Cases of myocarditis and pericarditis have been reported following the receipt of Covid-19 mRNA vaccines. As vaccination campaigns are still to be extended, we aimed to provide a comprehensive assessment of the association, by vaccine and across sex and age groups. Using nationwide hospital discharge and vaccine data, we analysed all 1612 cases of myocarditis and 1613 cases of pericarditis that occurred in France in the period from May 12, 2021 to October 31, 2021. We perform matched case-control studies and find increased risks of myocarditis and pericarditis during the first week following vaccination, and particularly after the second dose, with adjusted odds ratios of myocarditis of 8.1 (95% confidence interval [CI], 6.7 to 9.9) for the BNT162b2 and 30 (95% CI, 21 to 43) for the mRNA-1273 vaccine. The largest associations are observed for myocarditis following mRNA-1273 vaccination in persons aged 18 to 24 years. Estimates of excess cases attributable to vaccination also reveal a substantial burden of both myocarditis and pericarditis across other age groups and in both males and females.
Important excerpt from the Discussion Section:
In conclusion, this study provides strong evidence of an increased risk of myocarditis and of pericarditis in the week following vaccination against Covid-19 with mRNA vaccines in both males and females, in particular after the second dose of the mRNA-1273 vaccine. Future studies based on an extended period of observation will allow to investigate the risk related to the booster dose of the vaccines and monitoring the long-term consequences of these post vaccination acute inflammations.
Version 1 : Received: 21 June 2022 / Approved: 22 June 2022 / Online: 22 June 2022 (07:53:57 CEST)
How to cite: Mörz, M. A Case Report: Multifocal Necrotizing Encephalitis and Myocarditis after BNT162b2 mRNA Vaccination against Covid-19. Preprints2022, 2022060308 (doi: 10.20944/preprints202206.0308.v1). Mörz, M. A Case Report: Multifocal Necrotizing Encephalitis and Myocarditis after BNT162b2 mRNA Vaccination against Covid-19. Preprints 2022, 2022060308 (doi: 10.20944/preprints202206.0308.v1).
The current report represents a case of a 77-year-old man with Parkinson’s disease who died three weeks after receiving his third COVID-19 vaccination in January 2022. The patient was first vaccinated in May 2021 with the ChAdOx1 nCov- 19 vector vaccine, followed by two more doses with the BNT162b2 mRNA vaccine in July and December 2021. The family of the deceased requested an autopsy due to the ambivalent clinical features noted before death. The underlying illness (Parkinson’s disease) was confirmed by autopsy. However, no sign of a florid COVID-19 was discovered. Meanwhile, the immunohistochemical staining of the brain and heart revealed previously undiagnosed conditions. The brain, in distinctive, revealed multifocal necrotizing encephalitis with massive inflammatory lymphocyte infiltrates. In addition, the heart showed signs of serious myocarditis. Finally, immunohistochemical staining revealed that the SARS-CoV-2 spike protein was evident in the tissues investigated. Based on these immunohistochemical findings, it appears that the inflammatory changes in the patient’s brain tissues are most likely the result of immunological processes. Concurrently, the absence of SARS-CoV-2 nucleocapsid-protein was evidenced, indicating that the detected spike-protein is unrelated to a SARS-CoV-2 infection. If such an infection was the cause of the spike protein, the SARS-CoV-2 nucleocapsid protein would also be detectable. As a consequence, the confirmed presence of the spike protein had to be attributed to the previous vaccination with the BNT162b2 mRNA vaccine that the deceased patient had received.
Finding the spike protein in the tissues investigated puts to rest the fact it goes systemically into the body, and not only the injected arm. This was admitted by a vaccine researcher over a year ago but is still denied/ignored by ‘the powers that be’
It accumulates in ovaries, the liver, spleen, bone marrow, and adrenal glands
It can bind to receptors that are on our platelets & the cells that line our blood vessels
It can cause platelets to clump, leading to clotting, or bleeding which explains the many reports of heart issues after these injections
I ended up in the cardiac ICU. It was a rare complication of Lyme disease
I thought I was healthy — but when my heart rate dropped randomly, it seemed I’d need a pacemaker. Then a doctor suspected Lyme carditis.
I thought I was healthy so when I started to have signs of a heart block, I felt stunned that I had become so sick. I later learned that I had a rare complication of Lyme disease. Courtesy Eric Miller
By Eric Miller, as told to Meghan Holohan
Eric Miller, 43, teaches principles of design and fabrication, maker classes and programming at a boarding school in Northeastern Ohio. As he started his new position last summer, he experienced a health crisis and landed him in the cardiac ICU with a dangerous arrhythmia. Doctors soon learned that he had Lyme carditis, a rare complication of Lyme disease. He shared his story with TODAY.
During employee orientation for my new job, I started feeling really rundown in the evenings. At first, I dismissed it as stress and nerves from starting a new career. (See link for article)
My father developed end-stage heart failure in his early 60’s, despite decades of top cardiology care. They said heart transplant was his only hope—They were wrong. Treating an overlooked infection permanently fixed his heart, averting the need for heart transplant. He passed away on 5/9/2021, 1 year ago today, at almost 90, may he rest in peace. This work is dedicated to my Dad. May it save someone you love.
Heart failure is the heart’s inability to ably pump blood. When the heart muscle chronically weakens, it’s called cardiomyopathy. Myocarditis describes acute inflammation of the heart—Sometimes also causing heart failure.
A major cause of disability and death, the many causes of cardiomyopathy include coronary artery disease, hypertension, and diabetes. But up to 50% of cases (75% when it’s in kids!) are “idiopathic,” of unknown origin. I dislike that word. It makes me feel like the medical profession is trying to compensate for its ignorance by using a lofty Latin-derived term to …
(See link for article)
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The infection Dr. Phillips’ dad had was a tick-borne illness and is a perfect example of why treating the infection(s) is so important. As it stands, the ‘powers that be’ want us all to fall for the belief that those of us chronically/persistently infected are just dealing with some simple immune issues that need ironing out.
Well, that’s true in a sense because infections cause immune issues.
To sum it up best I will repeat what Dr. Hoffman (RIP) one of the most experienced WI LLMD’s told me on the issue:
If you treat the infection(s), most if not all symptoms just disappear.
This has certainly been the case for myself, my husband, and thousands of others, but this simple, logical fact is completely ignored by mainstream medicine and current scientific inquiry. It’s sad that we are still dealing with an issue Polly Murray writes about in the 70’s in “The Widening Circle,” a story about a woman’s observation of severe illness in her entire family and the Connecticut area in which they lived.
For more on tick-borne illness causing heart issues: