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Updated: 1/18/23

Marty Ross MD Discusses Rifamycins, Azoles and Methylene Blue for Bartonella Brain Fog

Bartonella Treatment in Lyme Disease—A Lot Has Changed

The latest laboratory experiments suggest effective Bartonella treatments must do more than kill growing germ forms—they should also kill hibernating persister forms of Bartonella and include agents to remove biofilms and fibrin nests.

Persisters and Biofilms

Research published in 2019 and early 2020 is changing the approach I take to treat Bartonella. Previously, research showed Bartonella has rapidly growing germ forms; therefore, the antibiotics I recommended in the past treated growing forms only. New research shows that Bartonella also has non-growing forms called persisters. Think of a persister as a hibernating form of the germ that ignores most antibiotics and immune system attacks. This new research also shows that Bartonella forms protective sugar-slime coverings called biofilms. These biofilms can block the immune system and antibiotics from reaching Bartonella. This new research is based on petri-dish laboratory experiments.

Bartonella-Fibrin Nests

In addition to these key findings, some with chronic Bartonella may require enzymes to break up nests of Bartonella and the blood clotting protein called fibrin that can form in narrow and small blood vessels. These Bartonella nests limit blood flow to tissues and may block antimicrobials and the immune system from killing these germs. Lumbrokinase, a group of enzymes that come from earthworms, is very effective at breaking up fibrin. Other options include nattokinase or serapeptase, but these enzymes are much weaker than lumbrokinase.  (See link for article)


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