Archive for the ‘Testing’ Category

How to Spot the Symptoms of Lyme Disease in Dogs

https://www.thesprucepets.com/lyme-disease-in-dogs-3384701

How to Spot the Symptoms of Lyme Disease in Dogs

Lyme disease is caused by a bacteria called Borrelia burgdorferi and is spread by ticks. Ticks become infected with the bacteria by feeding on infected mice and other small animals. When an infected tick bites other animals, it can transmit the bacteria to these animals. Lyme disease is transmitted by the deer tick (black-legged tick) and a small group of other closely related ticks. The deer tick is small and may bite animals and people without being detected. Lyme disease affects a variety of species, including dogs, cats, and people. Up to 95 percent of dogs infected with B. burgdorferi do not develop symptoms (people are much more likely to become ill with Lyme disease).

There is no evidence that Lyme disease is spread by direct contact with infected animals. However, keep in mind that ticks can hitch a ride home on your pets and move on to the humans in the household. **Please see my comment at end of article**

Risk Factors

Dogs that spend a lot of time outdoors, especially in the woods, bush, or areas of tall grass are most commonly infected with Lyme disease. However, ticks can be carried into yards on other animals, and dogs can become infected anywhere ticks are found.

Infections occur during tick season (usually spring through early fall), but the time between infection and the appearance of Lyme disease symptoms can be up to 2-5 months. **Please see comment**

Lyme disease is seen across the US and in many other parts of the world. In the US. Lyme disease is most common in the northeastern US, along with the Pacific coast, and in the midwest.

Signs of Lyme Disease

When clinical signs do develop, they may be transient or recurrent, and can include:

  • Fever.
  • Decreased appetite.
  • Swollen, painful joints (dogs may be reluctant to move).
  • Lameness — limping which may be mild at first, then worsen, and may also shift from one leg to another.
  • Lethargy.
  • Swollen lymph nodes.

Some dogs with Lyme disease may develop kidney disease.

Signs of kidney disease may include depression, vomiting, loss of appetite, and increased thirst and urination (sometimes a lack of urination will develop). Dogs who develop kidney disease can become very ill and may not respond to treatment.

Neurological disease (behavioral changes, seizures) and heart complications, which are sometimes seen in humans, are rare in dogs.

Diagnosis of Lyme Disease

The diagnosis of Lyme disease must be based on a combination of factors, including history (tick exposure), clinical signs, finding antibodies to B. burgdorferi bacteria, and a quick response to treatment with antibiotics.

A positive antibody test is not enough to make a diagnosis on its own, because not all dogs that are exposed to B. burgdorferi get sick, and antibodies can persist in the blood for a long time after exposure.

Other diagnostic testing, such as blood and urine tests, x-rays, and sampling of joint fluid, may be done to check for signs of kidney disease and to rule out other conditions with similar signs and symptoms.

Treating Lyme Disease

Treatment with antibiotics usually produces rapid improvement in symptoms (antibiotics will be continued for a few weeks). Treatment may not be completely clear the bacteria, but produces a state where no symptoms are present (similar to the condition in dogs that don’t have symptoms from infection).

Kidney disease may develop some time after the initial infection, so is it a good idea to regularly check for excess protein in the urine of dogs that have had Lyme disease. Catching the kidney disease early in its course offers the best prognosis. If kidney disease is present, a longer course of antibiotics along with additional medications to treat the kidney disease is usually necessary.

Preventing Lyme Disease
  • Tick Control is extremely important for the prevention of Lyme disease (and many other diseases that can be transmitted by ticks). Check your dog daily for ticks and remove them as soon as possible, since ticks must feed for at least 12 hours (possibly 24-48 hours) before transmitting the bacteria causing Lyme disease. This is especially important in peak tick season and after your dog spends time in the bush or tall grass (consider avoiding these areas in tick season).  Products that prevent ticks such as monthly parasite preventatives (e.g., Frontline®, Revolution®) or tick collars (e.g., Preventic®) can be used; be sure to follow your veterinarian‘s advice when using these products. Keep grass and brush trimmed in your yard, and in areas where ticks are a serious problem, you can also consider treating your yard for ticks.  **Again, please see my comment at end of article**
  • Vaccines for Lyme Disease: Vaccination against Lyme disease is a controversial topic and is something that should be discussed in depth with your veterinarian. Many specialists do not recommend routine vaccination because so few dogs develop symptoms of Lyme disease, and when Lyme disease does occur in dogs, it is usually readily treated. Additionally, because arthritis and kidney problems associated with Lyme disease are at least partly related to the immune response to the bacteria (rather than the bacteria itself), there is concern that vaccination may contribute to problems. Vaccination is also not 100 percent effective, and it’s only helpful in dogs that have not already been exposed to B. burgdorferi. However, vaccination before exposure can help prevent dogs from getting Lyme disease and also prevent them from becoming a carrier of the bacteria. Where vaccines are used, it is usually recommended to start vaccinating dogs as young puppies (e.g., at around 12 weeks, with a booster 2-4 weeks later). The vaccine does not provide long-lasting immunity, so annual re-vaccination (ideally before tick season) is necessary. The recombinant form of the vaccine is considered to have less potential for side effects than the bacteria form of the vaccine.
Please note: this article has been provided for informational purposes only. If your pet is showing any signs of illness, please consult a veterinarian as quickly as possible.
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**Comment**
While there are many useful take-aways from this article, a number of myths continue to be propagated.  
  1. There is evidence that there is transmission by direct contact with animals.  http://www.lymerick.net/Transmission-Bb-contact.htm  (Here we see evidence of Bb in feces, urine, tick excretes, cow milk, food, in utero, transplacental, sexual, semen, and mucus membranes.)
  2. My vet treated my dog for longer than a couple of weeks.  I think that wise knowing the organism reproduces slowly.  They also have the canine equivalent of probiotics but they are designed for a dog’s micro biome so don’t give him yours.
  3. The fallacy of it taking 24-48 hours to be transmitted, is just that – a fallacy.  Please read more about transmission time here:  https://madisonarealymesupportgroup.com/2017/04/14/transmission-time-for-lymemsids-infection/
  4. Transmission can occur at ANY TIME of the YEAR.  I have buddies pulling live ticks off their dogs in Northern Wisconsin in February.

Also, please note the comments about the vaccine.  They always want to state how great it is in animals but I see many comments that suggest extreme caution – similarly to the human Lyme vaccine.  First, it doesn’t provide lasting immunity, it causes obvious side-effects, it’s not 100% effective, and vaccination can make things worse for dog exposed to Bb.  Since this can be transmitted congenitally, it’s pretty hard to know what dogs already have Bb.  It’s Russian Roulette with dogs just as much as with humans.  Buyer beware.

Aluminum in the Brain in Multiple Sclerosis: Regulatory and Funding Agencies Silent, Complicit

https://jameslyonsweiler.com/2018/08/18/aluminum-in-the-brain-in-multiple-sclerosis-regulatory-and-funding-agencies-silent-complicit/

By James Lyons Weiler in Cures August 18, 2018

https://jameslyonsweiler.com

MEDICAL SCIENCE proceeds along a hierarchy of evidence; often, patients are studied individually (case studies), or a small collection of patients are examined and characterized together (case series studies). Case series studies typically have smallish sample sizes and it is generally understood that larger studies will be necessary to determine more accurately the characteristics being studied.

In a new case series study, brain tissue from 14 donors with a diagnosis of MS was studied in a case series by Mold et al (2018) using transversely heated graphite furnace atomic absorption spectrometry. The study found high aluminum content (>10 ug/g dry weight) in all areas of the brain studied, with some areas exceeding 50 ug/g. They found aluminum both with cells and in the interstitium between cells. They found aluminum co-localised with structures known to be present in Secondary Progressive Multiple Sclerosis (SPMS) in the frontal cortex of one donor with SPMS.

There are a number of critical lines of evidence that make this fundamental finding critically important. Patients with MS have lower amounts of aluminum in their hair, suggesting depressed detoxification, and higher amounts are found in urine on chelation challenge testing (Fulgenzi et al., 2014). Chelation with EDTA is known to significantly reduce aluminum intoxication (Fulgenzi et al. 2014), and consumption of silica-rich mineral waters also increase urinary excretion of aluminum from patients with SPMS (Jones et al., 2017).

Extremely plausible direct mechanisms of the cause of MS from aluminum are known and animal studies routinely induced MS using aluminum hydroxide injections. So much evidence exists that points to aluminum as a source of strange new conditions of unknown causes, such as MMF and Gulf War Syndrome, one would think that calls to reconsider the use of aluminum in vaccines would be answered. The same team had previously found high amounts of aluminum in the brains of people with autism, and and in patients with Alzheimer’s disease. The latter result, while also important, is not surprising, as it has long been known that amyloid is part protein and part aluminum. Finally, when France brought on HepB vaccination, cases of MS following vaccination increased; when they stopped recommending the HepB vaccine, which contains aluminum hydroxide, the rate of HepB vaccine-associated MS cases dropped to near zero.

francems

Complicity

What is surprising is the lack of action on the part of the US FDA to put an end to the use of this dangerous metal in vaccines, and that the NIH is not funding more studies like this. NIH should fund studies to determine how to most safely remove aluminum from anyone exposed via vaccines; brain stem amyloidosis is a non-trivial concern. Approaches like ketogenic diet, silica-rich mineral waters, hyperbaric oxygen, EDTA, intranasal insulin and intranasal deferoxamine (to prevent brain stem amyloidosis) should all be tested in randomized clinical trails in clinical populations known to be afflicted with aluminum intoxication (autism, Alzheimer’s, MS).

What is also surprising is that the CDC and ACIP remain blithe to the morbidity and mortality their continued approval of vaccines that contain metals like aluminum and mercury. They make decisions on behalf of us all, and yet every member of ACIP with the exception of one military member has conflicts of interest with vaccine manufacturers. ACIP should review all of the literature on aluminum and make recommendations on how to phase it, and thimerosal, out of vaccines completely.

Further inaction on the part of these regulatory and funding agencies, and active denialism at this point will surely be seen by future generations as both callous disregard, and where conflicts of interest reside, complicity.

The study, conducted at the Keele University, was funded in part by the Children’s Medical Safety Research Institute.

References

Jones K et al. EBioMedicine. 2017 Urinary Excretion of Aluminium and Silicon in Secondary Progressive Multiple Sclerosis. 26:60-67. doi: 10.1016/j.ebiom.2017.10.028.

Fulgenzi A, Vietti D1, Ferrero ME. Aluminium involvement in neurotoxicity. Biomed Res Int. 2014;2014:758323. doi: 10.1155/2014/758323.

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**Comment**

https://www.nationalmssociety.org/Symptoms-Diagnosis/Other-Conditions-to-Rule-Out/Lyme-Disease

Lyme disease can cause delayed neurologic symptoms similar to those seen in multiple sclerosis (MS) such as weakness, blurred vision caused by optic neuritis, dysesthesias (sensations of itching, burning, stabbing pain, or “pins and needles”), confusion and cognitive dysfunction, and fatigue. Lyme disease symptoms may also have a relapsing-remitting course. In addition, Lyme disease occasionally produces other abnormalities that are similar to those seen in MS, including positive findings on magnetic resonance imaging (MRI) scans of the brain and analysis of cerebrospinal fluid (CSF).

These similarities in symptoms and test results have led some people with MS to seek testing for the presence of antibodies to Borrelia, to determine if their neurologic symptoms are the result of Lyme disease or truly MS. The distinction is important because Lyme disease, especially when treated early, often responds to antibiotic therapy, whereas MS does not.

Studies examining Lyme disease & MS
Two studies have examined the overlap in diagnosis of MS and Lyme disease. The studies were conducted in parts of Long Island, New York, an area where Lyme disease is endemic, or regularly found.

In the first study, people who had Borrelia antibodies in their blood as well as a variety of neurologic symptoms considered to be “MS-like,” were evaluated with MRI, evoked potentials (EP) and CSF analysis, including a test for the presence of Borrelia antibodies in the spinal fluid.

While those with the MS-like illness had the highest incidence of abnormal MRIs and were the only ones among those studied to have abnormal EP and oligoclonal bands in their spinal fluid (indicating an abnormal immune response), they did not prove to have any Borrelia antibody in their spinal fluid.

The researchers concluded that the few patients with the MS-like symptoms probably had these symptoms due to MS and had also been exposed to the Borrelia bacterium.
A companion study looked for the presence of Borrelia antibodies in the blood of 100 people with the diagnosis of possible MS. Of 89 people who in fact turned out to have definite MS, only one had Borrelia antibodies. The researcher concluded that “…infection with Borrelia is infrequent in MS patients who live in an endemic area. Lyme disease is unlikely to be a significant factor in the differential diagnosis of MS.” Furthermore, the presence or antibodies to Borrelia does not prove that Borrelia is causing the neurological symptoms, only that there has been previous infection with the organism.

Just remember, “rare” is only “rare” if it isn’t you.

 

Our Battle Ongoing: Lyme Disease in Australia

 Approx 1 hour 30 min.

Our Battle Ongoing: Lyme Disease in Australia | Full Documentary

Directed by Andy Smith
Published on May 25, 2017

Our Battle Ongoing takes a look into the lives of Australians across the nation currently suffering from Lyme disease or a Lyme-like illness. Every individual has their own unique story as symptoms differ from person to person, but they are all united in a common cause of seeing recognition come to those suffering now and for the generations to come. With little to no help currently available, some have to take drastic measures in order to receive treatment. Our Battle Ongoing brings to light the tragedy of living with an invisible illness in Australia.

www.ourbattleongoing.com

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**Comment**

The number of afflicted Australians is growing yearly just as it is worldwide.  To deny this pandemic is truly an “Emperor has no clothes” story.  You’ve got thousands of people with Lyme-like symptoms, most of who improve with appropriate treatment, but denial from the establishment.  For every patient who can high-tail it to Germany for treatment, you have 1,000 who can’t afford to.

Devastating.

While it is bad here in the U.S., it is truly abysmal in Australia.

Recently I posted this:  https://madisonarealymesupportgroup.com/2018/08/20/tick-borne-diseases-causing-autonomic-dysfunction/ and declared it a literal rap sheet for Lyme.  Please compare this article to the patients you see in this documentary.  They are identical.  

While I agree that Australia is a unique area with its own flora and fauna and pathogens, don’t kid yourself – migrating birds have no problem transiting ticks to and from Australia or anywhere else on the planet.  To say otherwise is burying your head in the sand.  Time to wake up and quit blaming the climate and look around at the integrated world in which we live with animals and humans moving in and out of areas with ticks on them on a daily basis.  For more on why the climate is NOT responsible for this pandemic:  https://madisonarealymesupportgroup.com/2018/08/13/study-shows-lyme-not-propelled-by-climate-change/

More on Lyme in Australia:  https://madisonarealymesupportgroup.com/2016/11/03/ld-not-in-australia-here-we-go-again/

https://madisonarealymesupportgroup.com/2017/09/19/tbis-in-australia/

https://madisonarealymesupportgroup.com/2018/08/08/an-invasive-new-tick-is-spreading-in-the-u-s/  Known in Australia as bush ticks and in New Zealand as cattle ticks, long-horned ticks can multiply rapidly and suck so much blood from a young animal that it dies. The ticks bloat up like fat raisins until their tiny legs are barely able to support them.  After a blood meal, females can lay hundreds of fertile eggs without mating.

https://madisonarealymesupportgroup.com/2018/08/02/12-scary-diseases-you-can-catch-at-the-beach-lyme-is-one-of-them/  Ticks have been found on the beach, in caves, underneath picnic benches, on rocks, have blown from trees into swimming pools, have blown onto my house by a lawn mower and ended up crawling up the interior basement walls.  Can we just say ticks can be virtually everywhere with the need for extreme diligence?

https://madisonarealymesupportgroup.com/2018/03/23/australian-lyme-disease-research-pilot-funded/  The study utilises a proprietary capture methodology that has not previously been employed in the detection of tickborne pathogens in Australia.

While the authorities don’t believe you, we do.  

First Identification in China of Guertu Virus From Ticks

https://www.ncbi.nlm.nih.gov/m/pubmed/29802259/

A novel tick-borne phlebovirus, closely related to severe fever with thrombocytopenia syndrome virus and Heartland virus, is a potential pathogen.

Shen S, et al. Emerg Microbes Infect. 2018.

Abstract

Tick-borne viral diseases have attracted much attention in recent years because of their increasing incidence and threat to human health. Severe fever with thrombocytopenia syndrome phlebovirus (SFTSV) and Heartland virus (HRTV) were recently identified as tick-borne phleboviruses (TBPVs) in Asia and the United States, respectively, and are associated with severe human diseases with similar clinical manifestations. In this study, we report the first identification and isolation of a novel TBPV named Guertu virus (GTV) from Dermacentor nuttalli ticks in Xinjiang Province, China, where TBPVs had not been previously discovered. Genome sequence and phylogenetic analyses showed that GTV is closely related to SFTSV and HRTV and was classified as a member of the genus Phlebovirus, family Phenuiviridae, order Bunyavirales. In vitro and in vivo investigations of the properties of GTV demonstrated that it was able to infect animal and human cell lines and can suppress type I interferon signaling, similar to SFTSV, that GTV nucleoprotein (NP) can rescue SFTSV replication by replacing SFTSV NP, and that GTV infection can cause pathological lesions in mice. Moreover, a serological survey identified antibodies against GTV from serum samples of individuals living in Guertu County, three of which contained neutralizing antibodies, suggesting that GTV can infect humans. Our findings suggested that this virus is a potential pathogen that poses a threat to animals and humans. Further studies and surveillance of GTV are recommended to be carried out in Xinjiang Province as well as in other locations.

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**Comment**

More and more research showing viruses in ticks is coming out.  I pray authorities are taking note.  While the viruses may or may not cause direct symptoms, they certainly must be considered in patient cases as the overall immune system will be impacted and have the potential to make cases more severe.  We desperately need research in this area.

Many practitioners find patients improve when anti-viral medications are used and the immune system is strengthened.

**For more on Thrombocytopenia Syndrome** https://wwwnc.cdc.gov/eid/article/20/11/14-0888_article

(SFTS) is a newly emerging infectious disease. Symptoms and laboratory abnormalities are fever, thrombocytopenia (low platelet count), leukocytopenia (low white blood cell count), and elevated liver serum enzyme levels. Multiorgan failure occurs in severe cases, and 6%–30% of case-patients die. The syndrome is caused by the SFTS virus (SFTSV) (genus Phlebovirus, family Bunyaviridae). SFTS case-patients were first reported in China (1) and more recently were reported in Japan (2) and South Korea (3). Two case-patients with symptoms consistent with a similar virus, Heartland virus, were reported in the United States (4).

Ixodid tick species are implicated as vectors of SFTSV (1,5,6). One study described a SFTSV prevalence in Haemaphysalis longicornis ticks, a major vector of SFTSV, of 0.46% minimum infection rate in South Korea (7); in another study, SFTSV was detected in ticks that had bitten humans (6). From these studies, we realized that SFTSV was common throughout the country. We aimed to evaluate the prevalence of SFTS in South Korea and isolate the SFTSV to analyze its phylogenetic properties.
The major signs and symptoms of the 35 case-patients, including fever (100%), gastrointestinal symptoms (74%), fatigue (74%), thrombocytopenia (100%), and leukocytopenia (100%), were similar to those of case-patients in China and Japan (9).

It is mentioned that the “Asian” SFTSV and the “U.S.” HRTV have similar clinical manifestations.

Please know that ticks do not regard borders and are being transited everywhere by migrating birds and other mammals and even reptiles.

https://madisonarealymesupportgroup.com/2018/06/08/hemorrhagic-fever-virus-found-on-ticks-on-migratory-birds/  An example of Hemorrhagic fever virus on ticks on migratory birds.

https://madisonarealymesupportgroup.com/2018/08/19/monster-ticks-found-in-germany-threaten-europe-with-deadly-disease-crimean-congo-fever/  This recent article shows a tick with a disease that shouldn’t be in Germany but is.  They also found one tick to have a tropical form of tick typhus.

https://madisonarealymesupportgroup.com/2017/08/11/death-from-tick-borne-virus-sfts/  1st recorded death in Japan from SFTS and the patient didn’t even have a tick bite but rather a cat bite demonstrating the first recorded mammal to mammal transmission.

The aforementioned haemaphysalis longicornis (Asian Longhorned tick or bush tick) tick is in now in at least 7 U.S. states:  https://madisonarealymesupportgroup.com/2018/07/19/rutgers-racing-to-contain-asian-longhorned-tick/.  So again, although it’s considered an Asian tick it’s here which means the potential to transmit the diseases considered “Asian” could be here as well.

Milford Pathologist Fires Broadside at CDC Motion to Discuss

https://www.change.org/p/1120418/u/23141062?

Milford Conn. Pathologist Fires Broadside at CDC Motion to Dismiss

Carl Tuttle
Hudson, NH
AUG 15, 2018 —

MILFORD MOLECULAR DIAGNOSTICS
2044 Bridgeport Avenue
Milford, CT 06460
www.dnalymetest.com
August 15, 2018
Media Contact: Kevin Moore, 203-788-8497

FOR IMMEDIATE RELEASE

Milford Conn. Pathologist Fires Broadside at CDC Motion to Dismiss in Groundbreaking $57.1 Million Lyme Disease Lawsuit

Demonstrates that CDC relied on “unreliable” Wikipedia as source to discredit Dr. Lee

Milford, Conn… Sin Hang Lee, M.D., the Connecticut pathologist who, in May, filed a $57.1 million lawsuit against the Centers for Disease Control, in a legal opposition to the CDC’s motion to dismiss his lawsuit, informed the U.S. Court of Federal Claims, that the CDC had relied on unverifiable, non-peer reviewed Wikipedia as a source for informational support to back its motion to dismiss.

To suppress direct detection tests for Lyme disease,” said Dr. Lee, referring to the Sanger DNA sequencing testing method that he employs, and, which is at the heart of his lawsuit, “the CDC is willing to exhaust all of its administrative remedies. When its patented metabolomics technology could not stop Sanger sequencing in science, the CDC told its lawyers to look up Wikipedia for help in a motion to dismiss my lawsuit.”

In 2013, after testing two panels of Lyme disease reference serum samples from the CDC by Sanger sequencing, Dr. Lee informed the CDC that some of the archived serum samples taken from patients with Lyme disease in fact were positive for Borrelia miyamotoi and a novel unnamed relapsing fever borrelia, and published the data in a peer-reviewed article. Years later, the CDC claimed in social media that Dr. Lee published “inconsistent results”  https://www.change.org/p/the-us-senate-calling-for-a-congressional-investigation-of-the-cdc-idsa-and-aldf/u/15796418  while promoting its own, newly patented, unproven metabolomics technology for diagnosis of Lyme disease.

In order to deny the facts that clinical Lyme disease may be caused by a diversity of borrelial strains of bacteria, the CDC instructed its lawyers to quote Wikipedia, an online encyclopedia with no peer review, as the alternative science to discredit Dr. Lee’s work in the U.S. Court of Federal Claims. The CDC attorneys then filed the following with the U.S. Court of Federal Claims:

“However, Borrelia miyamotoi is not a causative agent of Lyme disease. Wikipedia, https://en.wikipedia.org/wiki/Borrelia_ miyamotoi p. 1 (“Although infection [with Borrelia miyamotoi] can cause some similar symptoms [as Lyme disease] including fever, headache, fatigue, and muscle aches, acute Lyme disease often presents with rash, while infection with B. miyamotoi does not; it remains unclear whether B. miyamotoi causes a relapsing fever syndrome”).

In the PLAINTIFF’S RESPONSE TO MOTION TO DISMISS filed on August 13, 2018, Dr. Lee’s attorney, Mary Alice Moore Leonhardt, countered with the following statement:

“The Defendant relied on the inherently unreliable website Wikipedia in its attempt to discredit Dr. Lee, rather than the CDC’s own data. The CDC expressly acknowledges that Borrelia miyamotoi causes hard tick relapsing fever and Borrelia miyamotoi infection causes fever, chills and headache which are common symptoms in Lyme disease, and may cause skin rash in about 8% of the patients (4/51). (CDC, Borrelia miyamotoi Disease, available at https://www.cdc.gov/ticks/tickbornediseases/borrelia-miyamotoi.html
CDC, B. miyamotoi, available at https://www.cdc.gov/ticks/miyamotoi.html Thus, Dr. Lee’s test results detected the presence of two tick-borne illnesses, including Lyme disease and a disease that presents in similar ways to Lyme disease. These results were 100% accurate as confirmed through the DNA sequencing with the Gene Bank.”
The CDC should come out to debate the science and technology in direct detection testing for the diagnosis of Lyme disease instead of hiding behind a wall of lawyers and Wikipedia encyclopedia,” said Dr. Lee. “Accurate diagnosis of Lyme borreliosis must not be stopped by CDC lawyers.”
###

* The official documents with Appendix filed in the U.S. Court of Federal Claims can be accessed through the Drop Box. Complete filing here: https://www.dropbox.com/sh/zkcp96z7eua1fnn/AAA377iX8aZFQZs7BlQbUGb5a?dl=0

Milford Molecular Diagnostics Laboratory:  http://www.dnalymetest.com/lymediseasednatesting.html  
Milford Medical Laboratory offers the first reliable DNA test for Lyme disease bacteria and B. miyamotoi, the spirochete causing a Lyme disease-like infection.
http://www.dnalymetest.com