Lyme Disease And Blood Cancer: A Startling Connection Revealed In New Study
May 7, 2023
A groundbreaking study by renowned researcher Alan B. MacDonald reveals a hidden link between Lyme disease and Chronic Lymphocytic Leukemia (CLL), a slow-growing blood cancer.
The study found that some people had Borrelia (Lyme) infections in their blood for three years before being diagnosed with CLL, even though they didn’t have any symptoms.
The study discovered new findings, such as Borrelia bacteria getting into both healthy and cancerous white blood cells, highlighting the need to better understand the connection between Lyme disease and other health issues for more accurate diagnosis and treatment.
Introduction: Uncovering a Surprising Connection Between Lyme Disease and Blood Cancer
Imagine the shock when a groundbreaking study, published in the Journal of Clinical Review and Case Reports on May 15, 2023, unveils a hidden link between Lyme disease and cancer. In the eye-opening research, “Invasion of Chronic Lymphocytic Leukemia (CLL) Bloodstream Tumor Cells by Borrelia Spirochetes,” renowned researcher Alan B. MacDonald reveals astonishing insights into the connection between Lyme disease and CLL, a slow-growing blood cancer.
The Study: Examining a CLL Patient with Borrelia Infections In this study, Dr. MacDonald investigated the case of a 66-year-old woman with a family history of breast cancer and CLL. The patient had gradual onset of axillary lymph node enlargement (swelling of lymph nodes in her armpit area) in 2019, and subsequent tests confirmed a CLL diagnosis. Interestingly, the patient was found to have asymptomatic Borrelia (Lyme) infections in her bloodstream, including both Borrelia miyamotoi and Borrelia burgdorferi.
Findings: Lyme Infections Before CLL Diagnosis
The study found that the patient had Lyme infections in her blood without showing any symptoms three years before being diagnosed with CLL. Blood tests from 2016 using a special technique called Fluorescence In Situ Hybridization (FISH) showed that Lyme bacteria were sticking to a special type of white blood cell that helps fight infections. In 2022, when the patient’s white blood cells turned cancerous, the tests still showed an ongoing infection with two types of Lyme bacteria (Borrelia burgdorferi and Borrelia miyamotoi).
Significance: Unprecedented Observations in CLL and Lyme Infections This study shares some new discoveries about the connection between Borrelia (Lyme) and blood cancer (CLL):
Some people had Borrelia infections in their blood for three years before being diagnosed with CLL, even though they didn’t have any symptoms.
One person with CLL was found to have two different types of Borrelia bacteria in their blood at the same time.
Borrelia burgdorferi bacteria were seen sticking to and entering healthy white blood cells, something that was only seen in labs before.
Borrelia bacteria were also seen entering cancerous white blood cells, which is a completely new finding.
Borrelia miyamotoi infections have been found in patients with a specific type of blood cancer, but this is the first time they’ve been shown to actually enter the cancerous cells.
Groups of Borrelia bacteria were found in patients’ blood samples, both before and after developing leukemia, and these groups have been linked to cancer in other cases.
Summary: Borrelia Implications Beyond Lyme
A groundbreaking study by Dr. Alan B. MacDonald investigates the link between Lyme disease and a slow-growing blood cancer called Chronic Lymphocytic Leukemia (CLL). The research looked at a 66-year-old woman with CLL who had Lyme bacteria in her blood without any symptoms, three years before being diagnosed with CLL. The study discovered new findings, such as Lyme bacteria getting into both healthy and cancerous white blood cells. This highlights the need to better understand the connection between Lyme disease and other health issues for more accurate diagnosis and treatment.
About the Author: Dr. Alan B. MacDonald As an expert in Lyme disease and tick-borne illness complications, Dr. MacDonald has made several appearances on the Tick Boot Camp Podcast, discussing this groundbreaking study (PDF) and more. He is known for his cutting-edge research on Lyme disease and tick-borne illness complications. His research emphasizes the importance of understanding the link between Lyme disease and other medical conditions, such as CLL, to better diagnose and treat patients affected by these illnesses.
Photo Description The featured photo in this blog post depicts a lymphocyte cancer cell with two Lyme disease spirochetes attached. More specifically: “Immunohistochemistry with monoclonal antibody CB10 specific for protein OSP A of borrelia burgdorferi group sl spirochetes. Two borrelia spirochetes (black arrows) are attached to a large caliber leukemic lymphocyte. Magnification 1000x original.”
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**Comment**
Mark my words: this will only be shared between sick patients and the doctors who dare treat us.
While cancer cases are rising in younger people globally, no one – particularly the cancer industry mills, will even pause a moment to consider whether Lyme/MSIDS or heaven forbid, the mRNA gene therapy injections are implicated. The injections were not evaluated for carcinogenicity potential.
And speaking of cancer, watch this brief 2 minute video with Dr. Sucharit Bhakdi where he explains the COVID gene therapy injections alter your DNA forever and increase your risk for cancer.
“The integration of any foreign gene into your chromosome can cause cancer immediately,” warned microbiologist Dr. Sucharit Bhakdi.
“[It] can cause all sorts of inflammation and moreover will cause this gene to be transmitted to your offspring,” he explained.
“This is not a hate speech. This is a speech to make everyone wake up and see the dangers that they are facing because the WHO and the CDC and the FDA and all these guys up there are planning to introduce RNA vaccines worldwide.”
And for this bit of truth, Prof. Sucharit Bhakdi is charged with “incitement” and “trivialization of the Holocaust” for statements he made comparing the C19 vaccination program to 1930s/40s Germany. His hearing is scheduled for 23 May in Plön, Germany. More details here: https://doctors4covidethics.org/dr-bhakdis-legal-case/
SAPI2022AONM_221203_013821 Go here for Eva Sapi’s slides titled, “Potential Connection of Borrelia Infection and Breast Cancer“. She also addresses “Mixed Biofilm in Other Infected Skin Tissues.” There is a particular slide that shows where in the human body that Borrelia biofilm is found.
Why some people recover from Lyme disease, while others experience months, years or even decades of chronic symptoms has long puzzled doctors. New research offers some clues to an immune system marker in the blood that is elevated among people with lingering Lyme disease symptoms, even after they’d received antibiotics.
In the new study, published on May 9 in the Center for Disease Control and Prevention’s Emerging Infectious Diseases journal, researchers found an immune system marker in the blood called interferon-alpha was elevated among people who had been treated for Lyme disease but had lingering symptoms.
Interferon-alpha is one of a handful of key signaling proteins the body makes to tell immune cells to fight off bacteria or viruses. If the blood levels are too high, the immune system can overact, causing pain, swelling and fatigue — symptoms often seen with Lyme disease.
In patients with high levels of interferon-alpha, the immune response to the Lyme bacteria may cause chronic inflammation, even once the infection is gone, said Klemen Strle, an assistant research professor of molecular biology and microbiology at Tufts University and an author of the new study.
“We think this is a possible driver of persistent symptoms,” Strle said. And since a number of drugs are already approved to lower interferon-alpha, he suggested the research could mean a possible treatment option for lingering Lyme symptoms.
The study was small, including 79 people diagnosed with Lyme disease, and found only a link between the higher interferon-alpha levels and the persistent Lyme disease symptoms, not that the immune marker was itself causing the lasting symptoms. A larger clinical trial would be needed to affirm the connection.
Male and female adult black-legged ticks, Ixodes scapularis, on a sesame seed bun to show their relative size.CDC
Anywhere from 30,000 up to 500,000 people develop Lyme disease from a tick bite each year, according to the CDC. For most, the infection is mild and easily treated with antibiotics. About 10% experience symptoms like fatigue and brain fog along with muscle, joint and nerve pain that persists even after treatment.
The new findings represent a significant shift in understanding why some people infected with Lyme suffer chronic symptoms. Previously, some researchers believed that a specific strain of the spiral-shaped Borrelia burgdorferi bacteria that causes Lyme might be a cause. Others wondered whether undetectable low levels of infection lingered in the body after treatment. The new research suggests that the way the body reacts to the bacteria — not the bug itself — could result in long-lasting symptoms.
It’s still unclear why some people have elevated interferon-alpha, but Strle said he’s looking into a possible genetic cause. (See link for article)
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To reiterate:
This study consisted of 79 people diagnosed with Lyme based off of matched serum and CSF samples. Once again, no seronegative patients are represented.
A link between higher interferon-alpha levels and the persistent Lyme disease symptoms was found. This does not prove causality.
Far more than 10% experience these depilating chronic symptoms. They continue to regurgitate this number which only includes those diagnosed and treated early. It doesn’t include the far larger subset of patients that are diagnosed and treated late.
Go here to read Lyme advocate Carl Tuttle’s letter to Strle where he reminds him that while interferon alpha has also been found in patients with chronic hepatitis,it wasn’t blamed for chronic hepatitis but rather was correlated with the presence of viral replication, or persistent viral infection. He then reminds Strle of a 1995 case study on a Lyme patient who received multiple courses of IV and oral meds yet relapsed after each one and that the only way this patient stayed in remission was when kept on open ended clarithromycin. To cinch the deal, Tuttle reminds Strle of a 2018 study of 12 Canadian Lyme patients who were culture positive for infection even after multiple years on antibiotics. Tuttle simply but wisely asks the obvious question: would a chronic, persistent Lyme infection raise IFN-a levels?
‘The powers that be’ are desperate to continue the narrative because there’s a big Lyme “vaccine” in the pipeline that could make them lots of money. A chronic/persistent infection would not be conducive to a vaccine.
This year, 2,753 cases of Lyme disease (a 93% increase) and 47 cases (a 236% increase) of tick-borne encephalitis have been confirmed in Poland, according to the latest data on infectious diseases.
This covers the period from January 1 to April 15 this year. During the same time in 2022, there were 1,423 cases of Lyme disease and 14 cases of tick-borne encephalitis.
An educational campaign on the prevention of tick-borne diseases under the slogan “Don’t be attractive to ticks” has been launched. (See link for article)
Mycoplasmas, the smallest bacteria lacking a cell wall, can cause various diseases in both humans and animals. Mycoplasmas harbor a variety of virulence factors that enable them to overcome numerous barriers of entry into the host; using accessory proteins, mycoplasma adhesins can bind to the receptors or extracellular matrix of the host cell. Although the host immune system can eradicate the invading mycoplasma in most cases, a few sagacious mycoplasmas employ a series of invasion and immune escape strategies to ensure their continued survival within their hosts. For instance, capsular polysaccharides are crucial for anti-phagocytosis and immunomodulation. Invasive enzymes degrade reactive oxygen species, neutrophil extracellular traps, and immunoglobulins. Biofilm formation is important for establishing a persistent infection. During proliferation, successfully surviving mycoplasmas generate numerous metabolites, including hydrogen peroxide, ammonia and hydrogen sulfide; or secrete various exotoxins, such as community-acquired respiratory distress syndrome toxin, and hemolysins; and express various pathogenic enzymes, all of which have potent toxic effects on host cells. Furthermore, some inherent components of mycoplasmas, such as lipids, membrane lipoproteins, and even mycoplasma-generated superantigens, can exert a significant pathogenic impact on the host cells or the immune system. In this review, we describe the proposed virulence factors in the toolkit of notorious mycoplasmas to better understand the pathogenic features of these bacteria, along with their pathogenic mechanisms.
Hemotropic mycoplasma is caused by bacteria from the Mycoplasma genus, which lack a cell wall and are unable to replicate by themselves. To survive they have to infect red blood cells. Like the other bacteria discussed here, emerging species are found in bats. Hemotropic mycoplasma may be a co-factor in white nose syndrome, a fungal disease that is killing bats.