lymphocytes, may also contribute to ongoing tissue damage. Prior studies suggest that microglia are the main culprits behind lesion expansion, but the exact types of cells found near lesions and their biological mechanisms are elusive.

To better understand MS lesions, Dr. Reich and his colleagues used single-cell RNA sequencing, a powerful technique which enables researchers to catalog gene activity patterns in individual cells, to examine post-mortem brain tissue of five MS patients and three healthy controls. Samples were provided by the Netherlands Brain Bank, Netherlands Institute for Neuroscience, Amsterdam, the Netherlands, and the NINDS Neuroimmunology Clinic.

“Single-cell RNA sequencing technology allows us to do a much deeper dive into the types of cells present in MS lesions,” said Dr. Reich.

By analyzing the gene activity profiles of over 66,000 cells from human brain tissue, researchers created the first comprehensive map of cell types involved in chronic lesions, as well as their gene expression patterns and interactions.

Dr. Reich’s team found a great diversity of cell types in the tissue surrounding chronic active lesions compared to normal tissue, and a high proportion of immune cells and astrocytes at the active edges of those lesions. Microglia comprised 25% of all immune cells present at the lesion edges.

“Our dataset is very rich. The beauty of having such a detailed map is that now we have a better understanding of the entire network of cells involved in smoldering inflammation,” said Martina Absinta, M.D., Ph.D., a former post-doctoral fellow in Dr. Reich’s lab and current adjunct assistant professor at Johns Hopkins University, Baltimore, who led the study.

More detailed analyses revealed that the gene for complement component 1q (C1q), an important and evolutionarily ancient protein of the immune system, was expressed mainly by a subgroup of microglia responsible for driving inflammation, suggesting that it may contribute to lesion progression.

To determine the function of C1q, researchers knocked out the gene in the microglia of mouse models of MS and examined the brain tissue for signs of neuroinflammation. In mice lacking microglial C1q, they found significantly decreased tissue inflammation compared to control animals. Additionally, in another group of animals, blocking C1q reduced iron-containing microglia, revealing a potential new therapeutic avenue to treat chronic brain inflammation in MS and related neurodegenerative diseases.

According to the authors, it’s possible that targeting C1q in human microglia could halt MS lesions in their tracks.

In MS, the immune system attacks myelin, a protective layer that forms around nerve cells in the brain and spinal cord, leading to vision loss, muscle weakness, problems with balance and coordination, fatigue, numbness, and other debilitating symptoms. A subset of people develop progressive MS, resulting in extensive brain tissue damage and disability. Anti-inflammatory medications help patients manage their symptoms by dampening the responses of immune cells in the blood and lymph nodes. But treatments are not as effective for patients with chronic lesions who experience ongoing brain tissue inflammation.

“We have terrific therapies that block new inflammation but nothing to stop the inflammation that’s already there,” said Dr. Reich. “In order to make strides in developing new therapies for progressive MS, we’re going to need to pick apart the cellular and molecular mechanisms one by one.”

In 2019, Dr. Reich and his team reported that damaging, chronic active lesions may be a hallmark of progressive MS. The current study identifies microglia and C1q as promising targets for progressive MS and supports the use of chronically inflamed rim lesions as an MRI biomarker for disease progression.

There is no cure for MS, and no therapies that directly treat chronic active lesions. By gaining a deeper understanding of lesion features, this study may help pave the way toward early clinical trials to test new therapies for this aspect of the disease.

This study was supported in part by the Intramural Research Program at the NINDS.

NINDS is the nation’s leading funder of research on the brain and nervous system. The mission of NINDS is to seek fundamental knowledge about the brain and nervous system and to use that knowledge to reduce the burden of neurological disease.

About the National Institutes of Health (NIH): NIH, the nation’s medical research agency, includes 27 Institutes and Centers and is a component of the U.S. Department of Health and Human Services. NIH is the primary federal agency conducting and supporting basic, clinical, and translational medical research, and is investigating the causes, treatments, and cures for both common and rare diseases. For more information about NIH and its programs, visit www.nih.gov.

NIH…Turning Discovery Into Health®