This segment focuses on a legal challenge to California’s COVID-19 misinformation law, arguing that doctors should retain First Amendment protections when discussing medical information and treatment options with patients. Attorney Richard Jaffe tells Jimmy that after earlier rulings treated doctor-patient advice as regulable “conduct,” a later Supreme Court decision on protected therapeutic speech changed the legal landscape, leading a federal judge to rule in favor of three doctors challenging California’s restrictions.
Dr. Pierre Kory argues that the case has implications far beyond COVID-19 because allowing governments or medical boards to dictate what physicians may say could suppress medical dissent and independent judgment. The three characterize the ruling as a major victory for doctors’ and patients’ freedom of medical discussion, while also crediting legal organizations that supported the litigation.
Horowitz Dapsone Protocols: Risks vs Benefits Explained
About Dapsone & Persisters
Dapsone, a Leprosy drug, can help some with treatment-resistant Lyme and Bartonella due to persisters plus treatment resistant Babesia. In 2016, Richard Horowitz, MD, and Phyllis Freeman, PhD, published research on 100 patients. The study showed 59 percent of people had improvements with dapsone of 100 mg or less.
More recently, Dr. Horowitz has continued refining a higher-dose approach, publishing a more detailed double-dose/high-dose protocol in 2023.
However, my experience with dapsone is mixed. While it helps some, it is also a very difficult medicine for many to take.
In this article, I review:
my experience with this novel medicine,
a specific antibiotic protocol that includes dapsone as a normal-dose regimen,
how to decrease side effects and Herxheimer reactions on dapsone,
the risks of using dapsone,
the chances for treatment success using a dapsone regimen, and
a detailed look at, and critique of, Dr. Horowitz’s newer double-dose/high-dose protocol and why I don’t support it.
(See link for video and article)
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**Comment**
I always appreciate doctors who share their clinical experience. It’s sometimes the only way we can gather intel on whether or not we should try a certain treatment. Case in point – I wrote about my severe psychosis after taking disulfiram, followed by another practitioner’s experiences in ameliorating symptoms as well as an update on a few points of consideration.
So, while Dapsone may be a perfect fit for some, it may not for others.
This is another point that never makes the news. Reactions to treatment varies widely in Lymeland, probably because some have been infected for years and their bodies are simply overwhelmed, making another drug appear to be a toxin by the body.
Dr. Ross points out the following issues:
You shouldn’t take Dapsone if your G6PD levels are low.
Dapsone blocks folate metabolism which can lead to anemia
Dapsone suppressed bone marrow which can also lead to anemia
Dapsone is a harsh drug that causes severe herxes, allergic rash, methemoglobinemia, and many side effects
The article contains Horowitz’s 2016 Dapsone regimen. Ross states he does not recommend the Dought or high dose Dapsone protocol and doesn’t offer it in his practice. He also points out that Horowitz’s 2023 paper which offered HDDCT to 50 patients, but only 25 are considered in the results leaving one to ask how many dropped out due to side effects. Further, Horowitz is the treating physician, the sole study author, and one of three others from his practice who performed the validation and analysis. There was no control group, no blinding, no independent review, and no oversight.
Ross does feel; however, that some sho failed other treatments could consider the normal-dose dapsone 100mg protocol if carefully monitored.
Hadid said it was “intimidating” to explain her symptoms, which include brain fog, fatigue, pain, anxiety, infections, and more.
“You demand answers that no one can find. You fight. You finally have a few good days. You think you’ve found the right protocol, the right routine, the right treatment… and then a flare up comes back and all of a sudden nothing feels certain again,” the American model, who is of Dutch and Palestinian descent, wrote.
“Haven’t been able to shake off this flare up,” Hadid wrote in another Story, accompanied by a teary-eyed selfie. “Slept 11 hours. Again … Nap everyday. Took every protocol from any and every doctor I’ve seen. Still nothing helping. (See link for article)
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Bella Hadid health updates have taken a serious turn as she battles a brutal Lyme disease flare up, revealing her hardest fight yet. This video examines the latest news surrounding Bella Hadid and the reality of her ongoing health challenges. Despite the designer outfits and high-profile campaigns, this update offers a candid look at the physical toll her condition is taking behind the scenes. We break down exactly what she shared about her current state and why this specific Lyme disease update is being described as her most difficult period to date.
**Comment**
Sadly, this is the reality of Lyme/MSIDS.
It’s also proof that no matter your station in life, this monster can cripple you.
I do wish; however, that these news stories would take on the coinfection issue, that testing is abysmal, that it quite probably was tweaked in a lab to be more virulent and harder to find, that it remains extremely controversial, as well as the fact every case is different. We need to move past the shock and awe factor of someone being extremely ill to meaningful, helpful information, but the majority of journalists are beholden to their bosses whom are beholden to Big Pharma whom buy up ad spaces – shaping the narrative.
Go here for a a few well done pieces by Wisconsin Watch on the complexities of Lyme/MSIDS:
https://madisonarealymesupportgroup.com/2022/06/13/wisconsins-chronic-lyme-patients-embrace-alternative-treatments-rack-up-big-bills/I’m happy to report that since this was written in 2022, my husband and I have been in remission. After FIVE years of pulsed oral antibiotic treatment with a multitude of supplements and herbs, we relapsed 3-4 times requiring 2-3 month stints of treatment each time (for Bartonella); however, each time we retreated, we achieved a greater level of health than previously. I often wonder how many patients are never given this opportunity to “cycle” or retreat a complex illness that is relapsing in nature. I must also add that our main Lyme literate doctor (LLMD) did not feel comfortable treating us with antibiotics anymore. We had to switch to a nurse practitioner up North who treats entire families with this. She had no trouble retreating us as she’s seen the results with her own eyes. I say this because you need to know you will have to fight in every sense of the word to get well from this.
The one question I left standing in Part I — if it was the shots, why the tick at all? The scientific literature answers it in one word. Three questions that collapse the tick story from the inside.
In Part I, I built a fence. On one side, I put everything solid — Richet’s Nobel Prize, the route problem, the Japanese gelatin admission, the billing codes, and my own Amish patient base, covered in ticks, completely unvaccinated, and, at least as far as I can ascertain, free of alpha-gal — with a single imported exception I’ll come back to shortly. On the other side, I put my suspicions about a declassed clandestine program, telling you where the record stopped, and my hunches began.
Today, there will be no discussion of Fort Detrick, Plum Island, or other biolabs / bioweapon labs. There’s no need for that when answering a question that several readers asked via the comment section. Before I had finished my morning coffee…
If it’s really the shots that cause alpha-gal, then why is the tick needed at all?
I’m going to answer this by asking and answering three very specific questions from the tick research community’s own filing cabinet — CDC-funded papers and the industry-disclosed authors. From the scientists who are certain the Lone Star tick is the primary cause.
I’m going to let people far smarter than me answer these questions using their own studies…
Question One: Why Is a Tick Needed At All?
But the condition has no historical precedent. It appears in the literature at a specific moment in the late 1980s, in multiple countries simultaneously, at a time when gelatin-containing vaccines were being introduced into childhood immunization schedules worldwide. Japanese researchers documented, confirmed through intervention, and published the direct causal relationship between gelatin-containing DTaP vaccines and alpha-gal sensitization in children who had never been exposed to ticks. That evidence is in the peer-reviewed record. It has been there since 1996. -From Dr Travis Johnson’s June 2 piece in Medium(The Allergy That Shouldn’t Exist: Vaccines, Gelatin, and the Strange Origins of Alpha-Gal Syndrome)
To answer question one, it isn’t — at least not as the origin. Vaccines alone can do it — the tick isn’t required for the mechanism. But historically, before the schedule ballooned, cases clustered in tick country. And the scientific literature already told you why.
Go pull the 2021 paper from Frontiers in Cellular and Infection Microbiology that tick researchers themselves titled “Tick Saliva and the Alpha-Gal Syndrome: Finding a Needle in a Haystack”. Read that title again. A needle in a haystack. They picked the metaphor, but pointed it at the park instead of at the vial.
Let’s watch what the tick actually does and why the needle in the haystack might be apropos, but maybe not in the way the authors intended…
Another 2021 study, this one on a tick enzyme, describes the alpha-gal sugar as being — their word, not mine — “injected into humans from the lone-star tick bite”. Injected. Not eaten. Not digested. Injected — straight through the skin, past the gut and its digestive processes, and into areas where food is not supposed to be. And as I stated in Part I, this is extremely similar to the phenomenon I have referred to for over two decades as “The Leakies”. Example: (Systemic Leaky Barrier Syndrome (SLBS): A Systems-Level Framework for Chronic Disease).
Earlier studies localized the sugar to the secretory vesicles of the tick’s salivary glands — meaning it’s built to be squirted into a bite. In other words, it’s not wrong to think of ticks as syringes with eight legs — light enough to cross your skin without tripping pressure receptors, and armed with saliva loaded with painkillers, anti-inflammatories, and anticoagulants that let them latch and drink for days without ever setting off the itch that would make you look down and flick them off.
What these devilish little creatures are loading into you is galactose-alpha-1,3-galactose — alpha-gal for short; the “ose” tells you it’s a sugar, like glucose or sucrose — a bit of mammalian sweetness squirted straight under your skin. And here’s the kicker nobody thus far has answered…
When it comes to the animals the tick commonly feeds on — deer, cattle, dogs, rodents, goats, hogs, etc — alpha-gal is “endogenous”. In other words, they build it, they carry it, and their immune systems recognize it as native (“self”) so that it’s not attacked as an invader. So the best answer for why it’s in the saliva at all isn’t that it works some mechanical trick like the anticoagulants and painkillers do — it’s camouflage. Dress your spit in the host’s own sugar, and it reads as ‘self,’ letting the tick guzzle until 100X normal size, all under the immune radar. Which means the sugar is doing exactly its job on a deer. (See link for article)
The most important take-away is the fact that the Ozarks are full of deer, lone star ticks, and ‘vaccinated’ people, with the exception of the unvaccinated Amish whom are smack dab in the middle of the same area and free of AGS.
“Geography is an alibi the ‘forced vaccination community’ at large is hiding behind because ticks and the heavily vaccinated live side by side in so much of the country.” ~ Dr. Shierling
The good doctor also states he’s only come into ONE Amish person who got AGS, but that she grew up in Minnesota and received some vaccines as an infant…..
Another important take-away is that the younger the child and the larger the ‘vaccine’ dose(s), the more likely they are to develop sensitivity rather than tolerance. The Journal of the American Dental Association now quietly lists topical numbing gels, Gelfoam, prophy paste, catgut, bone graft materials — while noting manufacturers aren’t required to disclose animal-derived ingredients, as containing alpha-gal bearing excipients. This website has also posted the fact that graphene oxide was found in all three dental anesthetics tested.
Crohn’s and colitis are called genetic, autoimmune, and idiopathic. What if all three labels are wrong? A new paper tests each against current evidence.
What if Crohn’s disease and ulcerative colitis are caused by more than genetics alone?
Dr. Yusuf “JP” Saleeby, IMA Senior Fellow in Functional and Integrative Medicine, and gut health specialist Josh Dech take a closer look at what may contribute to inflammatory bowel disease, also known as IBD. The two recently co-authored a new paper published in the Journal of Independent Medicine. Their conversation traces how genetics, diet, gut health, and the environment may work together to shape both diseases.
Inflammatory bowel disease affects more than 7 million people worldwide and ranks among the fastest-growing chronic diseases globally. Nearly everyone diagnosed with Crohn’s disease or ulcerative colitis hears some version of the same three things: the disease is genetic, the immune system is attacking its own tissue, and the underlying cause is unknown. Those three explanations leave two treatments on the table, drugs and surgery, and they leave a patient nothing to investigate.
A new paper in the Journal of Independent Medicine argues that all three explanations fail against current evidence. Josh Dech and Dr. JP Saleeby, its co-authors, point out that each has been contested separately in the literature for two decades without anyone testing them as a set. Taken together, they conclude, the conventional model does not hold.
What replaces it is a disease that is partially heritable, environmentally activated, and immune-mediated, and the distinction is not academic for anyone living with one. If exposures determine whether susceptibility becomes disease, exposures can be found and changed. (See link for article, research paper and video)
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SUMMARY:
The authors found that genetics only explain about a quarter of disease risk for irritable bowel (IBD).
The authors argue that the autoimmune label, despite holding for decades, is based on the weakest evidence and that pathogenic transfer has never been demonstrated.
The serologic markers long cited as evidence for autoimmunity turn out to recognize microbial and fungal targets but are not autoantibodies in the classical sense.
A review of 53 meta-analyses across 71 risk factors shows the following exposures for IBD that are quantified and modifiable:
antibiotic exposure
oral contraceptives
breast-feeding was protective for Crohn’s and colitis
ultra-processed food
air pollution
psychological stress
mold
mycotoxins
The authors state current treatments complement but ignore the environmental exposures.
The authors give the following issues that should be questioned alongside a standard work-up:
Birth mode and feeding history
Early antibiotic courses, particularly before age 5
Water damage & mold exposure at home, work and in vehicles.
Adolescent diet, stressors, and infections
At this point in the article, they described a case report on a 14 year old whose Crohn’s progressed far enough that surgeons planned to remove most of his intestines & place a colostomy. Testing pointed to chronic Lyme disease and three months into treatment a repeated scope test found no lesions.
In short, they conclude the following answers for IBS: antibiotic stewardship, breastfeeding support, reducing ultra-processed food, and remediating indoor mold.
After reading the comments after the article, I would be remiss if I did not mention the ‘vaccine’ issue due to the fact they all introduce foreignsubstances the body recognizes as foe, priming it for later potential problems such as life-threatening allergies to many things including food, which many are also linking to Alpha Gal Syndrome (AGS), an allergy to animal products supposedly caused by ticks – with no solid proof, as well as the fact some get AGS without any known tick involvement. So while ticks play a part, they are obviously not the only ingredient required to get AGS.