Archive for the ‘research’ Category

70% of TN Embalmers Still Seeing White Fibrous Clots in 1 Out of Every 6 Corpses

https://gregreese.substack.com/p/70-of-tennessee-embalmers-still-seeing?

70% of Tennessee Embalmers Still Seeing Death by Clot Shot

And 39% are seeing a fourteen percent increase in Infant Death

Just around three years ago, as millions of people around the world were dying from strokes and heart attacks shortly after getting the experimental gene editing COVID shots, coroners and embalmers began finding large dense clots in the vascular system of the dead. Clearly large enough to explain the cause of death, but ignored by government and media. And the public, including children, are still getting these clot shots.

During the winter of 2023/2024, Thomas Haviland conducted the, “Worldwide Embalmer Blood Clot Survey,” where in embalmers from the United States, Canada, Australia and the UK were surveyed about the blood clots. The study found that the “White Fibrous Clots” were not seen by anyone Pre-Covid/jab, and over seventy percent were seeing them after the Covid/jab in twenty percent of their corpses. And about twenty percent of embalmers saw a twenty-five percent increase in Infant Death after the Covid/jab. (See link for article and video)

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**Comment**

Yet crickets from ‘public health,’ mainstream media, and mainstream medicine.

For more:

A Case For Chronic Lyme: A Medical Perspective

https://danielcameronmd.com/chronic-lyme-exist/

The Case for Chronic Lyme: A Medical Perspective With Dr. Cameron

The Chronic Lyme Disease Controversy 

I’ve been treating chronic Lyme disease for 37 years. In that time, I’ve witnessed firsthand the struggles of patients whose symptoms didn’t resolve after the standard course of antibiotics. I’ve also seen how the medical community has been deeply divided on the existence of chronic Lyme disease.

Many of my colleagues—some of whom I deeply respect—have argued with me over the years, insisting that chronic Lyme disease doesn’t exist. They believed that once a patient completes a prescribed course of antibiotics, any lingering symptoms must be the result of something else—an autoimmune response, lingering inflammation, or simply the wear and tear of everyday life. Some even suggested that the symptoms were psychological, a product of health anxiety rather than a persistent infection.

Their skepticism wasn’t entirely unfounded. The medical community places a high value on evidence-based medicine, and in the absence of a universally accepted diagnostic test for chronic Lyme disease, many physicians were unwilling to acknowledge what they couldn’t definitively prove. But for me, the proof wasn’t in a single test—it was in the patients I saw every day, the ones who continued to struggle with unrelenting fatigue, joint pain, cognitive dysfunction, neuropathy, and dysautonomia (POTS) long after their treatment ended.

A Shift in Perspective: Acknowledging Chronic Manifestations

While some colleagues remained rigid in their views, others began to recognize that Lyme disease could lead to serious, chronic complications. Over time, Lyme arthritis, Lyme encephalopathy, Lyme neuropathy, Chronic Neurologic Lyme, Neuropsychiatric Lyme, PANS, POTS, and PTLDS were increasingly acknowledged in medical literature. These weren’t vague, unproven conditions—these were well-documented manifestations of Lyme disease that had been observed in patients.

It was particularly interesting to see that many of my colleagues—who had once completely dismissed the idea of chronic Lyme disease—began to accept the concept of Post Treatment Lyme Disease Syndrome (PTLDS). They acknowledged that some patients remained ill after treatment, sometimes for years. They saw the ongoing fatigue, cognitive dysfunction, and pain that persisted long after the initial infection.

But even as they accepted PTLDS, many still refused to consider that a persistent tick-borne infection might be responsible for these symptoms. They viewed PTLDS as an immunologic response to a past infection rather than a sign that the bacteria were still present. This created a paradox—if they could acknowledge that patients were still sick, why couldn’t they entertain the idea that there was an active infection driving these symptoms?

The Importance of Keeping an Open Mind

There’s still so much we need to learn about Lyme disease, particularly in its chronic form. Dismissing the possibility of persistent infection without fully exploring the science doesn’t serve patients. Many Lyme patients feel abandoned by the medical community because they are told their symptoms aren’t real or that they shouldn’t still be sick. But I’ve seen too many cases where patients responded positively to additional treatment—sometimes with extended antibiotics, sometimes with a combination of therapies targeting co-infections like Babesia or Bartonella.

I’ve also seen how new research continues to challenge old assumptions. Studies have shown that Borrelia burgdorferi, the bacteria that causes Lyme disease, can persist in animal models even after antibiotic treatment. Other research suggests that biofilms and persister cells may allow the bacteria to evade treatment, potentially leading to chronic symptoms. While the debate continues, the evidence pointing toward persistent infection is growing.

What This Means for Patients

For patients who continue to suffer, the name of their condition—whether it’s called chronic Lyme disease, persistent Lyme infection, or PTLDS—is far less important than the reality they live with every day. What matters most is that we, as doctors, listen to our patients, acknowledge their suffering, and remain open to the possibility that Lyme disease is far more complex than we once believed.

The more I work with Lyme disease patients, the clearer it becomes—chronic Lyme disease is real. And for many, the right treatment can mean the difference between lifelong suffering and reclaiming their health.

We must continue asking the tough questions, challenging old beliefs, and keeping an open mind. Because at the end of the day, it’s not about proving a point—it’s about helping people get their lives back.

Related Articles:

Lyme disease as a cause of chronic illness for some patients

What is “chronic Lyme disease”?

Chronic Lyme disease patients dismissed by medical community

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**Comment**

Absence of proof isn’t proof of absence.

For more:

The Effects of Light on the Human Body

http://  Approx. 30 Min

March, 2026

Explore what a single photon of sunlight does to your body the moment it touches your skin — and why the physics your grandmother never knew explains everything she told you.

This video explores vitamin D photosynthesis, circadian entrainment, and nitric oxide release. Learn how one broken chemical bond controls over 200 of your genes, and find out why indoor living is quietly weakening your bones, disrupting your sleep, and raising your blood pressure.

Whether you are curious about what is really happening inside your body, fascinated by physics, or looking for explanations that actually make sense, this will change how you understand the sun, your health, and the biology you were built with. Watch now to discover what nobody tells you about sunlight.

CHAPTERS

  • 0:00 – Introduction
  • 0:42 – One Photon, One Bond
  • 2:21 The Vitamin D Factory
  • 5:23 – The Latitude Problem
  • 8:52 – Your Circadian Clock
  • 14:49 – Serotonin, Mood, and Sleep
  • 18:49 – Sunlight and Blood Pressure
  • 22:35 – The Real Risk: UV and DNA
  • 25:01 – Bones, Falls, and Immunity
  • 28:52 – Conclusion

https://gregreese.substack.com/p/the-effects-of-light-on-the-human?

The Effects of Light on the Human Body

Screen time leads to blindness and mind control
 
Article Excerpts:

The blue light emitted by LED is a frequency band of 400 to 500 nanometers. Exposure to this has been shown to suppress melatonin production, a hormone critical for sleep. Blue light exposure will shift brain activity away from relaxed theta-dominant states, and towards heightened alert beta states. And so exposure in the evening will further complicate sleep patterns, which will negatively effect health in many ways.

The red light emitted is a frequency band of 620 to 700 nanometers. And it has been shown to promote bone and tissue repair and regeneration. It reduces inflammation and body fat, and it alleviates chronic and acute pain. The main reason that red light improves overall health, is because it enhances mitochondrial function throughout the entire body system. This leads to increased ATP production, reduced oxidative stress, and modulated cytokine activity.  (See link for article and video)

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https://sayerji.substack.com/p/the-ultimate-human-superpower-you

The Ultimate Human Superpower You Never Knew You Had: Melanin — The Hidden Solar Panel in Human Biology

Beyond Skin Deep: Melanin as a Biophotonic Engine
 
Article Excerpts:

For two centuries, this idea stuck: melanin as nature’s UV filter, nothing more.

Yet even as textbooks dutifully repeated melanin’s sunscreen status, scientists kept stumbling on puzzles that didn’t fit the simple story. For one, melanin pops up in places where sunlight scarcely shines. Consider the human brain: certain neurons in the deep brain (the substantia nigra and locus coeruleus) are loaded with neuromelanin, giving these regions a dark hue. Why would brain cells – tucked inside the skull – bother to produce a UV-blocking pigment? The inner ear is another enigma: the cochlea has melanocytes (pigment cells), and their dysfunction can cause hearing loss. Again, no sunshine reaches there. Even stranger, melanin is found in the heart valves of some animals and the lungs of certain seabirds. These “internal melanized sites” not obviously subject to light have puzzled researchers for years. Could melanin be doing something else there?

Laboratory observations added to the mystery. In one experiment, skin cells rich in melanin were found to contain far fewer mitochondria – the tiny organelles that generate ATP energy – than their non-pigmented counterparts, yet they grew and developed just as well. In these heavily melanotic cells, mitochondrial number dropped a whopping 83%, and respiration (the usual oxygen-burning energy process) was 30% lower, without impairing cell growth. It was as if the melanin was somehow compensating for the lost mitochondria.

Clinicians also noted paradoxes: melanin in the skin reduces UV DNA damage (protective), but an abundance of melanin can correlate with melanoma (cancer) risk; neuromelanin in the brain might be protective in some ways but is lost in Parkinson’s disease, possibly contributing to degeneration. Melanin, the pigment, was behaving less like a static shield and more like a dynamic player – one with a dual nature that scientists didn’t yet fully grasp.

Such anomalies prompted a few intrepid thinkers to ask uncomfortable questions. Was it possible that melanin had a metabolic function – that it could, under the right conditions, act as an energy source or catalyst for life’s processes?

One of the early proponents [of melanin being an unrecognized bioenergetic molecule] was Geoffrey Goodman, who published a speculative paper in 2008 titled “Melanin directly converts light for vertebrate metabolic use”.

Dr. Arturo Solís Herrera, a Mexican ophthalmologist and biochemist, stumbled onto melanin’s secret while studying diseases of the eye. In the 1990s, Solís-Herrera was investigating the causes of blindness like glaucoma and diabetic retinopathy…..anatomists knew the back of the eye is pigmented. But what struck Solís-Herrera was a question: Why would nature put a dense ring of melanin 2.5 centimeters deep inside the head, effectively behind the light-sensitive retina? It’s like finding solar panels buried in a cave – seemingly out of place.  (See link for article)

SUMMARY:

After a 12 year research journey Herrera’s team:

  • found melanin granules can dissociate water molecules, using light energy to split H₂O into hydrogen and oxygen – essentially the first step of photosynthesis.
  • demonstrated melanin’s water-splitting in vitro and measured the currents and reaction products and laid out their findings that melanin represents over 90% of cell energy requirements – and that a vast majority of our ATP energy might come from light and water via melanin while glucose is used to build biomass.

Over time more concrete evidence trickled in:

  • fungi that ‘eat’ radiation: melanin enabled fungus to harness radiation for metabolic energy – behaving like a broad-spectrum solar panel.
  • melanin rich mouse skin cells could function with 83% fewer mitochondria and 30% lower respiration rate, yet developed similar to non-pigmented cells implying that melanin was compensating and providing energy through another route.
  • it is hypothesized that the painted turtle can survive underwater without oxygen for months during winter hibernation due to the shell and skin containing melanin.
  • seeds: life’s solar-powered time capsules. Why would dormant seeds buried in soil away from light need a ‘sunscreen’ pigment? 
  • birds’ solar-powered eyes: darkly pigmented gel in a birds’ eye might directly convert light into metabolic energy to support the retina during 100 hours of flight without landing or eating.
  • photomodulation in humans (hairlessness advantage): red and near-infared light boost mitochondrial activity and ATP, reduce inflammation, and activate certain genes related to metabolism.
  • experiments by Dadachova’s group found that irradiating melanin with gamma, UV, visible, or infrared light all boosted the electron transfer rate – and to a similar extent regardless of the photon energy. This is telling: it suggests melanin absorbs across a broad spectrum (it is known to absorb UV through visible and into IR) and converts that absorbed energy into driving chemical reactions.
  • a 2014 discovery showed mammals can absorb dietary chlorophyll metabolites which then accumulate in mitochondria, allowing cells to harvest light to enhance ATP production.
  • Dr. Gerald Pollack’s research into the 4th phase of water—known as EZ water (Exclusion Zone water) opens the door to understanding how light, water, and pigment form a functional trinity at the heart of cellular energetics¹⁰.

This has far-reaching implications for health.

  • A 2012 paper states: “a failure in [the] light/melanin/water system” was posited as a cause of AMD and Alzheimer’s disease¹¹. 
  • some researchers think melatonin might synergize with melanin’s function to maintain mitochondrial health in the brain and retina. CF, metabolic syndromes, and developmental issues might involve melanin’s activity, opening new avenues for treatment: enhancing melanin’s function or compensating for its decline through light and other therapies.
  • 2012 research showed that mice receiving just 50 mg/kg of melanin showed 100% greater survival at 30 days compared to untreated mice. Perhaps equally important, the melanin showed no toxicity even at double this dose (100 mg/kg), suggesting a remarkable safety profile.
  • Another 2012 study showed 90% of mice fed a melanin-rich fungus one hour before exposing them to a massive 9 Gray dose of Cesium-137 radiation survived, but all control mice died within 13 days.  When the mice were fed white porcini mushrooms (which lack melanin but contain all the other bioactive mushroom compounds), they died almost as quickly as controls, but when the white mushrooms were supplemented with melanin, the mice gained the same radioprotection as those eating the melanized mushrooms, proving it was melanin that made the difference. (Chaga, Reishi and Turkey Tail mushrooms are all melanin dense, demonstrating anti-cancer and immune modulating properties)
  • In 2015, researchers in Mexico demonstrated a prototype melanin battery which could lead to novel solar energy storage systems.

Bartonella and Babesia Detected in Patients With Chronic Illness

https://news.ncsu.edu/2024/07/bartonella-and-babesia-co-infection-detected-in-patients-with-chronic-illness/

Bartonella and Babesia Co-Infection Detected in Patients with Chronic Illness

For Immediate Release
Edward Breitschwerdt

A small pilot study has found evidence of human co-infections from Bartonella and Babesia odocoilei, a protozoal tick-borne infection primarily found in deer, moose and other cervids. The co-infections were detected in six of seven patients suffering from chronic, non-specific illness, who were enrolled in a Bartonella study at North Carolina State University. The work presents some of the first definitive DNA evidence of this strain of Babesia infection in humans, and could impact treatments for patients with BartonellaBorrelia or Babesia infections.

Bartonella are a group of vector-borne bacteria transmitted primarily via arthropods like fleas, lice and potentially ticks, but also by the animals that harbor them. There are at least 45 different known Bartonella species, of which 18 have been found to infect humans. Improved methods for detecting Bartonella infection in animals and humans have led to the diagnosis of bartonelloses in patients with a host of chronic illnesses, as well as in some patients with psychiatric symptoms.

In the U.S., the main Babesia species that infect humans are B. microtiB. duncani and B. divergens-like. Transmission occurs mainly by tick bite, but there are reports of transmission by transfusion of contaminated blood, organ transplantation and transplacental transmission. In addition to asymptomatic infection, babesiosis can be associated with non-specific symptoms such as fever, chills and night sweats, or with severe, life-threatening hemolytic anemia.

Babesia and Bartonella are often suspected as co-infections with Lyme disease, caused by the bacterium Borrelia burgdorferi.

“Doctors who work with Lyme patients often suspect co-infection with Babesia most often based on serology data and symptoms,” says Edward Breitschwerdt, Melanie S. Steele Distinguished Professor of Internal Medicine at NC State’s College of Veterinary Medicine and corresponding author of a paper describing the work. “So we decided to test for potential co-infections with this subset of our Bartonella study participants.”

The seven patients in the pilot study were already enrolled in a larger study aimed at detecting Bartonella in the blood of people with chronic illness and extensive animal contact. The participants’ ages ranged from two-and-a-half to 62 years old. Four were veterinary workers, one was a student and veterinary technician, one was a veterinarian’s daughter and one was a pre-school aged child. All submitted blood and tissue samples for testing.

Using digital droplet PCR testing, which identifies pathogens by their DNA sequences, the researchers discovered that all seven participants were infected with Babesia odocoilei, and that six of the seven were co-infected with at least one, and sometimes two or more, species of Bartonella.

“We were surprised by these findings for several reasons,” Breitschwerdt says. “First, this strain of Babesia – B. odocoilei – isn’t currently considered a cause of human infection. Second, these patients reside in locations around the U.S. and in Mexico, so the distribution may be geographically widespread. Finally, we were able to identify these seven infected individuals within a one-year time period, suggesting that this may be a more prevalent infection than currently appreciated.”

The findings could have implications for how patients with symptoms of vector-borne illnesses such as Lyme, babesiosis, or bartonellosis are tested and treated.

“The main symptom associated with babesiosis is acute, severe hemolytic anemia. None of these patients reported any such illness, a factor that could limit a doctor’s decision to test for Babesia infection,” Breitschwerdt says.

“This study primarily focused on patients with chronic illnesses and suspected bartonelloses. Most often, doctors don’t look for Babesia in these patients, but we now have good evidence that people can be infected by this organism, evidence that should be pursued in larger studies. Physicians who treat Lyme disease have often suspected co-infections with these pathogens, and it looks like their suspicions are correct, at least in a subset of patients.”

“We are committed to providing the best vector borne infectious disease testing results possible,” says Ricardo Maggi, research professor at NC State and first author of the study. “The cases presented in this manuscript reflect our team’s efforts to develop better approaches and diagnostic tools that can give answers to both patients and their doctors.”

The study appears in Parasites and Vectors and was supported by NC State’s Bartonella/Vector-Borne Diseases Research Fund and the Steven & Alexandra Cohen Foundation. NC State Ph.D. student Charlotte Moore, São Paulo State University Ph.D. student Ana Cláudia Calchi, and NC State research specialist Emily Kingston also contributed to the work.

For more:

Magic Trick Reveal: How To Get Perfect Efficacy From a ‘Vaccine’ That Doesn’t Work. Two White Papers Call to Halt COVID Shots, But FDA Approves Non-mRNA COVID Shot

https://kirschsubstack.com/p/the-big-magic-trick-revealed?

The big magic trick revealed!

How can you get near perfect efficacy from a vaccine that doesn’t work? Thanks to Professor Jeffrey Morris, I now know the magic trick too! This one had me fooled me for years.

The Secret Truth Behind Penn & Teller: Fool Us

I like Penn and Teller. But they don’t know jack about vaccines. Check out this video they did. They should apologize and re-do the video to tell the truth. YouTube will ban it though.

Executive Summary

If you have a policy of COVID testing where it is mandatory to test the unvaccinated 2x/week but the vaccinated can totally skip testing, this can massively skew all studies based on that data.

I just found out this was the case in the Czech Republic which is the only country where we have record level data.

That explains a lot, doesn’t it? A testing policy like that can make an ineffective vaccine close to perfect.

Bottom line: the only honest way to see if the vaccine worked is to compare the whole period all-cause mortality between those who got the shots and those who did not. Then you learn the truth.

Guess what? There isn’t a single study that does this.Zero. Zip. Nada.

So I calculated it for the Czech Republic. It shows that the vaccines should be stopped. But you already knew that, didn’t you?

And if you can’t get COVID, you can’t die from COVID!

BAM! The magic trick has been revealed. The next time you want to make a vaccine super effective, there is nothing like deploying a differential testing policy like this.

Therefore, in Czechia, you get a near perfect vaccine and can write papers about it and nobody is the wiser.  (See link for article)

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**Comment**

These tactics need to be continually exposed for what they are: false and misleading – constituting scientific fraud.  Those in public health should be reviewing these things before they roll out a drug or medical product of any kind.

Hirsch then shows a table (to access, click top link) that shows when you actually compare whole period all-cause mortality between the injected and the non-injected you discover the shots are killing people.  But, reality is already showing this for those paying attention.

For more:

People have been shouting about these death shots for years.  There’s plenty of published science showing the myriad of problems.

Two White Papers Call For COVID Shot Halt

The IPAK-EDU White Paper 2025-ENDEUA states that when corrected efficacy curves and suppressed risks are taken into account, the mRNA covid shots fail the EUA standard.  The shots were based upon interim data from large-scale Phase III trials. The legal standard under §564 of the Federal Food, Drug, and Cosmetic Act requires that a product may be authorized for emergency use only if it is “reasonable to believe” that the product “may be effective” against a life-threatening condition, that the known and potential benefits outweigh the known and potential risks, and that no adequate alternatives are availableThis report  demonstrates, through a rigorous retrospective analysis, that these criteria were never met.  

Go here to listen to Dr. Jeyanthi Kunadhasan, an Australian anesthesiologist and perioperative physician who was terminated from her hospital position after questioning the risk-benefit profile of COVID-19 vaccines in healthy individuals. Following her dismissal, she joined a group of international medical volunteers tasked with reviewing the 500,000+ pages of internal Pfizer documents released as part of a court-ordered FOIA request. This effort was coordinated by DailyClout and Dr. Naomi Wolf. As a result of that work, Dr. Kunadhasan has become one of the foremost experts on Pfizer’s pivotal mRNA vaccine trial, focusing specifically on discrepancies in reported deaths and adverse events.

What Dr. Kunadhasan uncovers raises grave questions about data integrity, regulatory failure, fraud, and the validity of the FDA’s EUA for the Pfizer-BioNTech COVID-19 shot.

And yet another White Paper is calling for an urgent halt on self-amplifying RNA ‘vaccine’ deployment until comprehensive safety studies are done to address contagiousness risks.  The paper discusses the significant theoretical risk including contagiousness through shedding, sexual transmission, and recombination with wild-type viruses.

FDA Commissioner Dr. Marty Makary and head of FDA’s Center for Biologics Evaluation and Research, Dr. Vinay Prasad, note that a number of other countries such as Australia and Germany only recommend the clot shots to certain populations while the U.S. has taken a ‘one size fits all’ approach because experts have argued Americans are too stupid to understand age-and risk-based recommendations.

But, even this approach ignores the elephant in the room: there are treatments for COVID which means a ‘vaccine’ isn’t even needed.  Then, there’s the fact they have negative efficacy.

What more is it going to take?

Evidently, a lot more……

The Food and Drug Administration issued an approval for Novavax’s Covid-19 ‘vaccine.’ This is the only non-mRNA vaccine.

The vaccine was approved for adults aged 65 and older and individuals aged 12 to 64 with at least one underlying health condition that increases their risk for severe COVID-19 outcomes. Reading the clinical trials prompts serious questions.

The trials included populations that had a single dose and populations that had two doses. The trials showed a reduction in symptomatic Covid 7 days after the second dose. But the FDA didn’t approve two doses. They only approved one. So is one dose even effective at all at preventing Covid? We don’t know, that wasn’t tested (Is this sounding familiar yet?)

There is also a noted risk of myocarditis and heart inflammation. There was also at least one vaccine recipient with “a serious event of Guillain Barré syndrome reported 9 days following the shot.  The FDA notes that Novavax has to follow up on any further cases if they observe it.

The FDA is also supposed to follow up on how the vaccine does when it is shipped to patients. They must submit a study called “Shipping Evaluation of SARS-CoV-2 rS (JN.1 Vaccine) Drug Product in Pre-Filled Syringe (PFS) Finished Good Presentation” by July of this year.

They approve vaccines not knowing how they will fare once they are shipped to doctor’s offices. They test it on us.

Don’t forget the low prevalence of Covid.

There is good news: there are at least 16 states with pending bills seeking to ban the clot shot mandates, establish a ‘vaccine’ bill of rights, or prohibit any immunization containing mRNA material.  Some bills would require manufacturers to label foods containing mRNA, according to data compiled by Bloomberg Government.

More than 2,500 ‘vaccine’-related bills have been introduced since 2021.

And top FDA official just disclosed she never got the clot shot due to her concerns about biodistribution.  She was concerned about excretion in breast milk since she was pregnant during the COVID ‘pandemic.’  When asked if information has emerged that validates her choice, said she thinks it does, but wanted to go on record that she was not speaking on behalf of the FDA.  

Researchers reported in a 2022 paper that messenger ribonucleic acid, which is in the Pfizer and Moderna COVID-19 shots, was detected in human breast milk. Another paper, in 2023, detailed similar findings.

Pfizer and Moderna did not return requests for comment.

And ‘Dilbert’ comic strip creator, Scott Adams, just admitted to having the same aggressive turbo cancer as former President Biden.

“The smartest, happiest people are the ones who didn’t get the vaccination, and they’re still alive.” ~ Scott Adams, Dilbert creator

It’s getting hot in the kitchen, folks.