This article, although a year old, has some great insights.

How Lyme and Hidden Infections Sabotage Our Clinical Outcomes

By Jason Bachewich, ND

What if I told you that as a clinician, you were potentially misdiagnosing a large percentage of your autoimmune patients?  What if the arthritis, Alzheimer’s, cancer, or Grave’s disease was actually caused by an infection?   The research is starting to show that perhaps our bodies are not flawed or simply have bad luck but rather sabotaged by chronic and hidden infections.  Our treatment plans would be different, and our outcomes more positive.  This is the beginning of a whole new understanding of chronic disease, and the potential is hugely exciting.

Lyme disease has been gaining a lot of attention in the media lately.   Doctors are becoming more aware of the symptoms, but why just look at Lyme disease?  There are multiple bacterial, viral, and other parasitic infections that can sabotage our clinical outcomes and have been ignored or assumed to be benign. This article is going to help you to identify those key symptoms to look for, how to test for the infections, and familiarize you with the most common hidden infections that we are not taught about in medical school. (See link for article)



Time for the one germ one drug paradigm to die.  Patients present with complex clinical pictures that can not be explained away simply, but demand astute observation and lengthy medical histories.  It is not uncommon for patients to have multiple things going on simultaneously.

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For the first time, Garg et al. show a 85% probability for multiple infectionsincluding not only tick-borne pathogens but also opportunistic microbes such as EBV and other viruses.

I’m thankful they included Bartonella as that one is often omitted but definitely a player.  I’m also thankful for the mention of viruses as they too are in the mix.  The mention of the persister form must be recognized as well as many out there deny its existence.

Key Quote:  Our findings recognize that microbial infections in patients suffering from TBDs do not follow the one microbe, one disease Germ Theory as 65% of the TBD patients produce immune responses to various microbes.”

Many patients also struggle with mold and mast cell issues:

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