healthcare-08-00157-v2 Full study Here
Presentation of Acrodermatitis Chronica Atrophicans Rashes on Lyme Disease Patients in Canada
John D. Scott
International Lyme and Associated Diseases Society, 2 Wisconsin Circle, Suite 700, Chevy Chase, MD 20815-7007, USA; jkscott@bserv.com; Tel.: +1-519-843-3646 Received: 13 May 2020; Accepted: 29 May 2020; Published: 4 June 2020
Abstract:
Lyme disease (Lyme borreliosis) is a complex multisystem illness with varying clinical manifestations. This tick-borne zoonosis is caused by the spirochetal bacterium, Borrelia burgdorferi sensu lato (Bbsl) and, worldwide, presents with at least 20 different types of rashes. Certain cutaneous rashes are inherently interconnected to various stages of Lyme disease. In this study, five Canadian Lyme disease patients from a multi-age range presented various phases of the acrodermatitis chronica atrophicans (ACA) rash. In each case of ACA, the underlying etiological pathogen was the Lyme disease spirochete. Although ACA rashes are normally found on the lower extremities, this study illustrates that ACA rashes are not directly correlated with a tick bite, geographic area, age, Bbsl genospecies, exercise, or any given surface area of the body.
- Case 4 provides confirmation for an ACA rash and gestational Lyme disease (club feet at birth). Both parents tested positive for Bbsl.
- One patient (Case 5) puts forth a Bbsl and Bartonella sp. co-infection with a complex ACA rash.
This study documents ACA rashes on Lyme disease patients for the first time in Canada.
**Comment**
The study states that about 63% of patients infected with Lyme develop chronic Lyme disease. Authorities keep telling us it’s only 10-20%. It also states Lyme colonizes in many immune-privileged sides including bone, brain, eye, ligaments & tendons, heart, kidney, bladder, liver, muscle, synovial cells, central nervous system, claim and neuronal cells, and fibroblasts/scar tissue. It states Lyme can be an insidious neurologic pathogenesis with demyelination, and even fatal.
The study points out that Bb is pleomorphic with diverse forms (spirochetes, round bodies, blebs, granules, and biofilms). The study also touches upon the hopelessness many patients can experience which can lead to despair and suicide. Lyme may be potentially transmitted via intimate relations as well as gestationally.
For more:
- https://madisonarealymesupportgroup.com/2018/06/19/33-years-of-documentation-of-maternal-child-transmission-of-lyme-disease-and-congenital-lyme-borreliosis-a-review/
- https://madisonarealymesupportgroup.com/2018/11/11/gestational-lyme-other-tick-borne-diseases-dr-jones/ A retrospective study showed 480 children with gestational Lyme/MSIDS. Diagnosis was based on clinical physical and history. Two cases of in vitro fertilization caused embryonic infection. Gustafason & Burgess demonstrated gestational Bb infection in dogs. Of the inoculated bitches, 80% became infected who then birthed mostly infected pups.
- https://madisonarealymesupportgroup.com/2020/02/19/how-can-mother-to-fetus-transmission-of-lyme-disease-be-rare-when-no-one-is-counting/
- https://madisonarealymesupportgroup.com/2019/02/22/why-mainstream-lyme-msids-research-remains-in-the-dark-ages/
- Scott uses sources that had an even lower percentage of patients with the EM rash. His study states only 9-39% have a rash. Authorities often state 80% have it. This is important because overemphasizing the rash makes doctors think it’s required to be infected when in my experience far fewer have a rash than is commonly thought.
- The ACA rash is noticeably different than the EM rash and is commonly caused by the B. afzelii strain (cultivated from the lesion), is most commonly found on the leg and can lead to atrophy, sclerosis, and ulceration. The majority of patients with ACA experience peripheral neuropathy while others may have severe localized pain. The study says while the EM rash can last 2 years, ACA rash can last longer than 10 years.
Various studies showing how many get the EM rash.
