Hopefully the inaccurate term ‘long covid’ will disappear just like the inaccurately deceptive term PTLDS will disappear.
After the link between the clot shots and ‘long covid’ has been finally gaining some acceptance, American virologist Dr. Robert R. Redfield, who served as the director of the CDC during the ‘pandemic,’ has admitted that reports of ‘so-called Long Covid’ are actually a cover-up for global surges of “mRNA vaccine injury.”
The evidence continues to mount showing the experimental gene therapy injections are maiming and killing people.
Another recent study admonishes against using the term Long COVID as the symptoms are no worse than those after the flu. In fact, PCR testing can’t distinguish between COVID and the flu. So, what in fact is causing lingering symptoms in some people and how severe are they actually? That is the important question.
The following information is at the heart of the “persistence” war. A few points:
- The following article is talking about viruses, not bacteria. It is widely known that certain bacteria can and do persist causing chronic illness. Viruses, on the other hand, supposedly can live peacefully all over our body lying dormant until stressors can cause them to reemerge. If the immune system is strong, no harm no foul.
- The only thing the authors actually found were pieces of viral RNA but no evidence that a person had become reinfected. This is important. Finding pieces of stuff floating around doesn’t mean you are sick.
- If you remember, the PCR test scam occurred for multiple reasons:
- It can’t differentiate between COVID and the flu
- It can’t identify variants, but tests for any strain, not just COVID – which means all data based upon this test is invalid. This too is important.
- It can not distinguish virus from harmless viral fragments
- A positive test didn’t mean you were sick
- Patients tested positive, then negative, then positive within hours
- Despite the attempt by the CDC to blame asymptomatic people with transmitting COVID, the vast majority of asymptomatic individuals who tested positive — 96% — did NOT transmit the virus. We were even warned by honest virologists about this back in 2020, as well as having the fact play out in reality over and over and over and over again but it was completely ignored by corrupt public health because it would have stifled their plan to make the asymptomatic public enemy #2 right after public enemy #1, the unvaccinated, who were blamed for extending the ‘pandemic’ when it’s actually the “vaccinated” that are forcing the virus to mutate. All of these known, verifiable facts were simply ignored by ‘public health’
- Meanwhile, Lyme/MSIDS patients have legitimate, continuing, severe symptoms as well as distinguishable bacteria and viruses lingering in their bodies, ZERO is being done about it other than gaslighting patients and threatening doctors.
- The authors state more research is needed to determine whether the persistence of these fragments drives long COVID and such associated risks as heart attack and stroke. I’m sure these authors and others will easily get tons of government grants to study this while Lyme/MSIDS gets ZERO.
- Sadly, these authors aren’t even entertaining the fact that the COVID gene therapy injections could be behind these heart attacks and strokes or the accepted link between the COVID shots and “Long COVID.” They probably won’t even make distinguish between the vaxxed and the unvaxxed in their study. Sadly, it’s simply easier and less polarizing to blame long COVID than an injection that has caused more adverse reactions and death than any other vaccine in the history of VAERS that only has a 1% capture rate.
- The institutional bias against Lyme/MSIDS is not new, nor has it changed.
Corrupt ‘public health’ has been effectively playing us. While fragments of viral RNA are uniformly blamed for lingering symptoms, those with chronic Lyme/MSIDS are kicked to the curb.
https://www.lymedisease.org/covid-virus-can-stay-in-body/
COVID virus can stay in the body more than a year after infection
3/10/24
The COVID-19 virus can persist in the blood and tissue of patients for more than a year after the acute phase of the illness has ended, according to new research from University of California, San Francisco that offers potential clues to why some people develop long COVID.
The scientists found pieces of SARS-CoV-2, referred to as COVID antigens, lingering in the blood up to 14 months after infection and for more than two years in tissue samples from people who had COVID.
“These two studies provide some of the strongest evidence so far that COVID antigens can persist in some people, even though we think they have normal immune responses,” said Michael Peluso, MD, an infectious disease researcher in the UCSF School of Medicine, who led both studies.
The findings were presented at the Conference on Retroviruses and Opportunistic Infections, held recently in Denver.
Evidence of long-term infection
Early in the pandemic, COVID-19 was thought to be a transient illness. But a growing number of patients, even those who had previously been healthy, continued having symptoms, such as, brain fog, digestive problems and vascular issues, for months or even years.
The researchers looked at blood samples from 171 people who had been infected with COVID. Using an ultra-sensitive test for the COVID “spike” protein, which helps the virus break into human cells, the scientists found the virus was still present up to 14 months later in some people.
Among those who were hospitalized for COVID, the likelihood of detecting the COVID antigens was about twice as high as it was for those who were not. It was also higher for those who reported being sicker, but were not hospitalized.
“As a clinician, these associations convince me that we are on to something, because it makes sense that someone who had been sicker with COVID would have more antigen that can stick around,” Peluso said.
Virus persists up to two years in tissue
Since the virus is believed to persist in the tissue reservoirs, the scientists turned to UCSF’s Long COVID Tissue Bank, which contains samples donated by patients with and without long COVID.
They detected portions of viral RNA for up to two years after infection, although there was no evidence that the person had become reinfected. They found it in the connective tissue where immune cells are located, suggesting that the viral fragments were causing the immune system to attack. In some of the samples, the researchers found that the virus could be active.
Peluso said more research is needed to determine whether the persistence of these fragments drives long COVID and such associated risks as heart attack and stroke.
But, based on these findings, Peluso’s team at UCSF is involved in multiple clinical trials that are testing whether monoclonal antibodies or antiviral drugs can remove the virus and improve the health of people with long COVID.
“There is a lot more work to be done, but I feel like we are making progress in really understanding the long-term consequences of this infection,” Peluso said.
SOURCE: University of California, San Francisco