Archive for the ‘Psychological Aspects’ Category

Neuropsych Disorders in Kids: An Interview with Co-Founder of The Stanford PANS Clinic – Dr. Kiki Chang

https://www.neuroimmune.org/dr-kiki-chang.html

Neuropsychiatric Disorders In Children: An Interview with Co-Founder of The Stanford PANS Clinic, Dr. Kiki Chang

10/22/2018

Please go to link for an informative interview with Dr. Kiki Chang, a child psychiatrist with over 22 years of experience whose specialty is working with youth and young adults at risk for serious mood disorders such as depression, bipolar, and PANS/PANDAS and related neuropsychiatric disorders.


 

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https://madisonarealymesupportgroup.com/2015/10/06/november-dr-brown-on-msids-pandas-pans/  According to Dr. Brown, 80% of his PANS patients have Lyme/MSIDS.

 

 

 

 

Autism Spectrum Disorder (ASD) The Question Every Parent Should Ask….Why is This Not Medical?

https://www.linkedin.com/pulse/autism-spectrum-disorder-asd-question-every-parent-ask-goldberg-md/

Autism Spectrum Disorder (ASD) The Question Every Parent should ask.… Why is This Not Medical?

Michael Goldberg, MD

There are different kinds of childhood disorders, yet none scientifically or medically can remotely affect the CDC reported 1:36 children and be thought of as genetic or developmental in origin, unless, medical science is ignored and that child is NOW labeled, ASD Autistic! However, given the horrific and increasing numbers of ASD affected children, there must be an underlying unidentified medical disease presenting with autistic like symptoms and behaviors i.e. an ASD “PHENOTYPE”!  

It is scientifically impossible to have an epidemic without a disease origin. Mistakenly labeling and then treating children as “psychiatric Autistic” is the failure of our medical system to recognize these children are really part of an enlarging, unrecognized medical pandemic affecting children and young adults. ASD (1:36) today is worse than the Polio epidemic (1:1500 – 1:2000) of the 1950’s.

As early as the 1960s, certainly in the 1970s, pediatricians were being taught at UCLA and other excellent medical schools that Psychiatric, DSM autism affected 1 – 2 in 10,000 children. Further, those 1-2 children, in order to be labeled Autistic must never be affectionate/ never normal! To have that number mysteriously begin to increase in the 1990s to the astounding level of 1 in 36 otherwise affectionate children begs the questions; “Is this epidemic? And if so “Why is this condition not considered and treated as medical?”  

Instead of being excited about the arrival of new baby, watching a child grow and prosper, sadly many mother’s today (expressed constantly when meeting new parents) live with perhaps worse fears than parents and families of the 40s and 50s. At that time the polio epidemic was a real threat, affecting 1:1500 – 1:2000 children, and the world mobilized to find a cure. Today ASD affects 1:36 and climbing! Where is the medical community? You cannot have an epidemic of psychiatric or developmental conditions!

I am proposing an answer to the question, nobody in public health, academic research or the pediatric medical community dare to acknowledge or ask: Is ASD in children and young adults a medical condition? Are they medically ill and thus treatable? 

 My answer: YES and the medically treatable symptoms and manifestations are directly related to Herpes viruses (potentially others) and the immune system! 

Medical School of the 1970s was eventful.  Professors opined we were entering a “golden age” of medicine, because “common” pathogens were being identified and eradicated. As new physicians we recognized we live in a sea of viruses. We were protected by our immune system and its adaption over thousands of years.

Nobel level professors taught there were differences between “normal” viral titers (markers) and “elevated” viral titers, indicating the presence of an active virus. Then in the early – mid 1980s very powerful medical leaders (CDC, NIH) unexplainably decided elevated Herpes viral titers in children and adults were meaningless! Lab evaluations of “normal” vs. elevated, are still carried over today, but “ignored” when elevated. As a practicing pediatrician, to suddenly be required to ignore the role of the Herpes virus was and still is beyond comprehension.  

The Medical Literature still support the significance of a fourfold change in viral titers. However, if a pediatrician does not consider or is precluded from testing for viral titers, how can the physician begin to evaluate or “rule out” if there is treatable viral activity? She/he cannot! How does that benefit the child or the family? It does not! For a physician to leave a child with overwhelming sensory issues, without a complete medical work up is unconscionable. In my opinion, to discount and ignore elevated viral titers in children and young adults remains one of the biggest travesties being perpetrated upon our children by the current medical system.

All of the ASD labeled children I am working up are affectionate. Many present with this ASD “phenotype”. Typically, their blood tests show elevated viral titers for the HHV6 herpes virus, Epstein Barr and/or CMV virus. In addition, many of these children also present with outright early developmental delays and motor issues. For these issues, I was taught by excellent professors to think of viruses, “rule-out viruses.”  

Most children having issues today become labeled as “on the ASD spectrum” without a proper medical workup and investigation for illness, chronic viral activation issues, etc. By artificially removing the medical criteria developed over decades past, the current “system” too quickly, attaches the label of ASD. The pediatrician unwittingly abandons the children and parents to the psychiatric community for behavior training and a life of isolation and despair. Parents are told to cope and forego their focus or desire to pursue real medical answers and real potential help for their children. How much worse can this get before parents and others step up and declare “enough is enough”!

In my professional opinion based upon over 40+ years of clinical experience, “Autism spectrum disorder” (not meeting strict Kanner criteria) with accompanying language impairment is in reality a medical disease (complex immune – complex viral) presenting as an encephalopathy (often viral) with “autistic” symptoms.

The language impairment in these ASD labeled children is part of the disease, not secondary to Autism Spectrum Disorder. Many of my patients (approx. 75+%) respond favorably to a medical protocol of anti-viral medications and diet modifications, eliminating known allergenic foods. This anti-viral component mitigates the effects of the viruses to the brain, while the diet changes reduce stress on the body, the brain, and the immune system. Additional improvement is achieved with the use of an SSRI (Selective Serotonin Reuptake Inhibitor). The SSRI is introduced not for “depression,” but as a pharmaceutical/medical way to treat temporal lobe hypo perfusion; a real, medically definable, physiologic CNS dysfunction evidenced on a NeuroSPECT scan

This medical protocol results in the elimination or severe abatement of the “autistic” like symptoms and behaviors and allows an increase in, a return of, higher cognitive function. The most common phrase I hear from parents of improving children is,

“It is as if a fog has lifted.”

and from speech and other therapists,

“This is not the same child I have been working with.”

These otherwise affectionate “typical/normal” (now much brighter cognitively) children are now placed in a position to be taught (not trained), allowed to “catch up” and progress with their peers. For whatever reasons, those in positions of authority refuse to acknowledge or investigate this treatable complex immune, complex viral medical problem. Instead, current focus and research is on “causation” with activated “gene expressions” or “complex genetic” ideas being proffered as the origin. The reality is a treatable underlying viral/immune process is being ignored! The “system” has become so biased, against the obvious, that good professors wishing to pursue research into a readily treatable complex immune, complex viral causation, have not only been refused funding, but fear losing their positions. These short-sighted money decisions at the clinical level mean the loss of the near immediate relief from ASD behaviors and mannerisms, improved, often excellent cognitive abilities, and improvement in the future quality of life for the child and family. Why?

Recently I met with a group of educators discussing the “differences” working with a medically treated ASD child (stressing an ASD “phenotype,” not developmental “autism”). They were aware the medically treated child was able to understand and be taught! These educators realized this was an emerging potentially regular child, not a child mysteriously born “miss-wired.” Thankfully, there are excellent academic professors who also know something is seriously wrong, literally acknowledging we are in a missed medical “pandemic”.

An appropriately focused and engaged medical community together with key medical and academic researchers, could create a pathway for a healthier future for the children and reduce the financial costs to all affected, including our social and educational systems.

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**Comment**

https://madisonarealymesupportgroup.com/2017/10/26/clinical-trial-shows-most-kids-with-autism-are-not-born-with-it/

The Duke study is remarkable in that 60-70% or more of children with Autism have de novo gene mutations (not found in either parent) that must have occurred after birth according to the results, not in the egg, sperm or early utero development as previously, and erroneously assumed.  This new finding reveals research should now be geared to finding out what environmental damage after birth leads to these mutations and/or what pathogens are acting as triggers.  Autism like Lyme/MSIDS is a pandemic and according to one Wisconsin LLMD, 80% of his Autistic patients are also infected with Lyme/MSIDS.

More and more is coming out about the pathogenic aspect of disease (even mental illness).  This is certainly true for Alzheimer’s & Dementia, as well as other autoimmune issues such as in this story:  https://madisonarealymesupportgroup.com/2017/10/01/panspandas-steroids-autoimmune-disease-lymemsids-the-need-for-medical-collaboration/  While Susannah Cahalan’s issue was truly autoimmune, we learn of Patrik, who had Lyme:

Boy’s Lyme Disease Morphs into Autoimmune encephalopathy. It took 10 years and 20 doctors to find out 12-year-old Patrik had Lyme disease. Just 4 months later the doctors discovered he also has a condition where his immune system attacks his brain. Dr. Souhel Najjar, Cahalan’s doctor, heroically saves the day again.

This is another great read regarding the pathogen element within Alzheimer’s:  https://madisonarealymesupportgroup.com/2018/09/11/its-time-to-find-the-alzheimers-germ/

Then there’s this gem:  https://madisonarealymesupportgroup.com/2016/06/03/borrelia-hiding-in-worms-causing-chronic-brain-diseases/  (Excerpt below)

MacDonald states that both worms and borrelia can cause devastating brain damage and that,

 

“while patients are wrongly declared free of Lyme and other tick-borne infections, in reality, too often they contract serious neurodegenerative diseases which can kill them.”

MacDonald made his discovery from 10 specimens from the Rocky Mountain Multiple Sclerosis Center Tissue Bank. All 10 showed evidence of borrelia infected nematodes. Five patients who died of Glioblastoma multiforme, a malignant brain tumor, and four patients who died of Lewy Body dementia also showed infected nematodes.  MacDonald used FISH, Fluorescent In Situ Hybridization, which uses molecular beacon DNA probes to identify pieces of borrelia’s genetic material which fluoresce under the microscope with a 100% DNA match.

In other words, this is no mistake.

 

 

 

 

School Related Difficulties With Lyme Disease

https://globallymealliance.org/school-related-difficulties-lyme-disease/

school lyme
October 2, 2018

School-Related Difficulties With Lyme Disease

by Jennifer Crystal

Can you imagine missing all of the second grade? Or having to leave college? Lyme disease can have a devastating impact on kids in school.

When I was a junior in college, I started getting headaches while doing my homework. Textbook print, which had always been easy for me to read, suddenly seemed small and difficult to focus on. I found myself straining my eyes and struggling to concentrate, often losing my place and having to re-read entire paragraphs a few times. Sometimes I would feel dizzy or nauseous after studying too long, like I was experiencing vertigo.

Thinking my eyesight had worsened, I went to see my ophthalmologist. He suggested magnifying glasses. The answer, I regret to report, was not that simple.

I was suffering from three undiagnosed tick-borne illnesses. Lyme disease, but also two tick-borne co-infections—babesia, and ehrlichia—which had crossed into my central nervous system. I’d been bitten by a tick two years earlier, but I was not subsequently tested for tick-borne infections.

My rash had been brushed off as nothing, and the on-and-off flu symptoms I had in college were also written off as me “just being run-down.”

For the rest of my college career, I wore magnifiers to read, which helped to a degree, but I also developed other, seemingly idiopathic neurological symptoms, such as hand tremors. It would be another six years before I was accurately diagnosed. And I was one of the lucky ones.

I still finished college on time, studied abroad, double-majored, and graduated with high honors. Later, when I was in the midst of treatment, bedridden and suffering from Herxheimer reactions, I couldn’t read at all. Sentences in a simple celebrity magazine swam before my eyes. I couldn’t type because my fingers and forearms ached too much. Writing more than a paragraph by hand exhausted me. Spending more than a few minutes in front of a computer or other screen would leave me with a screaming migraine.

Had I been in school then, I probably would have had to drop out. I might have never earned my degree.

This sadly is the case for many Lyme patients, whether they are diagnosed and under treatment, or suffering from debilitating symptoms which are not yet diagnosed. I know students who had to take a semester or a year off; college students whose illness(es) allowed them only to study part-time, and who still suffered for doing so; even high school students who were home-schooled, hoping to finish their studies in six years instead of four; and elementary school students who had missed two, three, four years of class.

Can you imagine missing all of the second grade? Not being able to socialize with your classmates?  Not having the chance to sit at your desk with fresh school supplies, learn math with your peers, and run around at recess? Missing lunch in the cafeteria, after-school sports tournaments, and evening dances? Aspects of school that I took for granted—and even complained about—but which feel like fantastic unattainable dreams to young patients with a tick-borne illness.

And those are the ones who know that they’re dealing with a tick-borne illness, and are under the care of a Lyme Literate Medical Doctor (LLMD), the ones who hope to regain at least some of their physical and cognitive capabilities one day. For college students like me, who don’t yet know they have tick-borne illnesses, all they know is that suddenly math equations that were easy last year are impossible this year. They are suddenly mixing up their letters, forgetting assignments. They can’t concentrate in class, and often have their heads on their desks because they’re so tired. They’d rather nap than run around at recess. They might have headaches, inappropriate outbursts, frustration and rage, or malaise.

From an outsider’s perspective, these symptoms might seem psychological or behavioral. And sometimes they, in fact, are. That’s why it’s important that children be clinically evaluated for all possible root causes of their struggles. But if a child seems to be suffering from common Lyme symptoms, or spends time playing outdoors, or who suddenly finds a tick bite or rash, they should also be evaluated for the possibility of tick-borne diseases as soon as possible.

Sadly, children ages 3-14 are at the highest risk for these diseases because they spend so much time outside (and are lower to the ground than adults, often rolling around in the grasses or running through the woods).

Early detection is key. Not only doctors but school nurses, teachers, and parents should know signs and symptoms to look out for, and should be aware of the many preventative measures such as doing daily tick checks, drying clothes at high heat, wearing light-colored clothing, and wearing long pants and high socks. Lyme disease that is caught right away can often be cleared up with three weeks of antibiotics. If you miss this early window for treatment your risk for chronic disease grows exponentially. Why, even of those treated in a timely fashion some 20% still advance to the chronic forms of the disease.

Had my tick-borne illnesses been diagnosed immediately, in all likelihood I would have had an easier time studying, and could have kept up my high-achieving momentum for more than just a couple years after college. Instead, I ended up losing the second half of my twenties. I don’t want that to happen to you or your child.


Additional Resources

jennifer crystalJennifer Crystal is a writer and educator in Boston. She is working on a memoir about her journey with chronic tick-borne illness. Contact her at jennifercrystalwriter@gmail.com

 

 

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For more:

https://madisonarealymesupportgroup.com/2018/09/26/more-awareness-needed-for-childrens-neurological-conditions/

https://madisonarealymesupportgroup.com/2018/08/23/caring-for-a-child-with-lyme-podcast/

https://madisonarealymesupportgroup.com/2018/07/24/diagnosed-with-cellulitis-child-had-lyme-docs-said-it-was-a-non-relevant-tick-bite/

https://madisonarealymesupportgroup.com/2017/08/12/lyme-disease-case-started-with-headaches/

https://madisonarealymesupportgroup.com/2017/06/30/child-with-lymemsidspans-told-by-doctors-she-made-it-all-up/

https://madisonarealymesupportgroup.com/2018/06/04/ld-diagnosis-took-forever-because-of-mental-health-stigma/

https://madisonarealymesupportgroup.com/2017/10/08/misdiagnosed-how-children-with-treatable-medical-issues-are-mistakenly-labeled-as-mentally-ill/

Dementia Misdiagnosed for PTLDS or Vice Versa? A Case Report

https://www.ncbi.nlm.nih.gov/m/pubmed/30282363/

Frontotemporal Dementia Misdiagnosed for Post-Treatment Lyme Disease Syndrome or vice versa? A Treviso Dementia (TREDEM) Registry Case Report.

Di Battista ME, et al. J Alzheimers Dis. 2018.

Abstract

We describe the case of a 61-year-old woman diagnosed with Borreliosis at the age of 57. Subsequently, the patient developed depression, anxiety, and behavioral disturbances. A lumbar puncture excluded the condition of Neuroborreliosis. The diagnostic workup included: an MRI scan, a 18F-FDG PET, a 123I-ioflupane-SPECT, an amyloid-β PET, a specific genetic analysis, and a neuropsychological evaluation.

Based on our investigation, the patient was diagnosed with probable behavioral-frontotemporal dementia (bvFTD), whereas in the previous years, the patient had been considered firstly as a case of Post-Treatment-Lyme Disease and, secondly, a psychiatric patient.

We believe that, in the present case, such initial symptoms of Borrelia infection may have superimposed on those of bvFTD rather than playing as a contributory cause.

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**Comment**

Here we have a woman with an actual Lyme diagnosis who goes on to develop depression, anxiety, and behavioral disturbances.  (All common symptoms with neuro-Lyme:  https://madisonarealymesupportgroup.com/2015/10/18/psychiatric-lymemsids/https://madisonarealymesupportgroup.com/2018/08/25/neuropsychiatric-lyme-borreliosis-an-overview-with-a-focus-on-a-specialty-psychiatrists-clinical-practice/https://madisonarealymesupportgroup.com/2018/06/04/ld-diagnosis-took-forever-because-of-mental-health-stigma/)

Despite mainstream knowledge of absolute proof of abysmal testing, they state they ruled out infection despite a prior diagnosis based on a lumbar puncture.

In this informative read from Columbia University, we learn that there are specific steps to be followed for lumbar punctures regarding Lyme as well as the fact that patients may have neurologic Lyme Disease but still test negative on the Lyme index (an index used with cerebrospinal fluid in a lumbar puncture)  https://www.columbia-lyme.org/diagnosis.

So this woman was handed a label and told, “Go home and be well.”

This scenario has played out so many times it’s like a skip in a record.

How about a clinical trial of antimicrobials known to have action against borrelia and then retest her (called a provocation test)?  Clinicians in the field understand how elusive this organism is.  A full work-up needs to be done on symptomology as it could possibly be a different pathogen altogether known to be transmitted by ticks and other bugs.  How about also testing for other tick borne pathogens known to give behavioral symptoms like Bartonella?  https://madisonarealymesupportgroup.com/2017/07/01/one-tick-bite-could-put-you-at-risk-for-at-least-6-different-diseases/ (It could be one of 18 and counting pathogens spread by ticks)

You see, something is causing this “probable behavioral-frontotemporal dementia (bvFTD).”  

All they’ve done here is slap a name to it but they haven’t found the cause.  Without the cause they will not treat appropriately.  

Somebody get this woman to Columbia University!

How many more are going to slip through the cracks and loose their minds due to poor testing?

For more on the abysmal testing:  https://madisonarealymesupportgroup.com/2017/08/15/reliability-of-lyme-testing/

https://madisonarealymesupportgroup.com/2018/09/08/whats-the-best-test-for-lyme-dr-rawls/

https://madisonarealymesupportgroup.com/2018/01/16/2-tier-lyme-testing-missed-85-7-of-patients-milford-hospital/

More on the relationship between Alzheimer’s, Dementia, ALS and Lyme:  https://madisonarealymesupportgroup.com/2016/06/09/alzheimers-byproduct-of-infection/  Kris Kristofferson was wrongly diagnosed with Alzheimer’s but had Lyme Disease. For years doctors told Kristofferson it was either Alzheimer’s or dementia, and may have been the result of blows to his head from boxing, football and rugby. The medication he was given gave him bad side effects and didn’t help.  Since starting treatment for Lyme Kristofferson “has made remarkable strides.” His wife Lisa said,

“all of the sudden he was back.” Although he still has some bad days, there are other days when he is “perfectly normal,” she said.

https://madisonarealymesupportgroup.com/2017/06/10/the-coming-pandemic-of-lyme-dementia/  Bacteria are usually ignored despite its historical and current significance in dementia research.  Today, the main bacterial threat to acquiring dementia comes from Lyme disease—a bacterium borrelia burgdorferi.

https://madisonarealymesupportgroup.com/2016/06/03/borrelia-hiding-in-worms-causing-chronic-brain-diseases/

https://madisonarealymesupportgroup.com/2016/08/09/dr-paul-duray-research-fellowship-foundation-some-great-research-being-done-on-lyme-disease/

Antidepressants Cause Severe Withdrawal Symptoms Like “Hallucination,” “Mania,” & Anxiety,” New Study Reveals

http://www.greenmedinfo.com/blog/antidepressants-cause-severe-withdrawal-symptoms-hallucination-mania-anxiety-new-

Antidepressants Cause Severe Withdrawal Symptoms like “Hallucination,” “Mania,” & “Anxiety,” New Study Reveals

Wednesday, October 3rd 2018 at 6:15 pm

Written By:  Sayer Ji, Founder

© [10/3018] GreenMedInfo LLC. This work is reproduced and distributed with the permission of GreenMedInfo LLC. Want to learn more from GreenMedInfo? Sign up for the newsletter here http://www.greenmedinfo.com/greenmed/newsletter.”

New research reveals severe withdrawal symptoms in over half of those who discontinue antidepressant drugs, including lasting and even permanent damage.

A concerning new study published in the journal Addictive Behavior and titled, “A systematic review into the incidence, severity and duration of antidepressant withdrawal effects: Are guidelines evidence-based?,” reveals that antidepressants are far more addictive and harmful than previously assumed, and vindicates the long time activism on this issue spearheaded by American psychiatrists like Kelly Brogan, MD and Peter Breggin, MD.

Highlights from the paper are as follows:

  • More than half (56%) of people who attempt to come off antidepressants experience withdrawal effects.
  • Nearly half (46%) of people experiencing withdrawal effects describe them as severe.
  • It is not uncommon for the withdrawal effects to last for several weeks or months.
  • Current UK and USA Guidelines underestimate the severity and duration of antidepressant withdrawal, with significant clinical implications.

This study aimed to assess the veracity of the the U.K.’s current National Institute for Health and Care Excellence and the American Psychiatric Association’s depression guidelines which state that withdrawal reactions from antidepressants are ‘self-limiting’ (i.e. typically resolving between 1 and 2 weeks).

In order to accomplish this goal the systematic review used the following methods:

“A systematic literature review was undertaken to ascertain the incidence, severity and duration of antidepressant withdrawal reactions. We identified 23 relevant studies, with diverse methodologies and sample sizes.”

The results were reported as follows:

“Withdrawal incidence rates from 14 studies ranged from 27% to 86% with a weighted average of 56%. Four large studies of severity produced a weighted average of 46% of those experiencing antidepressant withdrawal effects endorsing the most extreme severity rating on offer. Seven of the ten very diverse studies providing data on duration contradict the UK and USA withdrawal Guidelines in that they found that a significant proportion of people who experience withdrawal do so for more than two weeks, and that it is not uncommon for people to experience withdrawal for several months.”

Side effects were wide-ranging, lasting several months or longer (including permanent dysfunction), such as: 

“Typical AD withdrawal reactions include increased anxiety, flu-like symptoms, insomnia, nausea, imbalance, sensory disturbances, and hyperarousal. Dizziness, electric shock-like sensations, brain zaps, diarrhoea, headaches, muscle spasms and tremors, agitation, hallucinations, confusion, malaise, sweating and irritability are also reported (Warner, Bobo, Warner, Reid, & Rachal, 2006, Healy, 2012). Although the aforementioned symptoms are the most common physical symptoms, there is also evidence that AD withdrawal can induce mania and hypomania, (Goldstein et al., 1999; Naryan & Haddad, 2011) emotional blunting and an inability to cry, (HolguinLew & Bell, 2013) long-term or even permanent sexual dysfunction (Csoka & Shipko, 2006).”

The study concluded:

“We recommend that U.K. and U.S.A. guidelines on antidepressant withdrawal be urgently updated as they are clearly at variance with the evidence on the incidence, severity and duration of antidepressant withdrawal, and are probably leading to the widespread misdiagnosing of withdrawal, the consequent lengthening of antidepressant use, much unnecessary antidepressant prescribing and higher rates of antidepressant prescriptions overall. We also recommend that prescribers fully inform patients about the possibility of withdrawal effects.”

The researchers also noted that the rising numbers of antidepressant prescriptions used throughout the world may be fueled by the antidepressant drug withdrawal side effects themselves:

“As the lengthening duration of AD use has fuelled rising AD prescriptions over the same time period, we must understand the drivers of such lengthening use. The evidence set out suggests that lengthening use may be partly rooted in the underestimation of the incidence, severity and duration of AD withdrawal reactions, leading to many withdrawal reactions being misdiagnosed, for example, as relapse (with drugs being reinstated as a consequence) or as failure to respond to treatment (with either new drugs being tried and/or dosages increased). This issue is pressing as long-term AD use is associated with increased severe side-effects, increased risk of weight gain, the impairment of patients’ autonomy and resilience (increasing their dependence on medical help), worsening outcomes for some patients, greater relapse rates, increased mortality and the development of neurodegenerative diseases, such as dementia.”

The concerning implications of this study to millions around the world who are on antidepressants were immediately recognized by the media, as evidenced by mainstream reporting on the topic with the following headlines:

Thanks to a small but courageous group of professionals who have been raising awareness of the profound, unintended adverse effects of psychiatric drugs and the abject absence of objective criteria for determining “mental disease,” not only are there already resources available to the public today to better understand the dangers of psychiatric drugs, but there are also programs and protocols in place to help those who are on them to come off of them safely and with the support of others who have done the same already. For instance, the program put together by Dr. Kelly Brogan — Vital Mind Reset — has produced powerful outcomes. Take a look at the testimony wall here to learn from the first hand experiences of those who underwent the program and came out drug-free, often with their psychiatric symptoms and comorbid conditions reduced or completely put into remission.

Related reading: 

Sayer Ji is founder of Greenmedinfo.com, a reviewer at the International Journal of Human Nutrition and Functional Medicine, Co-founder and CEO of Systome Biomed, Vice Chairman of the Board of the National Health Federation, Steering Committee Member of the Global Non-GMO Foundation.

Disclaimer: This article is not intended to provide medical advice, diagnosis or treatment. Views expressed here do not necessarily reflect those of GreenMedInfo or its staff.