There are those who deny that aluminum is a neurotoxin. Paul Offit, for example, thinks it might be a nutrient, given that premature infants had higher amounts of aluminum body burden than full-term infants (yes, he really said that – in a book, even). There are those who have worked systematically to utterly confuse the American public on the toxicity of thimerosal – which is 50% ethylmercury by weight – by claiming that it is “safer” than methylmercury.
That’s like saying kerosene is safer to spread around your home because gasoline is more volatile.
In my course, “Environmental Toxicology: Ecosystem and Human Health”, we review the toxicology of many different classes and types of compounds. Many of them are established neurotoxins – that is, their effects on the human brain are established by science. In one lecture, I go into detail, especially on the developmental neurotoxic metals – lead, mercury, aluminum – when the human brain is most vulnerable.
This chart is from Grandjean & Landrigan (The Lancet Neurology 2014 13330-338DOI: (10.1016/S1474-4422(13)70278-3)) and it captures a well-known fact: the earlier in life people are exposed to neurotoxins, the more compounded the effects.
Another fact is that kids with autism are sponges for environmental toxins. Take this result from ref$, which shows that kids with autism have ADOS scores that are correlated with the mean serum concentration of toxins from common, everyday exposures: gasoline, pesticides, flame retardants, plasticizers, etc.:
Published in Nature, 2015, this study also found that kids without autism had far lower ADOS scores and that their scores were not correlated with mean serum concentration at all. The combined effects of these organic pollutants are clearly profound. Scott Faber, second author of the study, came to the book release party for “The Environmental and Genetic Causes of Autism” at the Hard Rock Cafe in Pittsburgh, PA, and showed me this result. It was so compelling I called the publisher and had them (literally) stop the presses to make sure we had this particular result in the book.
These pollutants no doubt have their own mechanisms of toxicity, setting the stage for synergistic toxicity. Now consider aluminum and fluoride. In a study of mice, one study found that the combined application of
“sodium fluoride and aluminum chloride-induced severe perturbation and imbalance in the neurotransmission systems under investigation. The pattern of change and severity differed with the different brain areas. The combination of the two pollutants exerted general synergistic impacts with different specific responses in the different brain areas.” (From: The potential roles of aluminum chloride and sodium fluoride on the neurotoxicity of the cerebral cortex, hippocampus, and hypothalamus of male rat offspring.
It turns out that historically, concern over aluminum and fluoride synergistic toxicity existed due to their effects on G-coupled proteins. Check out this resource from 2013:
“Fluoride and Aluminium Alum ( Aluminium ) is added to water to flocculate dirt and bacteria at water treatment plants. Fluoride is added at the last stage. Alumino -fluoride complexes (Al. Fx) form spontaneously in aqueous solutions containing fluoride and traces of aluminium ions and appear to act as phosphate analogs Fluoride anions have long been known to influence the activity of various enzymes and guanine nucleotide-binding proteins (G-proteins).”
and
“Fluoride and Aluminium Understanding the role of phosphate and G-proteins in cell signaling makes it possible to suggest a hypothesis that the synergistic action of fluoride and aluminum in the environment, water and food chains can evoke various and multiple pathological symptoms. The long-term synergistic effects of these ions in the living environment and their hidden danger for human health is not yet fully recognized.”
Now fast-forward to the post-COVID-19 world in which vaccines with mRNA capable of inducing the production of the Wuhan-1 spike protein are being, perhaps perpetually, pointed at everyone around the world.
Unfortunately for everyone taking the shot, epithelial cellular adhesion helps maintain the integrity of the Blood-Brain Barrier (BBB). This study shows that the Wuhan-1 spike protein activates RhoA, which leads to increased permeability of the blood-brain barrier.
This means:
The Wuhan-1 SARS-CoV-2 Spike Protein is THE UNIVERSAL SYNERGISTIC NEUROTOXIN
The toxicity of the Spike protein, however, is not restricted to neurologic tissues. In fact, nearly all of the adverse events from the vaccine are tied to the toxicity of the Spike protein breaking down the barriers between endothelial cells by creating connections call syncytia.
The amazingly objective journalist Jeremy Hammond has reviewed most of the literature on the toxicity of the spike protein (debunking the so-called “Fact Checking” opinion website Health Feedback). I recommend a complete read AND A SUBSCRIPTION (he is REALLY amazing). The studies show that the spike protein impairs the endothelial cells all around the body.
These effects are on top and in addition to the autoimmunity predicted and validated to be occurring by #PathogenicPriming.
All of this tells us that Wuhan-1 Spike Protein is THE UNIVERSAL SYNERGISTIC TOXIN.
Thanks to the brilliant people at the US FDA, future COVID-19 vaccines will continue to force people to produce the Wuhan-1 strain of the SARS-CoV-2 virus via their plan to include mRNAs for Wuhan-1, a long-extinct SARS-CoV-2 variant, known to cause antibody-dependent enhancement.
Unfortunately, people will have morbidity and mortality stemming from the toxicity of all types of toxins that will never be attributed to the spike protein encoding vaccines making it worse. We need to reduce total population exposure to ALL sources of toxins due to the problem of synergy. That’s up to us working together.
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