COVID Injections Drive COVID Variants & Nobel Prize Winner Urges Public to Reject Jabs

May 21, 2021

https://thenewamerican.com/french-nobel-prize-winner-warns-vaccines-facilitate-development-of-deadlier-covid-variants-urges-the-public-to-reject-jabs/

Nobel Prize Winner Warns Vaccines Facilitate Development of Deadlier COVID Variants, Urges Public to Reject Jabs

by Veronika Kyrylenko
Nobel Prize Winner Warns Vaccines Facilitate Development of Deadlier COVID Variants, Urges Public to Reject Jabs
France’s Luc Montagnier / AP Images

Luc Montagnier, a French virologist and recipient of the 2008 Nobel Prize in Medicine for his discovery of the human immunodeficiency virus (HIV), has recently exposed the dangers of the COVID-19 vaccines. Montagnier discussed the issue in an interview with Pierre Barnérias of Hold-Up Media earlier this month, which was exclusively translated from French into English for RAIR Foundation USA.

The vaccines don’t stop the virus, argues the prominent virologist, they do the opposite — they “feed the virus,” and facilitate its development into stronger and more transmittable variants. These new virus variants will be more resistant to vaccination and may cause more health implications than their “original” versions.

During the interview, professor Montagnier referred to the vaccine program for the coronavirus as an “unacceptable mistake.” Mass vaccinations are a “scientific error as well as a medical error,” he said. “The history books will show that, because it is the vaccination that is creating the variants.” Montagnier explained that “there are antibodies, created by the vaccine,” forcing the virus to “find another solution” or die. This is where the variants are created. It is the variants that “are a production and result from the vaccination.” (See link for article)

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**UPDATE**

Montagnier isn’t alone.  A French medical doctor is also stating that these injections are causing new infections and deaths and that the evidence is overwhelming. She also calls for a moratorium on them.

UPDATE: Montagnier HAS BEEN PROVEN TO BE CORRECT.

Nations participating in the “Zero COVID” movement with draconian edicts and mass vaccination campaigns have had explosions in COVID-19 cases across the board.

There are no longer any “success stories” involving nations using tyrannical means in an attempt to stop a virus. Source

**Comment**

Viruses and bacteria are adaptive.  This concept used to be widely understood, but since this truth doesn’t fit with the accepted narrative that we should all fear a killer virus, it’s ignored, by our corrupt public health ‘authorities’ and the bought-out media.  It’s an inconvenient truth that these COVID injections are causing the mutant (and typically more severe) strains cropping up all over the world.  But, we were warned.

Antibody Dependent Enhancement (ADE) is a very real thing we’ve been warned about.  ADE occurs in SARS-CoV1, MERS, HIV, Zika, and Dengue Virus vaccines, and data confirms it occurs in SARS-CoV-2, or COVID-19.

Montagnier states,

“You see it in each country, it’s the same: the curve of vaccination is followed by the curve of deaths.  I’m following this closely and I a doing experiments at the Institute with patients who became sick with Corona after being vaccinated.”

But rather than admitting this fact, our corrupt public health ‘authorities’ blame it on COVID. They don’t want to lose their revenue stream from the injections and they certainly don’t want to admit fault.

They are also now rigging the system by lowering PCR cyclesjust for the “vaccinated” to try and downplay the fact fully “vaccinated” people are testing positive and contracting COVID-19.  Further, they disingenuously call these “break-through” cases, which is very reminiscent of the fraudulent PTLDS moniker for Lyme patients. Words mean things and whole paradigms are constructed using these restrictive words. It’s a slight of hand the CDC continues to use and get away with.

The article then goes on to delineate Montagnier’s letter calling for suspension of COVID mass vaccination due to:

  1. Serious short term side effects
  2. Lack of vaccine protection
  3. Competition between natural antibodies and vaccine antibodies
  4. Antibodies by vaccination will lead to variants/mutants.  The virus will always win.
  5. Risk of viral RNA into the human genome

Last year Montagnier showed at least half a dozen mini-sequences of the HIV virus in the SARS-CoV2 genome.  This information was published under the title “Synthetic origin of Covid-19 and Evolution.”  Montagnier believes the coronavirus escaped when researchers were trying to develop a vaccine against HIV.

Montagnier is far from alone.  

  • Recently vaccine specialist Vanden Bossche also called for a halt to mass vaccination programs and states they actually drive viral immune escape.  He states that countries that have undertaken mass vaccination, (UK, Israel, USA), will initially experience a drop in infectivity rates, but they will inevitably suffer from a steep incline in severe COVID cases in the weeks to come.
  • The group Doctors For COVID Ethics also recently wrote a paper where they state COVID injections are “needless, ineffective and dangerous.” They delivered notices of liability for COVID-19 “vaccine” harms and deaths to every member of the European parliament.
  • ICAN has delivered emergency petitions to halt clinical trials until all adverse reactions are tracked.
  • Children’s Health Defense just filed a citizen petition to the FDA to revoke Emergency Use Authorizations for existing COVID vaccines and refrain from approving and licensing them
  • America’s Frontline Doctors just gave a motion for a temporary restraining order against the use of COVID vaccines in children
Hopefully it’s clear that despite the continued drumming of propaganda, there are concerned experts giving red flags. 
 

Category:

vaccines, Viruses

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Can Lyme Disease & Bartonella Trigger Eating Disorders?

https://www.lymedisease.org/lyme-bartonella-eating-disorders/

Can Lyme disease and Bartonella trigger eating disorders?

May 12, 2021

by Daniel A. Kinderlehrer MD

There are quite a few reviews in the medical literature documenting the neuropsychiatric conditions associated with Lyme disease, and most of them include the general category of “eating disorders.”  But when I searched for case histories, I could find only one, published in 1992.

Pachner et al. described a 12-year-old boy who initially presented with right knee swelling and tested positive for Lyme disease.1 He was treated with doxycycline for 30 days and his knee was better.

However, two months later, he became depressed and restricted his food intake, compulsively exercised, and lost 31 pounds. He still tested positive for Lyme disease and was treated with IV penicillin for two weeks, and soon thereafter he was no longer depressed, he ate normally, and gained weight.

This month, I published a case study about a second case of anorexia nervosa in association with Lyme disease.2 It is important to distinguish between simple anorexia, or loss of appetite, which is common with many medical conditions and infections, and anorexia nervosa, which is a disorder characterized by the obsessive need to lose weight by refusing to eat.

My patient Diane was raised in upstate New York, which is highly endemic for Lyme disease. In fact, she had multiple tick bites as a youngster with no known illness.

However, she did have some issues that stood out: at two years old she began screaming and pulling her hair out—a condition termed trichotillomania. This gradually morphed into generalized anxiety and irritability.

Food restrictions

When she was 14, Diane had another tick attachment that was not treated, and she developed fatigue, frequent sore throats, impaired cognition, and worsening depression, anxiety and irritability. At 15,  she began restricting her food intake and purging after meals.

This led to an admission to an eating disorder unit, where she was diagnosed with anorexia nervosa. She went in and out of the inpatient facility and an outpatient eating disorder program several times without success. She was also making serious suicide threats. Her mother is a savvy nurse and had her tested for Lyme disease. It was positive.

When I first met Diane, she was 16. She was refusing to eat and had a nasogastric tube in place, through which she received her nutrition. She maintained that she was still overweight and needed to restrict her diet—she was 5’6” and weighed 125 lbs. During her physical exam, I noticed Bartonella striae on her thighs—reddish striations that look like stretch marks, but not in the normal skin planes. Diane interpreted these as stretch marks, which further supported her belief that she was overweight.

Diane tested positive for Babesia and Mycoplasma in addition to Lyme disease. She tested negative for Bartonella, but the striae are virtually diagnostic for that microbe.

Bartonella and psychiatric symptoms

In a study posted in December 2020, Dr. Ed Breitshwerdt and colleagues described 29 patients with mental health issues who tested positive for Bartonella. Twenty-four of them developed skin rashes at the same time they developed neuropsychiatric symptoms. Most of those skin changes look the same as the ones on Diane.3The Bartonella striae are caused by new blood vessel formation in layers of the dermis, and biopsies of the striae have demonstrated Bartonella bacteria.4

We treated Diane with three months of intravenous ceftriaxone and a total of one year of oral antibiotics. Her eating disorder has stayed in remission for three years. It is interesting that when Diane began Bactrim, which attacks Bartonella, she had a Herxheimer reaction in which she again refused to eat. This suggests that Bartonella was triggering her eating disorder.

Traditionally, the etiology of anorexia nervosa is considered to be a combination of genetic and environmental factors, such as dysfunctional home circumstances, a history of emotional trauma and modern Western culture’s emphasis on thinness.5-8

PANDAS and PANS

However, there are several reports in the medical literature that describe the role of infections in the etiology of eating disorders.9-14 There is also a higher risk of eating disorders in children and adolescents with autoimmune disease.15 When you connect infection with autoimmunity, one of the outcomes is autoimmune encephalitis—i.e. PANDAS or PANS.

Susan Swedo initially described children who developed mental health issues following a streptococcal infection and coined the term PANDAS—Pediatric Autoimmune Neuropsychiatric Disease Associated with Streptococcal Infection.16,17 In particular, these kids had OCD, anxiety, and other mood and behavioral disorders. It wasn’t long before Sokol and Gray described anorexia nervosa in patients with PANDAS.18

Once it became clear that multiple microbes can trigger a PANDAS-like syndrome, the name was changed to Pediatric Acute-onset Neuropsychiatric Syndrome (PANS). The primary criteria for PANS established at the First PANS Consensus Conference include OCD or severely restricted food intake.19

Neuroinflammation

The dots are now connected. An infection can cause an autoimmune reaction that results in neuroinflammation—autoimmune encephalitis—that can manifest not only in OCD and other neuropsychiatric symptoms, but also trigger anorexia nervosa.

Diane probably had a PANS-like syndrome. When she was younger, she had trichotillomania (hair pulling), which has similarities to OCD. This was followed by anxiety and irritability.

When  she had yet another tick attachment at age 14, her symptoms worsened.  This exposed her to reinfection or to additional tick-borne pathogens.

It is noteworthy that Diane had four tick-borne infections: Lyme disease, babesiosis, mycoplasmosis and bartonellosis. In my clinical experience, Mycoplasma and Bartonella are the most likely pathogens to trigger PANS-like syndromes, and both of these have been documented as causing PANS.20-22 Wouldn’t it be a good idea if all patients with anorexia nervosa were screened for tick-borne infections and PANS?

With this in mind, Dr. Nancy Brown and I tested a group of adolescents in a residential treatment center who were suffering from severe mental health issues. In the next installment I’ll describe what happened with that study.

In the meantime, it’s good to keep in mind that PANS is not just for kids. Adults also suffer from autoimmune encephalitis with severe neuropsychiatric symptoms. And the onset is not always sudden—often the onset is gradual or stuttering. I suggest we change the name to Microbe-induced Autoimmune Neuropsychiatric Syndrome (MANS).23

Read the case study here.

Dr. Daniel Kinderlehrer is an internal medicine physician with a private practice in Denver, Colorado, devoted to treating patients with tick-borne illness. He is the author of  Recovery From Lyme Disease: The Integrative Medicine Guide to the Diagnosis and Treatment of Tick-Borne Illness.

References

  1. Pachner AR, Duray P, Steere AC. Central Nervous System Manifestations of Lyme Disease. Arch Neurol. 1989;46(7):790-795.
  2. Kinderlehrer DA. Anorexia Nervosa Caused by Polymicrobial Tick-Borne Infections: A Case Study. Int Med Case Rep J. 2021;14:279-287.
  3. Breitschwerdt, E.B.; Bradley, J.M.; Maggi, R.G.; Lashnits, E.; Reicherter, P. Bartonella Associated Cutaneous Lesions (BACL) in People with Neuropsychiatric Symptoms. Pathogens. 2020;9:1023.
  4. Maluki A, Breitschwerdt E, Bemis L, et al. Imaging analysis of Bartonella species in the skin using single‐photon and multi‐photon (second harmonic generation) laser scanning microscopy. Clin Case Rep. 2020;8(8):1654-1570.
  5. Yilmaz Z, Hardaway JA, Bulik CM. Genetics and epigenetics of eating disorders. Adv Genomics Genet. 2015;5:131–150.
  6. Tagay S, Schlottbohm E, Reyes-Rodriguez ML, Repic N, Senf W. Eating disorders, trauma, PTSD, and psychosocial resources. Eat Disord. 2014;22(1):33-49.
  7. Ferrell EL, Russin SE, Flint DD. Prevalence Estimates of Comorbid Eating Disorders and Posttraumatic Stress Disorder: A Quantitative Synthesis. J Aggress Maltreat Trauma. 2020;20:1-19.
  8. Becker AE, Fay KE, Agnew-Blais J, Khan AN, Striegel-Moore RH, Gilman SE. Social network media exposure and adolescent eating pathology in Fiji. Br J Psychiatry. 2011;198(1):43-50.
  9. Brown RF, Bartrop R, Beumont P, Birmingham CL. Bacterial infections in anorexia nervosa: delayed recognition increases complications. Int J Eat Disord. 2005;37(3):261-265.
  10. Favaro A, Tenconi E, Ceschin L, Zanetti T, Bosello R, Santonastaso P. In utero exposure to virus infections and the risk of developing anorexia nervosa. Psychol Med. 2011;41(10):2193-9.
  11. Raevuori A, Lukkariniemi L, Suokas JT, Gissler M, Suvisaari JM, Haukka J. Increased use of antimicrobial medication in bulimia nervosa and binge-eating disorder prior to the eating disorder treatment. Int J Eat Disord. 2016;49(6):542-52.
  12. Park RJ, Lawrie SM, Freeman CP. Post-viral onset of anorexia nervosa. Br J Psychiatry. 1995;166(3):386-9.
  13. Simon MW. Anorexia and failure to grow associated with Epstein-Barr virus infection. J Ky Med Assoc. 1998;96(1):13-15.
  14. Breithaupt L, Köhler-Forsberg O, Larsen JT, et al. Association of Exposure to Infections in Childhood with Risk of Eating Disorders in Adolescent Girls. JAMA Psychiatry. 2019;76(8):800–809.
  15. Zerwas S, Larsen JT, Petersen L, et al.. Eating Disorders, Autoimmune, and Autoinflammatory Disease. Pediatrics. 2017;140(6):e20162089.
  16. Swedo SE, Leonard HL, Kiessling LS. Speculations on antineuronal antibody-mediated neuropsychiatric disorders of childhood. Pediatrics. 1994 Feb;93(2):323-326.
  17. Swedo SE, Leonard HL, Garvey M, et al. Pediatric autoimmune neuropsychiatric disorders associated with streptococcal infections: clinical description of the first 50 cases. Am J Psychiatry. 1998;155(2):264–71.
  18. Sokol MS, Gray NS. Case study: an infection-triggered, autoimmune subtype of anorexia nervosa. J Am Acad Child Adolesc Psychiatry. 1997;36(8):1128-33.
  19. Chang K, Frankovich J, Cooperstock M, et al. Clinical Evaluation of Youth with Pediatric Acute-Onset Neuropsychiatric Syndrome (PANS): Recommendations from the 2013 PANS Consensus Conference J Child Adolesc Psychopharmacol. 2015;25(1):3–13.
  20. Piras C, Pintas R, Pruna D, Dessi A, Atzoril L, Fanos V. Pediatric Acute-onset Neuropsychiatric Syndrome and Mycoplasma Pneumoniae Infection: A Case Report Analysis with a Metabolomics Approach. Curr Pediatr Rev. 2020:16(3):183-193.
  21. Frankovich J, Thienemann M, Rana S, Chang K. Five youth with pediatric acute-onset neuropsychiatric syndrome of differing etiologies. J Child Adolesc Psychopharmacol. 2015;25(1):31-7.
  22. Breitschwerdt EB, Greenberg R, Maggi RG, Mozayeni BR, Lewis A, Bradley JM. Bartonella henselae Bloodstream Infection in a Boy with Pediatric Acute-Onset Neuropsychiatric Syndrome. J Cent Nerv Syst Dis. 2019;11:1179573519832014
  23. Kinderlehrer, D.A. Does Lyme Disease Cause PANS? J Biomed Res Environ Sci. 2021;2(3):126-131.

Category:

Bartonella, Lyme, research, Treatment

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Wildlife Borrelia Infection in Atlantic Canada

Wildlife Borrelia infection in Atlantic Canada:

Assessing the prevalence of Borrelia in wildlife hosts

Christopher Zinck

Mount Allison University

Lyme Research Network

Wildlife-Borrelia-infection-in-Atlantic-Canada-Assessing-the-prevalence-of-Borrelia-in-wildlife-hosts-Christopher-Zinck(1)  Slides Here

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Important findings:

  • Reservoir species are abundant
  • There are more Borrelia out there than B. burgdorferi (testing will miss all of them)
  • Different Borrelia species show different tissue tropisms (cells and tissues of a host which support growth of bacteria) in the body – B. miyamotoi is much more widely dispersed in the body in wild mice than Bb

Category:

Borrelia Miyamotoi (Relapsing Fever Group), Lyme, research

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Can You Get Lyme More Than Once?

https://danielcameronmd.com/can-you-get-lyme-disease-more-than-once/

CAN YOU GET LYME DISEASE MORE THAN ONCE? ASK THE LYME DOC.

Can-you-get-Lyme-disease-more-than-once

Patients often ask whether a person can become immune to Lyme disease or whether you can get Lyme disease more than once.

By

In 2014, a small study found that some people may, in fact, develop strain-specific immunity to Borrelia burgdorferi, the bacteria causing Lyme disease. “Once infected with a particular strain of the disease-causing bacteria, humans appear to develop immunity against that strain that can last six to nine years,” the authors wrote. [1] Ask the Lyme doc.

However, they also pointed out that at least 16 different strains of the Lyme disease bacterium have been shown to infect humans in the United States, so being bitten by a tick carrying a different strain of the disease is entirely possible.

Whether a person can develop immunity to Lyme disease is still unknown. Further studies are needed. There are certainly many instances of people contracting the infection more than once or redeveloping symptoms.

Re-infected through a new tick bite

There may be several reasons for this. A person can become re-infected with a new tick bite. Nadelman and colleagues described 17 patients who were re-infected multiple times with Lyme disease. [1]

Persistent infection 

Lyme disease symptoms can resurface in a person who has already been treated for the infection. Findings from both animal and human studies suggest that B. burgdorferi can cause a persistent infection. [2,3] Some doctors, however, dismiss the idea of a persistent infection. [4]

Lyme disease co-infections

Lastly, Lyme disease symptoms may return due to untreated co-infections. As of 2020, scientists have discovered 15 other diseases transmitted by ticks. [5] The list consists of viral, bacterial and parasitic infections and includes:

  1. Anaplasmosis
  2. Babesiosis
  3. Borrelia mayonii
  4. Borrelia miyamotoi
  5. Bourbon virus
  6. Colorado tick fever
  7. Ehrlichiosis
  8. Heartland virus
  9. Powassan disease
  10. Rickettsia parkeri rickettsiosis
  11. Rocky Mountain Spotted Fever (RMSF)
  12. STARI (Southern Tick-Associated Rash Illness)
  13. Tick-borne relapsing fever (TBRF)
  14. Tularemia
  15. 364D Rickettsiosis

Editor’s note: This is part of a Asl the Lyme doc series.  For the purposes of transparency, I’m an author on the International Lyme and Associated Diseases Society (ILADS) treatment guidelines, and I believe that symptoms can be due to a persistent infection in some patients. [6,7]

Related Articles:

Could Borrelia miyamotoi infections explain persistent symptoms in Lyme disease patients?

Persistent Lyme infection or inflammatory immune response?

What are Lyme disease co-infections?

References:
  1. C. E. Khatchikian, R. B. Nadelman, J. Nowakowski, I. Schwartz, G. P. Wormser, D. Brisson. Evidence for Strain-Specific Immunity in Patients Treated for Early Lyme Disease. Infection and Immunity, 2014; 82 (4): 1408 DOI: 10.1128/IAI.01451-13
  2. Sapi E, Kasliwala RS, Ismail H, et al. The Long-Term Persistence of Borrelia burgdorferi Antigens and DNA in the Tissues of a Patient with Lyme Disease. Antibiotics (Basel). 2019;8(4).
  3. Middelveen MJ, Sapi E, Burke J, et al. Persistent Borrelia Infection in Patients with Ongoing Symptoms of Lyme Disease. Healthcare (Basel). 2018;6(2).
  4. Shapiro ED. Repeat or persistent Lyme disease: persistence, recrudescence or reinfection with Borrelia Burgdorferi? F1000Prime Rep. 2015;7:11.
  5. Diseases Transmitted by Ticks. Centers for Disease Control and Prevention. https://www.cdc.gov/ticks/diseases/index.html Last accessed 12/12/20.
  6. Cameron D, Gaito A, Harris N, et al. Evidence-based guidelines for the management of Lyme disease. Expert Rev Anti Infect Ther. 2004;2(1 Suppl):S1-13.
  7. Cameron DJ, Johnson LB, Maloney EL. Evidence assessments and guideline recommendations in Lyme disease: the clinical management of known tick bites, erythema migrans rashes and persistent disease. Expert Rev Anti Infect Ther. 2014;12(9):1103-1135.

Category:

Lyme, research

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Study Shows COVID Injections Are Likely the Cause of Reported Deaths, Spontaneous Abortions, Anaphylactic Reactions, Cardiovascular, Neurological, and Immunological Adverse Events

May 20, 2021

https://www.americasfrontlinedoctors.org/frontline-news/study-analysis-suggests-the-vaccines-are-likely-cause-of-reported-deaths-spontaneous-abortions-anaphylactic-reactions-cardiovascular-neurological-and-immunological-adverse-events

Study: ‘Analysis suggests the vaccines are likely cause of reported deaths, spontaneous abortions, anaphylactic reactions, cardiovascular, neurological, and immunological adverse events’

May 19, 2021
 
 

This is a summary of a study by Dr. Jessica Rose, PhD, MSc. BSC, recently completed, submitted for publication, and accepted, entitled: A report on the U.S. Adverse Events Reporting System (VAERS) of the COVID-19 Messenger RNA (mRNA) biologicals.

The goal is make the public aware of the soaring Adverse Event reports in the context of the COVID-19 biologicals being administered en-masse prior to scientifically respectable safety and efficacy studies being completed.

The study concludes:

“This work summarizes VAERS data to date and serves as information for the public and a reminder of the relevance of any adverse events, including deaths, that likely occurred as a direct result of vaccine administration.

“Based on analysis of the VAERS numbers, it may appear that AEs are not currently imposing a significant burden on the fully vaccinated population; however, the weekly releases of VAERS data do not include all of there reports made to date — they are all the reports the CDC has processed to date — and the backlog is likely to be staggering.

“Thus, due to both the problems of under-reporting and the lag in report processing, this analysis reveals a strong signal from the VAERS data that the risk of suffering an SAE following injection is significant and that the overall risk signal is high.

“Analysis suggests that the vaccines are likely the cause of reported deaths, spontaneous abortions, and anaphylactic reactions in addition to cardiovascular, neurological and immunological AEs.

“Based on the precautionary principle, since there is currently no precedent for predictability with regards to long-term effects from mRNA injections, extreme care should be taken when making a decision to participate in this experiment. mRNA platforms are new to humans with regard to mass injection programs in the context of viruses. There is currently no way to predict potential detrimental outcomes with regards to SAE occurrences in the long-term. Also, with regards to short-term analysis, this data is limited based on reporting that likely significantly underestimates actual events.”

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**Comment**

The Presentation only included VAERS events as of April 30 – nearly a month ago. Deaths and adverse reactions have only increased since then. It’s important to keep in mind that historically VAERS only captures 1% of actual events as well as the fact it is backlogged by months. This means actual numbers are much, much higher.

According to Dr. Rose, 6% of all “breakthrough cases” (people contracting COVID after being fully “vaccinated”) resulted in death. This fact challenges manufacturer’s claims that the injections prevent death from COVID. Also, the injections do not stop transmission of COVID. This disingenuous term is meant to confuse the public that the injections don’t stop you from becoming ill, just as the PTLDS moniker for Lyme is meant to permanently end the issue of chronic/persistent infection.

Further, the CDC is up to its old shenanigans by lowering the PCR cycle threshold only for those getting the injections to downplay the fact many “fully vaccinated” are contracting COVID.

If Youtube censors this information, you can see it on rumble: https://rumble.com/vhb28z-phd-researcher-analyzes-vaers-data-and-concludes-covid-shots-are-causing-de.html

For more:

Category:

Activism, vaccines, Viruses

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