Archive for the ‘Pregnancy’ Category

Canada Acknowledges Maternal-Fetal Transmission of Lyme Disease


This Picture:  Left to Right – Steven Sternthal (Director General for Zoonosis Division), Dr. Howard Njoo (Deputy Chief Public Health Officer), Tamara House (Vice-President of Families and Children, LymeHope), Kim Elmslie (Vice-President Infectious Diseases Prevention and Control), Dr. Siddika Mithani (President, Public Health Agency of Canada), Jennifer Kravis (Co-Founder, LymeHope), Dr. Nick Ogden (Senior Research Scientist, Zoonosis Division PHAC), Jane Bailey (Lyme Advocate), Dr. Robbin Lindsay (Research Scientist PHAC, National Microbiology Laboratory) and Sue Faber (RN, Co-Founder of LymeHope).  We were also joined via teleconference with Dr. Charles Ray Jones, (Pediatric Lyme Specialist, New Haven Connecticut), Dr. Ralph Hawkins (Internal Medicine Physician), Dr. Lisa Waddell (Epidemiologist with the National Microbiology Laboratory, PHAC) and Dr. Matthew Gilmour (Scientific Director General of National Microbiology Laboratory, PHAC).

Breaking News: Canada Acknowledges Maternal-Fetal Transmission of Lyme Disease!


“What is the value of one child’s life?”
As Canadians, we soon will be coming together to celebrate a cherished national holiday Thanksgiving.   At LymeHope we are genuinely thankful for the incredible news which we will now share with you.

This news pertains to all Canadians suffering from or concerned about Lyme disease, but specifically those who are concerned about the issue of maternal-fetal (in-utero) infection also described as transplacental transmission.  Lyme disease is not only transmitted through a tick-bite, it can be passed from human-to-human, mother to child in pregnancy as reported in the scientific and medical literature.

This morning we met with senior scientists and executive officials at our Federal Public Health Agency of Canada as part of our ongoing discussions about transplacental transmission of Lyme disease.  We had started these discussions over 18 months ago with the Agency anchored in a respectful, transparent and collaborative approach – doors opened, we walked through them.

We were incredibly honoured to have U. S. Dr. Charles Ray Jones, MD, the world’s foremost expert in pediatric and adolescent tick-borne diseases join in this meeting.  Dr. Jones shared his experience of treating over 30,000 children, from every continent in the world and every Province of Canada, over 60 years of practicing medicine and saving lives. He talked about the hundreds of congenitally infected children he has seen, and more importantly, how he has been able to cure them of their tick-borne diseases and allow them to live healthy lives.  A short 1 minute video about Dr. Jones pioneering work here:

32 years ago, in 1988, our Public Health issued a bulletin stating that transplacental transmission of Lyme disease had been documented, and yet somehow, despite 30 years going by, the majority of Canadians and medical professionals remain unaware of this.  Even worse, many experts have stated, to patients and publicly it “doesn’t exist”.  We have been determined to change this.

Today, our senior most officials at Public Health acknowledged that transplacental transmission of Lyme disease can occur, AND that they will be updating their website to reflect this.

As mothers with suspected congenitally infected children, tears streamed down our face and hope rose in our hearts, that this truly is the beginning of a new era in Canada, a breath of fresh air.  This means mothers who are told “maternal-child transmission of Lyme disease doesn’t exist” can now update their medical teams of this acknowledgement by our very government, following on the heels of the recent acknowledgement of the World Health Organization.

We must urgently come together to do our very best thinking – an all hands-on-deck approach to research this alternate mode of transmission and help children and families currently affected, many of whom reach out to us personally on a day-to-day basis.

Each of you, through your signing and sharing this Petition, sharing your stories and comments, encouraging and touching our hearts, shares in this miraculous and well-deserved news.  Thank you from the bottom of our hearts.

LymeHope is already taking active steps to engage and assemble a multi-disciplinary working group of interested medical experts, scientists and meaningful patient representation.  We must now come together to re-examine this critical issue and help implement the necessary steps to create a new reality in Canada, where pregnant women and their medical professionals are aware of the risks of transplacental transfer and are taking steps to ensure our future generations are protected from these devastating diseases.  Dr. Jones today said he would be honoured to be a part of this initiative.

We have promised our own precious children and indeed the children of Canada that we will not stop our work until the day when they all are able to walk into a doctor’s office and receive appropriate care and treatment.  We are now a step closer to our dream becoming a reality.

There is much work to be done; however, today we want to share this wonderful news with you.  As we gather with our friends and family this weekend, we will be giving thanks for many things, including this first important step in changing the lives of Canadian mothers, babies and children.  We wish you a wonderful Thanksgiving weekend, and, as always, thank you for your continued engagement and support as change happens.

In closing, just as the two-hour meeting came to a close, Dr. Jones shared the last word.  He asked,

“what is the value of one child’s life?”  Without hesitation he answered with these words.  “It’s Priceless.”

We all agreed and so, we step forward with renewed hope.

Follow us on Twitter and Facebook @lymehopecanada

If you haven’t already, please continue to sign, comment and share this petition.


Sue Faber
Jennifer Kravis
Tamara House
(the “LymeMoms”)

# Thankful


For More:

Congenital Transmission of Lyme – Myth or Reality?

July 22, 2018



1.) Dr. Lapenta, J Medic Surgeon, Specialty Dermatology, 24 years of exercise. Highly trained in the field of research; University of Carabobo, Venezuela. CEO DERMAGIC EXPRESS.

2.) Dr. Lapenta, JM. Medic Surgeon. University of Carabobo. Diplomat in Facial Aesthetics Occupational Medicine and Prehospital Auxiliary. Resident Doctor Ambulatorio Del Norte Maracay Aragua State. COO DERMAGIC EXPRESS.


Lyme disease or Erythema Migrans, described many years ago, and previously known under the name of Lyme Juvenile Arthritis, is produced by a spirochete transmitted by the bite of a family tick. Ixodidade, Ixodes scapularis and many others; discovered by the scientist Willy Burgdorfer in the year of 1981, being named Borrelia Burgorferi, in honor of its discoverer. Apart from the numerous cutaneous and organic manifestations attributed to Borrelia, it is nowadays discussed in the scientific field if it is capable of crossing the placenta and causing fetal damage. In this review we will show you that this biological agent, as in syphilis, produced by Treponema Pallidum, another spirochete, crosses the placenta and can produce serious consequences to the fetus, including death.

Key words: Lyme disease, Borrelia burgdorferi, congenital lyme, fetal damage and pregnancy


The main objective of this work is to demonstrate that Lyme disease and their biological agent, spirochete Borrelia Burgdorferi, is not only an illness with cutaneous manifestations. It can cross the placenta during pregnancy and produce fetal damage that in severe cases can cause death in newborns.


1.) Describe the clinical manifestations in children born from positive Lyme mothers who did not receive treatment in pregnancy, or in those who received treatment with resistance to it.

2.) Alert the World community that there is indeed transplacental transmission of Borrelia Burgdorferi in pregnant Lyme positive women and if there is not adequate treated in time, both the mother and fetus can present clinical symptoms ranging from mild, to severe, even stillbirth.

3.) To call attention to the World Health Organization so that in the revision of the international codes of diseases (ICD-11), this year 2018, the code “Congenital Lyme” be  included in them.


The Center for Disease Control and Prevention (CDC) affirms in its website that the pregnant woman Lyme positive when making her treatment, the child will be born healthy and recommends for this, the use of the antibiotic amoxicillin or cefuroxime, because doxycycline, which is the antibiotic of choice, can cause damage to the developing fetus. [1]

Other antibiotics recommended by the CDC are the macrolides azithromycin, clarithromycin or erythromycin in case of allergy or intolerance to those previously mentioned. [2]

The CDC itself recognizes that Lyme disease and its causative agent Borrelia Burgdorferi can cross the placenta and cause stillbirths. [1-2]

The question here is what would happen if the Borrelia species, as in some cases, is resistant to amoxicillin or another antibiotic, or the antibiotic to which Borrelia is sensitive cannot be indicated because it would harm the fetus? And beyond, if the patient does not receive the treatment by omission or carelessness? [3]

As we said previously the Borrelia Burgdorferi was discovered by Willy Burgdorfer in the year 1981, and just two (2) year later, in 1983 the first study was published by Shirts SR, and Brown MS, Bobitt Jr, where it is suspected that this spirochete can cross the placenta. [4]

After this study others began to appear, who definitely showed that this spirochete is able to cross the placenta and cause fetal damage, of which we will present in chronological order the most important and confirm the above said.



The first suspicion described that ante partum fever may be caused by the Borrelia Burgdorferi species, was made in 1983 in two febrile pregnant women in the third-trimester. The two newborn survived, but the scientists suggested the establishment of early laboratory tests to identify the causative agent and the establishment of a rapid treatment to avoid future complications in the pregnant and the fetus. [4]


Really, the first study describing the maternal-fetal transmission of Lyme disease, Borrelia Burgdorferi was published in 1985 by Schlesinger PA, Duray PH, Burke BA, Steere AC, and Stillman MT., Where they describe a case of a pregnant woman who acquired Lyme Borreliosis and did not receive treatment with antibiotics. The child was born at 35 weeks of pregnancy and died of congenital heart disease the first week of life. The autopsy revealed the Borrelia Burgdorferi spirochete in the spleen, kidneys and bone marrow. [5]


In the year 1986, MacDonald A, describes 4 cases of abortions in pregnant women who tested positive for Lyme, and in whose fetuses the Borrelia Burgdorferi was found in their tissues. This same author does another work in 1989 about Lyme disease and its implications for the fetus and describes side effects such as fetal death, hydrocephalus, cardiovascular abnormalities, neonatal respiratory distress, hyperbilirubinemia, intrauterine growth retardation, cortical blindness, sudden death syndrome of the infant and maternal toxemia of pregnancy, and raises the similarity of these with neonatal syphilis. [6-7].


Later, the same Willy Burgdorfer the discoverer of the Borrelia Burgdorferi, together with Dr. Alan Mc Donald and Jorge Benach PhD, published in year 1987 (31 years ago) a work where they relate this disease with children born dead associated in pregnant Lyme positive.

Among the highlights the description of congenital malformations, fetal death, cardiac anomalies and alert the scientific community to investigate exposure during the first trimester of pregnancy in the presence of Borrelia Burgdorferi; and in this cases to determine if cardiac organogenesis is complete by the end of the first trimester of pregnancy. They also recommend starting treatment with Penicillin at the same dose of syphilis in those cases of pregnant women who show signs and early symptoms of the disease. [8]


In these 24 years a total of more than 80 papers were published where Lyme disease is effectively related to pregnancy with fetal damage, studies done in the countries: United States, Canada, Hungary, Germany, Italy, Switzerland, Africa, Turkey, Czech Republic, Poland and Belgrade former Yugoslavia, and others [10-52]

Another study that is worth noting is that carried out by the updated MEDLINE database for the year of July 2012, the last revision of November 2012 of 88 journal articles from the PUBMED database, which we summarize in this way

Maternal-fetal transmission of Lyme disease (Findings):

1.) Mothers with active Lyme disease, treated: 14.6% of pregnancies resulted in sequel (a morbid condition following or occurring as a consequence of having Lyme).

2.) Mothers with active Lyme disease not treated: 66.7% of pregnancies resulted in sequel.

3.) Positive Lyme mothers, the treatment is unknown: 30.3% resulted in sequel.

4.) Specific adverse results included:

– Cardiac 22.7%,

– Neurological 15.2%,

– Orthopedic 12.1%,

– Ophthalmologic 4.5%,

– Genitourinary 10.6%,

– Miscellaneous anomalies 12.1%. [53]

Now we will put a summary of the most frequent clinical manifestations described in a study of more than 100 children born to mothers with LYME positive disease, conducted in the year 2005.


1.) Low grade fever: 59% -60%
2.) Fatigue and lack of resistance: 72%
3.) Nocturnal sweating: 23%
4.) Pale, dark circle under the eyes: 42%
5.) Abdominal pain: 20-29%
6.) Diarrhea or constipation: 32%
7.) Nausea: 23%
8.) Cardiac anomalies: 23%: palpitations / PVC, heart murmur, mitral valve prolapse.
9.) Orthopedic disorders: sensitivity (55%), pain (69%) spasms and generalized muscle pain (69%), rigidity and / or retarded motion (23%).
10.) Respiratory infections of the superior tract and otitis: 40%
11.) Arthritic disorders and painful joints: 6% -50-%
12.) Neurological disorders:
A- Headaches: 50%
B-) Irritability: 54%.
C-) Bad memory: 39%
13.) Delay in development: 18%
14.) Seizure disorder: 11%
15.) Vertigo: 30%
16.) Tic disorders: 14%
17.) Involuntary athetoid movements: 9%.
18.) Earning disorders and humor changes: 80%
A-) Cognitive speaking: 27%
B-) Speech delay: 21%
C-) Reading-writing problems: 19%
D.) Problems of vocal articulation: 17%.
E-) Auditory / visual processing problems: 13%
F-) Word selection problems: 12%
G-) Dyslexia: 8%
19.) Suicidal thoughts: 7%
20.) Anxiety: 21%
21.) Anger or rage: 23%
22.) Aggression or violence: 13%
23.) Irritability: 54% -80%
24.) Emotional disorders: 13%
25.) Depression: 13%
26.) Hyperactivity: 36%
27.) Photophobia: 40-43%
28.) Gastroesophageal reflux with vomiting and coughing: 40%
29.) Secondary eruptions: 23%
30.) Other eruptions: 45%
31.) Cavernous haemangioma: 30%
32.) Ocular problems: posterior cataracts, myopia, stigmatism, conjunctive erythema (Lyme eyes), optical nerve atrophy and / of uveitis: 30%
33.) Sensitivity of skin and noise (hyperacuity): 36-40%
46.) Autism: (9%). [41]


In recent years the number of cases of Lyme disease has increased notably in North America, Europe, Asia and Africa; in the United States and Canada, it is the most commonly transmitted vector-borne disease reported today [54-62].

For the year 2017 the World Health Organization (WHO) in charge of “coding” diseases through the ICD-10 (International Classification of Diseases year 2.017), had not yet included and recognized the code “Congenital Lyme”. It is expected for this year 2018 that all codes of Lyme disease will be recognized by this organization. [63-68]

You can read this classification and codes here:  UNDERSTANDIG THE LYME DISEASE CLASSIFICATION AND CODES. [69]


1.) Lyme disease is caused by the bite of a tick transmitted by the Borrelia Burgdorferi spirochete, discovered by Willy Burgdorfer in the year 1981. Initially, skin, joint and cardiac manifestations were described in those affected, but not in pregnant women or the fetus.

2.) Two years later, in 1983, was described the suspicion of infection in pregnant women, and in 1985 it was found the first clinical manifestations in pregnant women and fetuses, highlighting congenital malformations and fetal death.

3.) We demonstrate scientifically that definitely Lyme disease, in addition to its multiple organic manifestations, its causative agent Borrelia Burgdorferi, crosses the placenta and reaches the fetus in pregnant women, producing the side effects already described.

4.) All pregnant women living in endemic areas of Lyme disease should take the tests to rule out Borreliosis of pregnancy, to establish immediate treatment in case of being positive.

5.) In addition to establishing adequate treatment, we alert the population to defend against the bite of possibly infected ticks.

6.) We urge the World Health Organization to recognize all the codes and classification of Lyme disease in ICD-11 (International Classification of Diseases year 2.018).


To the community of patients affected by Lyme disease.

To the organizations that fight for the recognition of this pathology as a public health problem World.



The Lyme community owes a debt of gratitude to the doctors Lapenta.  You will see many of their articles on my website as they have painstakingly combed through the research and condensed it for laypeople to clearly see the ramifications of Lyme and other tick borne illnesses.

This article makes plain that Lyme can be passed congenitally to infants.

For more on pregnancy with Lyme:




33 Years of Documentation of Maternal-Child Transmission of Lyme Disease and Congenital Lyme Borreliosis – A Review

33 Years of Documentation of Maternal-Child Transmission of Lyme Disease and Congenital Lyme Borreliosis – A Review

by Sue Faber, RN, BScN


‘Transplacental transmission, adverse outcomes and reports of congenital infection of Borrelia Burgdorferi have been clearly documented over the last 33 years (1985 to 2018) by multiple international physicians, researchers, scientists and other experts. As entire families worldwide are affected by Lyme borreliosis resulting in serious debilitating illness and complex multi-systemic chronic infection, we must take this alternate mode of transmission – from mother to child in pregnancy, seriously.

For Lyme disease to be passed from mother to child in pregnancy drastically changes the narrative, we know that, it opens up new issues and challenges – however, recognizing it for what it is, is the right thing to do. It means upheaval and reordering and re-prioritizing in what has been taught and rethinking many areas of concern which perhaps have been looked over – but we must remember – we have no choice but to act with the highest integrity and honesty.

We have no option but to constructively engage, discuss and determine solutions and a clear path forward which will be a light for those who suffer, a beacon of Hope and healing. We need to prevent more miscarriages, stillbirths and babies from being born with Lyme and tick-borne illnesses – potentially leading to chronic pervasive, persistent and often disabling illness’. Sue Faber, RN.  (Excerpt below.  Please see link for more studies)

“Now we have found a spirochete capable of spreading transplacentally to the organs of the fetus, causing congenital heart disease and possible death of the infant.”

Dr. Willy Burgdorfer – The Enlarging Spectrum of Tick-Borne Spirochetoses: R. R. Parker Memorial Address, Reviews of Infectious Diseases. Vol 8, No 6. November-December 1986.

“It is clear that B. Burgdorferi can be transmitted in the blood of infected pregnant women across the placenta into the fetus. This has now been documented with resultant congenital infections and fetal demise. Spirochetes can be recovered or seen in infant’s tissues including the brain, spleen and kidney. The chronic villi of the placenta show and increase in Hofbauer cells as in luetic placentitis. Inflammatory changes of fetal or neonatal changes are not as pronounced as in the adult, but cardiac abnormalities, including intracardiac septal defects, have been seen. It is not known why inflammatory cells are so sparse from maternal transmission, but it is possible that an immature immune system plays a role.”

Dr Paul Duray and Dr Allen Steere – Clinical Pathologic Correlations of Lyme Disease by Stage. 1988.

“It is anticipated that more infants and fetuses with complications related to gestational Lyme borreliosis will be diagnosed in the future as the diagnosis is more frequently considered; it eventually will be possible to better describe the various clinical manifestations of congenital Lyme borreliosis.

“ order for infants with congenital Lyme borreliosis and therefore initiation of prompt antibiotic therapy of the congenitally infected infant usually depend on suspicion or confirmation of Lyme borreliosis in the mother. Therefore, in order for infants with congenital Lyme borreliosis to be recognized, it is essential for clinicians caring for newborns and infants to become familiar with the various manifestations of Lyme borreliosis in the adult, as well as in the congenitally infected infant.”

(serology) does not appear to be a sensitive method of diagnosis and reliance on sero-positivity leads to misdiagnosis of the majority of congenitally infected infants.”

“Large-scale, prospective studies of sufficient numbers of patients with Lyme borreliosis with follow-up to determine the pregnancy outcome of each enrolled patient; B burgdorferi-specific evaluation of any fetal or neonatal demise; and long-term follow-up of each infant born to determine the occurrence of possible early and late sequelae are needed.”

Dr. Tessa Gardner, Pediatric Infectious Disease MD, trained at Harvard University, 2001. Gardner, T. Lyme disease. In: Remington JK, J. editor. Infectious Diseases of the Fetus and Newborn. 5th ed: Saunders; 2001

“Transmission of Borrelia infection occurs via both zoonotic vectors and other humans. Congenital transfer is an established fact. Maternal to fetal transfer of Borrelia, can furthermore be clinically silent or unrecognized, and if not successfully treated, infection can be life long and latency, late activation and reactivation can occur.”

O’Brien J, Hamidi O. Lyme Disease ( Infection with Borrelia: Implications for Pregnancy, Nov. 2017.

“Intra-human transfer of Borrelia can be initially silent or unrecognized’

‘The similarities of the clinical presentation of congenital syphilis to pregnancies with acute Lyme disease helps guide ante partum management. Due to the severity of previously documented cases, there should be a low threshold of suspicion to diagnose cases of Lyme disease in pregnancy.

O’Brien J, Hamidi O. ‘Borreliosis Infection during Pregnancy’. Ann Clin Cytol Pathol 3(8). Oct. 2017.

“Pregnant women who are acutely infected with Borrelia burgdorferi (the primary cause of Lyme disease) and do not receive treatment have experienced multiple adverse pregnancy outcomes including preterm delivery, infants born with rash and stillbirth.”

“In obstetric patients acutely infected during the first trimester, a fetal echocardiogram is reasonable, given the demonstrated high potential of fetal cardiac abnormalities.’

O’Brien J, Baum, J. Case Report. The Journal of Family Practice. Vol 66, No 8, Aug, 2017.

“It was stated and proved transplacental transfer of borrelia

“We need serious studies among pregnant women and newborn children in endemic regions…and in the future such patients should be monitored throughout pregnancy and after childbirth. Children born to these women should be examined for tick-borne infections at least during the first two years of life.”

Utenkova EO. Lyme disease and Pregnancy. Kirov State Medical Academy, Kirov Russia. Journal of Infectology, Volume 8, Number 2, 2016. *translated from Russian

“a new acronym is needed to include other, well-described cause of in utero infection: syphilis, enteroviruses, varicella zoster virus, HIV, Lyme disease (Borrelia burgdorferi) and parvovirus.”

In utero infection and intrapartum infections may lead to late-onset disease. Such infections may not be apparent at birth but may manifest with signs and symptoms weeks, months or years later.”

Maldonado Y, Nizet V, Klein J et al. Current Concepts of Infections of the Fetus and Newborn Infant (Chapter 1). Found in Remington and Klein’s Infectious Diseases of the Fetus and Newborn Infant, 8th ed., 2016.

“Histological observations have confirmed the presence of Bb in children with congenital Lyme disease. It is interesting that spirochetes may exist in the spleen, kidney, bone marrow and nervous system.”

“The ability of long term survival of Bb sl in tissues and spreading of spirochetes in the body despite antibiotic treatment can contribute to intergenerational infection with Lyme disease.”

Jasik K, Okla H, Slodki J et al. Congenital Tick-Borne Diseases, is this an alternative route of transmission of tick- borne pathogens in mammals? Vector- Borne and Zoonotic Diseases, Volume 15, Number 11, 2015.

“these documented cases strongly suggest that transplacental transfer occurred via identification of Borrelia Burgdorferi in fetal tissues by culture, immunohistochemistry or indirect immunofluorescence.”

“the outcome of a pregnancy affected by Lyme disease remains relatively unknown and unstudied. However, it is still important to equip obstetrical patients with information that will help protect them against Lyme disease and provide treatment options if a suspected case of Lyme disease occurs during pregnancy.”

O’Brien JM, Martens MG. Lyme disease in Pregnancy, a New Jersey Medical Advisory. MD Advisor, 2014;7:24- 27.

Borrelia Burgdorferi does appear to cross the placenta and infect the fetus. There are data to suggest an increased incidence of spontaneous abortion, stillbirth and congenital malformations associated with Lyme disease.”

“Adverse pregnancy outcomes are also more likely in women with untreated Lyme disease.”

Dotters-Katz S, Kuller J, Heine P. Arthropod-Borne Bacterial Diseases in Pregnancy. Obstetrical and Gynecological Survey, Vol 68(9). 2013.

“The parents of the five children in the study could not pinpoint an exact date of infection, but their treating physician suggested that the Bb bacteria could have been transmitted congenitally since all five of their mothers were diagnosed with Lyme disease and Bb has been shown to be transmitted congenitally in infected mothers. If the Bb bacteria were transmitted congenitally and this latency period presented itself in the infected children it could lead to an explanation of their late onset autistic symptomology.”

Kuhn M, Grave S, Bransfield R, Harris S. Long term antibiotics therapy may be an effective treatment for children co-morbid with Lyme Disease and Autism Spectrum Disorder. Medical Hypothesis (2012)

“The clinical picture of a fetus infected by B Burgdorferi is similar to that seen in the course of a syphilis infection. Most frequently they are: premature birth, intrauterine foetus death and malformation

“In the second stage of the illness, B. Burgdorferi traverses the placental barrier. Apart from foetal death, the following occur most frequently: syndactyly, sight loss, premature birth, neonatal rash, heart, liver, kidney damage or damage to the central nervous system.”

Relic, M, Relic, G. Lyme borreliosis and pregnancy. Vojnosanit Pregl 2012; 69(1):994-998. *translated from Polish

For more:




A Tale of 3 Metals, the Fate of Western Civilization, & What We Can Do About it

A Tale of Three Metals and The Fate of Western Civilization

by jameslyonsweiler



The Romans drank beverages prepared in lead vessels, and brought spring water into their homes through lead pipes. Lead poisoning undoubtedly hastened the fall of the Roman empire. So when we think about the evidence that we are harming ourselves, and our children, with lead in the water, mercury in the air, mercury in flu vaccines, and aluminum in many other vaccines, one has to wonder: what are the likely effects on society?

  1. African Americans will suffer the most. Due to Vitamin D deficiency, African Americans at northern latitudes can be expected to be most sensitive to toxins because they rely on dietary Vitamin D to drive their cellular detoxification systems. The fix? Measure blood Vitamin D levels, and absent any mutation that would preclude increased doses of Vitamin D, improve brain health via addition Vitamin D supplementation.
  2. Young adults (millenials) will have different sociality, and higher rates of early-age psychological disorders such as schizophrenia. They may also experience higher rates of early age of onset Parkison’s disease, Alzheimer’s disease, and other neurodegenerative diseases. The fix? Filter aluminum out of the water, try silica-rich mineral waters, silica drops, with a preference for sources with the more biologically available silicic acid. In short, detoxify their food, water, and everything in their environment, and more (see below).
  3. Young children with special education needs will tend to be more violent and brain studies will show increased presence of amyloid precursor protein, the kind responsible for Alzheimer’s disease.
  4. There will be a population-wide downward shift in IQ.
  5. There will be a plague of multiple chemical sensitivity.
  6. Academics will be stretched thin and the curriculum dumbed down to the point where schools will have to stop giving grades. When 20% of the class can no longer function academically to take and exam, the rest will be asked to “help” their classmates learn.
  7. Families will become increasingly stressful social units. Divorce rates will skyrocket.
  8. People will become increasingly dependent on the State (Nanny State).
  9. Those most able to withstand the toxic effects of accumulating neurotoxins will become increasingly taxed because their income and property will have to sustain an increasingly demanding medico-government empire.
  10. When they, too, begin to fall apart, the tax base will falter.
  11. Violence will become increasingly common. Those most damaged will tend to kill and injure those who are capable.
  12. America will tear itself apart from within.

This doomsday scenario is not inevitable. So what can we do to prevent this?

  1. Listen to the mothers. They have experience in what works. NIH has avoided real research on neurodevelopment disorders that address neurotoxic metal exposure since the CDC worked so hard to defraud the public on the vaccine/autism link. They gambled, lost, and we now pay the cost.
  2. These solutions must be tested in combinations in clinical studies to insure safety, and also to validate them (if they do help). They must be studied NOW, before it’s too late.

Option 1. Environmental Detoxification. Remove all neotoxins from your home. Use reverse osmosis water filters, and use filtered water for everything – even cooking – because aluminum is used to condition the water coming from the tap. Fluoride is another issue, and your filtration should also remove fluoride. Eat organic foods and nothing out of aluminum containers. Certainly never cook in aluminum pots.

Option 2. Get the Aluminum Out. Consider using high silicic acid mineral water, or adding silicic acid drops to your filtered water to bind any aluminum from food. Other possibilities include malic acid, magnesium, and acetoacetic acid:

Principles of Orthomolecularism. R.A.S. Hemat: “Aluminum can be effectively complexed and excreted with silicon, a complex of malic acid and mg, and acetoacetic acid.”

Precise combinations that work best and are safe are not yet determined. That’s why we need studies.

Doctor Toni Bark, MD informs me that ketogenic diet can also help reduce brain inflammation and reduce the effects of toxic metals from the body and the brain – including the reduction of brain amyloid. And Dr. Richard Frey’s research on intranasal insulin and intranasal deferoxamine seems very promising for the actual removal of iron and aluminum from the brain. Care should be taken to conduct any such research under the direct care of a physician.

Option 3. Up the Vitamin D3, watch the A, Avoid Folic Acid. Dr. Keith Baggerly, MD, has determined that the FDA flubbed in it recommended daily Vit D intake:–+K+Baggerly+2016-2017 As a result, most Americans are Vit D deficient. Increased Vitamin D3 can be expected to improve many aspects of health by helping our cells properly fold proteins. Vits A and D are antagonistic, and so watch all sources of Vit A and make sure you and your child are not taking in too much Vitamin A. Read The Big Vitamin D Mistake: and Grant Genereux’s resources on Vit A toxicity [1] [2].

Much of our population has MTHFR mutations that cause problems with Folic Acid. Moms taking prenatal vitamins should seek methyl folate or folinic acid: instead of folic acid. Children’s vitamins with methyl folate are also available.

Option 5. Reduce Brain Inflammation. Chronic low-grade inflammation is a hallmark of autism. Powerful brain antioxidants include N-acetylcysteine and glutathione. It seems likely that everyone with a brain could benefit from less brain inflammation.

Option 6. Improve the Gut. The commensal (helpful) bacteria in the large intestine can become significantly altered after antibiotic use to treat ear infections, most likely caused by harm from to the immune system from thimerosal. Pro-biotics may help, as will eating organic.

Option 7. Keep This Handy for Bad Head Days. Brain dysfunction from metal-induce excitotoxicity involves high glutmate levels in the brain. Oxaloacetate can reduce blood glutamate levels, allowing the excess glutamate in the brain to spill into the blood. Oxaloacetate is used after stroke to reduce the exitotoxic brain injury. Research is needed to determine if it should be used after vaccination to reduce the incidence of vaccine-induced excitotoxicity. And aluminum should be removed from vaccines because the schedule results in toxic doses in infants.

Option 8. HBOT is HOT. Consider Hyperbaric Oxygen Therapy (HBOT). HBOT can increase de novo neurogenesis. If the brain has suffered a loss of neurons due to toxic exposure, increased neurogenesis – at the right time in development – could ultimately be shown to increase IQ.

Option 9. Avoid Thimerosal. If you choose to use a flu vaccination, ask the doctor for the type of flu shot that does not contain thimerosal.

Option 10. Tell Congress You Want Research Reform.

No studies of the synergistic toxicity of aluminum, lead and mercury have been conducted at doses reflecting the vaccine schedule and daily exposure due to leaching of lead from pipes into homes.

We know which homes have lead pipes. Departments of Health should consider telling parents of children in those homes to avoid exposures to mercury and to aluminum – in other words, to skip vaccines that contain these neurotoxic metals. The children will become more educated, better behaved, make better decisions, commit fewer crimes, and overall have better lives. Toxicity of lead, aluminum and mercury is synergistic.

No studies of the options and combinations of options listed above have been conducted to determine if we could improve overall brain health in children and adults. This research is badly needed. YOU can make it happen.

Make an appointment with your Congressional Representative and ask them to create the Brain Health 2030 initiative designed to reverse the ill effects of the past 30 years of industry and medicine on brains, and on our childrens’ brains. These interventions are not intrusive. Studies could be done also with the Department of Education to determine whether reports of violence decrease, grades increase, drop-out rates decrease if entire SCHOOLS – including administrators – are enrolled in Healthy Brain programs, which could incorporate aspects of mindfulness.

You can join the Neurodevelopment Research Reform group on Facebook where ideas on the Brain Health 2030 initiative are shared. And you can support our efforts to compile the most promising evidence-based approaches to improving brain health by supporting IPAK’s Neurodevelopment Research Reform Initiative.

This article is a call for research reform. These statements have not been evaluated by the Food and Drug Administration. This product is not intended to diagnose, treat, cure, or prevent any disease. Check with your physician before changing any mode of medical treatment for your child, or yourself.

Further Reading:

Lyons-Weiler, J and R. Ricketson. 2018. Reconsideration of the Immunotherapeutic Pediatric Safe Dose Levels of Aluminum. Journal and Trace Elements in Medicine and Biology 48:67 73.

Thanks to Tim Lundeen for the Vit A information and of course to the outstanding medical doctor, Dr. Toni Bark, MD for permission to cite her expertise.

Dr. Lyons-Weiler is a research scientist and author of three books, the latest of which is “The Environmental and Genetic Causes of Autism”. He is available for speaking engagements and book signing events at your location. To contact, follow on twitter @lifebiomedguru, email ebolapromo[at], and connect via LinkedIn


For more:





The Eleven Year Old Boy Who Knows More About Lyme/MSIDS Than Most Doctors

  Approx. 6.5 Min

Published on Mar 6, 2018

An introduction to Lyme Disease, Symptoms, Treatment, Prevention, and challenges from the perspective of an 11 year old who started his own advocacy group. The information has been vetted for accuracy by biologists and doctors working in Lyme research and treatment.
Removal of Tick using tweezers:… (University of Manitoba)
Please check out these Lyme Organizations and Friends:

LivLyme Foundation: LymeHope: International Lyme and Associated Disease Society: G. Magnotta Foundation Lyme Out Loud Kids:…



I am so impressed.  Kudos to Daniel Stimers for speaking out for patients too weak to do so themselves!

Don’t you find it a bit sad when a kid knows more than most doctors regarding the most common vector borne disease in the U.S.?



Lyme/MSIDS patients are always looking for relief.  The pain we endure is indescribable.  We clearly understand most of the pathogens we are dealing with are persistent, despite the CDC/IDSA/NIH denial. Our experience shows that we function well off treatment for a while and then the dreaded symptoms return. We cycle in and out of treatments which are expensive, time consuming, and often have significant blow-back. While antibiotics kill or disable bacteria, we all know they disrupt the microflora of the gut and damage mitochondria. Similarly to other treatments for other diseases, say cancer for instance, sometimes the treatment is as bad as the disease and some are affected more negatively than others.

This article is about two substances that you should learn about and discuss with your medical practitioner. These two substances are described as “therapeutic principles,” – not drugs.  In many aspects they are quite similar and one is derived from the other.

It’s important to condense and overlap treatment modalities as much as possible for two reasons:  time and money.  Another lesson Lyme/MSIDS patients learn is this disease can take over your life and cost you everything you have since it’s a long-term treatment – perhaps life-long.  I believe the substances in this article to be worth real consideration as they do so many things simultaneously, and are cheap & effective for most who use them.

As always, this article is meant for educational purposes only and not meant in any way to diagnose or treat.


Dimethyl Sulfoxide (DMSO) is a colorless, odorless, transparent substance obtained from wood; however, small amounts are naturally present in common foods such as milk, tomatoes, tea, coffee, & beer, among others.

First discovered by Russian chemist, Alexander Mikhaylovich Zaitsev in 1867, today DMSO is obtained as an industrial by-product from lignin in paper manufacturing. Because of its polarity and low acidity, it is a highly aprotic (doesn’t yield protons) solvent that can be mixed with other substances to increase their effect. It’s ability to penetrate biological membranes and transport other substances with it has also made it an excellent carrier.

Hartmut P.S. Fischer has explained in great detail in his groundbreaking book, “The DMSO Handbook: A New Paradigm in Healthcare,” the structure of DMSO and its chemical properties as well as the following brief summary of pharmacological properties on humans and animals:

*regenerator *anti-inflammatory *analgesic *diuretic *loosens connective tissue *penetrator *carrier *protector *modulator *relaxant *vasodilatory *antioxidant *anticoagulant *healing *anti-sclerotic *oxygenator *anti-anaemic *induces histamine release by mast cells

These qualities play out in reducing pain, alleviating inflammation, diuresis, vascular dilation, free radical scavenging, wound healing, and muscle relaxation. All issues Lyme/MSIDS patients deal with at some point.

The father of the medical use of DMSO, Dr. Stanley Jacob, enjoyed notoriety in the 60’s but admits the reception was short-lived due to DMSO’s long list of pharmacological properties and its being labelled a “miracle medicine.”  In other words it did too many things to be taken seriously.  The FDA approved DMSO for preservation of stem cells, bone marrow cells and organs for transplant, as well as for therapy of interstitial cystitis and cancer radiation protection – by prescription.  It’s also used under medical supervision to treat several other conditions, including shingles.  DMSO is available without a prescription in gel, cream, or liquid forms. It can be purchased in health food stores, by mail order, and on the Internet.  Great article explaining MTHFR & its relation to Lyme/MSIDS and the fact some patients can not properly detox.  Here’s another:

MSM, which I explain next and is derived from DMSO, is considered a methyl donor and reduces homocysteine levels suggesting a role in the methylation process and in reducing oxidative stress.  Since many patients struggle with high homocysteine which leads to inflammation and neurological problems, this is another boon for Lyme/MSIDS.

Speaking of Methyl donors, I would be amiss if I didn’t mention their importance in the issues of mood & energy:  (I have no affiliation with any products)

For an excellent list of DMSO studies:

A “60-Minutes” interview with Jacob on DMSO, one of the most researched substances in history:

Approx. 16 Min.

A 2014 video by Integrative Medicine Orlando on DMSO IV Therapy

Approx. 2 Min

DMSO has been used for cancer and an article by Camelot Cancer Care states the reason it protects against radiation damage and side-effects of traditional cancer treatments is due to how it stimulates parts of the immune system and scavenges hydroxyl radicals that promote tumor growth.  Since this usage is considered “off label,” insurance companies can not be billed for it.  Go to link for more info on how it’s used for cancer.  Review Article:  Medicinal Use of DMSO

  • Cutaneous manifestations of scleroderma (an autoimmune rheumatic disease) appear to revolve following topical applications
  • IV DMSO may benefit amyloidosis (an abnormal protein builds up in tissues & organs)
  • Dermal application provides rapid, temporary, relief of pain in arthritis and connective tissue injuries
  • Animal studies indicate IV DMSO is as effective as mannitol & dexamethasone in reversing cerebral edema (brain swelling) and intracranial hypertension (a neurological disorder where cerebrospinal fluid pressure is high in the skull)– a human clinical trail in 11 patients supports this
  • DMSO is used with mixtures of idoxuridine in the UK for topical treatment of herpes zoster
  • Adverse reactions are related to the concentration of DMSO and are usually minor  This article explains how DMSO helps head trauma:

According to Dr. Jacob, “DMSO is a potent free-radical scavenger and diuretic that reduces swelling and improves blood supply to the brain… “we observed that when the human brain was treated with intravenously administered DMSO after a head injury, the swelling could be reduced within five minutes.  No other treatment comes close to acting that quickly.

Astonishingly, however, the Food and Drug Administration (FDA) has not approved any new pharmacological agent of significance for the treatment of traumatic brain injury in more than three decades. With so much attention focused on the plight of severely injured soldiers returning home from war, Dr. Jacob is leading the charge to gain FDA approval of DMSO to treat this type of injury. He believes that DMSO would be more effective than some current therapies such as removing parts of the brain to reduce swelling.

Dr. Jacob and his colleagues previously sponsored preliminary clinical trials of DMSO on traumatic brain injury patients in Europe. The results of the trial were remarkable, with an 80% survival rate (about twice the historical rate of 30-40%) and 70% of the patients experiencing a favorable outcome (far higher than the historical rate of less than 10%).1  The effects of DMSO make it potentially useful in the treatment of medical disorders involving head and spinal cord injury, stroke, memory dysfunction, and ischemic heart disease.

How’s all this relate to Lyme/MSIDS?  Glad you asked.

First, many with neuro-Lyme have brain swelling.  Excruciating head pain is a hallmark symptom – often worsened with Babesia & other coinfections.  Second, many Lyme/MSIDS patients suffer with various viruses – the herpes virus being one.  Some patients struggle with dermal issues such as Morgellons and various rashes.  Third, nearly all patients suffer with inflammationpain.  Pain, pain, pain and more pain.  Fourth, DMSO is a strong anti-oxidant and powerful free radical scavenger.  Both are helpful for immune health and fighting pathogens.  Fifth, DMSO is known as the “Supreme detoxifier,” and assists in heavy metal detoxification.  If patients can’t detox all they are killing, they will not improve. For more on this important issue:

Fischer has a section in his book about DMSO usage and Lyme. He, unlike the CDC/IDSA/NIH, acknowledges that the borrelia bacteria is very persistent and that standard treatment is either very long and/or mostly unsuccessful.  He recommends a combination of an oxidative bactericide (MMS, MMS2, or hydrogen peroxide) and DMSO as a carrier to ensure penetration into the favorite hiding places of borrelia as well as deals with the various forms and stages of the pathogen.  DMSO is an excellent detoxifier which is a boon for eliminating the endotoxins caused by borrelia when it is destroyed.  This last element is just as important as killing organisms as the die-off can make patients miserable and cause significant symptoms.  Whatever bactericide is used needs to be pure as DMSO will take it directly into the body.

IF your brain works like mine you are thinking, “If DMSO is such a great carrier, why don’t we use this with antibiotics to make them more effective?”  Great question.  Ask your doctor.  It could also be said that it could take essential oils into the body as well…..  See where this is going?  It also penetrates the blood, brain barrier.

While Fischer recommends combining DMSO with an oxidizer for Lyme, DMSO alone is bacteriostatic, antiviral, and antifungal.  Between 30-40% aqueous solutions have inhibited the growth of Pseudomonas, Staphylococcus aureus, and Escherichia coli.  Other tests have proven diluted DMSO fight bacteria, viruses, and fungi and improves distribution of other antimicrobial substances and enhances their effects.

Speaking of oxidizers, I want to share how ozonated olive oil completely healed up a basal cell carcinoma MOHS surgery.  I was told I’d need two successive plastic surgeries but I used ozonated olive oil with a tad of petroleum jelly to fix it into place.  See results:

 Approx 2 Min.

You can clearly see how the ozonated olive oil infuses surrounding tissue with oxygen and heals the wound from the inside out.  I’m currently using ozonated olive oil with a tad of DMSO on another suspicious spot.  I’ll keep you informed of the outcome….

Safety:  DMSO has a LD50 value and is safer than ibuprofen, aspirin, caffeine, and cooking salt!  Study after study has proven its safety record on both animals and humans.  If you search for DMSO on the U.S. National Library of Medicine (, you’ll get almost 30,000 indexed results, making it one of the most studied compounds of our time. Yet, we are led to believe that DMSO can’t pass the required regulations for its approval in other medical conditions even though its effectiveness and low toxicity profile is unquestionable.

The above link also completely refutes a 1960 animal study that DMSO caused eye lens trouble.  In fact:

As far as eyes are concerned, the evidence on DMSO is quite to the contrary. When several patients treated with DMSO for muscular problems reported to Dr. Jacob that their vision had improved, he sent them to Dr. Robert O. Hill, ophthalmologist at the University of Oregon Medical School. Confirming the favorable changes, Dr. Hill began his own experiments with DMSO (after it was known that the lens changes did not happen in humans). His research showed drops of 50% DMSO to be effective in retinitis pigmentosa and macular degeneration, and presented a report on this at the New York Academy of Sciences symposium in 1971. (Haley, 2000)  Great read on the importance of purity and cleanliness.  Please read.

Regarding pregnancy:

Dimethyl sulfoxide caused teratogenic responses in animals when administered intraperitoneally at high doses. Oral doses did not cause problems of reproduction in animals. In one study topical doses produced terata in animals, but in another study topical dose failed to produce any abnormalities. There are no controlled data in human pregnancy. FDA pregnancy category C: Animal reproduction studies have shown an adverse effect on the fetus and there are no adequate and well-controlled studies in humans, but potential benefits may warrant use of the drug in pregnant women despite potential risks.

Contraindications:  According to Jacob in “The Miracle of MSM The Natural Solution For Pain,” DMSO has been found to counteract platelet aggregation.  He advises caution for those taking blood thinning agents such as heparin, aspirin, or dicumarol.  Accelerated blood thinning can not be ruled out.  Indications that that is occurring would be bruising or increased bleeding from hemorrhoids.

Now for the tricky part…….

When applied externally DMSO opens capillaries.  This can lead to temporary redness of the skin.  It can also burn and itch.  Evidently, not everyone experiences this but I did.  To me it felt a lot like taking niacin.  Wowza, but worth it.


The reduction in pain and swelling will be dramatic.  It’s worth the initial discomfort.  For me the pain went away in seconds.  Other patients have told me it took a number of days for noticeable improvement.

The next tricky part is understanding how DMSO breaks down into components.  Most of the DMSO will be broken down to MSM in the body; however, there is a small part (about 1%) that breaks down into a substance that causes a temporary odor.  Some say it smells like garlic.  I say it smells like oysters.

The viscosity of DMSO is double that of water which means it drips off the skin easily which is why many like gels and lotions; however, Fischer cautions that gels/lotions are made from raw materials such as polyacrylic acid derivatives which will be taken directly into the body.  He recommends the pure DMSO liquid that will  take a tad longer to be absorbed than the lotions.  If you choose gels or creams make sure you are confident about ALL the ingredients as they will go directly into your body.  Please keep this in mind.  Essential oils, antibiotics, anything applied with DMSO needs to be PURE.  Also, many EO’s can cause skin irritation so you need to work with a knowledgable practitioner.  I include a recipe on how to make your own DMSO gel a bit further down in the article.

And that’s another point that needs to be made.  DMSO MUST COMPLETELY DRY before anything touches it.  Again, because DMSO is a solvent/carrier, it will take everything and anything directly into your body such as perfumes, dyes, germs, etc.  So application must be done over an unbleached white towel, with clean hands and instruments and allowed to completely dry before covering with clothing or anything else.  Also note what the DMSO is stored in.  I would only get DMSO stored in glass as immune disruptors in plastics can leach into your body.

The next tricky part: Just like you will find detractors for DMSO, you will find detractors on MMS and the other oxidizers Fischer recommends for Lyme/MSIDS.  This is where you need to do your own research, talk to plenty of health professionals and make up your own mind.  Perhaps I’ll take on the subject of oxidizers in another article, but for now I’m just going to cover DMSO and MSM.

For a great read on why some put DMSO down:

In essence, between the fact it does so many things which would interfere with the sale of many other drugs, and the fact it smells a bit, which makes it virtually impossible to complete double blind studies as the smell would give it away, big pharma tossed it to the sidelines.  But read the article for yourself as there’s great info in it, including what it can do for you.  **Update on smell**  Recently I applied the pain gel on a larger area on my husband a couple times a day for a few days (listed below) on my husband.  Our kids stated, “What’s that old lettuce-like smell?”  After searching throughout the house we figured out it was my husband.  So it’s not horrible but it’s noticeable and if you are in a cramped office space, trust me, they’ll smell it.  But, desperate times turn to desperate measures, right?


by Gabriela Segura, M.D.

Quick Guide and Ailments

DMSO is generally applied to the skin in a gel, cream, or liquid. It can be taken by mouth or as an intravenous injection, in many cases along with other drugs. It has also been administered subcutaneously, intramuscularly, intraperitoneally, intrathecally, by inhalation, instilled into the eye, on the mucous membranes, and into the urinary bladder. Strenghs and dosages vary widely.

If you are just dealing with pain or an injury, use a topical application. Don’t drink it. Drinking it is for serious detoxing and other internal necessities. If you use a rose scented DMSO cream, chances are that nobody will be able to smell DMSO’s garlic-like smell. (My comment:  Be careful here – unless you know for certain that smell came from a pure source, don’t use it.  I called this company and heard nothing back which says everything to me.  I won’t be using the rose cream!)

The usual oral dose of DMSO is one teaspoon per day of DMSO 70% (Morton, 1993). But since it can trigger detoxification reactions and DMSO’s total excretion from the body can take several days, it is best to do it only once a week to begin with.Start with half a teaspoon of DMSO 50% and increase to a teaspoon of DMSO 70% only if any possible detoxification reaction is well tolerated.

When you use liquid DMSO in the skin, let it dry for over 20 to 30 minutes before wiping the rest out (with an unbleached white towel). The skin must be clean, dry, and unbroken for any topical use of DMSO. The face and the neck are more sensitive to DMSO and no higher concenrations than 50% should be applied there. Topical concentrations of DMSO should be kept below 70% in areas where there is a reduction of circulation. When 60 to 90% DMSO is applied to the skin, warmth, redness, itching, and somtimes local hives may occur. This usually disappears within a couple of hours and using natural aloe vera, gel or cream, will help counteract or prevent this effect. When 60 to 90% DMSO is applied to the palm on the hand, the skin may wrinkle and stay that way for several days.

Chronic pain patients often have to apply the substance for 6 weeks before a change occurs, but many report relief to a degree that had not been able to obtain from any other source. In general, the greater the chronicity of the disorder, the longer the treatment with DMSO must be employed in order to achieve palliation (Steinberg , 1967).

Common health problems for which people will apply topical DMSO at home include acute musculoskeletal injuries and inflammations. The earlier DMSO is used, the more dramatic the result. A 70% concentration of DMSO mixed with water in volumes ranging from 8 to 12 ml, applied on and around the injury in a wide area at least three times daily, will have a healing affect in 4 out ot 5 people.

Arthritis, Sprains, Strains

It provides rapid amelioriation of pain and increased mobility and reduction of inflammation when used topically. You can see a positive response within 5 to 20 minutes and usually lasting for 4 to 6 hours. (Steinberg, 1967).

http://  Approx 27 Min.

Lyra Nara Inc.
Published on Sep 28, 2017

The first 15 Min is purely information on DMSO and how to use it.  Much of the information is repeated from what is written above.  From then on they explain and market the Herbeso line, which I am not affiliated with whatsoever.  It is interesting material and worth your time just for the information.


You can make your own gel: (make sure hands, utensils, and body are completely clean) Mix 1oz 70% pure DMSO and 3 oz Aloe Vera gel (at least 99.5% pure) in a glass container & store in a cool place.  It’s best to mix right before application.  I like this idea much better than the creams/gels on the market with questionable preservatives.

**For my personal recipe that has been used successfully for pain, please continue reading as it’s in the MSM section as MSM is one of the ingredients**

http:// Approx. 8 Min

I include this video to show there are DMSO creams on the market; however, I am concerned about it being stored in plastic as well as the added ingredients such as perfumes and dyes as they will go directly into the body.


According to Fischer in the DMSO handbook, due to the long half-life of DMSO, levels increase as you continue taking it.  Fischer recommends starting at a low dose of 3.5g (1 tsp = roughly 3ml) in a glass of water and to observe symptoms.  If pain is relieved and it is well tolerated, remain at that low dose.  If not, he recommends increasing in increments of 3.5g per day.

Interactions:  DMSO has been found in studies to counteract platelet aggregation.  If you are taking blood thinners, please consult your physician before starting DMSO.

Oral directions:

  • Pour 3.5g DMSO into clean glass (about 300ml or 10 oz)
  • Fill with water or chosen drink (DMSO releases histamines from body cells so he advises against tomato juice, but that cooled tea, or grape juice work well)  DMSO is bitter in water so some prefer something to mask it.  I say “tough it out” and use water.  You don’t need all that added sugar.
  • Mix well as DMSO will sink to bottom of glass
  • This gives you a 1-2% DMSO solution
  • Many find doing this after breakfast agreeable
  • DMSO has a diuretic effect.  Taking before bed is not advisable unless you like to get up to go to the bathroom a lot!
  • 3.5ml of DMSO at a density of 1.1 g/ml is equal to 3.85g giving you a dose of about .05g DMSO per kilogram of body weight if you weigh 75kg.  This is a long way from quantities that are labeled as clinically safe in most clinical trials and toxicity tests.

There is no promise of a cure; however, and there are some “non-responders.”  And of course, it’s important to work on all aspects of health such as proper rest/sleep, nutrition, exercise, stress reduction, addressing hormonal, nutritional, mineral, and emotional imbalances.   Take some time off regularly from DMSO use

Dimethyl sulfoxide exits the body in about 24 hours. Nevertheless, in both acute and chronic cases, it is recommended that you take some time off on a regular basis, say two days in a row each week. For example, just take Saturday and Sunday off every week. If you are using it daily, long term, take two to four weeks off, in a row, every six months. Or, you could do 30 days on and 30 days off.

Personal usage:  I have personally used a 70% DMSO gel purchased on-line topically myself as well as on numerous Guinea pigs – i.e., my family.  Once we got past the initial redness, burning, and desire to itch, pain & swelling reduced within minutes.  Dr. Jacob states this pain reduction lasts for 4-6 hours and that was our experience as well.  The smell of oysters in this case was very mild as the amount of DMSO used was small.  My LLMD has used IV DMSO and states that really smells – but the results were worth it.

One other note:  I have a suspicious spot under my eye that looks remarkably like another cancerous spot I had removed a few years ago.  I did have it treated with silver nitrate first, but after that healed, I applied the 70% DMSO gel on it.  I went back for a checkup and the dermatologist stated it looked great but to keep observing it.  I then told him about my experiment with DMSO to which he replied he just read an old study the previous night about the effectiveness of DMSO on cancer.  He also said to keep using it.


Before you try DMSO you may want to try MSM first as it is devoid of the odor, and doesn’t cause redness and itching when used topically.  Since it worked so well for me (as well as numerous other patients) I never have had to even take DMSO internally.  I have only used it topically when I’ve had specific pain and it worked like a charm.

MSM stands for Methylsulfonylmethane and is a naturally occurring sulfur compound which is 34% sulfur by weight and is a metabolite of DMSO.  It is a dietary mineral element that is an odorless, white crystalline powder that is somewhat bitter tasting. It was approved as a Generally Recognized As Safe (GRAS) substance in 2007 and is well-tolerated by most.

MSM is produced naturally as part of the Earth’s sulfur cycle involving algae, phytoplankton, and marine organisms, where it is absorbed into the soil, taken up by plants or soil bacterium and is expressed in minute amounts in many fruits, vegetables (broccoli, cauliflower, cabbage, garlic, onions), coconut & olive oil, eggs, pasture-fed meats, and grains. It is destroyed when pasteurized or heated at high temperatures and is also volatile when frozen or irradiated.

It’s synthetically produced through the oxidation of DMSO with hydrogen peroxide and then purified via crystallization or the preferred method of distillation which particular method yields no detectable differences from the naturally produced product. The synthetic method allows patients to ingest far more than possible through food alone.

According to Stephanie Seneff PhD for the Weston Price Foundation,

“Sulfur is the eighth most common element by mass in the human body, behind oxygen, carbon, hydrogen, nitrogen, calcium, phosphorus and potassium. The two sulfur-containing amino acids, methionine and cysteine, play essential physiological roles throughout the body. However, sulfur has been consistently overlooked by those addressing the issues of nutritional deficiencies. In fact, the National Academy of Sciences has not even assigned a minimum daily requirement (MDR) for sulfur…..

Experts have recently become aware that sulfur depletion in the soil creates a serious deficiency for plants,17 brought about in part by improved efficiency in the U.S. agricultural industry, which has steadily consolidated into highly technologized mega-farms.

It is estimated that humans obtain about ten percent of their sulfur supply from drinking water. Remarkably, people who drink soft water have an increased risk of heart disease compared to people who drink hard water.2 Many possible reasons have been suggested for why this might be true, and just about every trace metal has been considered as a possibility.3 However, I believe that the real reason may simply be that hard water is more likely to contain sulfur….

I recently came upon a remarkable article in a 1997 issue of FASEB11 which develops a persuasive theory that low blood serum levels of two sulfur-containing molecules are a characteristic feature of a number of disease conditions. All of these diseases are associated with muscle wasting, despite adequate nutrition. The authors have coined the term “low CG syndrome” to represent this observed profile, where “CG” stands for the amino acid “cysteine,” and the tripeptide “glutathione,” both of which contain a sulfhydryl radical “-S-H” that is essential to their function. Glutathione is synthesized from the amino acids cysteine, glutamate and glycine, and glutamate deficiency figures into the disease process as well, as I will discuss later.

The list of disease conditions associated with low CG syndrome is surprising and very revealing: HIV infection, cancer, major injuries, sepsis (blood poisoning), Crohn’s disease (irritable bowel syndrome), ulcerative colitis, chronic fatigue syndrome and athletic over-training….

In summary, a number of different arguments lead to the hypothesis that sulfur deficiency causes the liver to shift from producing cholesterol sulfate to producing arginine (and subsequently nitric oxide). This leaves the intestines and muscle cells vulnerable to oxidation damage, which can explain both the intestinal inflammation and the muscle wasting associated with Crohn’s disease.”

Doctors Jacob, Lawrence, and Zucker in their book “The Miracle of MSM – The Natural Solution for Pain,” explain that Sulfur is necessary for the proper formation of proteins and helps produce amino acids, connective tissue, enzymes and hormones.  And that  sulfur insufficiency is probably related to many disease states – perhaps including Lyme/MSIDS.

It all stems back to the 70’s when chemists from Crown Zellerback Corp, and doctors Herschler and Jacob of Oregon Health and Science University experimented with MSM to determine if it had similar therapeutic uses to DMSO. In 1981 Herschler obtained a patient to use MSM for skin, nails, and as a blood diluent. There were further patents claiming to relieve stress, pain, treat parasitic infections, increase energy, boost metabolism, enhance circulation and improve wound healing, despite little scientific proof. Current research has shown proven clinical improvement in arthritis, inflammatory disorders like interstitial cystitis, allergic rhinitis, and acute exercise-induced inflammation.

Both DMSO and MSM get into tissue due to their small size.

http://  Feb. 2013, Approx. 29 Min

Dr. Mercola interviews Dr. Rod Benjamin on MSM

Story at-a-glance

  • The clinical use of sulfur as an adjunct in our diet is becoming progressively more recognized as an important tool for optimizing health. MSM is already well-known for its joint health benefits, but may also be helpful for other conditions related to chronic inflammation and damage due to oxidation
  • MSM, which is a metabolite of DMSO approved for use in humans, primarily impacts your health by reducing inflammation. It’s widely used as a supplement for arthritic conditions. Like DMSO, MSM also appears to improve cell wall permeability, so it can be used to help deliver other active ingredients
  • MSM may be providing a missing link for optimal health, which appears to be related to sulfur. It also affects sulfur metabolism in the human body, although it’s still not entirely clear how
  • Sulfur also plays a critical role in detoxification, as it is part of one of the most important antioxidants that your body produces: glutathione. Without sulfur, glutathione cannot work
  • Toxicity studies have shown that MSM is extremely safe and can be taken at very high doses. Even if you have a very rich diet full of raw vegetables and MSM-rich foods, you can still supplement and not hit that toxicity level. Clinical research studies have found that the effective amounts range from about 1.5 grams to 6 grams

*Reduces cytokines & inflammation(in vitro studies show MSM reduces IL-6 (a marker implicated in chronic inflammation as well as suppressing NO and prostanoids) *antioxidant *free radical scavenger *kills gastrointestinal, liver, and colon cancer cells *restored normal cellular metabolism in mouse breast cancer and melanoma cells *helps wounds heal *increases blood flow *reduces muscle spasms*antiparasitic properties(especially for giardia) *normalizes the immune system *cholinesterase inhibitor *alleviates allergy symptoms *increases energy *improves condition of hair, nails, and skin


Dr. Michael T. Murray discusses MSM in under 2 minutes.


Karlene Karst, registered dietician, gives a 3.5 Min supplement review on MSM.  

Like DMSO, MSM is more of a therapeutic principle than a drug and seems to be providing some kind of missing link within the body.


Toxicity studies have shown that MSM is extremely safe and can be taken at very, very high doses. Even if you have a very rich diet full of raw vegetables and MSM-rich foods, you can still supplement and not hit toxicity. Clinical research studies have found that the effective amounts range from about 1.5 grams to 6 grams, although at higher doses, potential side effects include:

Intestinal discomfort
Swelling of the ankles
Mild skin rashes

These are likely detoxifying effects that can typically be mitigated or minimized by cutting back on the initial dosage, and slowly working your way up. In that case, you might want to start out with half a gram (500 milligrams) for a couple of weeks and then slowly increase until you get up to the desired dose.

Pregnancy:  Information regarding safety and efficacy in pregnancy and lactation in humans is lacking; however, according to the MSM book by Jacob, he states, “Clinical experience indicates MSM is safe for pregnant women.  We recommend; however, that you consult first with your physician before taking this or any other supplement or mediation.”  (p. 47)

Regarding children:  Jacob states that healthy children do not usually need MSM; however, those with allergies, asthma, or an inflammatory illness should consider using it.  He also states many kids have taken MSM – some in high amounts without problem.

Contraindications:  While DMSO has been found to counteract platelet aggregation, MSM has not been similarly tested in studies; however, clinical observations indicate it may also have a blood-thinning, aspirin-like effect.  Discuss MSM supplementation with your doctor before taking it.

MSM & blood tests:  Dr. Jacob recommends stopping MSM before a liver function test as it may interfere with the accuracy of the test and produce a false positive.  Resume supplementation after the test.

How to Select a High–Quality MSM Supplement

There are two methods of purification of MSM:


For MSM, distillation is by far superior. But crystallization is less expensive, and a lot less energy-intensive. According to Dr. Benjamin, only two companies that produce MSM use distillation. Mr. Benjamin explains why you should consider a product that has been purified using distillation.

“A lot of the problems with [crystallization] is you’re essentially crystallizing it out of a parent solvent or liquid. If there are any impurities, which could be salts of heavy metals, you could have aromatic hydrocarbons in that… It’s actually the parent solvent. It’s usually water. It is dependent upon water quality.” Distillation brands for MSM Immune and allergy research Inflammation/oxidation research Safety and metabolism research

I recently posted this:  Lyn-Genet Recitas, NMT, Sports Nutritionist, Holistic Health Pracitioner, RYT, and author of “The Plan,” calls MSM the wonder supplement for your gut. It can alleviate allergy symptoms, helps with detoxification, eliminates free radicals, and improves cell permeability. She states that with given time, MSM will start to actually repair damage caused by leaky gut – a common problem with Lyme/MSIDS patients. It can also help the body’s ability to absorb nutrients from food. Many Lyme patients struggle with paralysis of the gut where the muscles of the stomach and intestines stop being efficient. MSM helps this muscle tone as well.

For a great MSM guide: This article gives a current 2017 review of MSM as well as studies and 195 references. MSM has been studied for decades.


Similar to DMSO you can take MSM topically and internally.  It is recommended to start at a low initial dose and allow the body to acclimate. You can slowly increase the dose after a week. It is also stated that those with chronic conditions may take up to 6 or more months to notice a difference.


MSM comes in creams, gels, and lotions. Make sure you read about the other ingredients and if the MSM is made from distillation. Like any other supplement, the devil’s in the details.  Recently I made my own MSM cream, which was quite easy and I loved how it worked as a skin cream as well as for a pain cream.

  • Get pure 100% MSM powder made by distillation.  (Should have OptiMSM patent on it)
  • Get pure aloe vera gel (99.5% or higher)
  • Mix 1/2 Cup aloe vera with 1-2 Tablespoons MSM – a tiny whisk or stirring stick works best.  **Update** I’ve reduced the amount of MSM in half due to the drying effects for the facial cream.  Play with this amount for your own needs.  The pain cream obviously has much more MSM in it – read below.
  • (Optional) Add your favorite pure, organic therapeutic grade essential oils –  I used 3 drops lavender and 3 drops frankincense for a facial cream.  Guys this is for you too.  It is non greasy, tightens pores, & smells great.  **Update** I switched this to a synergy blend from Plant Therapy called Zit Fighter – 6 total drops for half C aloe.  
  • This same cream can be used for pain relief but add another tablespoon of MSM (total of 3 Tbsp).  Desired EO’s include Capaiba for inflammation (3 drops), Lavender for skin conditioning (3 drops) and peppermint as a cooling and driving oil taking the MSM deeper (4 drops).  Mix all well.  **Update**  I’ve switched this to a Plant Therapy synergy blend called Organic Rapid Relief  for the pain cream – 10 drops, and I’ve added liquid DMSO – 10 drops.  You can play with the amount of DMSO for your own personal preference.  I’ve used this on many and they all have responded with fairly immediate pain reduction, even a person who reactivated pain from an old foot fracture.  One word; however, sometimes a sheen of the white MSM crystals remain on the skin after it dries.  Just brush them off once it’s dry.  Also, since DMSO has been added, make sure this all dries before touching it to clothing or anything.  For tougher pain, I will take the appropriate amount of pain cream in my hand & add 10 or more extra drops of DMSO.  Immediately it heats up in your hand.  This has almost always worked for the worst kind of pain.  Of course the more DMSO you use the higher the potential for that lovely smell… it’s a balancing act if you are around people.
  • Store in glass with a tight fitting lid like a small wide mouthed mason jar in a cool, dark place.  I would also make sure the level of cream is such that it doesn’t touch any part of the lid – be it plastic or metal.  If it’s glass, that should be fine.  But it needs to be air-tight.


If you take 2-3g a day or less, capsules are convenient.  For higher doses crystals are cheaper and easier.

You should take the least amount to achieve the desired benefit.  More is not necessarily better.  According to the MSM book a dosage of 2g (2,000mg) is adequate; however, higher doses are often necessary to experience therapeutic effects.  You may need 3-4g of MSM to control allergy symptoms and for deep-seated severe conditions, you may have to even go higher.  It is also recommended to divide the dose throughout the day, but since MSM increases your energy, it’s best not to take it before bedtime.

For pain and inflammatory conditions, Jacob recommends topical and internal MSM.

Personal usage:  Currently, most of my family uses MSM internally, daily – including the dog.  We all started at 1/4 tsp once a day for a a few days then increased to twice a day.  After that, it was individual preference.  I increased to 1/2tsp twice a day and all of my pain is GONE.  My husband takes 1 tsp twice a day.  We simply put the crystals in a few ounces of waters, stir it up, and slam it.  It’s a bit bitter but I’ve had far worse.

My daughter struggles with Mast Cell issues and this has helped her a lot.  As you read it reduces or eliminates allergy symptoms and for her it has reduced mucus production and has boosted her immune system as well.  

I’ve used a manufactured MSM cream with glucosamine chondroitin in it; however, it does have the caprylic ingredient along with parabens and other nasty ingredients Fischer warns about so I won’t be purchasing more, just making my own.  I will say it worked for pain – within minutes.

Update:  I’ve been taking the MSM internally for months now with complete resolution of pain.  The only negative side-effect I had was my skin broke out due to the strong detoxification effects.  I broke out on my face, chest, and back just like a teenager.  Due to that I lowered the MSM to about half of what I took to give my body some help in clearing debris.  Within a couple of weeks that went away and I upped my dose back to 1/4tsp twice a day.  The pain never returned.  Please know this has been miraculous as nothing else has worked.  **Progress update** After a year or longer we are back on antibiotics as numerous symptoms came back.  This demonstrates once again the need to remain open-minded and adjust.  For us it’s pain in the C7 with accompanying occipital pain (right in the juncture between the base of the skull & the neck), stiffness of the spine, painful bottoms of the feet, migrating pain sometimes affecting the hip and/or other areas, as well as pain in the center of the palm (think crucifixion).  These are all past symptoms popping back up and and the body never forgets cellular memory!  It appears we are still dealing with Lyme (borrelia) and Bartonella.**

Depending upon your goals, if your doctor gives you a thumbs up, I would try MSM first as it is easy to obtain and has no side-effects or smell.  If the MSM works (primarily for pain, inflammation, detox, & leaky gut) then you are home-free.  If it doesn’t, you may want to then move on to get the OK from your doctor to try DMSO as it demands more knowledge, effort, attention to detail, and has a smell, depending upon amount used.  FYI:  I have never noticed the smell for the amount used topically in my recipes.  For those who desire to try DMSO and an oxidative bactericide for a full Lyme/MSIDS treatment, please work under the direct supervision of a practitioner.

And remember what your granny told you: “Necessity is the mother in invention.”  

And finally, after reading my last update, never feel badly about needing to return to anti-microbial treatment, be it antibiotics or others.  This is the nature of this beast.  It is persistent, it is stealthy, and it is tenacious.  You and I need to decide we are stronger and do what is necessary to get back on top.

Do not let the bugs get you down!


Transplacental Transmission & Fetal Damage With Lyme Disease


The two sides of the coin in pregnancy and LYME DISEASE

Hello friends of the network DERMAGIC EXPRESS, I bring you today another topic for what I call the “SAGA” on LYME DISEASE, in this case the controversial issue of TRANSPLACENTAL TRANSMISSION AND FETAL DAMAGE AND DEATH in pregnant women and infected with the feared BORRELIA BURGDORFERI.

I have found numerous references; most claim that BORRELIA in pregnant women with LYME DISEASE traverses the placenta and reaches the fetus and can cause multi organic damage, including the death of the same, intrauterine or a few hours or days after birth. Other authors say that this is false.

The CDC (Center of Control of Infectious Diseases) affirms that if the pregnant woman with LYME does her treatment, the child will be born healthy and recommends the use of the antibiotic AMOXICILLIN, because DOXYCYCLINE can cause damage to the developing fetus. The question here is what would happen if the BORRELIA species is resistant to AMOXICILLIN? Or the antibiotic to which BORRELIA is sensitive cannot be indicated because it would harm the fetus?

BORRELIA BURGDORFERI was discovered in 1982 by the aforementioned Willy Burgdorfer, the causal agent of the ERYTHEMA MIGRANS or LYME DISEASE, and only 1 year later the first study in 1983 described that it is suspected that this ESPIROCHETE can cross the placenta and infect the fetus, study published by Shirts SR, Brown MS, and Bobitt JR. under the name of “Listeriosis and borreliosis as causes of antepartum fever”. (8)

Later in the year 1985 Schlesinger PA, Duray PH, Burke BA, Steere AC, Stillman MT published a paper called “Maternal-fetal transmission of the spirochete of Lyme disease, Borrelia burgdorferi” where they report a case of a woman who developed LYME DISEASE and did not receive treatment with antibiotics. The child was born at 35 weeks of pregnancy and died of congenital heart disease the first week of life. The autopsy revealed the LYME ESPIROCHETE in the SPLEEN, KIDNEYS AND BONE MARROW. (2)

Later, the same WILLY BURGDORFER the discoverer and “father” of the ESPIROCHETE BORRELIA, who along with Dr. Alan Mc Donald and Jorge Benach PhD, published in the year 1987 (31 years ago) a work they called “stillbirth following maternal LYME DISEASE.” and I quote from the conclusions of these scientists: (24.)

“… Two cases of transplacental transmission of the BORRELIA BURGDORFERI were found associated with fetal death and congenital malformations, different anomalies were detected in each case …”

“… We recommend that pathologists study the tissues of stillborn fetuses in search of BORRELIA BURGDORFERI especially those with cardiac anomalies, and clinical doctors investigate the exposure during the first trimester of pregnancy to BORRELIA BURGDORFERI and in these cases determine if cardiac organogenesis is complete by the end of the first trimester of pregnancy”

“… We believe that there is enough evidence to alert women living in endemic areas of LYME DISEASE and doctors to recognize the early signs and symptoms of the disease and to start treatment with PENICILLIN at the same dose of SYPHILIS as used in pregnant women in the first trimester, regardless of the results of the laboratory tests … “

Another study that is worth noting is the one made by the MEDLINE database updated for the year of July 2012, the last revision of November 2012 of 88 journal articles from the PUBMED database, which I summarize as follows:

Maternal-Fetal Transmission of Lyme Disease


1.) Mothers with active Lyme Disease,Treated: 14.6% of the pregnancies with sequelae,
2.) Untreated: 66.7% of the pregnancies with sequelae,
3.) Unknown as to treatment: 30.3% with sequelae.
4.) Specific adverse outcomes included: cardiac 22.7%, neurologic 15.2%, orthopedic 12.1%, ophthalmic 4.5%, genitourinary 10.6%, miscellaneous anomalies 12.1%, 2nd trimester demise 12.1%. Highest rate of adverse outcome (72.7%) in women with infection acquired prior to or during first trimester.)

Now I will put a summary of the most frequent clinical manifestations described in the studies of children born to mothers with LYME disease, LYME positive


1.) LOW GRADE FEVER: 59% -60%
5.) ABDOMINAL PAIN: 20-29%
7.) NAUSEA: 23%
15.) VERTIGO: 30%
20.) ANXIETY: 21%
21.) ANGER OR RAGE: 23%
23.) IRRITABILITY: 54% -80%
25.) DEPRESSION: 13%
27.) PHOTOPHOBIA: 40-43%
46.) AUTISM.

There are numerous studies showing a clear EVIDENCE that the BORRELIA BURGDORGFERI in pregnant women is able to cross the placenta and infect the fetus. I could get tired here of giving you the description of each of them. But I will give you ALL the BIBLIOGRAPHIC REFERENCES that I found from the year 1983 until the year 2017, first the ones I found and then a chronology of ALL of them.

I close this issue which is HIGHLY DISCUSSED TODAY, with a post by Angélica Johansson, a great fighter against THIS PLAGUE that I found in my LINKEDIN network about the future of the planet and the LYME DISEASE … I quote:

“…”1 million people are predicted to get infected with Lyme disease in the USA in 2018. Given the same incidence rate of Lyme disease in Europe as in the USA, then 2.4 million people will get infected with Lyme disease in Europe in 2018. In the USA by 2050, 55.7 million people (12% of the population) will have been infected with Lyme disease. In Europe by 2050, 134.9 million people (17% of the population) will have been infected with Lyme disease. Most of these infections will, unfortunately, become chronic.  

The estimated treatment cost for acute and chronic Lyme disease for 2018 for the USA is somewhere between 4.8 billion USD and 9.6 billion USD and for Europe somewhere between 10.1 billion EUR and 20.1 billion EUR. If governments do not finance IV treatment with antibiotics for chronic Lyme disease, then the estimated government cost for chronic Lyme disease for 2018 for the USA is 10.1 billion USD and in Europe 20.1 billion EUR.

If governments in the USA and Europe want to minimize future costs and maximize future revenues, then they should pay for IV antibiotic treatment up to a year even if the estimated cure rate is as low as 25%. The cost for governments of having chronic Lyme patients sick in perpetuity is very large….”

But what you see every day is a fight between IDSA and ILADS, CDC and others on the subject of whether it is a simple tick bite and you take an antibiotic and you cure or that it is a disease of difficult diagnosis and high cost of treatment. Between believers and non-believers to summarize. The truth is that it is spreading all over the world in leaps and bounds.

And if you have doubts that this ESPIROCHETE may or may not harm the fetus of pregnant women, cause birth defects, and many other consequences including, stillborn babies, read this “MOUNTAIN” of references that I leave here.

   Approx. 25 Min.

Sue Faber, RN and Co-Founder of LymeHope speaks to pregnancy and Gestational Lyme at the LymeHope Education Event, Oakville, Ontario on November 3, 2017.  Published on Jan 18, 2017

Dr. Elena Frid, a board-certified NYC neurologist and specialist in Lyme disease & other vector-borne diseases, discusses congenital Lyme disease.  

**Comment**  In reference to Dr. Frid’s comment that congenital Lyme is rare, I would disagree.  We have not been keeping track of numbers and there are probably way more than is being acknowledged.

CONCLUSION: BORRELIA BURGDORFERI, not only transmitted by the tick bite, is TRANSMITTED by sexual contact, fluids and can also colonize the fetus of pregnant women if there is no effective treatment able to eradicate it during the same. And it is not exclusive to the Northern Hemisphere. The BORRELIA is also in the Southern Hemisphere.

Greetings to all.

Dr. José Lapenta.

1.) Lyme disease during pregnancy. Infect Dis Clin North Am. 1997 Mar;11(1):93-7. ]PUBMED] Silver HM1.

2.) Maternal-fetal transmission of the Lyme disease spirochete, Borrelia burgdorferi. Ann Intern Med. 1985 Jul;103(1):67-8. [PUBMED]. Schlesinger PA, Duray PH, Burke BA, Steere AC, Stillman MT.

3.) Borrelia burgdorferi in a newborn despite oral penicillin for Lyme borreliosis during pregnancy. Pediatr Infect Dis J. 1988 Apr;7(4):286-9. [PUBMED]. Weber K1, Bratzke HJ, Neubert U, Wilske B, Duray PH.

4.) Neonatal skin lesions due to a spirochetal infection: a case of congenital Lyme borreliosis? Int J Dermatol. 1997 Sep;36(9):677-80. [PUBMED]. Trevisan G1, Stinco G, Cinco M.

5.) Confirmation of Borrelia burgdorferi spirochetes by polymerase chain reaction in placentas of women with reactive serology for Lyme antibodies. Gynecol Obstet Invest. 1996;41(4):240-3. [PUBMED]. Figueroa R1, Bracero LA, Aguero-Rosenfeld M, Beneck D, Coleman J, Schwartz I.

6.) Detection of Borrelia burgdorferi DNA in urine of patients with ocular Lyme borreliosis.
Pleyer U1, Priem S, Bergmann L, Burmester G, Hartmann C, Krause A. Br J Ophthalmol. 2001 May;85(5):552-5. [PUBMED]

7.) Culture and identification of Borrelia spirochetes in human vaginal and seminal secretions [version 1; referees: 1 not approved]. Marianne J. Middelveen1, Jennie Burke2, Eva Sapi3, Cheryl Bandoski3, Katherine R. Filush3, Yean Wang2, Agustin Franco2, Arun Timmaraju3, Hilary A. Schlinger1, Peter J. Mayne1, Raphael B. Stricker1
Source: F1000 RESEARCH

8.) Listeriosis and borreliosis as causes of antepartum fever. Obstet Gynecol. 1983 Aug;62(2):256-61. [PUBMED]. Shirts SR, Brown MS, Bobitt JR.

9.) Maternal-fetal transmission of the Lyme disease spirochete, Borrelia burgdorferi.
Schlesinger PA, Duray PH, Burke BA, Steere AC, Stillman MT.

10.) Gestational Lyme borreliosis. Implications for the fetus. Rheum Dis Clin North Am 1989 Nov 15:657-77. MacDonald AB. Source: . Rheum Dis Clin North Am 1989 Nov 15:657-77

11.) transplacental Lyme borreliosis infant mortality. Arthritis Rheum 1987; Volume 30, Number 4, 3(Suppl):S50. Lavoie PE;Lattner BP;Duray PH; Barbour AG; Johnson HC.

12) Lyme Borrelia positive serology associated with spontaneous abortion in an endemic Italian area.) Acta Eur Fertil. 1988 Sep-Oct;19(5):279-81. [PUBMED]. Carlomagno G1, Luksa V, Candussi G, Rizzi GM, Trevisan G.

13.) Infection with Borrelia: Implications for Pregnancy. James M O’Brien 1. and 2 Odessa P Hamidi. Division of Maternal Fetal Medicine, Pennsylvania College of Medicine, USA. Department of Obstetrics and Gynecology, Pennsylvania College of Medicine, USA.

14.) MEDLINE results for: borrelia pregnancy AND human. 88 journal articles in the PubMed
database BDH, July 2012, Latest Revision Novemb
er 2012,

15.) Infants born to mothers with antibodies against Borrelia burgdorferi at delivery. Eur J Pediatr. 1989 Feb;148(5):426-7. [PUBMED]. Nadal D1, Hunziker UA, Bucher HU, Hitzig WH, Duc G.

16.) Human fetal borreliosis, toxemia of pregnancy, and fetal death..Amanda B Macdonald
Published .1986 in Zentralblatt fur Bakteriologie, Mikrobiologie…Hyg A.1986 Dec;263(1-2):189-200

17.) Congenital relapsing fever (Borrelia hermsii).Blood, 15 November 2000, Vol. 96, No. 10, pp. 3333-3333William A. Dittman. Sr, Sacred Heart Medical Center, Spokane, WA.

18.) Lyme Disease and Pregnancy. James M. Alexander and Susan M. Cox. Department of Obstetrics and Gynecology, University of Texas Southwaestern Medical Center, Dallas, TX

19.) Teratogen update: Lyme disease. Teratology. 2001 Nov;64(5):276-81. [PUBMED]. Elliott DJ1, Eppes SC, Klein JD.

20.) Borreliosis during pregnancy: a risk for the unborn child?. Vector Borne Zoonotic Dis. 2011 Jul;11(7):891-8. doi: 10.1089/vbz.2010.0102. Epub 2010 Oct 6. [PUBMED]. Mylonas I1.

21.) Intrauterine transmission of Borrelia burgdorferi in dogs. Am J Vet Res. 1993 Jun;54(6):882-90. [PUBMED]. Gustafson JM1, Burgess EC, Wachal MD, Steinberg H.

22.) Fetal outcome in murine Lyme disease. Infect Immun. 1995 Jan;63(1):66-72. [PUBMED] Silver RM1, Yang L, Daynes RA, Branch DW, Salafia CM, Weis JJ.

23.) The association between tick-borne infections, Lyme borreliosis and autism spectrum disorders. Med Hypotheses. 2008;70(5):967-74. Epub 2007 Nov 5. [PUBMED]. Bransfield RC1, Wulfman JS, Harvey WT, Usman AI. The full text here:

24.) Gestational Lyme Disease Case Studies of 102 Live Births. by Charles Ray Jones, M.D., Harold Smith, M.D., Edina Gibb,. and Lorraine Johnson, JD, MBA

25.) Stillbirth following maternal LYME DISEASE. N Y State J Med. 1987 Nov;87(11):615-6.
[PUBMED] MacDonald AB, Benach JL, Burgdorfer W. Source: full text:

26. ) The Enlarging Spectrum of Tick Borne Spirochetoses; R.R. Parker Memorial address. Reviews of Infectious Diseases, vol.8, no.6 (Nov-Dec 1986), pp.932940 Source fulle text:

27.) Teratogenic effects of the bacteria Borrelia sp. on the fetuses of pregnant women with Lyme disease. Sliwa, Leopold. Nowa Medycyna 04/2011. (Translation of above article)

28.) Lyme disease in pregnancy: case report and review of the literature. Obstet Gynecol Surv. 2007 Jan;62(1):41-50. [PUBMED] Walsh CA1, Mayer EW, Baxi LV.

29.) Borreliosis During Pregnancy: A Risk for the Unborn Child? VECTOR-BORNE AND ZOONOTIC DISEASES. Volume 11, Number 7, 2011. Mary Ann Liebert, Inc..DOI: 10.1089/vbz.2010.0102. Ioannis Mylonas. Source full text:

Lyme Disease and Pregnancy, Maternal Fetal Transmission of Lyme Disease:

1983 Shirts SR, Brown MS, Bobitt Jr. Listeriosis and borreliosis as causes of antepartum fever. Obstet Gynecol 1983;62:256.

1985 Schlesinger PA, Duray PH, Burke BA, Steere AC, Stillman MT. Maternal fetal transmission of the Lyme disease spirochete, Borrelia burgdorferi. (1985) Ann Intern Med, 103, 67-8.

1985 MMWR. Update: Lyme Disease and Cases Occurring during Pregnancy—United States. Vol. 34, No. 25 (June 28, 1985), pp. 376- 378, 383-384

1986 MacDonald A. Human fetal borreliosis, toxemia of pregnancy, and fetal death. Zentralbl Bakteriol Mikrobiol Hyg A. 1986 Dec;263(1-2):189-200.

1986 Burgdorfer, W., The Enlarging Spectrum of Tick Borne Spirochetoses; R.R. Parker Memorial address. Reviews of Infectious Diseases, vol.8, no.6 (Nov-Dec 1986), pp.932940

1986 Markowitz LE, Steere AC, Benach JL, et al. Lyme disease during pregnancy. JAMA.(1986); 255(24), 3394-6.

1987 MacDonald AB, Benach JL, Burgdorfer W. Stillbirth following maternal Lyme disease. N Y State J Med. 1987 Nov;87(11):615-6.

1987. Lavoie PE, Lattner BP, Duray PH, Barbour AG, Johnson HC. Culture positive seronegative transplacental Lyme borreliosis infant mortality. (1987) Arthritis Rheum, 30(4), 3(Suppl):S50.

1988 Weber K; Bratzke HJ, Neubert U, Wilske B, Duray PH. (1988) Borrelia burgdorferi in a newborn despite oral penicillin for Lyme borreliosis during pregnancy. Pediatr Infect Dis J, 7:286-9.

1988 Carlomagno G, Luksa V, Candussi G, et al. (1988) Lyme Borrelia positive serology associated with spontaneous abortion in an endemic Italian area. Acta Eur Fertil 19(5), 279-81.

1988 Medici F, Benach J, Williams C. Lyme Disease during Pregnancy A Cord Blood Serosurvey. Annals New York Academy of Sciences. Volume 539, Lyme Disease and Related Disorders Pages 504–506.

1988 Health and Welfare Canada. Canada Diseases Weekly Report, June 4, 1988. Lyme disease in Canada.
1988 Lyme disease in Canada. Epidemiologic Report. CMAJ Vol. 139, Aug 1, 1988
1989 MacDonald A. Gestational Lyme borreliosis. Implications for the fetus. Rheum Dis Clin North Am. 1989 Nov;15(4):657-77.

1989 Halperin JJ., Dattwyler R., et al. A Perspective on the treatment of Lyme Borreliosis. Reviews of infectious diseases. Vol. 11 Supp 6. Sept/Oct 1989. S1518-1525

1989 Nadal D, Hunziker UA, Bucher HU, et al. (1989) Infants born to mothers with antibodies against Borrelia burgdorferi at delivery. Eur J Pediatr 148(5), 426-7.

1989 Steere et al. Lyme Seropositivity and pregnancy outcome in the absence of symptoms of Lyme disease. Scientific Abstracts June 12-17, 1989. 53 Annual Meeting of American College of Rheumatology.

1991 Lakos A. Lyme Borreliosis in Hungary in the years 1984 through 1989. Parasit hung., 24;5-51, 1991

1992 ACOG Committee Opinion. Lyme disease during pregnancy. Int J Gynecol Obstet 1992, 39; 59-60.

1992. Bracero LA, Wormser GP, Leikin E. Tejani N. Prevalence of seropositivity to the Lyme disease spirochetes during pregnancy in an epidemic area: A preliminary report. J Matern Fetal Investig. 1992(2): 265-268

1993 Hercogova J, Tomankova M, Frosslova D, Janovska D. Early-stage lyme borreliosis during pregnancy: treatment in 15 women with erythema migrans. Ceska Gynekol 58(5):229-232.

1993 Strobino BA, Williams CL, Abid S, et al. (1993) Lyme disease and pregnancy outcome: a prospective study of two thousand prenatal patients. Am J Obstet Gynecol 169(2 Pt 1), 367-74.

1994 Gasser R. et al. A Most Unusual case of a whole family suffering from late Lyme Borreliosis for Over 20 years. Angiology Vol. 45, No. 1: 85-86.

1994 Trevisan G. Lyme Borreliosis; A general survey. Acta dermatovenerologica A.P.S. Vol 3, 94, No. 1/2 4-12

1994 Elsukova LV, Korenberg EI, Kozin GA., [Pathology of pregnancy and the fetus in Lyme disease] [Article in Russian]. Med Parazitol (Mosk). 1994 Oct-Dec;(4):59-62

1995 Gardner T. Infectious Diseases of the Fetus and Newborn, 4th edition, New York, NY. W.B. Saunders Company (1995) Chapter 11, Lyme Disease. page 447 – 528.

1995 Williams CL, Strobino B, Weinstein A, et al. (1995) Maternal Lyme disease and congenital malformations: a cord blood serosurvey in endemic and control areas. Paediatr Perinat Epidemiol 9(3), 320-30.

1995 Schmidt, B. et al. Detection of Borrelia burgdorferi DNA by Polymerase Chain Reaction in the Urine and Breast Milk of Patients with Lyme Borreliosis. DIAGN MICROBIOL INFECT DIS 1995;21:121-128.

1995 Alexander, J. Cox, S. Lyme disease and Pregnancy. Infectious diseases in Obstetrics and Gynecology 3?256-261 (1995)

1996. Figueroa R. et al. Confirmation of Borrelia burgdorferi Spirochetes by Polymerase Chain Reaction in Placentas of Women with Reactive Serology for Lyme Antibodies. Gynecol Obstet Invest 1996; 41?240-243

1996. Maraspin V, Cimperman J. Treatment of Erythema Migrans during Pregnancy. Clinical Infectious Diseases 1996; 22?788-93

1997 Silver H. (1997) Lyme Disease During Pregnancy. Inf Dis Clinics of N. Amer. Vol 11, No 1.

1997 Trevisan G, Stinco G, Cinco M. Neonatal skin lesions due to a spirochetal infection; a case of congenital lyme borreliosis? International Journal of Dermatology 36; 677-99

1999 Norris C., Gardner T. Aseptic Meningitis in the Newborn and Young Infant. Am Fam Physician 1999 May 15;59(10):2761-2770

2001 Elliot D, Eppes S., Klein J. Terratogen Update; Lyme disease. TERATOLOGY 64?276 – 281 (2001)

2001 Gardner T. Chapter 11, Lyme Disease. Remington and Klein: Infectious diseases of the Fetus and Newborn, Fifth edition. New York, NY. W.B. Saunders Company 2001 pgs. 519-641

2001 Gardner T. Lyme disease in pregnancy. Program and abstracts of the 14th International Scientific Conference on Lyme Disease and Other Tick-Borne Disorders; April 21-23, 2001; Hartford, Connecticut.

2003 Goldenberg, R. L and C. Thompson (2003). “The infectious origins of stillbirth.” Am J Obstet Gynecol 189(3): 861-73.

2003 Salvato, WT, Salvato P. Lyme disease: ancient engine of an unrecognized borreliosis pandemic? Medical Hypotheses 60(5): 742-759.

2005 Onk G, Acun C, Kalayci M, Cagavi F, et al. (2005) Gestational Lyme disease as a rare cause of congenital hydrocephalus. J Turkish German Gynecology Association Artemis, 6(2), 156-157.

2005 Jones CR, Smith H, Gibb E, Johnson L (2005) Gestational Lyme Disease: Case Studies of 102 Live Births. Lyme Times. Gestational Lyme Studies 34-36

2005 Goldenberg et al. Maternal Infection and Adverse Fetal and Neonatal Outcomes. Clin Perinatol 32 (2005) 523–559.

2006 Walsh et al. Lyme disease in pregnancy. Obstetrical and Gynecological Survey. CME Review Volume 62, Number 1.

2007 Bransfield, Robert C. et al. The association between tick borne infection, lyme borreliosis and autism spectrum disorder. Medical hypotheses (2007)

2008 Hercogova J, Vanousova D. Syphilis and borreliosis during pregnancy. Dermatol Ther 21(3), 205-9.

2008 Theiler RN, Rasmussen, S. et al. Emerging and Zoonotic infections in women. Infect Dis Clin North Am 2008 December ; 22(4): 755–viii

2009 Lakos et al. Maternal Lyme borreliosis and pregnancy outcome. International Journal of Infectious Diseases 14 (2010) e494–e498

2009. Hulinska D, Votypka J, Vanousova D, Hercogova J, Hulinsky V, Drevova H, Kurzova K, Uherkova L. Identification of Anaplasma phagocytophilum and Borrelia Burgdorferi sensu lato in Patients with Erythema Migrans. Folia Microbiol. 54(3), 246-256 (2009)

2011 Mylonas I. Borreliosis During Pregnancy: A Risk for the Unborn Child? Vector Borne Zoonotic Dis. 11?891-8.

2011 Sliwa, Leopold. Teratogenny wplyw bakterii Borelli sp. Na ploy matek chorujacych na borelioze z Lyme. Zaklad Biologi Rozwoju Czlowieka. Instytus Pielegniarstwa.

2011 Sliwa, Leopold. Teratogenic effects of the bacteria Borrelia sp. on the fetuses of pregnant women with Lyme disease. Nowa Medycyna 04/2011. (Translation of above article)

2012 Relic, Milijana, Relic, Goran. Lyme borreliosis and pregnancy. Vojnosanit Pregl 2012; 69(11): 994–998.

2014 Onyett, H . Lyme disease in Canada: Focus on Children. Paediatr Child Health 2014;19(7):379-83

2014 OʼBrien, JM. Martens MG. Lyme disease in pregnancy; a New Jersey medical advisory. MD advisory, Winter 2014, pgs 24-27

2015 Krysztof PJ et al. Congenital tick borne Diseases: Is this an alternative route of transmission of tick borne pathogens in Mammals? Vector-Borne and Zoonotic Diseases Vol 15, Number 11, 2015.

2015 Hu LT, Tsibris AM, Branda JA. Case Records of the Massachusetts General Hospital: Case 24-2015; A 28 year-old pregnant woman with fever, chills, headache and fatigue. N Engl J Med. 2015 Jul 30;373(5):468-75.

2016 Maldonato, Y, Nizet, V, Klein, J, Remington, J, Wilson, C. Current concepts of Infections of the Fetus and Newborn Infant. Chapter 1. page 6. Infectious Diseases of the Fetus and Newborn Infant. 8th Edition. 2016

2017 OʼBrien, JM. Baum JD. Case Report. The Journal of Family Practice. August 2017; 66(8) pg E9-10 Updated and printed by JC on November 2, 2017

2017 March of Dimes. Lyme disease and Pregnancy. Retrieved from:

2017 Centers for Disease Control,USA. Pregnancy and Lyme Disease. Retrieved from:

Compiled Dec 6, 2017 – by JC and Sue Faber RN

Produced by Dr. Jose Lapenta R. Dermatologist
Maracay Estado Aragua Venezuela 2.018
Telf: 02432327287-02432328571