Archive for the ‘Treatment’ Category

The Great Alzheimer’s Scam & the Proven Cures They’ve Buried For Billions

https://www.midwesterndoctor.com/p/the-great-alzheimers-scam-and-the

The Great Alzheimer’s Scam and The Proven Cures They’ve Buried for Billions

Decades of neglect in understanding the actual causes of dementia have transformed it into one of the most costly diseases in existence.
A Midwestern Doctor
Oct 05, 2025

• Alzheimer’s disease is commonly thought to result from abnormal plaque buildup in the brain that gradually destroys brain tissue. As a result, almost all Alzheimer’s research has been directed toward eliminating amyloid, even after the basis for much of this work was shown to stem from fraudulent research.

•As such, despite decades of research and billions of dollars spent, this model has completely failed to produce useful results. The costly “groundbreaking” Alzheimer’s drugs only slightly slow dementia progression—at the expense of causing brain bleeding and swelling in over a quarter of those treated.

•In contrast, numerous affordable treatments have been developed for Alzheimer’s disease that target the root causes of the disorder, producing significant benefits at a fraction of the cost and without any toxicity.

•One neurologist, for example, proposed that amyloid serves a protective function in the brain and treats Alzheimer’s by identifying the underlying process causing dementia (which can often be diagnosed through symptoms). Remarkably, despite the method being proven in clinical research, awareness of it or the fact there are completely different types of “Alzheimer’s disease” which require different treatments remains almost nonexistent.

•Likewise, a strong case can be made that impaired cerebral circulation, along with impaired venous and lymphatic drainage, plays a pivotal role in Alzheimer’s disease.

•This article will review the common causes of cognitive impairment and dementia (e.g., cells becoming trapped in a shocked state where they no longer function) along with the forgotten treatments for neurodegenerative disorders—some of which, like DMSO, have extensive evidence supporting their use.

Dr. Dale Bredesen detailed in The End of Alzheimer’s, a few pivotal conclusions:

  • Amyloid protein is a protective mechanism the brain uses to protect itself from other stressors that endanger brain tissue.
  • Rather than there being one type of Alzheimer’s, there are actually three with different symptoms, and it is essential to recognize which type someone has before initiating treatment.
    • Type 1: Inflammatory – often metabolic or infectious
      • Type 1.5 Glycotoxic (hybrid of types 1&2) insulin resistance and chronically elevated blood sugar
    • Type 2: Atrophic – deficiences in nutrients, hormones, and other factors
    • Type 3: Toxic – exposure to mold, chronic infections such as Lyme disease, Herpesvirus, cytomegalovirus, fungal infections, etc. heavy metals, and household chemicals
    • Type 4: Vascular – hypertension, atherosclerosis, or anything causing chronic restriction of cerebral blood flow
    • Type 5: Traumatic – brain injury
  • The brain is designed to be able to adapt to the needs of life, so it is always creating or pruning neural connections and brain cells. Alzheimer’s, in turn, results from the loss of signals that sustain brain cells, and the dismantling of neural connections outweighing the formation of new ones, gradually compounding over the decades.

As such, his formula was fairly straightforward: identify the type of Alzheimer’s someone has, eliminate its causes, and gradually wait for the physiological momentum to shift from neurodegeneration to neurodevelopment.  (See link for article)

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**Comment**

Many anecdotal reports of using DMSO successfully for dementia are given in the article.

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Category:

Alzheimer's, Cancer, Lyme, Mold, Treatment, Viruses

ACTION: COVID Justice Resolution

https://covidjustice.org/

COVID Justice Resolution

For years, Americans have waited for an honest reckoning with the COVID-19 response—one grounded in reality rather than excuses or institutional gaslighting. Instead, the nation endured the most sweeping suspension of civil liberties in modern history, driven by emergency powers that often lacked evidence, silenced dissent, and inflicted lasting harm, especially on children, small businesses, the elderly, and the working class.

This proposed Senate resolution is a first step toward clarity.

Developed by collaborators associated with organizations including Autism Action Network, Children’s Health Defense, Health Freedom Defense Fund, Stand For Health Freedom, and The Brownstone Institute, it formally repudiates the most destructive COVID-era policies and establishes binding principles to ensure they are never repeated.

This resolution does not assign blame. It does not trade in fear. It reasserts constitutional limits. It stands as notice to the future: emergency powers must never again override liberty, evidence, or human dignity.

Read the resolution. Remember what happened. Ensure it never happens again.

The COVID Justice Resolution

To affirm the permanent lessons of the COVID-19 response, to repudiate certain emergency measures as incompatible with constitutional liberty, and to establish binding principles for any future public-health emergency.

Whereas the COVID-19 pandemic of 2020–2023 occasioned the most widespread and prolonged suspension of civil liberties in American history;

Whereas many measures taken in the name of public health, at both the federal level and most states, lacked sufficient evidence of efficacy, in some cases were arbitrary, imposed disproportionate harm on the poor and working classes, and violated foundational principles of limited government;

Whereas the Senate now judges, with the benefit of hindsight and exhaustive subsequent reflection, that certain categories of intervention must never be repeated;

Now, therefore, be it

Resolved, That the Senate—…..(See top link for entire Resolution)

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Category:

Activism, Treatment, vaccines, Viruses

Do I Need IV Antibiotics?

https://danielcameronmd.com/iv-antibiotics/

Do I Need IV Antibiotics?
Dec22

Do I Need IV Antibiotics?

Posted By: Dr. Daniel Cameron

He Asked What Many Eventually Ask

He sat across from me and voiced something countless patients eventually reach: Doctor… do I need IV antibiotics?

The patient struggled with cognitive slowing, neuropathic pain, persistent headaches, sleep disruption, and mild dizziness. He was improving slowly with oral antibiotics — but anxious he was missing something.

He had seen images of patients with IV poles on social media and heard stories of recovery only after patients had been treated with IV antibiotics. He believed IV therapy represented the “real treatment.” He wasn’t alone. Many assume IV antibiotics are stronger, faster, or required.

What IV Antibiotics Can (and Can’t) Do in Recovery

IV antibiotics can help in some cases of Lyme disease, especially when there is significant neurological involvement and oral treatments haven’t brought noticeable improvement or symptoms keep getting worse.

However, IV therapy is not simply a stronger version of oral treatment. It carries catheter-related risks, varies in what it covers, and does not guarantee better outcomes. For some patients, it offers benefit; for others, progress occurs through different strategies. IV is a tool — not a universal upgrade.

Why Some Patients Don’t Improve Even After IV Therapy

Many patients aren’t aware that the most commonly used IV antibiotic for Lyme — ceftriaxone — doesn’t treat co-infections like Babesia, Bartonella, or Anaplasma.

If one of these infections is present, IV therapy alone may not help, even when it’s given correctly.

In many cases, it’s a mismatch in coverage, not a lack of effort, that explains why some people don’t get better after IV treatment.

How I Decide When IV Antibiotics Makes Sense

Many antibiotics offered in IV form — including doxycycline and azithromycin — exist orally with good tissue penetration and far fewer catheter-related risks.

Other IV antibiotics are still being studied, so I usually use them only in specific situations rather than as a first step or automatic next step.

Even when IV therapy is appropriate, drug selection matters. Ceftriaxone, for example, can affect the gallbladder, so in patients with prior concern, cefotaxime (Claforan) may be an alternative — though it requires far more frequent dosing.

The Turning Point Was Not IV — It Was Correct Treatment

Because Babesia was clinically suspected, we adjusted the patient’s oral regimen to include azithromycin (Zithromax) and Malarone.

His cognition sharpened. Neuropathy eased. Headaches settled. Sleep stabilized. Dizziness quieted.

A few weeks later he said, “I thought IV was my only shot. I didn’t realize I could get better without it.”

He never required IV treatment — he needed the right treatment, not a different route.

Sometimes progress isn’t escalation — it’s correction.

“Do I Need IV Antibiotics?”

When someone asks, “Do I need IV antibiotics?” the answer depends on symptoms, function, and treatment response.

There are situations where IV therapy is justified. But many improve when treatment is refined, not intensified.

If improvement stalls despite appropriate care — or neurological involvement is suspected — IV therapy may be considered.

But many regain ground when treatment targets co-infections and physiologic drivers.

If you have wondered whether you truly needed IV antibiotics, you are not alone. Share your experience below — someone else may feel less alone reading it.
Resources
  1. Columbia University. Lyme and Tick-Borne Diseases Research Center.
  2. CDC. Chronic Symptoms and Lyme Disease.
  3. Johns Hopkins Lyme Disease Research Center.
  4. Dr. Daniel Cameron: Lyme Science Blog. What Is the Best Treatment for Lyme Disease?
  5. Dr. Daniel Cameron: Lyme Science Blog. Lyme disease: One size does not fit all

**Comment**

I’m sure at some point every single patient asks this question. The sad truth about this complex illness is that it takes savvy, experience, and a whole lot of patience. It also typically takes more than one brain working on it and the challenge is being able to decipher what to use, when, and how much.

If ever there was an illness that is completely individualized, this is it!

It can also take YEARS to treat and this is really hard to wrap your mind around at first.  It took FIVE YEARS of highly individualized, expensive, and painful treatment for both my husband and me.  Painful due to the herxheimer reactions treatment causes and individualized because of the many coinfections that are often involved.

It’s also the reason why RCTs are futile, a waste of money, and why we desperately need N of 1 trials to be respected and accepted.

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Category:

Activism, Lyme, Treatment

Understanding Alpha-gal Syndrome and Its Growing Geographic Overlap With Lyme Disease

https://www.globallymealliance.org/blog/understanding-alpha-gal-syndrome-and-its-growing-geographic-overlap-with-lyme-disease?

Understanding Alpha-gal Syndrome and Its Growing Geographic Overlap with Lyme Disease

by Dr. Eleanor Saunders on January 15, 2026

<span id="hs_cos_wrapper_name" class="hs_cos_wrapper hs_cos_wrapper_meta_field hs_cos_wrapper_type_text" style="" data-hs-cos-general-type="meta_field" data-hs-cos-type="text" >Understanding Alpha-gal Syndrome and Its Growing Geographic Overlap with Lyme Disease</span>
Learn about alpha-gal syndrome, a tickborne allergy to red meat, its causes, symptoms, testing, and relation to Lyme disease- as well as prevention tips and current research insights.

The Basics 

Alpha-gal syndrome is a more recently identified (c. 2009) tickborne disease. It differs from Lyme disease, babesiosis, anaplasmosis and ehrlichiosis in that it is not a tickborne infection – it is a tickborne allergy. Alpha-gal syndrome is an allergy to red meat and other products containing alpha-gal, including dairy and gelatin for those with more sensitive allergy.

Alpha-gal syndrome’s best recognized cause is tick bites, and it has been described on 6 continents, with the culprit tick species varying across the globe. Lone star tick (Amblyomma americanum) bites are the primary cause of alpha-gal syndrome in the United States. Recently, rare cases linked to blacklegged and western blacklegged ticks (Ixodes scapularis and I. pacificus) have been reported in Maine, Washington state, and the upper Midwest, well outside of the lone star tick range (Thompson et al. 2023). Despite this early evidence that blacklegged ticks and western blacklegged ticks can cause alpha-gal syndrome, they are thought to be an uncommon cause given how few cases have been recognized in high-incidence Lyme regions, particularly of the northeastern United States.

The Timeline: tick bite to food allergy

It is not intuitive to connect how a tick bite can cause food allergy.

To begin with, a typical timeline of the development of allergy is as follows: a tick of a culprit species bites a human. (It is not yet known why some bites do and others do not cause alpha-gal syndrome.) Sometimes the tick bite that preceded new allergy is described as leaving an erythematous, inflamed, and itchy “bite site” lasting weeks. Many tick bites go unnoticed.

Weeks to months after the tick bite, a person who previously ate meat without incident has a meal containing red meat, such as a steak. However, they do not react right away. The allergic symptoms – which can include a combination of hives, facial and throat swelling, wheezing and difficulty breathing, vomiting and other gastrointestinal distress, and anaphylaxis – occur 2-6 hours after eating red meat.

The “classic” story of an initial reaction is someone who eats red meat for dinner, and then wakes up itching in the middle of the night, looks in the mirror, and is surprised to see hives and sometimes facial swelling. There are also less classic clinical presentations, such as people with isolated gastrointestinal distress who eat red meat frequently and may have a hard time connecting the two. Vegetarians and vegans who consume or are exposed to mammalian products may also manifest symptoms of alpha-gal syndrome. Tragically, the first case report of a death from alpha-gal syndrome has been recorded (Platts-Mills 2025).

The alpha-gal molecule and delayed reaction

Alpha-gal syndrome is an allergy to alpha-gal, which is a carbohydrate molecule. (Most food allergies are to proteins.) Human ancestors lost the ability synthesize alpha-gal tens of millions of years ago, but most mammals other than humans – including those that humans eat – do produce alpha-gal. Therefore, “red meat” – or meat from cows, pigs, sheep, deer, and other game – contains alpha-gal. (Fish and birds do not produce alpha-gal.) The alpha-gal carbohydrate in meat is attached to both fats and proteins. The fatty form, glycolipids, take time to be metabolized and enter the bloodstream. That’s why allergic symptoms often appear 2–6 hours after eating, rather than immediately.

In addition to mammals, ticks also have alpha-gal in their saliva, without ever biting a mammal. Why? One compelling explanation is molecular mimicry. Ticks have many ways of trying to disguise their bite to avoid being detected, so expressing alpha-gal may be one additional way to look like their hosts (deer, mice, and other mammals whose cells express alpha-gal). Of tick species in the United States, lone star ticks, blacklegged ticks, brown dog ticks (Rhipicephalus sanguineus) and the invasive Asian longhorned tick (Haemaphysalis longicornis) have been shown to have alpha-gal in their saliva.

Testing for alpha-gal syndrome

Only if you have allergic symptoms, or unexplained gastrointestinal symptoms, should you be tested for alpha-gal syndrome. The test for alpha-gal syndrome is a serum test for alpha-gal IgE. IgE is a type of antibody that the immune system produces in response to allergens. A positive does not necessarily mean you have the allergy. Instead, it shows that your body has made IgE antibodies against alpha-gal, a state called being “sensitized” to an allergen, in allergy terminology.

A high percentage of adult populations screened for alpha-gal IgE in areas with lone star ticks are sensitized to alpha-gal, in the realm of 20-30% and even higher in heavily tick-exposed populations such as forestry and outdoor workers. However, most sensitized individuals in groups that have been screened are “sensitized only” and do not report allergy symptoms.

Alpha-gal syndrome and Lyme disease

There is no established connection between alpha-gal syndrome and Lyme disease in the United States. That’s partly because lone star ticks are the primary cause of alpha-gal syndrome whereas blacklegged ticks transmit the Lyme bacteria. It is important to note that western Europe is different: there, a single tick species—Ixodes ricinus—can both trigger alpha-gal syndrome and transmit Lyme bacteria. Even there, however, being bitten by one of these ticks doesn’t mean a person will develop both conditions. A Swedish study (Tjernberg et al. 2017) found no link between Lyme disease history and having alpha-gal antibodies.

[Ixodes ricinus is commonly known as the castor bean tick or the sheep tick]

Considerations for Lyme-endemic regions of the United States

It is important to recognize that the lone star tick range is expanding, particularly northward and eastward, and prominently along the northeastern coastline. Lone star ticks are now well-established in eastern Long Island, where there are also blacklegged ticks and Lyme disease. Lone star ticks are also increasingly found on Martha’s Vineyard. They are considered an aggressive human-biting tick. Deer are an important host for lone star ticks, whereas white-footed mice (Peromyscus leucopus) are not.  EPA-registered insect repellents such as DEET and picaridin for skin and clothing and permethrin for clothing and gear remain important for lone star tick bite prevention, as for blacklegged and other tick bites. An important distinction from Lyme disease is that alpha-gal syndrome can likely be caused by a tick attached for as little as a few hours. The metric of removing a tick within 24 hours, while good advice for Lyme disease, should therefore not be considered protective for alpha-gal syndrome.

Tick bite avoidance

Not only is avoiding tick bites important to avoid developing alpha-gal syndrome, but it remains important for those who have the allergy. Over time (years), some patients with alpha-gal syndrome who avoid tick bites have declining alpha-gal IgE levels that correspond to a remission of their allergy and the ability to reintroduce red meat into their diets. Reintroducing red meat is a very individualized decision to be made with a knowledgeable healthcare provider and incorporating safety considerations. If a patient returns to eating red meat, new tick bites could cause allergic symptoms to return.

Current unknowns and research questions

Much of what is currently understood about alpha-gal syndrome, outlined above, comes from excellent, collaborative research. Yet important questions remain:

  • What percentage of people bitten by lone star ticks develop alpha-gal syndrome?
  • What percentage of people sensitized to alpha-gal go on to develop alpha-gal syndrome?
  • What genetic and immunologic factors determine whether someone sensitized to alpha-gal develops alpha-gal syndrome?
  • Why are some ticks (i.e., lone star ticks) more effective in sensitizing to alpha-gal and causing alpha-gal syndrome than others (i.e., blacklegged ticks)?
  • What compounds in tick saliva along with alpha-gal provoke the human immune system to produce allergic antibodies (IgE)?
  • What aside from ticks (and possibly chiggers, and Ascaris roundworms) can sensitize a person to alpha-gal? (Stoltz et al. 2019, Murangi et al. 2022)

There has been differing evidence about whether the molecule alpha-gal is produced by the tick itself or is synthesized by bacteria that are part of the tick microbiome. In either case, scientists have asked whether bacteria living in ticks could affect the amount of alpha-gal produced in tick saliva (Kumar et al. 2022, Cabezas-Cruz et al. 2018).

New treatments and future directions

For patients suffering from alpha-gal syndrome, the mainstay of management is avoiding red meat and—for some—dairy and other ingredients containing alpha-gal. For those patients sensitive even to minor exposures to alpha-gal, there also now exists a medication called omalizumab that has been effective in decreasing symptoms. It is an anti-IgE monoclonal antibody, and so works not only for alpha-gal syndrome but for IgE-mediated food allergy more broadly. Omalizumab may also be appropriate for those with unavoidable occupational exposures, such as those working in kitchens with skin and fume exposures to meat, and those who birth animals or dress deer and may be exposed to large amounts of body fluids containing alpha-gal (Nuñez-Orjales et al. 2017).

For patients who crave red meat but are allergic, GalSafe® pork is made from a genetically modified pig that doesn’t express alpha-gal, and so can be consumed by patients with alpha-gal syndrome. The technology of gene-editing mammals could also lead to medical products like gelatin and heparin (a blood thinner) being made without alpha-gal. Although reactions to these products are rare, concerns about alpha-gal have complicated medical care for some patients.

Tick control strategies

New strategies to control lone star tick populations are needed, both environmental controls and interventions under study such as a universal tick vaccine. Alpha-gal syndrome has reanimated some of these goals, both through the threat of people no longer being able to eat meat and dairy; through a growing understanding of how ticks interface with the human immune system; and through geography, which unites a growing swath of the United States population in a campaign against ticks and tickborne disease.

Short and sweet

A simple way to explain alpha-gal syndrome to others is double delay, double avoidance. There is a delay of weeks to months from tick bite to the first allergic reaction, and there is a delay of hours from eating red meat to when allergic symptoms appear. The treatment for alpha-gal syndrome is to avoid red meat and avoid further tick bites.

References:

Cabezas-Cruz A, Espinosa PJ, Alberdi P, Šimo L, Valdés JJ, Mateos-Hernández L, Contreras M, Rayo MV, de la Fuente J. Tick galactosyltransferases are involved in α-Gal synthesis and play a role during Anaplasma phagocytophilum infection and Ixodes Ixodes scapularis tick vector development. Sci Rep. 2018 Sep 21;8(1):14224.

Kumar D, Sharma SR, Adegoke A, Kennedy A, Tuten HC, Li AY, Karim S. Recently Evolved Francisella-Like Endosymbiont Outcompetes an Ancient and Evolutionarily Associated Coxiella-Like Endosymbiont in the Lone Star Tick (Amblyomma americanum) Linked to the Alpha-Gal Syndrome. Front Cell Infect Microbiol. 2022 Apr 12;12:787209.

Maldonado-Ruiz LP, Reif KE, Ghosh A, Foré S, Johnson RL, Park Y. High levels of alpha-gal with large variation in the salivary glands of lone star ticks fed on human blood. Sci Rep. 2023 Dec 4;13(1):21409. 

Murangi T, Prakash P, Moreira BP, Basera W, Botha M, Cunningham S, Facey-Thomas H, Halajian A, Joshi L, Ramjith J, Falcone FH, Horsnell W, Levin ME. Ascaris lumbricoides and ticks associated with sensitization to galactose α1,3-galactose and elicitation of the alpha-gal syndrome. J Allergy Clin Immunol. 2022 Feb;149(2):698-707.e3.

Nuñez-Orjales R, Martin-Lazaro J, Lopez-Freire S, Galan-Nieto A, Lombardero-Vega M, Carballada-Gonzalez F. Bovine Amniotic Fluid: A New and Occupational Source of Galactose-α-1,3-Galactose. J Investig Allergol Clin Immunol. 2017;27(5):313-314.

Platts-Mills TAE, Workman LJ, Richards NE, Wilson JM, McFeely EM. Implications of a fatal anaphylactic reaction occurring 4 hours after eating beef in a young man with IgE antibodies to galactose-α-1,3-galactose. The Journal of Allergy and Clinical Immunology In Practice. 2025 Nov.

Stoltz LP, Cristiano LM, Dowling APG, Wilson JM, Platts-Mills TAE, Traister RS. Could chiggers be contributing to the prevalence of galactose-alpha-1,3-galactose sensitization and mammalian meat allergy? J Allergy Clin Immunol Pract. 2019 Feb;7(2):664-666. 

Thompson JM, Carpenter A, Kersh GJ, Wachs T, Commins SP, Salzer JS. Geographic Distribution of Suspected Alpha-gal Syndrome Cases – United States, January 2017-December 2022. MMWR Morb Mortal Wkly Rep. 2023 Jul 28;72(30):815-820. 

Tjernberg I, Hamsten C, Apostolovic D, van Hage M. IgE reactivity to α-Gal in relation to Lyme borreliosis. PLoS One. 2017 Sep 27;12(9):e0185723. 

Guest Writer

Dr. Eleanor Saunders

Guest Writer

Opinions expressed by contributors are their own. Dr. Eleanor Saunders is an Infectious Diseases physician at the University of North Carolina at Chapel Hill. Dr. Saunders received her MD & MPH from the UNC School of Medicine and UNC Gillings School of Global Public Health, completed residency in Internal Medicine at Bellevue Hospital/NYU Langone Health, and completed fellowship training in Infectious Diseases at UNC. Dr. Saunders works on the epidemiology of alpha-gal syndrome with Dr. Scott Commins, one of the foremost experts on AGS.

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Category:

Alpha Gal Meat Allergy, Ticks, Transmission, Treatment

Measles Highly Treatable & Latest ‘Outbreak’ Shaped by Missing Data Points. Therapeutic Candidates for Measles

https://imahealth.substack.com/p/major-new-study-measles-is-highly?

Major New Study: “Measles Is Highly Treatable” IMA Peer-Reviewed Study Published in Antiviral Research

Comprehensive analysis highlights evidence-based therapeutic strategies and calls for a broader public health conversation about measles.
Independent Medical Alliance
Feb 11, 2026
 
Article Excerpts:

The study, titled “Acute Management of Measles: A Systematic Review of Therapeutic Strategies,” appears in the journal Antiviral Research and was authored by Amandeep Kaur, Ugo Alaribe, Joseph Varon, Sidra Hassaan, and Matthew Halma. Key Takeaways:

  1. Measles highly treatable. With a combination of strengthening the immune system and supportive supplements, Measles can be treated with a high degree of success.

  2. Vitamin A supplementation is supported by multiple randomized controlled trials and meta-analyses showing reduced measles-related mortality, particularly in vitamin A–deficient populations.

  3. Ribavirin and Interferon-α have demonstrated clinical benefit in severe or high-risk cases, especially among immunocompromised individuals.

  4. Supportive therapies, including intravenous immune globulin (IVIG), antibiotics to prevent secondary bacterial infections, and immune-supportive nutrients such as vitamins C and D, may reduce complications.

  5. Investigational antivirals and monoclonal antibodies show promise in preclinical studies.

https://popularrationalism.substack.com/p/whats-really-going-on-in-south-carolinas?

What’s Really Going On in South Carolina’s Measles Outbreak?

Key data points are missing that would allow a rational assessment.
James Lyons-Weiler, PhD
Feb 10, 2026

As of early February 2026, South Carolina remains the center of the largest measles outbreak in the U.S. in over 30 years. With 920 confirmed cases and over 90% reportedly occurring in “unvaccinated” individuals, headlines suggest a crisis of vaccine refusal. But beneath the headlines lies a more complex picture—one shaped by data classification, eligibility confusion, and methodological blind spots that public health authorities have failed to address.

This article unpacks the numbers behind the outbreak, highlights structural flaws in case reporting, and shows why surface-level interpretations of vaccination status are no substitute for scientific analysis.  (See link for article)

A few examples of the shamwizardry:

  1. Classification flaws: “Unvaccinated” includes infants under 12 months that are not eligible for MMR, conflating ineligibility with refusal
  2. No Denominators: Vaccine effectiveness (VE) requires knowing how many vaxxed and unvaxxed were at risk, exposed, or susceptible
  3. Misclassification: Those vaxxed who developed a rash within 21 days and were not genotyped via testing may have been declared ‘measles cases’ even though they are not contagious
  4. Confirmation Method not disclosed: measles outbreaks in high-vaxxed settings rely heavily on epi-linkage which means ONE PCR-confirmed case can result in DOZENS of cases being confirmed simply by association – without any testing.
  5. No breakdown of hospitalizations by vax status or age group.  Without cross-tabs, no assessment can be made.
  6. Most moms were vaxxed, not infected and antibody titers passed to babies wane far earlier leaving infants vulnerable the first year. Blaming parents for not vaxxing ignores the change in measles immunity
  7. No public access to raw case data.  What’s up with that?  Without public scrutiny any claims made are assertions not conclusions.
  8. Misapplication of surveillance architecture designed to detect outbreaks not evaluate efficacy.
  9. No audits on time, location, vaccine lot, etc revealing a logistical problem not an immunological problem

New Study Reviews Therapeutic Candidates for Measles

A new peer-reviewed study from IMA researchers reviews therapeutic candidates for measles, including Vitamin A. Why isn’t this part of the conversation?
Independent Medical Alliance
Feb 10, 2026
acute management of measles hero

If you’ve followed the news this year, you might think measles is an unstoppable force. Headlines warn of “surges” and “outbreaks,” case counts are tallied like a scoreboard, and the message is clear: be afraid.

But there’s a part of the story that rarely makes it into the coverage. In developed countries, serious complications from measles are rare. And perhaps most importantly: promising therapeutic candidates exist.

A new peer-reviewed study co-authored by IMA President Dr. Joseph Varon and Director of Research Matthew Halma adds to a growing body of research aimed at exploring therapeutic candidates for measles. Published in Antiviral Research, this systematic review compiles clinical evidence for treatment options—the kind of information that could help patients and physicians make informed decisions. So why isn’t it part of the conversation?

“Adjunctive therapies, including Vitamin A, Ribavirin, and Interferon-α, and emerging antiviral candidates, play an important role in reducing complications… Continued research is urgently needed to validate novel antivirals and immunomodulatory treatments.” — Study authors

📖 Read and Download the Full Paper

Acute management of measles: A systematic review of therapeutic strategies Authors: Amandeep Kaur, Ugo Alaribe, Joseph Varon, Sidra Hassaan and Matthew Halma

(See link for article)

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**Comment**

I love it when people who swallow the red pill decide to act in their sphere of influence.  Be thankful for the COVID train-wreck.  It woke a lot of people up!

Please read the entire article above but it’s worth pointing out a few salient facts:

  • Measles was nearly eradicated before a vaccine was introduced.  Deaths dropped from about 13 to .2 per 100,000.
  • Like any other disease, the outcome depends heavily on pre-existing health.
  • Vitamin A deficiency is a major factor (92% of hospitalized measles patients are deficient)

The following treatment candidates should be considered:

  • Vitamin A
  • Antivirals (Ribavirin and Interferon-a)
  • Supportive therapies (IVIG, antibiotics, vitamin C and D)
  • Investigational therapies (ERDRP-0519 and Monoclonal antibodies)

For more resources on measles, check out these guides from IMA:

For more:

Download the Measles Fact Sheet

 

 

 

Category:

research, Treatment, vaccines, Viruses