Archive for the ‘Viruses’ Category

Former NC Senator Has Powassan

http://www.witn.com/content/news/Former-Sen-Kay-Hagans-illness-linked-to-virus-spread-by-ticks-413508473.html  By Associated Press | Posted: Sat 9:31 PM, Feb 11, 2017

RALEIGH, N.C. (AP) – Former North Carolina Sen. Kay Hagan’s family says the brain inflammation that she’s recovering from was caused by a virus that’s spread by ticks from animals to humans.

A family spokeswoman on Saturday described the cause of Hagan’s encephalitis, citing a doctor who had been treating her. Recently received test results indicated Hagan contracted the Powassan virus.

Hagan’s husband says his wife has improved since she was moved to an Atlanta rehabilitation center three weeks ago, but that the recovery process will be long.

The former senator initially was in a Washington hospital in December and was later in Chapel Hill. She was elected to the U.S. Senate in 2008 and served through 2014.

Contact your US Senator and US Representative to insist that our
governmental agencies make this one of the TBD priorities. Find yours at:
http://www.whoismyrepresentative.com/

Tick research must be funded to protect us, our children, our pets and
other critters to prevent ALL Tick-borne diseases! In 41 years since Lyme
disease was found in CT, ticks and TBDs have been allowed to spread to over 49%
of the counties in the USA.

Please pass this on to others and contact your US Congressional Reps.
Calling them is very easy and takes a minute or so for each of the three. Your
voice is power, especially when they hear from many constituents!

 

Category:

Activism, Viruses

NTZ – Game Changer for Lyme Patients?

https://www.linkedin.com/pulse/nitazoxanide-known-killer-parasites-borrelia-cysts-booster-ozimek?trk=v feed&lipi=urn%3Ali%3Apage%3Ap_flagship3_feed%3BjOKiznIW%2BSs5upN50KaGRg%3D%3D

Is nitazoxanide (NTZ)- a real game changer for Lyme patients?
Written by:  Wojciech Piotr Ozimek, independent lecturer and researcher specialized in parasitic and vector-borne diseases.  Used with permission.

Nitazoxanide, a thiazolide compound, and its desacetyl derivative, tizoxanide, have antimicrobial properties against protozoa, cestodes, nematodes, trematodes, bacteria and viruses.

A retrospective review of charts of patients treated with nitazoxanide for infections caused by giardia lamblia, cryptosporidium parvum, blastocystis hominis, entamoeba histolytica, cyclospora, isospora and babesia caused infections demonstrated it’s high efficacy.

Unfortunately most of the studies showed that nitazoxanide is rather ineffective for the treatment of trichomoniasis.

Because the treatment of Helicobacter pylori infection may be jeopardized by metronidazole resistance, nitazoxanide and tizoxanide were tested in vitro and in vivo studies. Conclusions? It may be used as a single agent in the treatment of Helicobacter pylori and other Campylobacter sp. caused infections. It is also effective against Clostridium difficile caused infections.

There has been a study that claims there is some limited evidence for efficacy for SIBO (small intestinal bacterial overgrowth) in the context of an open label study, but no systematic review and all claims should be taken lightly.

Development of resistance to the two main classes of drugs used to treat intestinal nematode infections of humans has been reported. We need new and more effective drugs and ways to improve the efficacy of the old drugs. And again the promising alternative drug is nitazoxanide (NTZ). NTZ shown to have therapeutic activity against nematode (ascaris), cestode (taenia, hymenolepsis) and trematode (fasciola) infections. In addition NTZ combines synergistically with other classes of anthelmintic drugs, i.e. albendazole and pyrantel, making it a good candidate for further studies on its use in drug combination therapy of parasitic infections.

Lateef et al.. conducted a study in India that evaluated the effectiveness of nitazoxanide in the treatment of beef tapeworm (Taenia saginata) infection. They concluded that nitazoxanide is a well-tolerated drug for the treatment of niclosamide- and praziquantel-resistant beef tapeworm (Taenia saginata) infection.

It is also considered to be a “cyst busting” drug by some researchers, so can theoretically impact Borrelia, too. For these reasons, it is one of my favorite drugs, and in my opinion it should have a broader application for Lyme disease patients.

As if the above-mentioned benefits of nitazoxanide weren’t enough for some skeptics, the drug also impacts biofilm formation. This was shown by research published in Oxford’s Journal of Antimicrobial Chemotherapy, in a study entitled, “Nitazoxanide inhibits biofilm formation by Staphylococcus epidermidis by blocking accumulation on surfaces.” Several other studies also demonstrate nitazoxanide’s inhibition of biofilm forming.

And lastly, nitazoxanide has even been shown to inhibit numerous viruses, including hepatitis B and C viruses, rotavirus and influenza A virus (in vitro).

To summarize, nitazoxanide can be used to kill protozoa, cestodes, nematodes, trematodes, bacteriae (incl. cysts & biofilm forms) and viruses.

It’s no wonder that Lyme patients, who are often afflicted with many of mentioned above infections, can feel so much better as a result of using nitazoxanide.

Nitazoxanide should be one of the most multi-talented and broadly applicable drugs available to Lyme sufferers, and I consider it to be one of the most important medicines presented in the last years.

Nitazoxanide is an effective, inexpensive, and generally safe and well tolerated medicine. In the clinical trials no serious adverse events were reported. Adverse events occur in less than 1% of the patients but physician monitoring is still advisable.

Nitazoxanide is sold under the brand names Nizonide, Nitaxide, Nitax, Zox, Netazox, Niazid, Toza, Daxon, Dexidex, Kidonax, Mitafar, Pacovanton, Paramix, Alinia, Adonid, NT-TOX, Nitamax,and Annita.

 

Category:

C-diff, Lyme, research, Treatment, Viruses

A Bug for Alzheimer’s?

https://aeon.co/essays/how-microbial-infections-might-cause-alzheimers-disease

Please read the article above, written by Melinda Wenner Moyer, edited by Pam Weintraub.

A brief summary:

Robert Moir, a neurologist at Massachusetts General Hospital in Boston, believes that beta-amyloid, a key player in Alzheimer’s, might be a good guy who is actually protecting the brain from pathogens.

This idea is coming from numerous corners of the world and has been labeled ‘pathogen hypothesis.’ Others pointing this out are pathologist Alan MacDonald, neuropathologist Judith Miklossy, and microbiologist Tom Grier.

Moir has published mouse studies showing that their brains create amyloid plaques within hours of contracting infections and they actually kill pathogens.

This observation flies in the face of accepted dogma about beta-amyloid and it is rarely discussed in AD groups.

A meta-analysis of 25 published studies has shown that infected folks are 10 times more likely to develop AD, leading international researchers to co-sign an editorial begging others to consider pathogens in relation to AD.

But, the cabal isn’t having it. Moir’s 2016 paper was rejected six times without even a review before finally getting the nod.  https://madisonarealymesupportgroup.com/2017/01/13/lyme-science-owned-by-good-ol-boys/

The author reminds the reader that infections in the brain are nothing new and a short list of them includes: Syphillis, Herpes simplex encephalitis, tick borne disease, HIV, Toxoplasma gondii, Chlamydia pneumoniae, HSV-1, and Zika.

https://madisonarealymesupportgroup.com/2016/04/10/bugs-causing-alzheimers/

https://madisonarealymesupportgroup.com/2016/06/03/borrelia-hiding-in-worms-causing-chronic-brain-diseases/

https://madisonarealymesupportgroup.com/2016/08/09/dr-paul-duray-research-fellowship-foundation-some-great-research-being-done-on-lyme-disease/

http://www.huffingtonpost.com/david-michael-conner/man-diagnosed-with-als-di_b_8891262.html

The journalist also points out that pathogen causation is not proven and that Alzheimer’s patients might be prone to infection but that some studies suggest the infections came first. She also says that the majority of folks suggesting the ‘pathogen hypothesis’ do not feel the infections work alone but rather can cause a domino effect that over time can accumulate causing AD.

And lastly, if beta-amyloid causes AD then removing these plaques should get rid of symptoms, but when 145 beta-amyloid-reducing drugs were tested, not one slowed progression of the disease.

Once again, proving a science cabal exists, Moir recounts how at a Korean conference, attendees were asked to raised hands if they thought infections played a part in AD and a majority of hands went up.

“Ten years ago, it would have been four guys in a corner, all huddled together, not talking to anyone else,” Moir says.

https://madisonarealymesupportgroup.com/2017/01/02/fake-science/

Isn’t that sad?

Category:

Alzheimer's, Lyme, research, Syphilis, Toxoplasmosis, Viruses, Zika

2016 Vaccine News

MSIDS patients are hit with an onslaught of vaccine messages, often from the very pharmacies they must call to order life-saving drugs for tick borne infections.  These messages are always one-sided, telling patients to get their vaccine(s) now.

MSIDS patients often feel so incredibly lousy that they literally have a challenge putting one foot in front of the other.  The very thought of needing to research vaccine information sends the very stoutest to bed for a nap, and even if they manage to do a little sleuthing, they can’t remember what they have read just 10 minutes later.

It is especially for you that this post is written.

Published on Dec 10, 2013
Doctors say the human papillomavirus vaccine, Gardasil, may prevent cervical cancer, but two sisters say they believe the drug made them infertile.  They are now suing the manufacturer.

For more on Gardisil:

https://madisonarealymesupportgroup.com/2016/07/19/motor-and-sensory-findings-in-girls-who-received-gardasil/

https://madisonarealymesupportgroup.com/2016/04/24/gardasil-and-bartonella/

Deaths from live Polio Vaccine:

http://www.thevaccinereaction.org/2016/12/two-children-die-after-polio-vaccination-four-in-critical-condition-in-pakistan/

“Two children in the Tehsil Bara area of the Khyber Agency tribal region in Pakistan reportedly died last week ‘moments’ after receiving the live oral poliovirus vaccine. Another 11 children, who had been administered the vaccine in Tehsil Bara, fell unconscious and were taken to Hayatabad hospital in the city of Peshawar. Four of the 11 children remain in critical condition.  

OPV has long been known to actually cause vaccine strain polio paralysis in some individuals who get the vaccine, as well as cause paralysis in some who come in close contact with recently vaccinated persons shedding vaccine strain polio virus in saliva, urine and other body secretions. According to the U.S. Centers for Disease Control and Prevention (CDC), “Cases of vaccine-associated paralytic poliomyelitis cases do occur in countries using oral poliovirus vaccine.”

While the US does not use the live polio vaccine any more, there has been an alarming increase in Acute Flaccid Paralysis (AFM), a neurological disease that closely resembles the poliovirus in the U.S.  http://www.thevaccinereaction.org/2016/11/mysterious-polio-like-illness-plaguing-children-stumps-cdc/.  Eighty-nine people across 33 states have confirmed AFM.

“AFM affects a person’s nervous system, particularly the spinal cord. Patients present with a fever or respiratory illness, then develop temporary paralysis. Among the 121 cases reported in 2014, only three children recovered fully, although 85 percent recovered partially.”

The question begging to be asked is does the shedding of the live polio vaccine actually cause the AFM we are seeing in the U.S.?

http://www.thevaccinereaction.org/2016/12/mercks-ebola-vaccine-a-christmas-gift-for-the-world-really/

Media claims “New Ebola Vaccine Gives 100 Percent Protection.”

What really happened:

**5,643 adults and 194 children in the West African country of Guinea were in contact with people with Ebola.

**65% were given a single dose of the rVSV-ZEBOV vaccine.  They reported no cases of Ebola after a designated window of 9 days.  Some did actually come down with Ebola but they weren’t counted because they assumed they were infected before vaccination.  (Remember the axiom, “Assuming makes an Ass out of u and me.”)

**35% were given the vaccine 21 days later.  These folks along with those who never received the vaccine had 23 cases of Ebola.

**The incubation period for Ebola is 2-21 days with an average of 8-10 days. http://www.who.int/mediacentre/factsheets/fs103/en/.  Is it good science to stop observation after 9 days on a disease that has a 25%-95% fatality rate?

**Two different sized groups are being compared. The group with no Ebola contains 16% less people than the group with 23 cases and may actually be greater than that, given that an unknown portion actually came down with Ebola but were not counted.

This is reminiscent of how the CDC suddenly decided that two fetal samples proves that Zika causes microcephaly:https://madisonarealymesupportgroup.com/2016/12/21/how-zika-got-the-blame/

http://www.forbes.com/forbes/welcome/?toURL=http://www.forbes.com/sites/stevensalzberg/2016/12/20/anti-vax-movement-to-blame-for-quadrupling-of-mumps-cases-this-year/&refURL=  A recent article in Forbes blames those choosing to forego the Mumps vaccine with causing mumps outbreaks even though most that contracted it had been fully vaccinated!

http://www.thevaccinereaction.org/2016/12/scapegoating-anti-vaxxers-for-the-mumps-outbreaks-how-predictable/  Here’s a great article showing the Forbes author doesn’t present any facts to bolster his claim but in typical form belittles anyone who choses informed consent in regards to vaccinations.

The take home:  Do your reading.  You are chronically ill with pathogen(s) that are already giving your immune system a run for its money.  Consider carefully what you allow into or onto your body.  As Dr. Burrascano has stated, “Now is the time for the very best of health habits.”  http://www2.lymenet.org/domino/file.nsf/UID/guidelines

It’s your body.  You decide.

 

 

Category:

vaccines, Viruses, Zika

How Zika Got the Blame

http://www.thevaccinereaction.org/2016/12/cdcs-rasmussen-paper-on-zika-and-microcephaly-poor-case-for-smoking-gun/ Article in link by Marco Caceres.

In early 2016, the CDC began to suspect a link between Zika and Microcephaly due to two placenta samples of babies who sadly died, but they admitted that additional studies with lab testing that may take years to complete was needed. As of April 1, 2016, the CDC, NIH, PAHO, and WHO, all agreed there was insufficient evidence to state that Zika causes Microcephaly.

Magically, in two weeks time, after numerous papers were published stating that large prospective studies following pregnant women infected with Zika were needed, they made an about face and stated emphatically that the existing evidence was now sufficient to state Zika causes microcephaly.

What happened in two weeks?

Were hundreds of thousands of hours spent in the lab testing women and babies?

No.

Here’s what happened.

They took one paper (Rasmussen, Jamieson, Honein & Petersen)  http://www.nejm.org/doi/full/10.1056/NEJMsr1604338?query=featured_home&  and plopped it into a scientific formula called Shepard’s criteria which was developed by a pediatrician in 1994. Ironically, even Dr. Petersen, the director of the CDC’s division of vector-forne diseases, and one of the four authors in the paper initially denied a causal link.

Oh, by the way, did I mention that all the authors are CDC employees?

What is Shepherd’s criteria? Glad you asked.

1) Proven exposure to the agent at one or more critical times during prenatal development.
2) Consistent findings by two or more high-quality epidemiological studies, with control of confounding factors, sufficient numbers, exclusion of positive and negative bias factors, prospective studies if possible, and relative risk of six or more.
3) Careful delineation of clinical cases; a specific defect or syndrome, if present, is very helpful.
4) Rare environmental exposure that is associated with rare defect.
5) Teratogenicity in experimental animals important but not essential.
6) Association should make biologic sense.
7) Proof in an experimental system that the agent acts in an unaltered state.

The researchers agreed that only 1, 3, and 4 of the criteria had been met.

There have been numerous Brazilian studies and reports questioning their conclusion.
http://www.thevaccinereaction.org/2016/09/cdc-bets-farm-on-zika-based-on-conclusion-of-rasmussen-jamieson-honein-petersen-paper/

The Author of the opinion piece asks some important questions: should criteria from twenty years ago be used to prove causality when numerous other factors have been proven to cause birth defects, and why should a historically harmless virus become abruptly virulent?

I’d like to add to the list of questions: why is the CDC hellbent on whooping Zika up into a national health crisis when we have plenty of health topics that are far more prevalent (Lyme Disease/MSIDS, opioid addiction, and antibiotic resistant bacteria, for a start). How can the CDC on one hand determine causality from two fetal samples when there are literally 230 peer-reviewed studies that show borrelia persistence despite antibiotic therapy? http://norvect.no/230-peer-reviewed-studies-show-evidence-of-persistent-lyme-disease/  Why are ancient studies on everything from possible vectors/reservoirs to transmission times, to diagnostic myths such as mandatory EM rashes held in sacred honor and used to continually refute any new information to the contrary?

And when are scientists finally going to admit they are often being used to further agendas that are neither impartial nor transparent?

For more on Zika:  https://madisonarealymesupportgroup.com/2016/03/08/fixation-on-zikapolio/

https://madisonarealymesupportgroup.com/2016/07/17/zika-in-the-land-of-oz/

https://madisonarealymesupportgroup.com/2016/10/26/zika-puzzling-scientists/

https://madisonarealymesupportgroup.com/2016/03/04/health-policy-recap/

https://madisonarealymesupportgroup.com/2016/04/08/zika-ebola-zombies-and-the-cdc/

 

Category:

Activism, mosquitoes, Viruses, Zika