CDC director acknowledges hospitals have a monetary incentive to overcount coronavirus deaths

U.S. Centers for Disease Control and Prevention Director Robert Redfield agreed that some hospitals have a monetary incentive to overcount coronavirus deaths as they do deaths for other diseases.

“I think you’re correct in that we’ve seen this in other disease processes, too. Really, in the HIV epidemic, somebody may have a heart attack but also have HIV — the hospital would prefer the [classification] for HIV because there’s greater reimbursement,” Redfield said during a House panel hearing Friday when asked by Rep. Blaine Luetkemeyer about potential “perverse incentives.”  (See link for article)



I posted on this in April:

Due to Dr Jensen this became public knowledge, but he paid for it, as have many doctors who dare defy the narrative: (I include the fact the State Medical Board came after Jensen in the comment section. Thankfully the case was dropped)

This same CDC has monetary incentives to undercount Lyme disease.  They also NEVER, EVER will admit people can be chronically infected because that would stop lucrative vaccine development and manufacturing.

They can do whatever they please because they have no accountability and are allowed to own patents and have financial conflicts of interest but still determine public health policy.  A true case of the fox guarding the hen-house.

The rest of the world is finally getting a taste of what Lyme patients have dealt with for over 40 years.

The People With Hidden Immunity Against COVID-19

While the latest research suggests that antibodies against Covid-19 could be lost in just three months, a new hope has appeared on the horizon: the enigmatic T cell.

The clues have been mounting for a while. First, scientists discovered patients who had recovered from infection with Covid-19, but mysteriously didn’t have any antibodies against it. Next it emerged that this might be the case for a significant number of people. Then came the finding that many of those who do develop antibodies seem to lose them again after just a few months.

In short, though antibodies have proved invaluable for tracking the spread of the pandemic, they might not have the leading role in immunity that we once thought. If we are going to acquire long-term protection, it looks increasingly like it might have to come from somewhere else.  (See link for article)



The article points out that the researchers tested blood samples years before COVID and found T Cells specifically tailored to detect proteins on the surface of it.  They found some could test negative for antibodies but positive for T cells that identify COVID.  The finding is prevalent – 40-60% of unexposed individuals had these cells.

They now believe immunity is twice as common as previously thought.

Highly specific T cells hang around in the blood years after an infection and continue to identify and kill pathogens contributing to the immune system’s “long-term” memory, especially when it recognizes an old foe.

The article explains that the innate immune system, made up of white blood cells and chemical signals, is the first line of defense which then starts the production of antibodies weeks later. A little less than a week after infection, T cells get activated and start recognizing infected cells. The T cells or other parts of the immune system they recruit then eliminates the invaders before they can create viral factories that churn out copies.

This explains why antibody testing is fruitless at the beginning of infection, as Lyme/MSIDS patients are painfully aware.  Also, here is a picture of borrelia (Lyme) living quite happily with a macrophage:

There are 4 types of T cells:

  • Helper T cells cooperate with B cells in antibody production and activation of macrophages and inflammation.
  • Memory T cells persist in the blood stream to provide protection for future infections.
  • Suppressor T cells protect healthy tissues.
  • Killer T cells kill virus infected cells directly.

When it is proven & accepted that Bb infects white blood cells, it will give a potential answer to some late-disseminated Lyme cases as an immune-deficiency syndrome demonstrating why some patients have frequent and prolonged infections and may acquire new allergies as well as Mast Cell issues.

This is extremely important work that must be done if the Lyme community is to move forward.  We don’t need more climate data, we need research on pathogen persistence, better testing, better treatment, and a host of other crucial issues that will actually help Lyme patients.  Make sure you express this to Congressman Frank Pallone as well as demanding a hearing on the issues of Lyme before HHS gets another dime of tax-payer money:

July 6, 2020

By Dr. Darin Ingels N.D.

When speaking with children who are chronically ill, it can be difficult to understand what is causing their symptoms.

Children, naturally, might not know how to accurately describe their pains or illness. When there are multiple symptoms, it can be even more challenging as they grow and change so quickly.

For children with autism or other language disorder, they may be limited or unable to communicate why they feel the way they feel. Autism Spectrum Disorder (ASD) and Lyme disease are examples of what seem to be entirely different diseases, but they share an overlap of symptoms.

While autism is usually seen as a developmental disorder and Lyme disease and infectious disease, the two have more in common than you might think. There are interesting connections between the two, especially when diagnosed in children.

Sad boy with symptoms of lyme disease

Symptoms shared by both Autism and Lyme:

  • Neurological symptoms that include difficulty with communication and confusion, disorientation, muscle twitching, sensitivity to light, brain fog, and delayed development.
  • Psychological problems that impact behaviors, obsessive-compulsive disorder, an increased sense of doom, anxiety and outbursts.
  • Physical health issues such as muscle weakness, arthritis, and rashes.
  • Gut health issues including food allergies, bloating, constipation or diarrhea, and abdominal pain.

These symptoms are common features of autism and Lyme disease.

Coincidentally, many of these symptoms are also displayed in auto-immune disorders.

Tests for Lyme can be misleading, as they have a poor accuracy. A specialist is always needed in order to get a better sense of other treatment options because both autism and Lyme can have long-term issues.

However, there are treatments that benefit Lyme and autism alike. Focusing on gut health has been an important part of treatment for both conditions. This is because we are seeing the benefits of specific diets in patients with autism and/or Lyme.

Nutritional support strengthens the integrity of the intestinal membranes, balances the billions of bacteria in our gut and improves digestion and elimination.

All of this help support the immune function of the gut, which ultimately affects brain function.

An effective nutritional protocol would support the immune system, reduce symptoms, calm the nervous system and strengthen the body’s ability to fight infections.

Autoimmune conditions such as autism and Lyme disease benefit greatly from proper diet and lifestyle modifications.


Removing casein, dairy, sugar, processed foods and gluten from the diet will allow the body to heal and aid in the detoxification process, naturally.

Reducing environmental factors like external and emotional stressors are extremely important for both Lyme and ASD.

Stress responses increase the load on the immune & nervous system, which can lead to exhaustion and further relapse into symptoms.

Identifying these triggers help you to work around them and eventually train your nervous system to create new patterns and get rid of the old ones. Autoimmune conditions have very unique impacts on the immune system, especially Lyme and autism.

Consider speaking to a specialist about your symptoms, especially if they mimic other autoimmune conditions. And never be afraid to get a second or even third opinion, as it may be necessary in order to get to the root of problem.


For more:

Leah Lamonte, vector control specialist for the Allegheny County Health Department, looks for blacklegged ticks in their nymph stage after dragging a cloth along a trail in Hartwood Acres on July 30, 2020, in Allison Park.

Allegheny County’s tick collector warns of Lyme disease risks

Pam Panchak / Post-Gazette

The governor said he felt confident in the results of a negative test that was taken hours after he tested positive while being screened to greet President Trump.

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Gov. Mike DeWine tested negative for the coronavirus hours after a positive rapid-result test had prevented him from welcoming President Trump to Ohio on Thursday, a whiplash reversal that reflected the nation’s increasingly complex state of testing.
In a high-profile example of a new testing frontier, Mr. DeWine first received an antigen test, which allows for results in minutes, not days, but has been shown to be less accurate. The positive result came as a “big surprise,” said Mr. DeWine, a Republican, who had not been experiencing symptoms other than a headache.
Later on Thursday, he was tested using a more standard procedure known as polymerase chain reaction, or P.C.R., an accurate but time-intensive method that requires samples to be processed at a laboratory. His wife, Fran, and staff members also tested negative.  (See link for article)
And this is where testing remains…..
Important excerpt:
Public health experts say that widespread, rapid testing is necessary for quarantining and contact tracing to effectively control the virus.
This, not the virus, should frighten us.
They have been and will continue to use abysmal testing to take peoples’ freedoms away.

Are you infectious if you have a positive PCR test result for COVID-19?

August 5, 2020

Tom Jefferson, Carl Heneghan, Elizabeth Spencer, Jon Brassey

PCR detection of viruses is helpful so long as its accuracy can be understood: it offers the capacity to detect RNA in minute quantities, but whether that RNA represents infectious virus may not be clear.

During our Open Evidence Review of oral-fecal transmission of Covid-19, we noticed how few studies had attempted or reported culturing live SARS-CoV-2 virus from human samples.

This surprised us, as viral culture is regarded as a gold standard or reference test against which any diagnostic index test for viruses must be measured and calibrated, to understand the predictive properties of that test. In viral culture, viruses are injected in the laboratory cell lines to see if they cause cell damage and death, thus releasing a whole set of new viruses that can go on to infect other cells.

We, therefore, reviewed the evidence from studies reporting data on viral culture or isolation as well as reverse transcriptase-polymerase chain reaction (RT-PCR), to understand more about how the PCR results reflect infectivity.  (See link for article)



In one of the best reviews I’ve read on PCR testing so far the authors point out the fly in the ointment: few studies have cultured live SARS-CoV-2 virus from human samples.  This is a big deal.  BIG.  Without injecting live viruses into cells lines to determine infectivity, it’s all theoretical.  And I’ll add one more to that: these viruses must be not only isolated but purified from all else.  In the case of COVID, to my knowledge, this has not been done.  According to David Crowe, all they have is pieces and parts they are labeling “virus.” This is an important distinction and quite fundamental.  For a great read on this:

Back to the paper on PCR testing.Viral cultures for COVID-19 infectivity assessment. Systematic review. Tom Jefferson, Elizabeth Spencer, Jon Brassey, Carl Heneghan medRxiv 2020.08.04.20167932; doi:

The authors reviewed 14 studies that they labeled of “moderate quality” due to being inadequately sized, lack of protocols, standardized methods and reporting and reporting bias. They hit on some interesting issues like time of testing in relation to symptom severity, viral shedding, etc.  They also pointed out that time of testing is important because:

The lower the cycle threshold level the greater the amount of RNA (genetic material) there is in the sample. The higher the cycle number, the less RNA there is in the sample.

What does this mean?

This detection problem is ubiquitous for RNA viruses detection. SARS-CoV, MERS, Influenza Ebola and Zika viral RNA can be detected long after the disappearance of the infectious virus.

In other words, the test is picking up RNA material but the patient isn’t infected any more. The authors point out that this material can linger for weeks in the body.

The authors then sum it up by stating that the 14 studies provided limited data of variable quality of PCR results and are unlikely to predict viral culture from human samples.  They state:

Insufficient attention may have been paid how PCR results relate to disease. The relation with infectiousness is unclear and more data are needed on this.

And the most important point:

If this is not understood, PCR results may lead to restrictions for large groups of people who do not present an infection risk.
BINGO!  This is exactly what is happening.

For more:

Right here in Wisconsin, Governor Evers is pushing ‘contact tracing’ as part of the Badger Bounce-Back Program based on faulty testing:  DHS is coordinating the amount of tracers with the number of projected tests and positive cases with the goal of having 1,000 statewide tracers.

PCR testing:


I’m skeptical that a PRC test is ever true. It’s a great scientific research tool. It’s a horrible tool for clinical medicine.Dr. David Rasnick, bio-chemist, protease developer, and former founder of an EM lab called Viral Forensics

Rasnick’s advice for people who want to be tested for COVID-19.