Archive for the ‘research’ Category

Bulletin: 14 Year Fugitive From Justice Poul Thorsen Arrested in Germany For Defrauding the US Government: What This Means For the Vaccine/Autism Question

https://popularrationalism.substack.com/p/bulletin-fourteenyear-fugitive-from?

BULLETIN: Fourteen‑Year Fugitive from Justice Poul Thorsen Arrested in Germany for Defrauding the US Government: What This Means for the Vaccine/Autism Question

Resources. As always, we present the facts without speculation. Background and facts you can use.

James Lyons-Weiler, PhD
Sep 16, 2025

Poul Thorsen, a Danish physician and researcher prominently involved in vaccine‑autism epidemiology, was indicted in 2011 in the U.S. for allegedly misappropriating more than $1 million in CDC grant funds. For over a decade he was a fugitive who face, name and charges were openly displayed on the US OIG Most wanted. Breitbart News has reported Thorsen was arrested in June under INTERPOL red notice.

This POPULAR RATIONALISM BULLETIN article reconstructs Thorsen’s academic contributions, the criticisms of his methodological work, the legal findings, and the implications for public policy and scientific trust. It places legal culpability in the context of scientific reliability/implications for other Danish studies, identifies where those studies he co‑authored have been called out for design weaknesses or uncorrected biases, and outlines what must be done to restore clarity.  (See link for timeline and article)

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Category:

Activism, research, vaccines

KU Parters With Biotech Lab For Lyme & Chlamydia Vaccines

https://molecularbiosciences.ku.edu/news/article/ku-partners-with-biotech-lab-producing-vaccines-for-lyme-disease-and-chlamydia

KU partners with Biotech Lab producing vaccines for Lyme disease and Chlamydia

Mon, 12/02/2024

Logan Pierson

The University of Kansas is helping to develop both a Lyme disease vaccine and a Chlamydia vaccine with Lawrence born biotechnology lab, Design-Zyme, and the projects all started with help from a KU graduate student.

Peter Petillo, Design-Zyme founder and CEO, and Professor Scott Hefty, from the KU school of molecular biosciences, act as the foundation for a partnership between university and industry. They said that they have known each other for almost 15 years and met through a mutual colleague at the University. Hefty said that their collaboration between commercial lab and academia gives graduate students and faculty that are early in their careers a lot of opportunity to learn.

Petillo agreed and added, “Most of the students coming out of the program are not going to work in academics. They’re going to work in some sort of an industrial setting.”

This partnership led to fifth year PhD candidate Lexie Cutter obtaining an internship at Design-Zyme, through Hefty.

While Hefty and Petillo had started a Lyme disease vaccine project, and Hefty had introduced the idea of developing a Chlamydia vaccine, it was Cutter’s work through the internship with Design-Zyme and Petillo which pushed the initial Lyme disease vaccine project forward. Cutter said Hefty helped put together the internship with Petillo two summers ago and that her project there was to produce the proteins which formed the Lyme disease vaccine.

KU Students, Faculty and Partners Progressing Vaccine Production

According to Petillo, Design-Zyme’s method for creating a vaccine consists of taking one protein from the disease-causing bacteria and bonding it to another. The proteins are meant to stimulate the immune system and build up a response to the bacteria, which causes a disease.

Petillo said the process for adding proteins together to create vaccines can be more difficult depending on the proteins and that it has been more difficult for the development of the Chlamydia vaccine. This has led to the Chlamydia vaccine ending up behind in development compared to the Lyme disease vaccine. As a result, Cutter is currently working on the continuing production of the Chlamydia vaccine, using the same method that she used when helping to create the Lyme disease vaccine.

However, the Lyme disease vaccine is progressing much faster and other faculty and students on the project have separate roles which contribute to the testing and research of the vaccine. This includes KU Lab Technicians Dominique Jaramillo and Nancy Schwarting.

Jaramillo and Schwarting said they handle mouse injections for the Lyme disease vaccine. This process involves directly injecting a mouse with the vaccine before using another syringe to inject the borrelia burgdorferi bacteria or the bacteria that causes Lyme Disease, into the mouse.  (See link for article)

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**Comment**

Interestingly, the next step will be the ‘tick challenge study’ where they will collect ticks that have the ‘right form of the bacteria.’  This is an admission that the ‘vaccine’ will only be based upon this singular ‘right’ form.  There’s numerous strains of borrelia that people have been infected with.  This is a problem in itself, not to mention the fact patients are often coinfected with other pathogens simultaneously.

The author then regurgitates the accepted narrative that ticks have increased due to ‘global warming,’ which has been proven to be false.  The article then goes on to state that there hasn’t been a Lyme vaccine since 2002 when LYMRrix was withdrawn by its manufacturer citing poor sales, as the reason – but adding as an afterthought that a ‘small portion’ who took it experienced arthritis.

Well, that’s the understatement of the year. 

See top link below for the real story.

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Category:

Activism, Lyme, research, Ticks, vaccines

Abyssinian in Crisis – Cured By MMS

https://pierrekorymedicalmusings.com/p/report-a-case-of-persistent-feline?

Abyssinian in Crisis: How One Cat’s Mysterious Illness and an Unconventional Therapy Transformed a Family — A Physician-Patient Case Report

One of my favorite cancer patients shared her increasing anxiety over Pearl, her new Abyssinian cat that was dying of viral infections. An offhand suggestion led to a complete recovery.

Pierre Kory, MD, MPA
Sep 02, 2025

This is the third report in a growing series of cases successfully treated with chlorine dioxide therapy. The first two were of my own personal illnesses (infectious colitis and paronychial abscess), and this third one is of… a cat named Pearl.

The following case report is co-authored by me and my patient, who has requested anonymity (I will refer to her as Laura).

In a recent follow-up visit with Laura, whom I treat for the prevention of recurrence of breast cancer (which she has had 4 separate times), one of the many topics we covered was her glowing update on the condition of her cat Pearl. Meet Pearl:

SUMMARY:

Dr. Kory’s case report written for an academic journal (AI-assisted).

Title

Clinical Report: A Case of Persistent Feline Calicivirus and Mycoplasma felis Infection in an Abyssinian Kitten and the Use of Chlorine Dioxide (MMS) Therapy

Abstract

We report the clinical course and management of a blue-ticked Abyssinian kitten (“Pearl”) presenting with chronic gastrointestinal, respiratory, and ocular disease following adoption from a multiple-cat household and a recent vaccination for FVRCP. Diagnostic workup established infections with Feline Calicivirus and Mycoplasma felis. Despite conventional therapy, the patient’s symptoms persisted. Off-label use of chlorine dioxide (MMS) was initiated by the parents, along with nutritional support and adjunct therapies. Clinical improvements were observed, including resolution of gastrointestinal symptoms and improved activity. This case highlights the complex interplay between vaccination, pathogen persistence, and adjunctive therapies in feline medicine.

Clinical Outcome

Within days of initiating MMS and supportive nutrition, Pearl demonstrated improved appetite, resolution of diarrhea and vomiting, normalization of respiratory effort, and increased mobility. No serious adverse effects were reported, save for transient gastrointestinal upset during dosage increases (interpreted as Herxheimer reaction by parents). By ten months, Pearl exhibited vigorous activity and weight gain (7 lbs 10 oz), with persistent ocular sight impairment but otherwise full return to health. Co-housed littermate Clio remained asymptomatic following similar supportive care. (See link for full article)

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**COMMENT**

Cats AND humans can be infected with Mycoplasma felis, the smallest known organism capable of free existence that does not possess a cell wall.  It can cause pneumonia, UT issues, conjunctivitis, bite wound abscesses, and other diseases. Mycoplasma can cause mycoplasmosis, which can lead to inflammation of several joints, such as knees, ankles, hips, or shoulders. Other symptoms include long-term lethargy, difficulty moving, fever, discomfort, and respiratory issues like sneezing and coughing.

Antibiotic treatment for mycoplasma infections can last a long time, so it is important to remain patient and follow the veterinarian’s or doctor’s instructions. Source

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Category:

Activism, Mycoplasma, research, Transmission, Treatment

Highlights: How Fenben and Meben Disrupt Cancer Pathways

https://drturner.substack.com/p/cancer-fighters-fenbendazole-and?

Cancer Fighters: Fenbendazole and Mebendazole

Repurposing Parasite Drugs to Target Tumors and Disrupt Cancer Pathways

 
Michael Turner M.D.
Aug 26, 2025
Article Excerpts:

Mebendazole, Fenbendazole and Albendazole all belong to a class of medications called “benzimidazoles” which were originally developed as treatment for parasites. Turns out they also do nasty things to cancer cells. (What follows is not an exhaustive list, but rather highlights some of the most prominent mechanisms.)

1. Induce Apoptosis (Programmed Cell Death)

Did you know that all cells have the ability to eliminate themselves by entering a process called “apoptosis”? Apoptosis is a form of guided, programmed cell death that allows the body to remove damaged, unnecessary, or potentially harmful cells in a controlled and orderly way.

For example, during embryonic development, it helps sculpt structures like fingers and toes by removing unneeded cells. In the immune system, apoptosis eliminates infected or malfunctioning cells, such as those with DNA damage, preventing them from becoming cancerous.

Turns out that these medications trigger apoptosis in cancer cells by activating pathways such as p53 and BAX.

Apoptosis gets rid of cells by breaking them into bits your body recycles or destroys

2. Prevent cancer cells from dividing.

A cell is a 3-dimensional object that has an internal skeleton (think of it as a “scaffold”) around which the parts of the cell are attached. In order for a cell to divide, the genetic material replicates, a scaffolding system forms on the opposite end of the cell, and the the scaffold from each side reaches out and pulls the genetic material apart equally.

These pieces of scaffolding are called “microtubules” and if they can’t replicate and move properly, the cell is frozen — unable to divide.

Guess what?

These medications disrupt cancer cell microtubule function!

What about normal cells?

Cancer cells divide rapidly, depending heavily on microtubules for mitosis. Interruption of microtubule function thus has a disproportionately higher impact on these cells.

3. Kill cancer stem cells.

Cancer stem cells are the “seeds” that spawn further cancer growth — but they are not typically targeted by traditional approaches including radiation, surgery or chemotherapy; hence their failure rates.

Watch the video in the link above for more.

What this means, practically, is that measures should be taken to destroy cancer stem cells wherever they may be hiding.

Turns out these medications directly kill cancer stem cells.

(Extra credit: so do Ivermectin, doxycycline, metformin, atorvastatin, green tea extract (EGCG), melatonin, vitamin D3, curcumin, berberine, omega-3 fatty acid, resveratrol, aspirin, diclofenac, and phosphodiesterase 5-inhibitors)

For more reading: https://imahealth.org/cancer-stem-cells/

4. Interfere with the tumor’s ability to form new blood vessels.

In order for a tumor to grow, two essentials are needed: blood supply and energy supply (typically as glucose or glutamine; more on that next..)

So the tumor can only grow to the extent to which it can surround itself with new blood vessels. This process of recruiting blood vessels is called “angiogenesis”.

Guess what?

These medications inhibit angiogenesis (particularly by inhibiting a molecule called vascular endothelial growth factor (VEGF). So by cutting off the nutrient supply, these medications induce tumor starvation and shrinkage.

5. Inhibit Glucose Metabolism (Warburg Effect).

Normal cells have a choice of three main sources of biochemical energy: glucose, fatty acids and ketones. Cancer cells predominately rely on glucose (aka blood sugar) and glutamine—which is a vulnerability that can be exploited.

What if we interfered with the cancer cell’s ability to absorb and utilize glucose as a fuel?

Exactly.

These medications interfere with glucose uptake and utilization by “downregulating”glucose transporters such as GLUT1 and possibly hexokinase; the result is the cell is starved of energy.

Normal cells, which are less dependent on glucose as a fuel source, are less affected by these metabolic disruptions.

Here is Dr. Seyfried talking about the importance of glucose and glutamine for cancer growth:

And finally…

6. Disrupt cancer signaling pathways.

Imagine your body as a city with a traffic light system. Imagine that traffic light system operates during rush hour in a newly developing city (youth). Once the city is built (adulthood), the system mostly shuts down. But in certain rogue neighborhoods (tumors), this traffic controller comes back online and starts creating bypasses, shortcuts, and green lights for dangerous drivers (cancer cells), allowing them to speed through red zones, avoid checkpoints, and grow unchecked.

One of these traffic control systems is called the “Hedgehog (Hh) Pathway”.

Now you already know what I’m going to tell you next, right? 🙂

These medications decrease the activity of the Hedgehog pathway. (Extra credit: so does Ivermectin, Doxycycline, Vitamin D, Curcumin, and Sulforaphane.)

Treatment:

  • These medications should only be obtained from a licensed pharmacy (quality assurance) and taken under the supervision of a healthcare professional, as they are very bioactive, the dosing for cancer is different than for parasites, they interact with other medications and supplements, and require lab monitoring.

  • Mebendazole is the human form (pricey and difficult to find), while Fenbendazole is the veterinary form (a bit harder on the liver but much more affordable and accessible).  (See link for article and references)

Further Reading:

IMA cancer protocol, page 94

http://

The Metabolic Theory of Cancer: Glucose, Glutamine & Anti-Parasitics, Fenbendazole — Thomas Seyfried

The Moss Report
Aug. 30, 2025
 
In this groundbreaking conversation, Professor Thomas N. Seyfried, PhD (Boston College), author of Cancer as a Metabolic Disease, joins Ralph W. Moss, PhD and Ben Moss to discuss the Mitochondrial and Metabolic Theory of Cancer — and why it challenges decades of conventional thinking. Dr. Seyfried explains how cancer cells depend on glucose and glutamine as their dual fuel supply — and how cutting off both pathways may be the key to shutting cancer down.
 
He details the role of the Glucose Ketone Index (GKI), nutritional ketosis, and new interest in anti-parasitic drugs like fenbendazole and mebendazole, which appear to target cancer’s energy metabolism.
 
The full article with links, resources and full transcript can be found here: https://themossreport.com/s5-e13-prof…
 
This episode explores: 
  • Why the somatic mutation theory no longer explains cancer’s true origin
  • How mitochondrial dysfunction drives tumor growth
  • The critical role of glucose and glutamine fermentation in sustaining cancer cells • Why targeting both fuels together is essential for effective therapy
  • Emerging research on fenbendazole, mebendazole, and DON as tools in metabolic therapy
  • The potential of a paradigm shift in oncology — from genes to metabolism
 

For more:

 

Category:

Cancer, research, Treatment

When a Tick Changes the Game: Jared Allen’s Battle with Alpha-Gal Syndrome

https://www.si.com/everyday-athlete/nfl-legend-jared-allen-s-tick-bite-diagnosis-every-athlete-needs-to-know-about

NFL Legend Jared Allen’s Tick Bite Diagnosis Every Athlete Needs to Know About

John Welbourn

When a Tick Changes the Game: Jared Allen’s Battle with Alpha-Gal Syndrome

Most athletes know the importance of diet when it comes to peak performance; what you eat fuels your training, recovery, and overall health. But what happens when something as small as a tick forces you to rethink how you fuel your body completely? That’s precisely what happened to former NFL legend Jared Allen, who recently opened up about his battle with alpha-gal syndrome, a tick-borne food allergy that has reshaped his lifestyle—and his plate.

What is Alpha-Gal Syndrome?

Alpha-gal syndrome (AGS) is an allergy caused by the bite of the Lone Star tick, commonly found in the southeastern and midwestern United States. Unlike typical food allergies that react to things like peanuts or shellfish, AGS is unique: it causes the body to have a delayed allergic reaction to red meat and other mammal-based products. That means beef, pork, lamb, venison, and even hidden mammal-derived ingredients in foods or supplements can trigger severe symptoms.

The reaction doesn’t always happen immediately after eating, which makes it tricky to diagnose. Symptoms can range from stomach pain and hives to life-threatening anaphylaxis hours after a meal.

Jared Allen’s Diagnosis

For Jared Allen—known for his grit and strength on the football field—the diagnosis meant he had to completely cut mammal meat out of his diet and switch to what he calls a “fins and feathers” lifestyle, sticking to poultry and fish. Imagine going from fueling your body with steak or burgers after grueling workouts to suddenly being told those foods could send you to the ER. That’s a massive change for anyone, let alone a professional athlete used to finely tuned nutrition. (See link for article)

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**Comment**

My son was recently bitten by a Lone star tick.  Well, I should say he was nibbled on by a LS tick, leaving a minuscule red pin prick.  The tick was not remotely engorged.  I received the frightening text from him but admitted I needed to brush up on all of this as so far Wisconsin patients are still mostly dealing with black legged ticks and Lyme/MSIDS even though Lone Star ticks have been found here.

But, the nibble was enough to cause profound illness in 2 weeks time.  (Yes, I’m kicking myself for not demanding prophylactic treatment, but we all grow slack at some point and need a wake-up call.  This was it!) 

His symptoms sounded exactly like Lyme but he was worried he had also developed Alpha Gal as he would get diarrhea within a few hours of eating red meat.  Thankfully this dreaded symptom quickly went away.

All I initially remembered was that LS ticks transmit not only Alpha Gal Syndrome (AGS) the meat allergy the NFL star got, but also STARI, which looks, smells, and acts just like Lyme disease, despite the fact at least 9 transmission experiments involving B. burgdorferi in Lone Star ticks have failed to demonstrate vector competency.  The offending agent of STARI is B. lonestari not B. burgdorferi, but the illness looks the same.  Go here for the nuts and bolts.

BTW: STARI is also called Masters’ disease, named after famed rebel Dr. Ed Masters who took the CDC on single-handedly and outwitted them.  All of Masters’ patients improved dramatically with extended antibiotic treatment despite the CDC’s belief that antibiotics should be used sparingly, if at all.

So, what to do?

Well, I figured if this looked and felt exactly like Lyme, it would respond to Lyme treatment.  My son went on the following (reminder: I’m not a doctor and I don’t diagnose or treat anyone):

  • 100mg minocycline, twice daily for two weeks; however when discontinued his symptoms returned, signaling that a layered approach was needed.  This is common.
  • he then pulsed 500mg tinidazole once a day for two successive days weekly
  • he then layered in 12mg ivermectin every other day
  • he did daily red light and sauna therapy
  • he did two rounds of EBOO (extracorporeal blood oxygenation and ozonation) 3 weeks apart.  He said the EBOO completely knocked him on his butt and he had to take a day off work to sleep, but that shortly he felt the best he had felt since starting treatment.
It took every bit of that treatment for three months to finally knock it.
 I’m happy to report he has remained symptom free.

On a side note, ivermectin and/or fenbendazole has:

This was not a fun experiment but I know how important it is to share our experiences, as that is often all we patients truly have – each other.

Category:

Alpha Gal Meat Allergy, Alzheimer's, Autism, Lyme, Parasites, research, Testing, Ticks, Transmission, Treatment