Archive for the ‘Adrenals’ Category

Seasons of Lyme

By Jennifer Crystal


In the past couple weeks, many people have told me that the recent change of the clocks to Daylight Savings Time is really getting to them. They feel tired and sluggish. While the Equinox should mark a positive shift towards spring and sunlight, the transition can actually be difficult for healthy and sick people alike.

I used to hear the term “spring fever” and assume it meant I was supposed to feel restless and excited for the upcoming season, like the people I saw running and playing outside. But I just couldn’t summon their energy. For me, spring fever manifested itself as malaise and, sometimes, an actual fever. All I wanted to do was sleep. I was not alone.

I’ve learned that while some people are able to jubilantly jump into a new season, it’s more upsetting for others, especially Lyme patients. For those of us suffering sleep disturbances, changing the clocks just one hour can seriously impact our circadian rhythms. I always experience an uptick in my sleep-related symptoms when I shift to Daylight Savings or back to Standard Time. I have more difficulty with insomnia, I have more nightmares, and I have trouble falling asleep for—and then getting up from—my afternoon nap. It can take me a week or more to get used to the new schedule.

But even without a time shift, simply the jump from season to season can be especially trying for Lyme patients. In my case, it may be my system is so compromised and is working overtime to fight infection, that it overreacts to the slightest changes. If I need to adjust a medication, I have to do so incredibly slowly, tricking my body into thinking that no dosage change is happening at all. If I get too hot or too cold, my body has a much stronger reaction than the non-Lyme sufferer would; I can overheat without warning, needing cool compresses all over my body, or I can get suddenly hypothermic.

Since climate change has altered the Earth’s ability to raise or lower temperatures based on the season, we’ve experienced heat waves in February followed by freezes and blizzards in March. That’s enough to send anyone’s system into shock, but for Lyme patients, these erratic weather patterns can cause the hyper-reactions I describe above. Many Lyme patients find that symptoms such as joint pain, fevers, and headaches—already difficult to control—become almost impossible to predict during seasonal fluctuations.

And it’s not just during the shift from winter to spring. Lyme patients struggle with every seasonal shift. I personally get more babesia symptoms such as air hunger and headaches in summer, while other Lyme patients might have worse joint pain in fall or winter.

Even though our bodies might react strongly to seasonal fluctuations, one advantage we Lyme patients have is resilience. If you really think about it, we experience internal season changes every single day. We can feel spring-like—energetic, full of possibility—for a morning, and then slip into a winter-like hibernation in the afternoon, often without warning. Lyme symptoms can vary daily if not hourly, and we have to learn to adapt to that unpredictability. If we can handle these shifts multiple times a day, we certainly can handle them a few times a year.

There are small steps we can take to help ourselves through seasonal fluctuations. I find it helps to be especially fastidious about my sleep hygiene during these times, making sure I stick to the same schedule, even if the clock changes by an hour. I allow myself a little extra time for rest during these periods. I wear more layers than most people do when it’s very cold outside, and cancel outdoor activities when the temperature rises above 90 degrees. I carry a water bottle with me where ever I go. I also sometimes have to change my medication or supplement doses seasonally, knowing that certain symptoms flare or subside depending on the weather.

Of course, it isn’t always easy to handle these shifts; they can be extremely frustrating. One hour we can be happy and hopeful, the next exhausted and depressed, later that day anxious and angry. We can experience summer and winter and fall and spring several times in a day, in no particular order, and that can make a patient feel really off-kilter.

If you’re in a tough season of Lyme, remember what we say in New England: wait five minutes, and the weather will change. Longer, brighter days really are ahead.

Opinions expressed by contributors are their own.

Jennifer Crystal is a writer and educator in Boston. She is working on a memoir about her journey with chronic tick-borne illness. Contact her at




Resilience for the Lyme/MSIDS patient…….not so much.  Our bodies don’t bounce back like they used to.

A reminder:  the climate has always changed.  This is not a new fact.  What is new for the Lyme/MSIDS patient is that these infections affect everything, including our adrenals and hormones that run our body’s thermostat, ability to sleep and feel rested, body temperature, sex drive (or lack thereof) and more.  My husband had horrible hypoglycemic attacks and temperature dysregulation.  He could be under 5 blankets with teeth chattering one minute, then sweating profusely which would initiate overall body itching, and then get hit with unwarranted anxiety over absolutely nothing, the next minute.

And so it goes…..everyone responding differently, yet crazily.

Many have trouble with the new moon due to parasite reproduction.

The thing is to be aware and try to accommodate these changes with preparedness and understanding for yourself.  Keep reminding yourself that your body’s in a war and under siege by pathogens that cause unbelievable havoc.  

For more on sleep tips:







Can’t Sleep? 18 Plants & Herbs Can Help

Natural Sleep Aids
18 plants and herbs for better sleep

by Beth Janes | Posted March 30th, 2018

We’ve all been there: Exhausted, but sleep just isn’t happening. Maybe your brain won’t shut down, or you can’t get comfortable. Or perhaps you did manage to drift off, but woke up feeling like you ran 8 miles, not slept 8 hours.

What gives? It’s likely a disruption in the normal tides of brain chemicals that are tuned into your circadian rhythms, says Dr. Bill Rawls, medical director of Vital Plan. And these rhythms are what either keep us awake or put us to sleep.

“During the day, levels of the stress hormone cortisol are elevated, which helps us get through day-to-day activities,” Dr. Rawls explains. In the evening, cortisol and its cohorts are supposed to ebb, making way for the flow of a new set of relaxing chemicals that induce and sustain sleep. However, stress and other factors, such as stuffy sinuses or aches and pains, can throw off the chemical tides—and your Zzzs.

While you may be tempted to pop a sleeping pill, they can come with dependency and other unwanted side effects. Instead, consider turning first to nature’s pharmacy. Research shows it’s stocked with plants that can promote a healthy sleep environment and may help you unwind, drift off, and wake up feeling energized and refreshed.

Here are three tips and a garden of options to try:

1. Bring nature into your bedroom

Not only do studies suggest that simply being around plants can help you feel calmer, certain varieties are especially effective at scrubbing the air of pollutants that cause sleep-disrupting symptoms, according to a paper in the journal Environmental Health Perspectives. Others, meanwhile, give off rest-promoting aromas.

• Air purifying houseplants

Take your pick of Areca, lady and bamboo palms, English Ivy, Boston fern, peace lily and Ficus. All are on the top-10 list of best houseplants for their ability to remove volatile organic compounds (VOCs) from indoor air, as assessed by a NASA researcher. Many building and household materials like paint, carpeting, and cleaning supplies release VOCs, which are known to irritate eyes and airways and trigger headaches and fatigue—in other words, symptoms that mess with sleep.

• Calming houseplants

Scents are known to affect the nervous system, and science shows that lavender, jasmine, and gardenia are especially calming. For example, researchers at Wesleyan University found that when people sniffed lavender oil before bed, they spent more time in deep sleep and felt more energized and refreshed in the morning. In another study from Wheeling Jesuit University, people were exposed to jasmine scents while sleeping, causing them to move around less, indicating better-quality sleep.

2. Sip your way to better sleep

There’s something immediately calming about cupping your hands around a warm mug of herbal tea and breathing in the steam that wafts up. But the right mix of steeped herbs in your cup could make the ritual even more effective.

Here are three teas to look for:

• Passion flower

“Passion flower helps bring on calm, and it also promotes muscle relaxation,” says Dr. Rawls. Those two benefits make this Amazonian plant especially effective for promoting sleep. In fact, people who drank passion flower tea for a week reported better sleep quality than when they drank a placebo tea, according to a study from Monash University in Australia.

• Chamomile and valerian

Perhaps the two most common herbal ingredients found in bedtime teas, their sleep-supporting benefits are well supported by research. For example, postnatal women who drank chamomile tea for two weeks reported less sleep interference from physical symptoms, according to a study in the Journal of Advanced Nursing.

Just be sure to listen to your body if you try these teas. While chamomile works well for many, it may keep others awake, Dr. Rawls says. Likewise for valerian: “About 25 percent of people who take it can feel agitated,” he says.

3. Use herbal supplements for temporary support

Certain herbs are believed to help you rest by affecting the brain’s gamma-aminobutyric acid (GABA), a key neurotransmitter that induces sleep, Dr. Rawls says. The caveat: They work best if taken only intermittently — a few nights in a row to deal with occasional sleep trouble.

“If you use anything that hits the GABA system every night, whether it’s herbs or drugs, it can suppress natural GABA over time,” says Dr. Rawls. “That can cause a rebound effect that makes insomnia worse.”

Here are a few Dr. Rawls recommends for occasional sleep support:

• Passion flower

Tea isn’t the only way to take advantage of the calming properties held in the leaves of this pretty plant. For instance, a study in the Journal of Anesthesia found that patients about to undergo spinal anesthesia who took passion flower extract felt calmer than those who received a placebo. Another study found that a combination of passion flower, valerian, and hops worked significantly well for improving occasional sleeplessness.

• Bacopa

An herb native to India, bacopa has been used for thousands of years and is best known to help support memory, focus, and mental function. But it’s also calming and has a mild sedative effect, Dr. Rawls says. One study, for example, showed the herb could help mitigate some of the effects of stress.

• Motherwort

Although it originated in central Eurasia, this member of the mint family has long been used in herbal medicine, and it now grows in gardens in temperate areas of the world. “It’s a nice, calming herb that affects dopamine and has sleep-promoting qualities,” Dr. Rawls says. Russian researchers found that in subjects with high blood pressure and sleep problems, 80% of those who took motherwort saw significant or moderate improvement in low mood and related sleep trouble.

• Ashwagandha, magnolia, and phellodendron

“The key to a good night’s sleep isn’t what you do at bedtime, but instead it’s what you’re doing during the day,” Dr. Rawls says. Herbs like ashwagandha, an adaptogen that hails from India and parts of Africa, as well as magnolia and phellodendron help moderate daytime stress and may set the stage for healthy sleep.

Utilizing houseplants, teas, and supplements may be all you need for a good night’s rest. But for the best and lasting results, Dr. Rawls recommends combining plants with lifestyle changes that are known to improve sleep long-term.

“Regular exercise and other stress-reducing activities, as well as practicing healthy sleep hygiene like limiting screen time at night, are also essential elements for enjoying optimal sleep.”

Lack of sleep is a huge problem with Lyme/MSIDS patients.  We slump through the day, feeling exhausted and depleted, and then roam the halls like zombies all night long.
I tried many, many things over the years and frankly, the best thing that helped me was Lyme/MSIDS treatment.  Get rid of the bugs and you can sleep again.
However, some other things that had limited effect were melatonin & valerian root tincture for me, and 5-HTP, & Gabapentin for my husband.  I know many patients who have to resort to stronger medications to get relief.
Some other hacks I learned through the years:
  • Try and determine if you can’t get to sleep or stay asleep, or both, as that will help your doctor pin point your problem and help you find the right remedy.
  • Get away from all blue-screens (computers, phones, iPads, etc.) preferably hours before bedtime, as that type of light tricks the body into thinking it’s day.
  • Establish a night time routine.
  • Perhaps take a nightly bath in epsom salts for detoxing and relaxing.
  • Read a book that will relax you – or even comics.
  • If you have a racing mind, keep a notebook and pen by your bed to write wandering thoughts or “to-do” lists so you can free your mind up.
  • Sleep in a completely blackened out room as any light will affect melatonin production.  If you can’t obtain that, wear a sleep mask.
  • If noises bother you, wear ear-plugs.
  • Exercise is important, but don’t do it too close to bedtime as it will rev you up.  And, speaking of exercise, do what feels good.  We have enough pain without adding more.  I walked.  Walking, as it didn’t give me pain, helped me tease out what was Lyme/MSIDS related pain as I knew it wasn’t due to walking.  If you are just starting up; however, you will notice it in your calves, shins, hips, and perhaps feet until your body adjusts.  Start by walking to the mailbox.  Add distance as you are able.  Wear good walking shoes.
Make sure you work with your doctor and be honest about inability to sleep as it is such an important aspect of healing.

Bb, Genetic, & Environment in Pathogenesis of Autoimmune Thyroid Diseases

Infections, genetic and environmental factors in pathogenesis of autoimmune thyroid diseases.

Shukla SK, et al. Microb Pathog. 2018.


In Autoimmune disease a combination of infection, genetic and environmental factors causes an autoimmune response to the thyroid gland (characterized by lymphocytic infiltrations), thyroid stimulating hormone receptor (TSHR) and different thyroid antigens. Graves’ and Hashimoto disease are autoimmune disorders with genetic predisposition. CD40 that stimulates the proliferation and differentiation of lymphocytes is an essential immunomodulatory component for follicular cells in the thyroid and the cell that present the antigen. CD40, PTPN22 and thyroid-specific genes are immunomodulating genes for the TSH receptor and thyroglobulin. CD40 used to be associated with Graves’s disease as positional candidate on the basis of Graves’ disease linkage study connecting with 20q11 genome chromosomal region.

The PTPN22 gene gives rise to a substantial risk of specific autoimmune phenotypes and frequent disease mechanisms. Infections have been implicated in the pathogenesis of AITD including Coxsackie virus, Yersinia enterocolitica, Borrelia burgdorferi, Helicobacter pylori and retroviruses (HTLV-1, HFV, HIV and SV40). Infectious hepatitis C agents are the strongest proof supporting an affiliation with AITD.

The essential environmental triggers of AITD are iodine, drugs, infection, smoking and perhaps stress. Autoimmune disease provide important facts on genetic mechanisms that influence the prognosis and treatment of the disease and by recent molecular techniques through gene expression study by quantitative Real Time-PCR and microarray, we can identify novel genes which are responsible for Graves’ and Hashimoto disease.


Insurance Claims Show Rural-Urban Divide With Lyme Disease   by Lisa Schnirring  July 27, 2017

Lyme disease insurance claims show disease rise, rural-urban divide


Lyme disease levels in the United States have been rising since 2007, and patterns show differences in rural and urban settings, according to new findings released today from a nonprofit health insurance group, which based its analysis on more than 23 billion privately billed insurance claims.

The study from FAIR Health, which studies healthcare costs and health insurance information, found that insurance claims involving Lyme disease diagnosis rose 185% in rural areas from 2007 to 2016, with a smaller 40% increase in urban areas. The group’s findings appear on its Web site.

Lyme disease, caused by Borrelia burgdorferi bacteria, is spread through the bite of blacklegged ticks. Symptoms include fever, headache, fatigue, and a characteristic skin rash. When untreated, the disease can lead to joint, cardiac, and neurological complications.

The US Centers for Disease Control and Prevention (CDC) says about 30,000 Lyme disease infections are reported from states each year, a number it says probably reflects only a fraction of the disease burden.

Timing, ages also vary between rural and urban Lyme claims

In its full report, researchers said it’s not clear how many Lyme disease diagnoses from urban areas are the result of exposure to ticks in city yards and parks or from visits to rural settings.

The peak for Lyme disease claims is June and July, which the group said isn’t surprising. However, it noted that claims during winter and early spring months are more common from urban areas, though the location of the medical visit may vary from the patient’s exposure location.

The age at which patients were diagnosed with Lyme disease varied between the rural-urban divide as well, with rural areas reporting higher numbers of claims for middle-aged and older people and urban areas reporting a larger share of younger people.

Regarding geographic distribution, the insurance claims analysis suggests that Lyme disease is spreading beyond the two traditionally hardest hit areas: the northeast and the upper Midwest. From the review of 2016 claims, North Carolina is now among the five states the highest percentage of claims, and FAIR Health said the disease has apparently found a foothold in the south.

Disease complication findings

To probe the relationship between Lyme disease and other health problems, researchers conducted a retrospective longitudinal study that looked at insurance claims between 2013 and 2017.

The most common diagnoses that came in the wake of a Lyme disease diagnosis were joint pain, fatigue, soft-tissue disorders, and hypothyroidism. When they compared the diagnoses by age to people not affected by Lyme disease, the same conditions still stood out as more common in those sickened by Lyme disease.

“Our data suggest that at least some autoimmune diagnoses correlate with Lyme disease,” they wrote, alluding to previous research studies that have suggested a possible autoimmune reaction link to disease complications.

For more:

PCOS & Lyme: My Story

***For those of you who have read anything I’ve ever written, you understand that by Lyme, I actually mean MSIDS or multi systemic infectious disease syndrome – as research has shown most of us are infected with far more than just borrelia, the causative agent of Lyme Disease:  Key Quote:  

“Our findings recognize that microbial infections in patients suffering from TBDs do not follow the one microbe, one disease Germ Theory as 65% of the TBD patients produce immune responses to various microbes.”

Ticks are coinfected – logic would indicate we are too:

And this great article shows that in fact we are:

Borrelia alone is a formidable, complex, pleomorphic bacteria(ish) that shape shifts to avoid medications and forms biofilm to evade our immune systems.  There are 300 strains and counting of borrelia worldwide, and 100 strains and counting in the U.S.  Current CDC Two-tiered testing tests for ONE strain.  So when I write about Lyme Disease, I mean the whole ball of wax.  Research, BTW is abysmal on all of this and dominated by what I call The Cabal.,  and,

OK, now that we’ve cleared that up….

My story


Around the same time our dog Bucky (Go Wisconsin Badgers!) tested positive for Lyme and we finally got to the bottom of my husband’s bizarre issues (he too had it), I suffered from severe pelvic pain. The pain appeared to originate in my ovaries but radiated everywhere leaving me hunched over like an old woman, holding my gut.  I was scheduled to undergo the knife for a small epigastric hernia but at the last minute the surgeon recommended an MRI which revealed two deflating cysts. No surgery needed, I continued to suffer in silence with pain that honestly felt like someone put a shot gun up my vagina and pulled the trigger.

In January one of my knees swelled up to twice its size, became red and hot to the touch, and I developed a fever. My old retired doctor told me I had “Washer Woman’s Knees”  from scrubbing the floor (You can’t make this stuff up), so I continued to suffer in silence. Six months later I saw flashing lights, had racing, erratic heart beats, insomnia, memory loss, migrating joint pain, colitis, severe occipital headaches, and a stiff and painful neck and spine, and frankly, stuff I’ve blocked out. I could no longer deny I had Lyme/MSIDS. The infamous quote by Dr. Jemsek,

“You either have 20 diseases or you have Lyme Disease,” certainly held true for me.

I diagnosed both of us by watching the documentary Under Our Skin, the best primer on Lyme Disease on the market.

I had done my reading by this point with my husband in Lyme/MSIDS treatment, and I got to a LLMD (Lyme literate doctor trained by ILADS), and tested positive on the IgeneX Extended Western Blot for Lyme.  Please see this wonderful presentation by IgeneX at our Lyme Support Group to learn why their testing is far more sensitive than the current CDC two-tiered testing that most GP’s use and that misses over half of all cases:

My LLMD told me to save my money on co-infection testing as he felt the testing was that poor and that his treatment would automatically treat co-infections. Of course, the rest is history, with over 5 years of an intense, painful, and expensive pulsed antibiotic regimen with IV blood ozone and UV light, herbs, IV vitamin C and other minerals, magnet therapy, chiropractic help, supplementation with numerous things including hormones (anabolic), LDN, and other modalities – to the tune of $10-$15 K out of pocket each year per person for over 4 years and counting.  I also must add that we both have relapsed three times necessitating 2-3 months of treatment each time.

But, I often wonder …

Would the story have turned out differently had I been aware of the very real probability that Lyme/MSIDS can be spread sexually?

Looking back, many symptoms were there that I intentionally blocked out as I was more concerned with my husband who appeared far worse than me at the time.  I also didn’t know what I know now. I find this tendency of one partner ignoring symptoms due to the seemingly more severe symptoms of the other partner to be a common thread in couples infected with Lyme/MSIDS.

I first became aware of the possibility of Lyme/MSIDS being sexually transmitted years ago when I started looking up PCOS (polycystic ovary syndrome) and Lyme. I discovered a hidden world of women who had severe pelvic pain, PCOS, and a Lyme diagnosis. And while I didn’t have a definitive PCOS diagnosis, the symptoms were quite similar.

I wasn’t alone!

Here’s just a few sites: (Soul Cysters – gotta love the name!);f=1;t=043160;p=0   According to the U.S. Department of Health and Human Services, PCOS is a common health problem in the ovaries caused by imbalanced reproductive hormones. Essentially the egg may not develop or be released normally. It can cause irregular menstrual periods and lead to cysts and infertility.   Some other symptoms include hair where men usually have hair, acne, thinning of hair, weight gain, darkening of skin, skin tags, depression and anxiety, decreased sex drive, and an increase in stress levels. Experts feel genetics, and high levels of androgens and insulin play a role. And while there is no single test to diagnose it, doctors typically perform physical exams including a pelvic exam with an ultrasound as well as blood tests looking at male hormone levels, thyroid, cholesterol, and test for diabetes. Once other conditions are ruled out patients may be diagnosed with PCOS if they have at least two of the following:

*Irregular periods, including periods that come too often, not often enough, or not at all
*Signs of high androgen levels – extra hair growth on face and body, acne, thinning hair
*Higher than normal blood levels of androgens
*Multiple cysts on one or both ovaries

I want to point out that cysts are common with Lyme/MSIDS.

Numerous coinfections cause skin tags (husband has them all over his back), many patients gain weight, lose head hair (this would be me), have bizarre menstrual issues, experience anxiety (husband had this) and depression (we both had this) as well as can’t tolerate stress (both had this), and could care less about having sex.

There’s a hidden world of women with a LD diagnosis who also have pelvic pain and PCOS-like symptoms.  An article put out by my favorite compounding pharmacy discusses numerous issues with hormones and LD including adrenal depletion, low DHEA levels contributing to low testosterone levels, thyroid issues, and cortisol issues. Nearly every Lyme/MSIDS patient I know is severely low in magnesium which is needed in over 300 biochemical reactions in the body.

While many LD male patients have told me their testosterone levels are through the basement floor, I haven’t heard from women about their hormone levels. Could LD/MSIDS cause inflammation in the pelvic area caused by borrelia? Could it alter/deplete hormone levels?

In this informative video, Dr. Phillips takes sexual dysfunction caused by Lyme/MSIDS head on:  Findings are:

  • painful testicles with recurrent Bartonella
  • Prostatitis is common, where the prostate becomes swollen, tender, and inflamed
  • anal and/or vaginal fissures (little painful cracks)
  • libido issues
  • Vaginal dryness
  • Psychological aspects
  • Low Testosterone levels
  • Extreme fatigue
  • Sexually induced severe headaches
  • A Pavlovian response in that if sex causes pain, it trains you to no longer desire it
  • Pre-menstral flares
  • Balanitus, a painful swelling of the foreskin, or head of the penis. Dr. Phillips believes this to be the result of an undiagnosed infection.  He’s also seen it as a herxheimer reaction. It can take time to resolve.

Once you go down the rabbit hole of gynecological symptoms of Lyme/MSIDS, the question of sexual transmission begs to be asked.

According to the CDC, Lyme is not sexually transmitted, despite being a spirochete that can burrow through any tissue and organ and despite being a cousin to Syphilis (T. pallidum). They also claim that the sole perp is the dastardly black legged tick.

I believe both of these tenants to be myths and time will reveal a much broader and complex picture.   Yet, Medical Daily reports a 2014 study in the Journal of Investigative Medicine that found Lyme (B. burgdorferi) in vaginal secretions of all women with Lyme and in the semen of half of the men with Lyme. One of the heterosexual couples with Lyme had the identical strain in their genital secretions.  Here is the updated article and the fact that 2 referees approved and 2 did not approve of the rigor of the study.  In essence, it was a draw but everyone just proclaims it isn’t a STD.

Lead author Marianne Middleveen stated, “Our findings will change the way Lyme disease is viewed by doctors and patients.”

That was in 2014, and……well….crickets

But, recently (May, 2019) a case study hot off the press showed live spirochetes in a genital lesion that could be grown in special culture broth:   Researcher Marianne Middelveen stated,

“The presence of live spirochetes in a genital lesion strongly suggests that sexual transmission of Lyme disease occurs,” said Middelveen. “We need to do more research to determine the risk of sexual transmission of this syphilis-like organism.”

This case study supports the 2014 pilot study and should be noticed by the world, but it isn’t.

Most recently, John Scott’s work not only found 63% of patients developed chronic Lyme disease, that Bb is pleomorphic, and that many patients experience despair and suicide, but also that his Case 4 provides confirmation for an ACA rash and gestational Lyme disease (club feet at birth).  Both parents tested positive for Bbsl, which is highly suggestive of Lyme being a STD, with the baby being born infected and with a birth defect indicative of Lyme infection:   Bizzarely, I found in this 2011 article a statement in quotations supposedly by the CDC which states,

“Conversely, we find no study supporting that sexual intra-human Borrelia burgdorferi transmission does not occur. Inferential data strongly suggest the possibility of human sexual transfer.”
“This data come from sound veterinary studies, the finding of Borrelia burgdorferi in human semen and breast milk, and by similarity to Treponema pallidum where sexual transfer is abundantly documented. Our clinical experience strongly suggests that predictable, possibly inevitable Borrelia burgdorferi transfer between sexually active couples occurs.

The article didn’t have a bibliography so I do not know where they found the CDC quotes.

Here’s what the current CDC website states as of 2019:

“There is no credible scientific evidence that Lyme disease is spread through sexual contact.Published studies in animals do not support sexual transmission (Moody 1991; Woodrum 1999), and the biology of the Lyme disease spirochete is not compatible this route of exposure (Porcella 2001). The ticks that transmit Lyme disease are very small and easily overlooked. Consequently, it is possible for sexual partners living in the same household to both become infected through tick bites, even if one or both partners doesn’t remember being bitten.”

Obviously the CDC changed their story at some point which is par for the course. The CDC flip-flops more than a waffle-maker.

Regarding breast milk the CDC states as of 2019, 

“There are no reports of Lyme disease being spread to infants through breast milk. If you are diagnosed with Lyme disease and are also breastfeeding, make sure that your doctor knows this so that he or she can prescribe an antibiotic that’s safe for use when breastfeeding.”

Please notice the dates for all those studies (1991-2001).  They have inches of dust on them, yet the same rhetoric is given:  Lyme is not a STD, is not spread congenitally or via breastmilk despite vastly increasing numbers worldwide & some very credible science, as well as clinical reality.

Many doctors and researchers disagree with the CDC.

Dr. Jones, a highly respected LLMD who has treated over 12,000 children from all over the world, has personally seen and treated cases of sexual, congenital, and breast milk transmission:  In this pdf there is a table of symptoms in children suspected of contracting Bb congenitally at or slightly after birth.

Dr. Jones spoke at the 2018 ILADS convention and his talk is summarized here:

Here’s 33 years of documentation of congenital Lyme collected by a Canadian nurse:  Due to pressure and proof from ticked-off moms of sick kids, Canada now recognizes congenital Lyme:

The U.S. didn’t seem to get the memo

Here’s another Bibliography on Gestational Lyme & via lactation:  From the CDC:  Untreated, Lyme disease can be dangerous to your unborn child.  Lyme disease that goes untreated can also cause you to have brain, nerve, spinal cord and heart problems.”  They also say there are NO reports of LD transmission from breast milk, which LLMD’s have found to be false (practitioners trained by ILADS – the International Lyme and Associated Diseases Society).  Bb DNA found in urine and breast milk of Lyme patients.  Here we see borrelia found in colostrumblood, synovial fluid, and urine in cows and horses way back in 1988 from our very own Elizabeth Burgess, a UW researcher.  Go Wisconsin!  Burgess also found:

This woman, bless her heart, attempted to warn the world, but nobody was listening.  Gregory Bach, DO, International Scientific Conference on Lyme Disease, April 2001

“Lyme spirochetes/DNA have been recovered from stored animal semen. Recovery of spirochete DNA from nursing mother’s breast milk and umbilical cord blood by PCR (confirmed by culture/microscopy), have been found in samples provided to my office.

Surprisingly, initial laboratory testing of semen samples provided by male Lyme patients (positive by western blot/PCR in blood) and the male sexual partner of a Lyme infected female patient were positive approximately 40% of the time. PCR recovery of Lyme DNA nucleotide sequences with microscopic confirmation of semen samples yielded positive results in 14/32 Lyme patients (13 male semen samples and 1 vaginal pap).  ALL positive semen/vaginal samples in patients with known sexual partners resulted in positive Lyme titers/PCR in their sexual partners. 3/4 positive semen patients had no or unknown sexual partners to be tested.”  Another link with helpful information mostly regarding infection in children but also has links on pregnancy and transmission.  This informative 2014 article gives case studies of numerous medical practitioners showing interesting findings:

“I’ve seen women who were infected long before they were pregnant and I’ve checked their children – the first-born is in good shape, but the third-born is badly infected,” Kriz said. I’ve seen it in several families.”

And, a woman with symptoms all her life who was finally diagnosed with Lyme possibly contracted in utero stated,

“All of the sudden, all my little quirks – the things that made me, me – those were the disease,” she said. “It wasn’t typical Lyme symptoms, it was little things, like severe anxiety, irrational fears, a poor memory.  I was bad with directions. I had a foggy brain. I thought everyone deals with these things.”  And, “Certainly, having a foggy brain in middle school and high school does not do a lot for your self-confidence,” she said. “It’s hard to feel confident in yourself when you can’t rely on your brain to do what you want it to do. It’s hard to make the commitment to decide what kind of career you want, or anything else.”   CONCLUSION: BORRELIA BURGDORFERI, not only transmitted by the tick bite, is TRANSMITTED by sexual contact, fluids and can also colonize the fetus of pregnant women if there is no effective treatment able to eradicate it during the same. And it is not exclusive to the Northern Hemisphere. The BORRELIA is also in the Southern Hemisphere.  This link shows everything from Alan McDonald’s finding of borrelia in a chronically infected male’s testicle in an autopsy to survival of borrelia in blood products to an ancient 1986 study by none other than the NIH of:

uninfected mice becoming infected with Bb within 42 days after being put in a similar cage with infected mice.  

Please pause here.  Let your eyes fall back.  Mice infected just by close proximity with infected mice.
Now granted, animals eliminate freely in the cage and are exposed to both urine and feces – unlike humans who have proper sanitation, but this study shows that no ticks are needed for transmission.  This link also states it is well established that Bb is found in urine and that Willy Burgdorfer, the discoverer of Bb, got Lyme from infected rabbit urine when it got into his eye.  

And somebody went to a boatload of work collecting animal transmission studies on Bb:  I found this collection of studies years ago and am happier than a lark it’s still available.  Please read the variety of ways Bb is spread in animal studies.

If this happens in animals (other mammals), it’s highly probable it happens in humans (other mammals).

And last but not least, I dug up this gem from 2005 of Lida Mattman PhD, who was an expert on spirochetes and who wrote the textbook on cell-wall deficient forms:  Excerpt:

Mattman isolated living Borrelia spirochetes in mosquitoes, fleas, mites, semen, urine, blood, plasma and Cerebral Spinal Fluid.

“We get to the ways that burgdorferi is transmitted. I laugh at all this stuff about looking for the Woodtick. That’s so ridiculous because most of the people who get Lyme disease have never heard or seen a tick. We know now it’s in tears and people wipe their eyes and then you shake hands with them. Or we don’t laugh so hard about the physician we had in the hospital who wouldn’t touch the doorknobs in the hospital without taking his white coat and handling the doorknob through a coat. Maybe he wasn’t so insane after all.

So we think this is spread by what is called fomites (an inanimate objector substance that is capable of transmitting infectious organisms from one individual to another) which is the pencil in the bag as you pick up a pen to write a check or anything you handle. So it’s in urine and in tears and it’s also spread by mosquitoes and who hasn’t had a mosquito bite?  We’ve tested the mosquitoes in Michigan and sure enough they can carry the Lyme spirochete.” 

Another woman researcher trying to warn us again.  Crickets….

The real head-scratcher of course is why isn’t the CDC/IDSA/NIH taking all of this into account?

One word: vaccine development.

Let’s suffice it to say the polarization continues unabated in the world of Lyme/MSIDS.

But, you are duly warned.

Lastly, it is my firm opinion that the science will only be settled on these issues when the tsunami of the infected do the work.  A perfect example is when independent tick researcher John Scott and his wife Kit, both infected for decades, rang the bell on how climate change has nothing to do with tick movement and the increasing Lyme cases:

“The climate change range expansion model is what the authorities have been using to rationalize how they have done nothing for more than thirty years. It’s a huge cover-up scheme that goes back to the 1980’s. The grandiose scheme was a nefarious plot to let doctors off the hook from having to deal with this debilitating disease. I caught onto it very quickly. Most people have been victims of it ever since. This climate change ‘theory’ is all part of a well-planned scheme. Even the ticks are smarter than the people who’ve concocted this thing. Climate change has nothing to do with tick movement. Blacklegged ticks are ecoadaptive, and tolerate wide temperature fluctuations…..It’s all a red herring to divert your attention.” –  John Scott

There’s lots of red herrings in Lymeland.