Press Release
08.27.18
In Recent Years, Upstate New York Has Seen An Explosion Of Ticks, Lyme Disease, And Other Tick-Borne Illnesses, Making New York The #1 Target For Ticks In The Country
Schumer’s Recently-Passed Amendment To The Health And Human Services Appropriations Bill Will Increase By Over $1 Million CDC Funding To Thwart Tick-Borne Illnesses; Funding Boost Will Be Used To Study, Identify, And Prevent Exposure To Vector-Borne Pathogens That Cause Lyme Disease And Other Tick-Borne Illnesses
Schumer: Funding Increase For CDC To Fight Lyme Disease Will Help Protect Against Tick-Borne Illnesses
U.S. Senate Minority Leader Charles E. Schumer today announced that the U.S. Senate has passed his amendment to the upcoming Health and Human Services (HHS) appropriations bill to increase by 12.15 percent FY2019 funding for the Centers for Disease Control and Prevention (CDC) to combat Lyme disease and other tick-borne illnesses. Specifically, the bill increases Lyme disease funding from FY2018’s level of $10.7M to $12M for FY2019.
Schumer’s amendment secures the first increase in CDC Lyme disease funding in five years. Schumer said that this long-overdue increase in funding will be used to support the all-out state and local war against tick-borne diseases, as New York is the #1 target for tick-related disease in the United States. Schumer called the increase in funding a major victory for communities across Upstate New York and vowed to shepherd the funding through Congress and onto the president’s desk.
“Upstate New York has been feeling the brutal bite of Lyme disease and tick-borne illnesses for years now, and thankfully this long-overdue increase in CDC funding will give us the resources we need to strike back,” said Senator Schumer. “New Yorkers and their children shouldn’t have to worry that spending time outside in their backyards will leave them with a debilitating ailment like Lyme disease, and this funding will help to prevent that. I’m proud to secure this crucial funding to combat the spread of tick-borne illnesses, and will now fight tooth and nail to see that it passes Congress and is signed into law.”
Schumer said that the increase in funding from the CDC will specifically be used to target vector-borne pathogens which cause diseases in human beings. The funding increase will help understand when, where, and how people become exposed to vector-borne pathogens, as well as help to prevent exposure to vector-borne pathogens and mitigate potential consequences of infection. Additionally, the funding will be used to help implement vector-borne disease diagnostics, surveillance, control, and prevention programs.
Lyme disease is a bacterial infection carried by deer ticks, which can be transmitted by a bite to a human or animal host. If left untreated, the bacterium Borrelia burgdorferi travels through the bloodstream, manifests itself in body tissues, and causes mild or severe symptoms, depending on the case. Lyme disease begins as a rash at the location of the tick bite and then spreads to the nervous system and joints. Early diagnosis and antibiotic treatment are crucial to recovery. Appropriate antibiotic use in the early stages of Lyme disease typically results in a swift and total recovery. Untreated and undiagnosed Lyme disease – which regularly occurs – can lead to debilitating effects on a person’s health.
Senator Schumer has long fought for federal funding for the prevention, diagnosis, and treatment of tick-borne illnesses like Lyme disease, which have seen a major spike in cases across New York State. Earlier in 2018, during an in-person meeting with CDC Director Dr. Robert Redfield, Schumer received a firm commitment from the director that he would be working to address Lyme disease in New York State. Additionally, Schumer played an integral role in securing funding for the CDC in this year’s FY2018 omnibus spending bill — Schumer, one of 4 negotiators, secured $8.005 billion in the federal funding for the CDC, a $900 million increase from FY17. Schumer also helped secure a historic $3 billion increase in National Institutes of Health (NIH) funding, which can be used to expand and build upon existing NIH-funded Lyme disease research that is already occurring at New York institutions, such as Stony Brook, Columbia University, in the SUNY system and at Hunter College. Schumer has traveled throughout New York State advocating for increased funding to fight tick-borne illnesses, and will continue to advocate for these funds for New Yorkers.
https://www.idse.net/Policy–Public-Health/Article/10-19/Finding-Link-Between-Infection-and-Alzheimer-s-Disease-Could-Be-Worth-1-Million/56312 Full article here
OCTOBER 15, 2019
By Tom Rosenthal
WASHINGTON—If you can present persuasive evidence that a particular microbe causes Alzheimer’s disease, there’s a $1 million prize waiting for you.
“This is an award for achievement; it is not a grant,” said Leslie N. Norins, MD, PhD, FIDSA, the founder and CEO of Alzheimer’s Germ Quest, Inc. (ALZgerm.org). The nonprofit group is funding the award to encourage more intensive research on the possibility that microorganisms are the root cause of the incurable disease afflicting 47 million people worldwide, including 5.6 million Americans aged 65 years and older.
“I’ve closely reviewed the scientific literature and personally believe it’s clear that one germ, possibly not yet discovered, is the root cause of most Alzheimer’s disease,” Dr. Norins said. “But we’ll see what researchers find out.”
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**Comment**
I posted on this a year ago when Leslie Norins, Ph.D. wrote this fantastic paper: https://madisonarealymesupportgroup.com/2018/09/11/its-time-to-find-the-alzheimers-germ/
Alzheimer’s has had a Cabal similarly to Lyme in that research has been hijacked by a group of individuals with blinders on. According to Norins, the 2017 Alzheimer’s Association had a conference in London where researchers from 70 countries could share progress. A keyword index of the presentations showed the largest entries, 110, were for amyloid/APP.The next most common item was tau, the tangled protein, with 85 entries. Inflammation—the body’s reaction to something—had 45 mentions. Presentations of germ importance had only single digit presence: prion proteins (8 entries), infectious disease (4 entries), bacteria (1 entry). Virus was not even listed as a keyword.
Yet numerous researchers have doggedly fought against the myopic focus and have found spirochetes in the brains of Alzheimer’s patients: https://madisonarealymesupportgroup.com/2016/06/09/alzheimers-byproduct-of-infection/, and Kris Kristofferson was diagnosed with Alzheimer’s but actually had Lyme disease: https://madisonarealymesupportgroup.com/2017/01/04/aluminum-alzheimers-ld/
https://madisonarealymesupportgroup.com/2017/01/18/a-bug-for-alzheimers/
https://madisonarealymesupportgroup.com/2016/06/09/alzheimers-byproduct-of-infection/
https://madisonarealymesupportgroup.com/2016/11/17/antibiotics-and-alzheimers/
https://madisonarealymesupportgroup.com/2018/03/25/a-brief-history-of-neuroborreliosis-research-dementia-an-inside-look-at-two-researchers/ Fantastic read by microbiologist Tom Grier.
https://madisonarealymesupportgroup.com/2017/06/10/the-coming-pandemic-of-lyme-dementia/
https://madisonarealymesupportgroup.com/2016/11/17/alzheimers-lyme/
https://madisonarealymesupportgroup.com/2018/07/28/herpes-viruses-implicated-in-alzheimers-disease/
NASHVILLE – The Tennessee Department of Agriculture, United States Department of Agriculture – Animal and Plant Health Inspection Services, Tennessee Department of Health, and University of Tennessee Institute of Agriculture today announced the detection of the invasive Asian longhorned tick in an additional six Tennessee counties: Knox, Jefferson, Claiborne, Cocke, Putnam, and Sevier. The tick was detected in Union and Roane Counties in May.
The Centers for Disease Control and Prevention reports that there is no evidence to date that the tick has transmitted pathogens to humans in the U.S. There are concerns that the tick may transmit the agent of Theileriosis in cattle, and heavy infestations can cause blood loss and lead to death.
It is important to be aware of this tick, as cattle and canines are particularly susceptible to tick bites. Livestock producers are reminded to be vigilant when purchasing animals, apply a tick treatment to cattle prior to bringing them to your farm, and always use best practices for herd health. Dog owners should provide their animals with a tick preventative and check for ticks.
“Tennessee has numerous animal hosts and a suitable habitat for this tick species,” Dr. R.T. Trout Fryxell, Associate Professor of Medical and Veterinary Entomology for UTIA, said. “While it is always important to be diligent and keep an eye out for all ticks, the unique biology of the Asian longhorned tick helps this species to establish quickly and become a problem.”
Tips to prevent tick bites in animals and livestock include:
· Coordinate with your veterinarian to determine appropriate pest prevention for pets and livestock.
· Check pets and livestock for ticks frequently.
· Remove any ticks by pulling from the attachment site of the tick bite with tweezers.
· Monitor your pets and livestock for any changes in health
If your animals are bitten by a tick, Dr. Trout Fryxell suggests putting the tick in a ziplock bag, writing down the date and where the tick was most likely encountered, and storing it in a freezer.
For additional information about the longhorned tick in the United States, click here. To find more information on tick-borne diseases, click here.
________________
**Comment**
The Asian Tick has been found in the following 12 states: Arkansas, Delaware, Kentucky, New Jersey, New York, North Carolina, Virginia, West Virginia, Pennsylvania, Maryland, Connecticut and Tennessee.
https://madisonarealymesupportgroup.com/2019/10/26/researchers-conclude-asian-longhorned-tick-contributes-minimally-to-lyme-disease-in-the-u-s/ This article admits it can transmit Lyme. It’s also suspected of transmitting the following: Anaplasma, Ehrlichia, Rickettsia, and Borrelia species.
For a great read: https://madisonarealymesupportgroup.com/2018/09/12/three-surprising-things-i-learned-about-asian-longhorned-ticks-the-tick-guy-tom-mather/
By Allison Caruana
President, The Mayday Project
The organism that causes chronic Lyme disease and STARI/Master’s disease is physiologically similar, but until now has not been defined. Borrelia burgdorferi (Bb) is the carrier of but not the cause of the longstanding symptoms of what is known as chronic Lyme disease. Borrelia burgdorferi B31 is a rickettsial agent. Helical spiroplasma that carries the Pandora Virus (circular plasmid 26), which transfers mutant virophages to the host, is the causative agent of chronic (not early) Lyme disease, as well as STARI/Master’s disease.
In an interview with Gary Engelman, Dr. Willy Burgdorfer made two important claims. The first, borrelia was the agent of Relapsing Fever, but is not the agent of Lyme disease.13 Relapsing Fever is a different species of borrelia2, whereas Borrelia burgdorferi B31 is Spiroplasma apis.21 Dr. Willy Burgdorfer goes on to state that these are spirochete-like organisms and that up until his discovery of Borrelia burgdorferi, spirochetes had never been found to exist in deer ticks.13 The same is not true for spiroplasmas, which are common in arthropods.16 Spiroplasmas are known to cause symptoms identical to Lyme disease, i.e., arthritis, heart murmur, swollen knee, and more.14
The Borrelia associated with Lyme disease are unlike other borrelia, as these organisms are known for an amorphous slime layer. Historically, this feature has been associated with spiroplasmas, not spirochetes.17, 20, 29, 30 Spiroplasmas (family spiroplasmataceae, order mycoplasmatales) are comprised of a group of helical prokaryotes whose external morphology differs from the spirochete. The spirochete is defined by an internal skeletal framework rather than an external peptidoglycan-containing cell wall.15 The organism we call the Bb spirochete has blebs and granules,6 which are attributes of spiroplasma and mycoplasma that occurs as a morphological response to environmental stress- which produce uncultivable mycoplasmas.20, 23, 29, 31, 32
Borrelia burgdorferi B31 is believed to be a pleomorphic spiroplasma within the family of Spiroplasma apis. This would explain Dr. Willy Burgdorfer’s statement regarding the “spirochete-like organisms” that are found in ticks.13 Additionally, Borrelia burgdorferi contain 12 linear and 9 circular plasmids, some of which have been determined to harbor mutant “bacteriophages”.4, 11, 12, 18 In theory, the term bacteriophage could be exchanged for a more proper term: “virophage”. This suggestion comes on the basis that the term “plasmid” and “chromosome” can be interchanged for “Giant viruses”.19 The circular plasmid 26 (cp26) of Borrelia burgdorferi contains 27 phages7, 11, 12 of which 19 are mutant phages (FIG. 6).18 If cp26 was not a Girus (Giant Virus), the plasmid would have burst due to the infection of a natural phage. However, cp26 is the only plasmid to be found in all isolates of Bb that has not experienced gene loss.7, 18
It is important to note that convoluted terminology has been previously experienced in discussions around Bb. The terms “spiroplasma”, “mycoplasma” and “spirochete” are used interchangeably,31 so this is understandable for “Giant virus”, “Plasmid” and “chromosome”. The Giant Virus is also said to be associated with amoebas, however, there are amoeba-associated bacteria which can exploit common mechanisms.26
Dr. Willy Burgdorfer reviewed and tested patient blood samples from Dr. Anderson for C9P09, which is a rickettsial helical Mycoplasma; P09 being a Rickettsia bellii and C9 being a Mycoplasma (FIG. 2).35 This is further supported by the theory that an endosymbiotic infection produces spirochetes that are uncultivable Mycoplasmas, which are also called spirochetes. 1, 22, 31, 32
The “Swiss Agent” is documented by Dr. Willy Burgdorfer (FIG. 3),25 who also wrote a speech on “Pandora’s Box”.5 Although the Swiss Agent paper is associated with the suspected African Swine Flu, Dr. Willy Burgdorfer appears to be famous for leaving clues throughout his work regarding Lyme disease. Upon reviewing the structure of the Lyme disease “Swiss Agent” and the “Pandora Giant Virus”, there is a striking resemblance that deserves further examination. This theory is supported by presentations FIG. 3 & 4, which show similar organisms. Additionally, this is supported by phylogenic information found on www.giantvirus.org regarding the genes found within the giant viruses (FIG. 5).
Giant viruses are also known to harbor a symbiotic colony of Sputnik or mutant virophages in archaeal viruses (plasmids).9, 27 These phages are found in cp26 and the multiple cp32’s, which are worthy of additional research.4, 7, 11, 12, 18 From a virology standpoint, the only way these phages could coexist in a symbiotic relationship within the bacteria would be if the plasmid was in fact a giant virus- able to transduce genetic information to ensure the survival of the host and the organisms.11, 12 The phages carried in cp26 are the only ones to be found in all isolates and never lost.7
The giant virus also brings into question why STARI and Master’s disease is noted by Dr. Willy Burgdorferi to be a Lyme disease agent that is found in Amblyomma americanum (FIG. 1). This is supported by the fact that flaB (a transcription gene for Bb) was found in all STARI patients he had tested.8, 11 The Lyme- like organism is a mimicking microbe that by scientific definition is a giant virus.9 The scientific differentiation has been made between “Early Lyme disease” and “STARI”/“Master’s disease”, but not “chronic Lyme disease”.24 There are numerous morphological similarities between chronic Lyme disease and STARI/ Master’s disease. In both organisms, uncultivable spirochetes can be found, in addition to the transcription gene, flaB being found in STARI. Being that flaB is a promoter gene of cp26,10 the giant virus of Borrelia burgdorferi would be found in STARI patients.
It is suspected that the rickettsial agents associated with the Lyme disease organism assists in a morphological transformation of the plasmid within the spiroplasma.2 This transition could cause the morphological transformation of the bacterial infection of early Lyme disease into the chronic Lyme/ STARI/ Master’s disease organism of uncultivable spirochetes.3, 31 In addition to the transformational aspect of early Lyme disease to chronic Lyme disease, we have a variety of ticks believed to carry this organism (FIG. 1). It is possible that the giant virus is still transferred to the patient, despite some ticks carrying an anti-borrelial agent in their salvia that eliminates the spiroplasma.1, 8, 24 All evidence supporting the theory that cp26 is in fact responsible for the uncultivable spirochetes found in human subjects,3 potentially the same for all three diseases in question in this research study.
Chronic Lyme (Figures found here)
References
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FIG. 1
Willy Burgdorfer’s research notes on established tick-borne disease of humans and/or animals in the United States. The Lyme disease agent, Borrelia burgdorferi is carried by Ixodes dammini, Ixodes pacificus, Ixodes scapularis, Amblyomma americanum.
FIG. 2
Willy Burgdorfer’s research notes on patients with rickettsial agents. The Swiss agent is shown to be a rickettsial helical mycoplasma, known as C9P09.
FIG. 3
A microscopic slide of the labeled Swiss agent
FIG 4
Figure B2 is a Pandoraviridae, showing a striking resemblance to that of the Swiss agent.
FIG 5
The phylogenetic tree of giant viruses that includes the RpoC, DN-directed RNA polymerase, beta subunit/160 kD Transcription of Borrelia burgdorferi
FIG 6
The mutant alleles on cp26 showing 27 total bacteriophages, of which 19 are mutant bacteriophages.
Contact Information
Allison.Caruana@TheMaydayproject.org
Acknowledgements: First and foremost, I acknowledge my Lord and Savior for saving me and giving me ability to do what I do to help others. To my five wonderful children that have supported my efforts and dealt with the hours upon hours of scientific studies. To my late husband Michael, who always believed in me and helped me to remain focused on what mattered the most. To my family of Lyme warriors, it is our suffering and our strength that has put the wind in my sails and given me the drive to see that future generations do not suffer the way we do.
